Discover and read the best of Twitter Threads about #ShootThatPoisonArrow

Most recents (11)

🩸 Blood and Sand ⌛️

gateway.pinata.cloud/ipfs/QmVc3SibR…
-COVID-19 is a blood and blood vessel disease.
-Current treatment protocols are actively harmful to patients
-Early treatment interventions have been suppressed in favor of vaccines and expensive patented drugs
- Natural immunity confers protection against all of the virus’s proteins, and not just one.
-Vaccines will do more harm than good.
-The vaccine and the virus were made by the same people.
Read 6 tweets
🚑 One Thing Leads to Another 🚑

Fire them...When you have a heart attack may there be no EMS (oops, no paramedics available!) and no staffed bed, thus.... hope you can fix your own cardiac blockage since you FIRED the people who would otherwise assist.
market-ticker.org/post=243699
According to the Office for National Statistics, since July 2 there have been 9,619 EXCESS DEATHS in England and Wales, of which 48% were NOT CAUSED by Covid-19.

So if all these extra people are not dying from coronavirus, what is killing them?>
Data from Public Health England shows that during
that period there were 2,103 extra death registrations with ISCHEMIC HEART DISEASE, 1,552 with HEART FAILURE, as well as an extra 760 deaths with cerebrovascular diseases such as STROKE>
Read 6 tweets
💔Heartbreaker💔

A marker for the severeness and disease progress of COVID is overexpression of serum amyloid A (SAA) to levels that in other diseases are associated with a risk for SAA AMYLOIDOSIS...SAA amyloidosis could also be a long-term risk of SC2 infections...we study>
the effect of a SC2 protein segment on SAA amyloid formation...we find that the presence of the 9-residue segment SK9, located at the C-terminus of the ENVELOPE protein, increases the propensity for SAA FIBRIL FORMATION by 3 mechanisms: it reduces the stability of the>
lipid-transporting hexamer shifting the equilibrium toward monomers, it increases the frequency of aggregation-prone configurations in the resulting chains, and it raises the stability of SAA fibrils.
pubmed.ncbi.nlm.nih.gov/34370466/
Read 16 tweets
I Can't Relax

"Only PMH was hypertension"
Just like with Spike from SC2, Spike from 💉is
lethal for patients with hypertension.
Spike is spike.
It is toxic for certain phenotypes/comorbidities.
Read 4 tweets
Myocarditis is NEVER mild.
The❤️muscles, the cells that make up the heart muscles NEVER regenerate.
If one dies, they’re done. It’s not like the liver or the kidney that regenerates. When a heart muscle dies it’s dead and it’s NEVER replaced.
Muscle cells in the heart will be dying...totally unpredictable in terms of its long term consequences. This is a most worrying development...exactly
the kind of complication that would have come out
of a normal clinical trial for a vaxx which typically takes a number of years.
Read 3 tweets
🐭Jambalaya🐭

...only the IV group developed histopathological
changes of myopericarditis as evidenced by CARDIOMYOCYTE DEGENERATION, APOPTOSIS and NECROSIS with adjacent inflammatory cell infiltration and calcific deposits on visceral pericardium, while evidence of>
coronary artery or other cardiac pathologies was absent. SARSCoV2 SPIKE antigen expression by immunostaining was occasionally FOUND IN INFILTRATING IMMUNE CELLS of the HEART or injection site, in cardiomyocytes and intracardiac vascular endothelial cells, but not skeletal>
myocytes. The histological changes of MYOPERICARDITIS after the first IV-priming dose PERSISTED for 2 wks and were MARKEDLY AGGRAVATED by a 2nd IM- or IV-BOOSTER DOSE.
Cardiac tissue mRNA expression of IL-1β, IFN-β, IL-6 and TNF-α INCREASED SIGNIFICANTLY from 1dpi to 2dpi in IV>
Read 5 tweets
High on spike protein, it calls to me

"This binding triggers a cascade of changes which disrupt normal cell function, and lead to the release of chemicals that cause inflammation. This happened EVEN WHEN the PROTEIN WAS NO-LONGER ATTACHED TO THE VIRUS.>
...evidence to suggest that the SPIKE PROTEIN can REMAIN IN THE BLOOD STREAM AFTER THE VIRUS HAS GONE and travel far from the site of infection. In this study, researchers only studied pericytes from the small blood vessels within the heart. However, pericytes are found within>
Read 7 tweets
Tiny Bubbles

"The clots I am talking about are microscopic. These are tiny…they are literally on a capillary level and they are scattered throughout your capillary network. They are not going to show on any scan. They are just too small and too scattered. So the only
way to find out for sure,if this predictable mechanism of clotting was actually happening was to do this blood test called a D-dimer...a blood test that shows a recent blood clot. It doesn’t show anything else other than a recent blood clot...I am still trying to accumulate more
information...the ones I have so far, 62% of them have EVIDENCE OF CLOTTING. Which means that these blood clots are NOT RARE. It means the majority of people are GETTING BLOOD CLOTS that
THEY HAVE NO IDEA that they are having"
Read 20 tweets
🕯️Lit Up🕯️

New ultra-accurate scans that can flag tiny blood clots, might be of benefit for post Covid and 💉patients.

The procedure involves a harmless dye being injected into the bloodstream which lights up ... mol.im/a/9935677#i-be…
The results of a LANDMARK TRIAL, presented at
European Society of Cardiology last week, showed
the SCAN IDENTIFIED 80 % OF DEADLY CLOTS,
while angiograms tend to spot about 60 %.

'They put the dye in my arm, and then I went under the scan. It was all done in an hour.’ -R.Klein
The DYE PICKED UP A SMALL CLOT FORMING in one of the arteries leading into Ronald’s heart, which doctors had not spotted originally.
researchgate.net/publication/34…
Read 4 tweets

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