Emma Hilton Profile picture
Sep 3, 2020 5 tweets 2 min read Read on X
Lots of clever people noticing the basic flaw with the current argument being presented to @WorldRugby.

If even ‘starved’ untrained transwomen retain 20-40% muscle/strength than matched females, how can one argue that trained transwomen will reach parity?
The premise of ‘nobody has studied changes in athletes’ implies that athletes:

1. will respond differently to the general population to T suppression.
2. will somehow become more similar to females than the untrained population, who retain a 20-40% over matched females.
The proposed biological mechanisms etc to support these implications are never clarified.

1. There is very solid rationale to believe that transwomen athletes will respond differently to the general population to T suppression.
That is: pre-trained transwomen are unlikely to follow the ‘starved model’ baseline, and newly-training transwomen are likely to mitigate muscle/strength loss, perhaps even gain.
Which deals with implication 2.

I want to hear biological arguments for the hypothesis that athletic transwomen will become more on par with females than will gym-shy, aggressively atrophying transwomen.

Does anyone have any? Seen any?

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More from @FondOfBeetles

Dec 16
Hello @michaelshermer

Please see the following links to various papers and commentaries I and others have published on sports categories.

Please follow me (obvs 😂), @TLexercise @Scienceofsport @runthinkwrite @cathydevine56 @BrowngaGreg @MaryOConnorMD @DrMJoyner and associated scientists for academic work.

Guys, add your papers below please.
Where it all started (academically): “the muscular advantage enjoyed by transgender women is only minimally reduced when testosterone is suppressed.”

There are a couple of letter responses linked to this too.

link.springer.com/article/10.100…
Tommy @TLexercise has worked with transgender women with an eye specifically on sports.

academic.oup.com/jcem/article/1…

onlinelibrary.wiley.com/doi/10.1111/jo…
Read 6 tweets
Dec 8
Let’s have a think what hormone categories looks like. And let’s assume that @neiltyson is considering a high/low T category. This has also been proposed by @AliceDreger

Thanks for the shoutout, Colin @SwipeWright
The proposal only works if you don’t deny evolution and sexual selection. Remarkably, there are academics who argue there is no biological basis for why males run faster than females. While it is plausible ongoing underinvestment in female sport means female athletes have not yet reached their full potential, it is frankly ridiculous to think this can explain the entirety of the performance gap.

See Sheree Bekker et al for more details on why, because one time, this one female figure skater won a medal, Usain Bolt should be allowed to race against females.
The proposal only makes sense if we recognise that the action of T on a body gives advantage in sport. This is by no means universally-accepted. Many humanities types argue T is not a key part of sports performance, citing males with low T and people registered as female with high T. Even though both phenomena are explicable by factors like illness, doping and male DSDs, still this argument persists.

See Veronica Ivy, Katrina Karkazis et al for why we should pretend that the stupidly high prevalence of weightlifting males with low T is not because they have just finished an off-period jacking up.
Read 17 tweets
Nov 13
Why male advantage in sport is not a social construct: height.

Height is a key difference between males and females. What is nature v nurture? What does that mean for sport?Image
Bigger skeletons are most obviously driven by longer bone growth. Key bones like those in your thigh (“long bones”) grow from their end to get longer, making you taller. Image
The site of bone lengthening is called the “epiphyseal plate” or “growth plate”. Here, cells divide/enlarge, making new tissue that pushes the bone ends apart. This tissue calcifies and is replaced by bone, leading to lengthwise growth. Image
Read 21 tweets
Nov 11
Ok, my charity wears off.

Bekker’s presentation of the “Hilton and Lundberg” argument is nonsense.

At no point have either of us, or anyone else we work with, reduced male advantage to simply muscle mass/strength. @TLexercise @Scienceofsport Image
In the contrary, we have consistently argued that male advantage stems from many physical then functional outcomes of male development.

We spent hours (actually days 😂) creating this graphic, trying to highlight key areas of physicality that underpin male advantage.

HowTF is this reduced to “it’s all muscle”?Image
In our original paper, we had a table upfront, highlighting (in a less pleasing presentation) the same type of metrics. Image
Read 7 tweets
Nov 11
I’m going to put my charitable hat on, and try to elucidate - maybe even, as good practice, steelman - an opposition argument.

Specifically, this one: Image
Let’s set a concrete example: the 10 second barrier (100m sprint).

Wiki - allowing for small errors - tells me that around 200 male sprinters have broken it. We know, of course, that no female sprinter has been close (Flo Jo record 10.49s).
For the following, I’m going to ignore the premise that humans might be close to biomechanical limits over a 100m sprint. It’s just an illustration.

If we follow world record progressions, we see trends (not just in sprinting, the graph below is from a swimming event). Image
Read 18 tweets
Nov 4
As the latest on Olympic boxer Imane Khelif is reported, a diagnosis of 5ARD is almost certain. I and others first raised the likelihood of this DSD a few months ago.

Understanding how the developmental biology of DSDs interacts with sports categorisation is crucial.
I spoke about this with Andrew Gold during the competition:

And I recently gave a talk at a meeting, on DSDs, male advantage and sports categorisation. I will add some slides below.
Read 15 tweets

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