three short chapters on gastrointestinal hypo-motility in critical care.
๐ฃthis topic often gets *ignored* until there's a serious complication
๐ฃearly attention to motility can avoid iatrogenesis & facilitate recovery...
(thread #1/4)
ICU gastroparesis
๐คฎ manifests as tube feed intolerance (but don't assume that feeding intolerance = gastroparesis!)
๐คฎ a post-pyloric feeding tube can treat this nicely. otherwise erythromycin +/- metoclopramide
๐คฎ treat this- don't just watch/wait (#2/4) emcrit.org/ibcc/gastroparโฆ
ICU ileus
๐คฎprevention is key- avoid opioids, early enteral nutrition, early mobility๐
๐คฎNG drainage *only* if needed for symptomatic relief
๐คฎprokinetic meds don't work, but *oral* naloxone might help among patients on significant opioid doses (#3/4) emcrit.org/ibcc/ileus/
colonic pseudoobstruction (a.k.a. Oglivie's syndrome)
๐ฉ progressive dilation of the colon with risk of perforation
๐ฉ mainstay of therapy is IV neostigmine
๐ฉ get comfortable treating this - early neostigmine may prevent progression (#4/4) emcrit.org/ibcc/oglivie/
โข โข โข
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how to place a consult: you MUST understand the five stages of consultant grief.
once you can understand this painful and natural process, requesting consults will make a LOT more sense
buckle up, it can be a little roughโฆ
๐งต 1/6โฆ
stage 1: denial
- You dont need a consult.
- You called the wrong service.
- 18 years old? consult pediatrics
- Iโm not actually on call now
- Everythingโs fine, just walk it offโฆ
stage 2: anger
- you should have consulted us earlier/later
- you should have checked this test before calling us
- youโre a terrible doctor/student/human being
this is much better than MINDS (which contained ~90% hypoactive), but probably still not ideal.
(at this point, does anyone actually think that haloperidol helps with hypoactive delirium ??)
other than dilution of the patient population by patients with hypoactive delirium (who are unlikely to benefit & might conceivably be harmed by over-sedation), the methodology seems pretty solid.
I think it's time for a difficult discussion, folks.
Let's talk about CSF lactate ๐ซฃ
CSF lactate has been shown to be *superior* to traditional CSF studies in sorting out viral vs. bacterial meningitis in several studies & meta-analyses...
a subset of patients with viral meningitis will initially have a *neutrophilic* pleocytosis.
this can lead to unnecessary admissions & antibiotics
some patients are subjected to repeat LPs ๐ฉ
a low CSF lactate could avoid all of this, allowing patients to go home from the ED
CSF lactate measurement is recommended in guidelines from the United Kingdom, Europe, and France.
(it's not recommended in the ID society of America guidelines, but they're from *2004* and require revisions)