ncbi.nlm.nih.gov/pmc/articles/P…
Look like ACE2-anti-C1q ScFv will work!

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More from @flavinkins

17 Oct
@BillyBostickson @HL3133 @MonaRahalkar @BatResearch @franciscodeasis @Ayjchan @AntGDuarte @babarlelephant Most importantly, the so-called “amplicons” were published long after the problems with the ILLUMINA was exposed—that no assembly can be made for RaTG13 using the SRA. These amplicons have strange things hanging off the ends and are artifacts from gene synthesis assembly errors
@BillyBostickson @HL3133 @MonaRahalkar @BatResearch @franciscodeasis @Ayjchan @AntGDuarte @babarlelephant They have a publishing date almost a month after the problems with the raw data was tipped off in April. They were made up in May—one month, synthesized on 2 plates, one for the forward reads, one for the reverse reads. They then had a problem with the S1 and have to make the
@BillyBostickson @HL3133 @MonaRahalkar @BatResearch @franciscodeasis @Ayjchan @AntGDuarte @babarlelephant Last 4 amplicons in a hurry—the result was that these amplicons had random, non-vector and non-Biological sequences hanging off the ends.
Read 11 tweets
8 Oct
link.springer.com/article/10.100…
羟氯喹对中共病毒应配合溴己新使用。对流感,鼻病毒,腺病毒,人冠状病毒可单独使用。bi.tbzmed.ac.ir/Files/Inpress/…
治疗症状8mg每次3次每天。
配合羟氯喹200mg每天
Read 8 tweets
6 Oct
sciencedirect.com/science/articl…
CR3022 ScFv fused to c terminus of ACE2. with a long linker. Antibody induced dissasembly of virion. binding to the wrong epitope leading to the virus being ripped apart.
A reason behind the absence of FCS in wild Sarbecovirii. CR3022 popped out in the first SARS outbreak. it is highly effective and catalytic in the dissasembly of the prefusion Spike solely due to the presence of a Furin cleavage site (FCS) in the CoV2 S. imagine what will happen
in a BAT that had this CR3022 (which, the generally highly effective immune system of bats and wild animals nearly always do so. the Epitope on the RBD is conserved in all Sarbecovirii.).
Read 5 tweets
3 Oct
@CBS_Herridge @CBSNews ncbi.nlm.nih.gov/pmc/articles/P…

statnews.com/2020/05/14/gil…

pubs.acs.org/doi/10.1021/ac…
Remdesivir is highly toxic—not by necessity but by malicious design. GS-441524 is not only simpler, have the exact same theraputic effect, but is also much less toxic than remdesivir. Upon injection,
@CBS_Herridge @CBSNews Remdesivir almost immediately hydrolyzes into GS-441524, the effective nucleotide and highly toxic phenol and isoamyl alcohol. The latter two quickly homes into the liver and the kidney, destroying their function and causes essentially prescribed renal failure that is designed to
@CBS_Herridge @CBSNews Keep the patient on the ICU and promote ventilator and dialysis use. All of which generated massive amount of money in the form of medical fees for the hospital. ncbi.nlm.nih.gov/books/NBK54231…
Read 12 tweets
2 Oct
This is how both drugs work together—8mg/dose 3 doses/day Bromhexine to block surface route. 200mg HCQ/day in one dose to block endosomal route. Quercetin is optional for blocking the 3CLPro.
Read 4 tweets
1 Oct
pnas.org/content/early/…
Death blow to KG anderson(background: the Superantigen at the S1-S2 interface only work as an aglycosylated peptide—if glycosylated, it won’t bind to TCR. In fact, the majority of the S1-S2 insert’s novel, experimentally confirmed functions, require naked
S1-S2)
ImageImage
Read 4 tweets

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