1) SARS-CoV-2 (SPIKE) ANTIBODIES MAY CAUSE UNIVERSAL VIRAL ANTIBODY DEPENDENT ENHANCEMENT VIA FcR IMPAIRMENT. THIS ALSO EXPLAINS THE EMERGENCE OF MUCORMYCOSIS.

The immune response to the spike protein antibodies of SARS-CoV-2 may be causing severe FcR receptor dysfunction.
2) These are the receptors that mediate Antibody Dependent Enhancement in disease. A very telling sign is that mice which have FcR's knocked out are susceptible to LETHAL FUNGAL infections while having STRONG IMMUNE RESPONSES to other types of pathogens.

In spite of the
3) deficiency, FcγRIIb−/− mice show highly effective immune responses against several types of pathogens, including bacteria, plasmodia and mycobacteria. The fungal immune responses of FcγRIIb−/− mice have been inadequately tested, despite the high susceptibility to
4) several fungal infections in patients with SLE. Indeed, a high prevalence of cryptococcosis among Chinese patients with SLE has been reported, and FcγRIIb loss-of-function polymorphisms are commonly associated with SLE in Asian populations.

Impaired FcR receptors are also
5) involved in impaired anti-cancer immunity as well.

Again, will doctors and public health administrators demand a pause in spike protein therapies until studies can be done regarding SARS-CoV-2 spike protein antibodies and FcR receptor functioning.

nature.com/articles/srep4…

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More from @Parsifaler

12 Jul
1) ANTIBODIES AGAINST CARDIOLIPIN. THE SPIKE CAUSES THE IMMUNE SYSTEM TO CREATE THESE. THIS IS WHY WE SEE THE PROBLEMS WITH COAGULATION, DIABETES, NEURODEGENERATION AND CANCER.

SPIKE PROTEINS (AND THERAPIES) INDIRECTLY TELL THE BODY TO SELF-DESTRUCT.

COVID mimics everything.
2) This is why the disease has been so diffiult to pin down. But what is the MECHANISM of this mimicry? The spike protein causes antibodies to be formed against Cardiolipin.

Early in the pandemic in NYC in April 2020, two cases of severe disease were closely observed and
3) it was found that anticardiolipin antibodies were raised among both the cases and could be a key factor determining the course of the disease.

So, what does Cardiolipin do? Among other things, absence of it, or dysregluation of it causes MITOCHONDRIAL DYSFUNCTION and
Read 8 tweets
12 Jul
1) Burkholderia pseudomallei, biowarfare, spike proteins and a recent outbreak.

An interesting idea: There have been three documented cases of Burkholderia pseudomallei in the US recently, yet this bacteria is not endemic to the US nor have the three recent cases traveled
2) outside the US.

If you research Burkholderia pseudomallei you will find that it is often spoken of as a potential bioweapon, given its high (~50%) mortality rate and its ability to mimic a vast array of diseases. In fact, it is known as "the great mimicker."

Could a
3) molecular geneticist please examine the spike protein of SARS-CoV-2 and compare it to Burkholderia pseudomallei?

What I find most fascinating is that this bacteria has the same modus operandi as SARS-CoV-2s spike protein. It causes the formation of giant multinucleated cells
Read 7 tweets
11 Jul
1) HOLY. MOTHER. OF. GOD.

AN EXPLANATION FOR THE OCCURRENCE OF SPONTANEOUS ABORTION POST SPIKE PROTEIN THERAPY - AND FUTURE IMPLICATIONS

Anti-ANXA2 antibodies (Annexin A2) were detected by ELISA in the sera from 46 women with obstetric morbidity, mainly recurrent miscarriage.
2) Once these antibodies develop, it may be difficult for women to maintain the attachment of their babies to the wall of the womb, resulting in repeated miscarriages.

SPIKE PROTEIN S2 ANTIBODIES ARE ANNEXIN A2 (ANXA-2) ANTIBODIES!

pubmed.ncbi.nlm.nih.gov/20015551/
Read 4 tweets
9 Jul
1) ARE THE SIDE EFFECTS OF THE SPIKE PROTEIN THERAPIES ACTUALLY VASCULITIS AND NOT THE "IMMUNE SYSTEM WORKING?"

Given the recent and abundant evidence that the S1 subunit of the SARS-CoV-2 spike protein damages blood vessels, is it perhaps more than likely that the spike is
2) being cleaved and the cleaved S1 unit is attacking blood vessels?

If one notes the "side effects" of the spike protein therapies, they are PRECISELY the same as VASCULITIS:

Hay fever
Fever
Loss of appetite and weight loss
Joint and muscle pain
Fatigue
Cough
3) Abdominal pain and gastrointestinal bleeding
Weakness, fatigue or a general feeling of being unwell
Rash or skin sores
Pain, numbness and tingling in your hands and feet
Severe abdominal pain
Shortness of breath

If the blood vessels in the heart are being attacked by the
Read 6 tweets
9 Jul
1) URGENT! Has the spike protein been created to hijack dendritic cells to cross the blood brain barrier and invade the CNS? Literally commandeering the DCs like a cavalry?

The spike has even HIGHER binding affinity for DC-SIGN than ACE2. DC-SIGN is a recepton on dendritic
2) cells that allows for their manipulation. They INVADE the central nervous system during periods of SEVERE INFLAMMATION.

Besides endothelial cells of the BBB, another endothelial cell type that has been extensively studied in leukocyte migration is the high endothelial venules
3) (HEVs). These specialized postcapillary venules allow exit of blood cells into secondary lymphoid organs. The role of HEVs with respect to leukocyte trafficking into the CNS has not been well studied. HEV differentiation markers, however, have been shown to be expressed in
Read 5 tweets
6 Jul
1) PLATELET FACTOR 4 AUTOANTIBODIES: THE MOST IMPORTANT RISK FACTOR

I believe I have found the most important risk factor for COVID-19. The frequency of preexisting PF4 antibodies is estimated at 1.4% in normal, 4.5% in elderly, 4.8% in pregnant and 8.6% in diabetic subjects!
2) THE DAMAGE DONE BY THE SPIKE PROTEIN TO THE ENDOTHELIUM MIMICS ACUTE CORONARY SYNDROME. THIS ACTIVATES THE IMMUNE RESPONSE TO PF4 AND EXPLAINS WHY HEPARIN MAKES THE CONDITION WORSE!

To explain the preexistence of HIT antibodies, it was proposed that heparin-like molecules
3) (glycosaminoglycans) on injured endothelium bind
with PF4 to form complexes with which the antibodies
react. Neoantigens appeared to cause conformational
change of PF4 to induce the production of heterogenetic antibodies in the Ig subclass in the presence of heparin like
Read 6 tweets

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