1) SARS-CoV-2 INFECTION MIMICS ACUTE SPINAL CORD INJURY VIA SPIKE PROTEIN a7 nAChR ANTAGONISM RESULTING IN HPA AXIS DYSREGULATION AND IMMUNE SYSTEM PARALYSIS

Severe acute spinal cord injury in mice causes massive reduction in a7 nAChR receptors and cholinergic system disruption.
2) The image shows a7 nAChR receptors after acute spinal cord injury in mice compared to (A) control.

The spike antagonistically binds to a7 nAChR recptors, mimicking the cholinergic response to severe spinal cord injury (SCI) - a cascading effect. One of the major impacts of
3) this cascade is to the function of the adrenal gland. In particular, SCI causes hypothalamic-pituitary-adrenal (HPA) dysregulation.

In mice, this dysregulation resulted in devastating immune cell death.

Interestingly, in this situation, “deactivation” of the adrenal glands
4) in mouse models was tried. This returned immune cell levels back to normal but failed to protect the mice from infection. However, transplanting healthy adrenal tissue into the affected mice did provide protection. This is because this tissue can produce necessary hormones
5) without being under the influence of the nervous system control mechanisms that are malfunctioning.

Again, the spike protein therapies must be paused while this interaction is investigated.

This explains the rapid deterioration in cases ~7 days post onset. The immune cells
6) have become hormonally paralyzed.

In fact, Hypocortisolism has been observed in recovering COVID patients, particularly when they should have been getting better.

Therapeutics addressing adrenal dysfunction may prove beneficial in treating and preventing severe COVID.
7) The Spike Protein may cause the nervous system damage by direct infection, as well.

ncbi.nlm.nih.gov/pmc/articles/P…

pubmed.ncbi.nlm.nih.gov/33521254/

nature.com/articles/nn.46…
8) This may be critical: The only thing that saved the SCI mice were new adrenal glands...

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More from @Parsifaler

20 Jul
1) HEAT SHOCK PROTEINS AND THE SPIKE

Prions and immune deificiency emerging simultaneously. Fungi and viral reactivation. The spike, I believe, is also affecting Heat Shock Proteins.

The fundamental problem of autoimmune diseases is the failure of the immune system to Image
2) downregulate its own potentially dangerous cells, which leads to destruction of tissue expressing the relevant autoantigens. Current immunosuppressive therapies offer relief but fail to restore the basic condition of self-tolerance. They do not induce long-term physiological
3) regulation resulting in medication-free disease remissions. Heat shock proteins (HSPs) have shown to possess the capacity of inducing lasting protective immune responses in models of experimental autoimmune diseases. Especially mycobacterial HSP60 and HSP70 were shown to
Read 8 tweets
15 Jul
1) SARS-CoV-2 (SPIKE) ANTIBODIES MAY CAUSE UNIVERSAL VIRAL ANTIBODY DEPENDENT ENHANCEMENT VIA FcR IMPAIRMENT. THIS ALSO EXPLAINS THE EMERGENCE OF MUCORMYCOSIS.

The immune response to the spike protein antibodies of SARS-CoV-2 may be causing severe FcR receptor dysfunction.
2) These are the receptors that mediate Antibody Dependent Enhancement in disease. A very telling sign is that mice which have FcR's knocked out are susceptible to LETHAL FUNGAL infections while having STRONG IMMUNE RESPONSES to other types of pathogens.

In spite of the
3) deficiency, FcγRIIb−/− mice show highly effective immune responses against several types of pathogens, including bacteria, plasmodia and mycobacteria. The fungal immune responses of FcγRIIb−/− mice have been inadequately tested, despite the high susceptibility to
Read 6 tweets
12 Jul
1) ANTIBODIES AGAINST CARDIOLIPIN. THE SPIKE CAUSES THE IMMUNE SYSTEM TO CREATE THESE. THIS IS WHY WE SEE THE PROBLEMS WITH COAGULATION, DIABETES, NEURODEGENERATION AND CANCER.

SPIKE PROTEINS (AND THERAPIES) INDIRECTLY TELL THE BODY TO SELF-DESTRUCT.

COVID mimics everything.
2) This is why the disease has been so diffiult to pin down. But what is the MECHANISM of this mimicry? The spike protein causes antibodies to be formed against Cardiolipin.

Early in the pandemic in NYC in April 2020, two cases of severe disease were closely observed and
3) it was found that anticardiolipin antibodies were raised among both the cases and could be a key factor determining the course of the disease.

So, what does Cardiolipin do? Among other things, absence of it, or dysregluation of it causes MITOCHONDRIAL DYSFUNCTION and
Read 8 tweets
12 Jul
1) Burkholderia pseudomallei, biowarfare, spike proteins and a recent outbreak.

An interesting idea: There have been three documented cases of Burkholderia pseudomallei in the US recently, yet this bacteria is not endemic to the US nor have the three recent cases traveled
2) outside the US.

If you research Burkholderia pseudomallei you will find that it is often spoken of as a potential bioweapon, given its high (~50%) mortality rate and its ability to mimic a vast array of diseases. In fact, it is known as "the great mimicker."

Could a
3) molecular geneticist please examine the spike protein of SARS-CoV-2 and compare it to Burkholderia pseudomallei?

What I find most fascinating is that this bacteria has the same modus operandi as SARS-CoV-2s spike protein. It causes the formation of giant multinucleated cells
Read 7 tweets
11 Jul
1) HOLY. MOTHER. OF. GOD.

AN EXPLANATION FOR THE OCCURRENCE OF SPONTANEOUS ABORTION POST SPIKE PROTEIN THERAPY - AND FUTURE IMPLICATIONS

Anti-ANXA2 antibodies (Annexin A2) were detected by ELISA in the sera from 46 women with obstetric morbidity, mainly recurrent miscarriage.
2) Once these antibodies develop, it may be difficult for women to maintain the attachment of their babies to the wall of the womb, resulting in repeated miscarriages.

SPIKE PROTEIN S2 ANTIBODIES ARE ANNEXIN A2 (ANXA-2) ANTIBODIES!

pubmed.ncbi.nlm.nih.gov/20015551/
Read 4 tweets
9 Jul
1) ARE THE SIDE EFFECTS OF THE SPIKE PROTEIN THERAPIES ACTUALLY VASCULITIS AND NOT THE "IMMUNE SYSTEM WORKING?"

Given the recent and abundant evidence that the S1 subunit of the SARS-CoV-2 spike protein damages blood vessels, is it perhaps more than likely that the spike is
2) being cleaved and the cleaved S1 unit is attacking blood vessels?

If one notes the "side effects" of the spike protein therapies, they are PRECISELY the same as VASCULITIS:

Hay fever
Fever
Loss of appetite and weight loss
Joint and muscle pain
Fatigue
Cough
3) Abdominal pain and gastrointestinal bleeding
Weakness, fatigue or a general feeling of being unwell
Rash or skin sores
Pain, numbness and tingling in your hands and feet
Severe abdominal pain
Shortness of breath

If the blood vessels in the heart are being attacked by the
Read 6 tweets

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