1) AN ACT OF WAR: HYPERMETABOLISM MULTIPLE ORGAN FAILURE SYNDROME. ARDS ASSOCIATED WITH COVID-19 IS NOT DUE TO LUNG INFECTION.

The pathophysiology of ARDS was not confined to lungs but is part of a systemic injury-response pattern now known as the hypermetabolism-organ failurr
2) syndrome.

THIS IS WHY PATIENTS SUDDENLY DETERIORATE ~ SEVEN DAYS INTO THE DISEASE, IF NOT CLEARED. THE BODY CANNOT KEEP UP WITH THE INDUCED HYPERMETABOLISM. METFORMIN IS USED TO TREAT HYPERMETABOLISM!

This particular syndrome was observed looking at those traumatized in WAR.
3) Does this explain the toxins? It seems the bioweapon is designed to REPLICATE SEVERE TRAUMA! Thereby inducing the hypermetabolism syndrome resulting in ARDS and multiple organ failure.

Is this also the purpose of the SPIKE PROTEIN?

What happens to the body ALSO happens in
4) SEVERE BURN PATIENTS.

SARS-CoV-2 INDUCES THE METABOLIC EFFECTS OF SEVERE TRAUMA. WE ARE TREATING IT WRONG. WE NEED TO TREAT PATIENTS AS TRAUMA PATIENTS, NOT INFECTED PATIENTS.

Please read the attached papers.

nature.com/articles/s4159…

journal.chestnet.org/article/S0012-…

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More from @Parsifaler

22 Jul
1) WHERE WE ARE AT. AN ASSESSMENT.

Is COVID-19 not so much an infection, but an injury? Could we compare it to a Rattlesnake bite, where the fangs (virus) are not removed from the body, allowing for continual envenomation?

The evidence is overwhelming about the toxicity of the
2) spike protein. So why "inject" it? Are we not injecting the very substance that causes injury?

It would seem that the intense binding of the spike to ACE2/cleaving of S1 could be the ENTIRE pathology of COVID-19. To quote a great researcher I know "If there is no TMPRSS2,
3) there is no game," which, the more I reflect on it, is ever so true. If the spike cannot cleave, not only is there no infection, there is no injury.

Deterioration about day seven is most likely when, if the body has not cleared the virus, the Peine dur et Forte of the spike
Read 5 tweets
22 Jul
1) ANSWERED! RT ASAP! I BELIEVE THE SPIKE PROTEIN MIMICS SEVERE TRAUMA BY ATTACKING THE ACE 2 RECEPTORS IN THE ENDOTHELIUM AND BLOOD BRAIN BARRIER, COMPROMISING THEIR INTEGRITY. THE BODY BELIEVES IT IS BLEEDING TO DEATH, EVERYWHERE. THIS IS THE IMMENSE DANGER OF INJECTING SPIKE Image
2) PROTEINS, OR HAVING THE BODY CREATE THEM.

THE IMMENSE BINDING AFFINITY TO ACE 2 MIMICS BLUNT FORCE!

In independent, but small, studies of trauma patients, we found that (i) high circulating syndecan-1, a marker of endothelial glycocalyx degradation,15 was associated with
3) inflammation, hypocoagulability, and increased mortality; (ii) the trauma-induced catecholamine surge was closely associated with biomarkers of tissue and endothelial damage, glycocalyx degradation, hypocoagulability including hyperfibrinolysis and independently predicted
Read 6 tweets
22 Jul
1) Evidence SARS-CoV-2 Mimics Trauma. Post-Traumatic Fatal Granulomatosis with Polyangiitis and Disseminated Intravascular Coagulation and Sustained Systemic Inflammatory Response Syndrome Predict Organ Dysfunctions After Trauma. Fatal Fungal Infection Post Trauma.

The image Image
2) attached looks EXACTLY like COVID pneumonia, doesn't it? It is actually an image taken after thoracic trauma. The physicians were so stunned by the similarity that they published a case report on it.

They found it curious. I find it definitive.

What is also definitive are Image
3) the findings of post traumatic disseminated intravascular coagulation and sustained systemic inflammatory response syndrome post trauma. Four days post trauma fibroningen levels are very high, and this leads to clotting issues. In fact, this is indicative of the further
Read 7 tweets
21 Jul
1) FIVE ALARM FIRE: TOXINS AND PRIONS

PROTEOTOXIC STRESS. MITOCHONDRIAL UNFOLDED PROTEIN RESPONSE. CACHEXIA. A UNIFYING THEORY FOR ALL OBSERVED OVER THE PAST 18 MONTHS.

WE ARE DEALING WITH A BIOWEAPON THAT WAS BEING DEVELOPED TO METABOLICALLY BURN ITS VICTIMS ALIVE .

We have Image
2) been looking at the wrong Unfolded Protein Response. The Mitochondrial Unfolded Protein Response (mtUPR) which is most important in SARS-CoV-2 infection.

Skeletal muscle cachexia resulting from a chronic imbalance between protein synthesis and breakdown leads to
3) significant long-term morbidity in burn survivors.

The data indicates profound PROTEOTOXIC stress within the mitochondria environment and, most importantly, suggest that this stress is transduced to the nucleus to help combat the accumulation of unfolded proteins within
Read 6 tweets
20 Jul
1) HEAT SHOCK PROTEINS AND THE SPIKE

Prions and immune deificiency emerging simultaneously. Fungi and viral reactivation. The spike, I believe, is also affecting Heat Shock Proteins.

The fundamental problem of autoimmune diseases is the failure of the immune system to
2) downregulate its own potentially dangerous cells, which leads to destruction of tissue expressing the relevant autoantigens. Current immunosuppressive therapies offer relief but fail to restore the basic condition of self-tolerance. They do not induce long-term physiological
3) regulation resulting in medication-free disease remissions. Heat shock proteins (HSPs) have shown to possess the capacity of inducing lasting protective immune responses in models of experimental autoimmune diseases. Especially mycobacterial HSP60 and HSP70 were shown to
Read 8 tweets
18 Jul
1) SARS-CoV-2 INFECTION MIMICS ACUTE SPINAL CORD INJURY VIA SPIKE PROTEIN a7 nAChR ANTAGONISM RESULTING IN HPA AXIS DYSREGULATION AND IMMUNE SYSTEM PARALYSIS

Severe acute spinal cord injury in mice causes massive reduction in a7 nAChR receptors and cholinergic system disruption.
2) The image shows a7 nAChR receptors after acute spinal cord injury in mice compared to (A) control.

The spike antagonistically binds to a7 nAChR recptors, mimicking the cholinergic response to severe spinal cord injury (SCI) - a cascading effect. One of the major impacts of
3) this cascade is to the function of the adrenal gland. In particular, SCI causes hypothalamic-pituitary-adrenal (HPA) dysregulation.

In mice, this dysregulation resulted in devastating immune cell death.

Interestingly, in this situation, “deactivation” of the adrenal glands
Read 9 tweets

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