1/The Spike Protein vaccines have potential to cause lasting states of inflammation that will affect longevity. In every immunogenic protein sequence that illicits t or b cell response, there are fragments of other pathogens such as proteobacteria.
2/ These fragments are either a result of unique convergent evolution of fungi, bacteria and virus genomes in an animal such as a bat that has survived millions of years in very "unsanitary" conditions. Or, as a result of bioengineered manipulation.
3/ The human immune system identifies a pathogen by the patterns of amino acids in these sequences. Our own evolution has programmed us to respond to these sequences with specific bio-chemical or immune cell signaling and activation.
4/ The Spike Protein vaccines will not only program our immune system to respond to the viral proteins but it will also program us to also respond to encountering bacteria and fungi which share homology with the Spike Protein sequence.
5/ The vaccine will program in "allergic reactions" to bacteria and fungi that we never had before because we are not genetically programmed to respond to pathogens that are found in a "bat cave" because we don't live in a "bat cave."
6/ Immune inflammatory response, whether pathogenic or auto-immune, is the ENEMY of health and longevity. These concepts are particularly important in a decision to vax a child whose adaptive immune system is being programmed to respond to the environment.
7/ We are risking causing brand new forms of allergy, asthma, and auto-immune disease in children. And we will not recognize that the response is to pathogens that were not in the past a cause of inflammatory response.
8/ Everything that I observe and report is DEFINITELY known by someone in our government health systems. What is the "risk taking" about? We do not need a vax for a <1% mortality in the not elderly. We are creating "altered" evolutionary immune systems that span generations.
8/ Here is an example. Image

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More from @Ducky68257909

24 Sep
I have just completed some research notes on the possible role of neutrophils and neutrophile elastase in COVID19. And the potential for elastase inhibitors for COVID therapies. I am continuing to update as I see more. If you would like to have this PDF, send me an email by DM.
Nagata N, Iwata-Yoshikawa N, Taguchi F. Studies of severe acute respiratory syndrome coronavirus pathology in human cases and animal models. Vet Pathol. 2010 Sep;47(5):881-92. doi: 10.1177/0300985810378760. Epub 2010 Jul 27. PMID: 20664013.
Here is the citation for the photo: Barnes et al: Targeting potential drivers of COVID-19: Neutrophil extracellular traps. J Exp Med 1 June 2020; 217 (6): e20200652. doi: doi.org/10.1084/jem.20…
Read 6 tweets
2 Sep
My opinion regarding COVID19 being a 💉💉 in progress for HIV doesn't involve the fragments of HIV that are causing all the controvery. More 👇 /1 connexionfrance.com/French-news/Di…
2/ Interesting to see what people admit and deny. Pasteur says yes there ARE HIV fragments but they are meaningless. I don't know one way or the other. The length of the amino acid fragments - don't help my theory really. pasteur.fr/en/home/resear… Image
3/ Papers on HIV 💉 research showed that they had knowledge of effective HIV antibody producing epitopes of certain important categories but they lacked CD8 t-cell epitopes which were specific to the genetic HLA-alleles for specific populations. There are NO universal epitopes. Image
Read 24 tweets
1 Sep
I am adding another phenomena. Macrophages recycle iron, a very important function. Cytokine storm completely debilitates macrophages in an ultra-inflammatory state - renders then unable to function in this role. COVID treatment MUST lower the level of cytokines to improve O2.
Then if you have high levels of Interleukin 6, you have a hepcidin caused anemia. We all know that severe COVID is associated with high IL-6. bmccancer.biomedcentral.com/articles/10.11…
Read 4 tweets

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