Walter M Chesnut Profile picture
Oct 13, 2021 17 tweets 5 min read Read on X
1) THE SPIKE PROTEIN: A PHARMACOLOGICAL “THERAPEUTIC” INTENDED TO INDUCE FATAL SYSTEMIC IRON DEPOSITION: DYSMETABOLIC HYPERFERRTINEMIA
First, please think about all we have read concerning COVID and its sequelae. Now please read this: Iron overload, irrespective of the underlying Image
Image
2) etiology, has varying manifestations, depending on the organs affected by the excessive iron deposit. It may present as fatigue, skin color changes, abdominal pain, joint pain, irregular menstruation, infertility, impotence, irregular heart rhythm, heart failure, new-onset Image
3) diabetes or difficulty controlling established diabetes and elevation in liver enzymes.
And what of Hypoxia? Pathological? Certainly. Also, a possible adverse effect of a drug intended to induce fatal systemic iron deposition. While we have been scratching our heads trying to
4) figure out WHY the HYPOXIA seen in COVID is UNRELATED to our LUNGS, the spike protein has been going about its “merry” business of forcing ever more iron uptake and deposition.
Both the rate of erythropoiesis and hypoxia regulate iron absorption. Expression of ferroportin and
5) Dcytb are upregulated in hypoxia and in a hypotransferrinaemic mouse which has chronic anaemia due to defective erythropoiesis. Increased expression of these genes is likely to account for the increase in iron absorption.
What most people, including many doctors, do not
6) realize is that the inherited disease Hemochromatosis is not the only cause of Iron Overload and Iron Deposition. There is also another condition, which I believe we are observing: Dysmetabolic Hyperferritinemia.
Dysmetabolic hyperferritinemia, also known as insulin resistance
7) associated with iron overload, is a much more common disorder than recognized clinically by physicians.
While we have been dancing around the related CNS involvement and apparent neurodegeneration, let’s spell it out now, once and for all: Iron dyshomeostasis appears to be a
8) central factor in neurodegenerative conditions such as AD, PD, ALS, Huntington’s disease, and Friedreich’s ataxia. For decades, deposits of iron have been detected in brain lesions of patients with these neurodegenerative conditions. How iron accumulates in the
9) diseased/injured brain is not known, however, heme-bound iron that is derived from the circulation and non-heme bound iron (i.e., all iron not bound to heme protein) are both present in affected tissue. It is likely that the deposition of iron into the brain parenchyma in
10) these conditions is the result of either hemorrhage/microbleeds (i.e., heme-bound iron), damaged cells (i.e., neurons, oligodendrocytes and microglia), and myelin, inflammatory processes, degradation of erythrocytes and heme proteins, and/or dysregulation of important
11) iron-related proteins (e.g., ceruloplasmin, ferroportin).
We have witnessed just in the past few days the collapse of Astra Zeneca’s Director of Medicines and an Austrian MP live on camera. This can be attributed to Cardiac Iron Overload. Deposition of iron may occur in the
12) entire cardiac conduction system, especially the atrioventricular node. Complete atrioventricular block caused by iron depostion may need implacement of a permanent pacemaker [8]. Iron deposition in the cardiac tissue causes nonhomogenous electrical conduction and
13) repolarization with atrial and ventricular tachyarrhythmias. Chronic iron overload reduces CaV1.3-dependent L-type Ca2+ currents, resulting in bradycardia, altered electrical conduction, and atrial fibrillation. Paroxysmal atrial fibrillation is the most common arrhythmia
14) observed in patients with cardiac hemochromatosis. The prevalence of ventricular arrhythmias increases with left ventricular dilation and low LVEF. Sudden cardiac death may develop.
I believe iron is also being deposited in the pancreas, as the cases of new onset diabetes
15) indicates. Excessive iron storage sometimes causes diabetes in patients with hemochromatosis, a disease caused by iron overloading.
I believe all organs and systems of the body are being overloaded with iron by the Hepcidin/Iron Metabolism interference of the Spike Protein.
16) Studies must be conducted ASAP to determine post spike protein therapy iron biomarkers. I urge a pause on all spike protein therapies until such studies may be conducted. The cost of antibodies for a disease with a 99%+ survival rate may be the ultimate cost.

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More from @Parsifaler

Jan 23, 2023
1) Although decreased translation fidelity causes protein misfolding and aggregation in both cardiomyocytes and Purkinje cells, the downstream pathways that lead to cell death in the two cell types may be quite different. Interestingly, a point mutation in the editing domain of
2) human mitochondrial AlaRS (AARS2) has been associated with infantile mitochondrial cardiomyopathy, suggesting increased errors in either cytoplasmic or mitochondrial protein synthesis can lead to cell death in the heart. In summary, our data show that a global reduction in
3) translational fidelity, rather than disruption of a specific protein, can induce defects in proteostasis in numerous cell types in the mouse. It is thus possible that proteinopathy can occur in the brain, heart, or even other tissues as a combination of genetic and/or
Read 4 tweets
Jan 3, 2023
On Damar Hamlin and Dr. Sutterer's "diagnosis" of Commodio Cordis
I am shocked that Commodio Cordis (CC) is named as the cause. Please examine the images carefully. In CC Ventricular fibrillation can be triggered by chest wall IMPACT ONLY OVER THE HEART, and predominantly occurs
with impact over the center of the left ventricle. Although CC usually involves impact from a baseball, it has also been reported during hockey, softball, lacrosse, karate, and other sports activities in which a relatively hard and compact projectile or bodily contact caused
impact to the person's precordium. Are we to believe that with the padding that professional football players wear, the location of the heart and the location of the impact, that this was actually CC?

Nonsense. Rapidly stated, narrative, ARRANT NONSENSE.
Read 6 tweets
Apr 11, 2022
1) UPDATED SYNTHESIS: EXTREMELY URGENT
IT’S COMPLICATED, BUT CLEAR – LOOKING ONE MOVE DEEPER
THE SPIKE PROTEIN PATHOGENIC ALGORITHM – DUAL PATHS TO TERMINAL SYSTEMIC FIBROSIS: IMMEDIATE FOR THOSE WITH SIGNIFICANT COMORBIDITIES, INDUCED FOR THOSE WITHOUT
The Spike Protein is Image
2) inducing terminal systemic fibrosis of all organs, including the blood, via two principal mechanisms.
The first is a direct, immediate path via binding to RGD-binding integrins, which includes several TGF-β -activating integrins. This this activates Myofibroblasts which ImageImageImage
3) induces Fibrosis. Indeed, in autopsies of COVID-19 patients with advanced disease, 38% collagen deposition was found in their lungs.
This is a rapid and certainly fatal circumstance.
But, this is not limited to the lungs. In a series of cardiac autopsies conducted in
Read 12 tweets
Apr 8, 2022
1) MORE THAN AMYLOIDOSES. ALL HUMAN TISSUES ARE BEING TRANSFORMED INTO FIBROUS MASSES. INCLUDING. THE. BLOOD.
AMYLOIDOSIS. FIBROSIS. | AUTOPSIES. INCREASED ORGAN WEIGHT. | CLOTS. AMYLOIDS.
The tissues of the body, INCLUDING THE BLOOD, are being either DEPOSITED WITH FIRBILS OR ImageImage
2) BEING TRANSFORMED INTO FIBRILS. Amyloidosis > Deposition of Fibrils. Fibrosis > Transformation into Fibrils.
The Spike Protein is transforming (at least) all human tissue (perhaps other species?) into non-functioning fibrous masses.
I was reading papers in the solarium after
3) dinner while having a cigar. And I kept thinking about Amyloidosis and Fibrosis. I have seen both. Is it one? Is it the other? After reading more papers – AND SOME AUTOPSY REPORTS - I came to a startling and incredibly disturbing conclusion. It is BOTH!
THE CLOTS WE ARE
Read 9 tweets
Apr 8, 2022
1) THE SPIKE PROTEIN IS THE “AMYLOID” BEING DEPOSITED AND INDUCING AMYLOIDOSES: A MAJOR FINDING MISSED
SEVERE COVID MAY BE DUE TO THE ADDED DEPOSITION OF COMPLEMENT WITH THE SPIKE
The paper “The histologic and molecular correlates of COVID-19 vaccine-induced changes in the skin” ImageImageImage
2) made a case for the immune response to the Spike Protein causing self-limited hypersensitivity reactions to the vaccine. However, if you study the paper carefully, you notice that the authors have missed a far more important finding.
The biopsy specimens of normal skin post
3) vaccine and of skin affected by the post-vaccine eruption showed rare deep microvessels positive for spike glycoprotein with no complement deposition contrasting with greater vascular deposition of spike protein and complement in skin biopsies from patients experiencing severe
Read 6 tweets
Apr 4, 2022
1) PLEASE READ THIS ENTIRE THREAD. IT IS URGENT.
Building on all recent work. By far my most important synthesis:
A NEW KIND OF CANCER: THE MARRIAGE OF ONCOGENESIS AND PRIONOPATHY
THE PROGRESSIVE DESTRUCTION OF TISSUE AND ORGANS BY THE AMYLOIDOGENIC AND OLIGOMERIC PROPERTIES OF Image
2) THE SARS-CoV-2 SPIKE PROTEIN
It has been proposed that Alzheimer's disease might be a 'whole body' problem, as amyloid-beta can travel, cancer-like, to brain from other parts of body.
Normal mice that had been joined (via bloodstreams) to genetically modified partners for a
3) year "contracted" Alzheimer's disease. The researcher song says the amyloid-beta traveled from the genetically-modified mice to the brains of their normal partners, where it accumulated and began to inflict damage.
Not only did the normal mice develop plaques, but also a
Read 11 tweets

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