1) THE SPIKE PROTEIN: A PHARMACOLOGICAL “THERAPEUTIC” INTENDED TO INDUCE FATAL SYSTEMIC IRON DEPOSITION: DYSMETABOLIC HYPERFERRTINEMIA
First, please think about all we have read concerning COVID and its sequelae. Now please read this: Iron overload, irrespective of the underlying
2) etiology, has varying manifestations, depending on the organs affected by the excessive iron deposit. It may present as fatigue, skin color changes, abdominal pain, joint pain, irregular menstruation, infertility, impotence, irregular heart rhythm, heart failure, new-onset
3) diabetes or difficulty controlling established diabetes and elevation in liver enzymes.
And what of Hypoxia? Pathological? Certainly. Also, a possible adverse effect of a drug intended to induce fatal systemic iron deposition. While we have been scratching our heads trying to
4) figure out WHY the HYPOXIA seen in COVID is UNRELATED to our LUNGS, the spike protein has been going about its “merry” business of forcing ever more iron uptake and deposition.
Both the rate of erythropoiesis and hypoxia regulate iron absorption. Expression of ferroportin and
5) Dcytb are upregulated in hypoxia and in a hypotransferrinaemic mouse which has chronic anaemia due to defective erythropoiesis. Increased expression of these genes is likely to account for the increase in iron absorption.
What most people, including many doctors, do not
6) realize is that the inherited disease Hemochromatosis is not the only cause of Iron Overload and Iron Deposition. There is also another condition, which I believe we are observing: Dysmetabolic Hyperferritinemia.
Dysmetabolic hyperferritinemia, also known as insulin resistance
7) associated with iron overload, is a much more common disorder than recognized clinically by physicians.
While we have been dancing around the related CNS involvement and apparent neurodegeneration, let’s spell it out now, once and for all: Iron dyshomeostasis appears to be a
8) central factor in neurodegenerative conditions such as AD, PD, ALS, Huntington’s disease, and Friedreich’s ataxia. For decades, deposits of iron have been detected in brain lesions of patients with these neurodegenerative conditions. How iron accumulates in the
9) diseased/injured brain is not known, however, heme-bound iron that is derived from the circulation and non-heme bound iron (i.e., all iron not bound to heme protein) are both present in affected tissue. It is likely that the deposition of iron into the brain parenchyma in
10) these conditions is the result of either hemorrhage/microbleeds (i.e., heme-bound iron), damaged cells (i.e., neurons, oligodendrocytes and microglia), and myelin, inflammatory processes, degradation of erythrocytes and heme proteins, and/or dysregulation of important
11) iron-related proteins (e.g., ceruloplasmin, ferroportin).
We have witnessed just in the past few days the collapse of Astra Zeneca’s Director of Medicines and an Austrian MP live on camera. This can be attributed to Cardiac Iron Overload. Deposition of iron may occur in the
12) entire cardiac conduction system, especially the atrioventricular node. Complete atrioventricular block caused by iron depostion may need implacement of a permanent pacemaker [8]. Iron deposition in the cardiac tissue causes nonhomogenous electrical conduction and
13) repolarization with atrial and ventricular tachyarrhythmias. Chronic iron overload reduces CaV1.3-dependent L-type Ca2+ currents, resulting in bradycardia, altered electrical conduction, and atrial fibrillation. Paroxysmal atrial fibrillation is the most common arrhythmia
14) observed in patients with cardiac hemochromatosis. The prevalence of ventricular arrhythmias increases with left ventricular dilation and low LVEF. Sudden cardiac death may develop.
I believe iron is also being deposited in the pancreas, as the cases of new onset diabetes
15) indicates. Excessive iron storage sometimes causes diabetes in patients with hemochromatosis, a disease caused by iron overloading.
I believe all organs and systems of the body are being overloaded with iron by the Hepcidin/Iron Metabolism interference of the Spike Protein.
16) Studies must be conducted ASAP to determine post spike protein therapy iron biomarkers. I urge a pause on all spike protein therapies until such studies may be conducted. The cost of antibodies for a disease with a 99%+ survival rate may be the ultimate cost.

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More from @Parsifaler

14 Oct
1) AN UNEXPECTED, YET FASCINATING TURN
THE SPIKE PROTEIN, THROUGH HEPCIDIN MIMICRY, INDUCES IRON OVERLOAD AND MITOCHONDRIAL PROTON/ELECTRON LEAK: THE OBSERVED “RADIATION POISONING” IS THIS PROTON/ELECTRON LEAK: CACHEXIA, AGING, ENDOTHELIAL AND CARDIOVASCULAR COVID PATHOLOGIES
2) EXPLAINED
Hepcidin: The Dark Crystal
Hepcidin, a small polypeptide synthesized and secreted by the liver, is considered a master regulator of body iron homeostasis. The physiological function of hepcidin is to decrease circulating iron levels through down-regulation of FPN1 in
3) macrophages. The action of hepcidin is tissue-specific: in enterocytes, hepcidin does not change FPN1 levels but decreases DMT1 expression. Hepcidin-induced FPN1 degradation is transient reaching a maximal effect at 2–6 h, with FPN1 levels returning to control values at 24.
Read 23 tweets
13 Oct
1) COVID AND TRANSFERRIN: UNITING IRON OVERLOAD, COAGULOPATHY, AGING, SENESCENCE, AND SHORTENED TELOMERES
A paper from May 2021 out of Wuhan noted that a previous study in Austria found that ferritin and transferrin were both associated with the risk for ICU admission in COVID-19
2) patients, and no significant relations were found in iron and other indices. This study indicated the relation between iron metabolism and the undesirable outcome of COVID-19, whereas the association with specific comorbidities warrants further investigation. In our study, we
3) found high serum iron level was associated with disease severity in COVID-19 patients.
Transferrin is a protein that binds iron and transports it throughout the body. It is the main iron carrier in the blood. Transferrin levels normally increase with iron deficiency. When iron
Read 12 tweets
12 Oct
1) LONG COVID AS A DISEASE OF (ACUTE?) IRON OVERLOAD
I will explore various aspects of Long COVID in the context of iron overload disease. We will now discuss iron overload and the weakness/exercise intolerance/weight loss/cachexia occurring in Long COVID.
Previously healthy mice
2) were injected with iron. The results are remarkably similar to what happens in Long COVID. If the body has too much iron, it upregulates Ferritin. Therefore, high ferritin in the ABSENCE of high serum iron may indicate that the muscles and organs are already overloaded.
Please
3) reference the graphic showing the ferritin levels of the mice injected with iron vs control.
How does this present clinically? Fatigue, exercise intolerance and muscle atrophy, which presents as weight loss. Please reference the graphic showing weight loss of the iron injected
Read 6 tweets
12 Oct
What if the spike protein, through its mimcry of Hepcidin and affinity for ACE2 receptors, caused such a massive efflux of iron into the bloodstream, that it mimicked an "iron injection?"

There is such a drug! Iron Dextran Injection.

What can happen?

Iron dextran injection may
cause severe or life-threatening reactions while you receive the medication. You will receive this medication in a medical facility and your doctor will watch you carefully during each dose of iron dextran injection. Tell your doctor if you experience any of the following
symptoms during or after your injection: shortness of breath; difficulty swallowing or breathing; wheezing; hoarseness; swelling of the face, throat, tongue, lips, or eyes; hives; itching; rash; fainting; lightheadedness; dizziness; bluish discoloration of the skin, lips,
Read 5 tweets
11 Oct
1) DELAYED SELF-POISONING (OR A QUICK DEATH VIA MACROPHAGE ACTIVATION SYNDROME)
THE SPIKE PROTEIN OF SARS-CoV-2 IS AN ENGINEERED BIOWEAPON DESIGNED TO CREATE MASSIVE, ALMOST CERTAINLY LETHAL (DELAYED, AS IS THE CASE WITH EXTREME IRON OVERLOAD) IRON EFFLUX FROM MACROPHAGES AND
2) RBCs. THE OBSERVED HYPOXIA IS PART OF THIS “PLAN.” THE BODY RELEASES YET MORE IRON UNDER HYPOXIC CONDITIONS: THE PARACRINE PARALLEL TO RADIATION POISONING.
A fascinating article appeared on ideas.ted.com in December of 2015. The chemist Rebecca Abergel realized the
3) way to cure people of radiation poisoning was to remove it from the body, as it spreads in a PARACRINE fashion.
At high doses, radiation blasts through tissues, ruptures DNA strands and alters the rhythms of cell division. Disrupted cells cause nausea, diarrhea and fever,
Read 11 tweets
9 Oct
1) COVID-19 IS AN IRON OVERLOAD-INDUCED STEM CELL DISEASE. IT IS FAR MORE DANGEROUS THAN ANYONE HAS PREVIOUSLY REALIZED. IF INTERFERON GAMMA CALLS ON hPSCs FOR “REINFORCEMENTS,” THE “TROOPS” SENT ARE DEFECTIVE. THIS IS WHY PEOPLE DIE. THJS IS WHY LONG COVID'S
2) CANCER/NEURODEGENERATION, ETC., DOES NOT MANIFEST FOR WEEKS/MONTHS POST SPIKE PROTEIN EXPOSURE. IT IS ONLY AFTER THE HEALTHY CELLS DIE AND ARE REPLACED WITH DEFECTIVE CELLS.
Interferon Gamma has been the Dr. Jekyll and Mr. Hyde of the COVID-19 pandemic. In moderate disease,
3) it has been found to be extremely therapeutic. In severe disease, it can be a marker for a fatal outcome. This is because it is the canary in the coal mine of COVID-19.
A paper from the Department of Pharmacy at the Second Affiliated Hospital - Harbin Medical University in
Read 11 tweets

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