I have been pondering on the Sacroiliac Joint and some clinicians fascination with it. I think that it is the source of more “evidence based ostriches” than any other joint.
Yes a brief thread
The SI joint and pelvis are not a “pubics cube” we do need to stop the madness.
That said yes the SI joint can be sensitized and produce nociceptive output when stressed. But so many hypotheses abound about positional faults, laxity, dysfunction and fixation that it is bizarrely paradoxical
Fact YOU can not alter the position of the SI joint by manipulation, you just can’t. Study here pubmed.ncbi.nlm.nih.gov/9615363/
You can’t alter the SI joint motion either by manipulation.
“Motion of the SIJ is limited to minute amounts of rotation and of translation suggesting that clinical methods utilizing palpation for diagnosing SIJ pathology may have limited clinical utility.”
Even pain provocation tests have recently been brought into doubt. They are not good for ruling in the SI joint as a source, but are good for ruling it out jospt.org/doi/pdf/10.251…
Imagine thinking that a joint that is a transit stress point for the hips and spine can be held together with a bit of stretchy Kinesiotape?
In 2008 I wrote a letter to the editor about the nonsense around the SI Ilial rotation. My thoughts are still the same except for maybe the manipulation CPR.
To be pretty blunt SI joint tests suck big time on their validity.
To sum up, the SI joint is built for stability and force transmission, it has extremely strong ligaments, there are no muscles that work directly to “move this joint” it is a creaking joint which protects the pelvis from rigidity and unwanted forces.
Time to stop the nonsense.
As always thanks for reading this far.
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I’m going to suggest that there are significant differences between mechanical compromise to a nerve root and a nerve trunk (nerve true). Hence we need to use radiculopathy for root and neuropathy for nerve, they are NOT the same.
Evidence shows that the nerve roots & nerves act differntly
“However, compression of the cauda equina and dorsal rhizotomy proximal to the DRG do not induce significant pain, whereas in the spinal nerve and peripheral nerve, injury distal to the DRG does induce neuropathic pain.”
When we talk about “exercise” what exactly are we talking about?
Systematic reviews often lump every man & his dog under “exercise”
I have long held that a movement performed for symptom modification compared to a movement done with resistance are like 🍏 v🍊
Self movement aimed at pain relief eg a preferred direction of loading movement that decreases your pain in the here and now, should be labeled “self treatment movements “ NOT exercise per se.
Self movement as shown in the diagram above is part of the empowerment Force/loading cycle.
It seems facile to compare a self movement aimed at treating one’s symptoms in the here and now, with a resistance exercise or cardiovascular exercise which have different intent.
A new thread 🧵 based on the last 48 hrs and me consistently saying that pain is reduced in many cases before or without any strength gains from resistance exercises. Is it true that weak muscles don’t mean pain? Do. You need to be stronger to get less pain?
Enjoy the ride.
So lets do LBP first, is there an association between decrease pain and improved strength and function? Apparently NOT
“However, noting that some practitioners and patients still consider this aspect an important part of the SMT experience, further research would be help- ful in fully comprehending the contribution to the per- ceived meaning of this phenomenon to both patients and practitioners”