1) AN EXPLANATION FOR THE SUDDEN CARDIAC DEATHS:
I believe the endocytosis of the Spike Protein is interfering with Ryanodine Receptors causing a calcium channelopathy resulting in Catecholaminergic Polymorphic Ventricular Tachycardia. This is a potentially fatal arrhythmia Image
2) which is induced when the heart rate exceeds 120 bpm.
The intracellular actions of the Spike Protein with Cathepsin L, a protease on the plasma membrane of host cells, increases Ca2+ release from the endoplasmic reticulum (ER) via the ryanodine receptors (RyRs). The associated ImageImage
3) elevation of cytosolic Ca2+ concentration, in turn, increases cathepsin L activity. Cathepsin L promotes virus fusion with host cells by cleaving and activating the spike (S) protein. High levels of extracellular and cytosolic Ca2+ concentrations are also necessary for virus
4) fusion and endocytosis. Cathepsin L in the endosome, under the condition of a high level of Ca2+ concentration, promotes virus RNA release into the cytosol. On the other hand, the increased cytosolic Ca2+ concentration due to the overactivation of RyRs activates calcineurin,
5) which dephosphorylates NF-AT and translocates into the nucleus for promoting transcription and virus replication. Excess Ca2+ release from ER via overactivation of RyRs in AD cells results in depletion of ER Ca2+ and associated ER stress, as well as the overloading of
6) mitochondria with Ca2+ and associated mitochondria damage.
The Spike Protein therapies should be paused while this mechanism is investigated.
ncbi.nlm.nih.gov/labs/pmc/artic…
pubmed.ncbi.nlm.nih.gov/29173400/
jbc.org/article/S0021-…
ncbi.nlm.nih.gov/labs/pmc/artic…
ncbi.nlm.nih.gov/labs/pmc/artic…

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More from @Parsifaler

16 Jan
1) AN ENERGY-BASED MITOCHONDRIAL EXPLANATION OF SPIKE PROTEIN PATHOGENESIS: SLOWLY TURNING THE POWER OFF
As we have not yet determined the primary pathogenesis of COVID-19, I propose that the disease is one of progressive Mitochondrial Dysfunction from a pathogen (therapeutic?)
2) delivered via the Endothelium and distributed throughout the body to all organs and tissues.
PREMISE: S1 and Trimer both induced mitochondrial damage including functional deficits in mitochondrial respiration.
For example: It is well established that the brain uses more energy
3) than any other human organ, accounting for up to 20 percent of the body's total haul. Therefore, it would only be logical that one of the first signs of mitochondrial dysfunction would be brain fog. Indeed, brain fog has been theorized as being an energy, or mitochondrial
Read 5 tweets
15 Jan
1) A CERTAIN PATHWAY TO SPIKE PROTEIN MEDIATED AUTOIMMUNE/FIBROTIC DISEASE
TGF-β DRIVEN IMMUNITY, SEVERE COVID-19 OR PROLONGED SPIKE PROTEIN EXPOSURE RESULTS IN AUTOIMMUNE/FIBROTIC DISEASE (VASCULITIS)
A very interesting paper from March of 2021 made a startling observation which
2) was concluded with the foreboding statement: Whether or not the antibodies generated in the continued immune reactions of COVID-19 patients in the ICU are harmless, or whether they may even cause detrimental immunopathology remains to be shown.
Why is this of interest now?
3) We have been searching for a satisfactory explanation for the observed myocarditis, autoimmune issues and vasculitis which are appearing ever increasingly.
What the paper demonstrated was that they analyzed samples from six patients within the first week after ICU admission,
Read 13 tweets
11 Jan
1) AN ONCOGENIC “FLASH DRIVE”: THE SPIKE PROTEIN OF SARS-CoV-2 AND CANCER
When I first began studying the SARS-CoV-2 virus I was initially concerned about its Spike Protein’s potential ability to induce prion disease, as the spike has homology with our Prion protein and it
2) induces a strong Unfolded Protein response.
However, almost two years after I reached that conclusion, I believe it is only part of the story. Yes, I am still concerned that it may induce Prion Disease. But, what concerns me more is that its interactions and mimicking of the
3) human Prion Protein may be more “effective” having a role in tumorigenesis and metastasis.
In fact, the more I look at the Spike Protein now, and with the now common knowledge that the Spike Protein mimics Trousseau Syndrome, I now believe that the Spike Protein may be a
Read 15 tweets
10 Jan
1) COVID-19 COAGULATION ABNORMALITIES AS TROUSSEAU SYNDROME
IS THE SPIKE PROTEIN INDUCING GENERALIZED CANCER AND COULD THOSE IMMEDIATELY EXPERIENCING COAGULATION ABNORMALITIES HAVE OCCULT CANCERS?
Malignancy affects the hemostatic system and the hemostatic system affects
2) malignancy.
Cancer's "purpose" is to invade organs and disrupt their normal functioning by rendering cells dysfunctional. These dysfunctional cells then spread, disrupting further cells, and so on until the victim dies.
The way cancer accomplishes this is to spread through
3) vessels. If it spreads through blood vessels, it must cross the barrier of the blood vessels to enter organs, including blood vessels themselves. Therefore disrupting (activating) the endothelium is essential to "breaking through" that barrier.
The close relationship between
Read 11 tweets
9 Jan
1) Guild Navigator:
You are transparent. I see many things. I see plans within plans. (DUNE)
VASCULAR DISEASE AS CAMOUFLAGE: THE SPIKE PROTEIN OF SARS-CoV-2 AS MICROTUMOR
Respiratory disease held the world in a false awe at the beginning of the COVID-19 pandemic. Health
2) authorities the world over reiterated the same narrative: We have a new Respiratory Virus. Those of us who looked closely at the disease from the beginning knew that wasn’t the true picture – or at least not the full picture.
Then, slowly, over the course of the first year of
3) the pandemic, everyone else began to realize this, too. Finally, towards the end of 2020, COVID-19 was universally recognized as a Vascular Disease. However, I have realized the virus’ deception does not stop there.
IT IS ONLY A VASCULAR DISEASE INSOMUCH AS THE SPIKE PROTEIN
Read 17 tweets
1 Jan
1) THE SPIKE PROTEIN OF SARS-CoV-2 RECOGNIZES THE BODY’S ENTIRE ENDOTHELIUM AS TUMOR ENDOTHELIUM. IT CAUSES THE BODY TO “TREAT” THE ENDOTHELIUM PRECISELY AS A CANCER THERAPY – BY DESTROYING IT WHILE CAUSING MASSIVE INLAMMATION AND IMMUNE CELL INFILTRATION.
First, let us look at
2) what Cancer does. Cancer wants an Anergic Endothelium. It wants an Endothelium which WILL NOT REACT. It will not cause the Endothelium to activate so that immune cells will not be “attracted” to it and remove it. It also wants to create new blood vessels to feed it.
3) This combination of “problems” it has can be easily remedied – by destroying it. Indeed, THIS IS A RECENT THERAPEUTIC UNDER DEVELOPMENT.
The following certainly is COVID-19. Would you not agree?
One important physiological function of normal endothelial cells is quiescence of
Read 16 tweets

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