1) COVID-19 COAGULATION ABNORMALITIES AS TROUSSEAU SYNDROME
IS THE SPIKE PROTEIN INDUCING GENERALIZED CANCER AND COULD THOSE IMMEDIATELY EXPERIENCING COAGULATION ABNORMALITIES HAVE OCCULT CANCERS?
Malignancy affects the hemostatic system and the hemostatic system affects
2) malignancy.
Cancer's "purpose" is to invade organs and disrupt their normal functioning by rendering cells dysfunctional. These dysfunctional cells then spread, disrupting further cells, and so on until the victim dies.
The way cancer accomplishes this is to spread through
3) vessels. If it spreads through blood vessels, it must cross the barrier of the blood vessels to enter organs, including blood vessels themselves. Therefore disrupting (activating) the endothelium is essential to "breaking through" that barrier.
The close relationship between
4) cancer and thrombosis has been known since 1865, when Armand Trousseau first described a clinical association between thrombosis and a yet undiagnosed cancer. Cancer favors the activation of blood coagulation with the appearance of a hypercoagulable state or chronic DIC in
5) these patients. Abnormalities in one or more coagulation tests are common in cancer patients, even without overt thrombotic and/or hemorrhagic manifestations. The results of laboratory tests demonstrate that a process of fibrin formation and fibrinolysis parallels the
6) development of malignancy, increasingly in those with metastases. Particularly, subtle hemostatic alterations are detected, such as high levels of plasma by-products of clotting reactions (i.e. prothrombin fragment 1+2 [F1+2], fibrinopeptide A [FPA], thrombin–antithrombin
7) complex [TAT] and D-dimer), or an acquired protein C resistance, as well as high levels of circulating microparticles (MP) shed by tumor cells and platelets. On the other hand, hemostatic proteins and reactions interdigitate the process of tumor growth and dissemination.
8) Indeed PF1.2 was elevated in most patients, but synchronously measured D-dimer was almost universally elevated. All biomarkers of Trousseau Syndrome are evident in COVID coagulation abnormalities.
Another very telling sign is that light weight molecular Heparin is successful
9) in treating COVID coagulation. This is precisely the same treatment used for Trousseau Syndrome.
Perhaps the most interesting aspect of this possibility is that thrombosis can also represent the earliest clinical manifestation of an occult cancer. Indeed, patients with
10) idiopathic VTE show a four- to seven-fold increased risk of being diagnosed with cancer in the first year after thrombosis, as compared with patients with VTE secondary to known causes, and the risk of cancer is even higher in patients with recurrent thromboembolism and in

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More from @Parsifaler

11 Jan
1) AN ONCOGENIC “FLASH DRIVE”: THE SPIKE PROTEIN OF SARS-CoV-2 AND CANCER
When I first began studying the SARS-CoV-2 virus I was initially concerned about its Spike Protein’s potential ability to induce prion disease, as the spike has homology with our Prion protein and it Image
2) induces a strong Unfolded Protein response.
However, almost two years after I reached that conclusion, I believe it is only part of the story. Yes, I am still concerned that it may induce Prion Disease. But, what concerns me more is that its interactions and mimicking of the
3) human Prion Protein may be more “effective” having a role in tumorigenesis and metastasis.
In fact, the more I look at the Spike Protein now, and with the now common knowledge that the Spike Protein mimics Trousseau Syndrome, I now believe that the Spike Protein may be a
Read 15 tweets
9 Jan
1) Guild Navigator:
You are transparent. I see many things. I see plans within plans. (DUNE)
VASCULAR DISEASE AS CAMOUFLAGE: THE SPIKE PROTEIN OF SARS-CoV-2 AS MICROTUMOR
Respiratory disease held the world in a false awe at the beginning of the COVID-19 pandemic. Health
2) authorities the world over reiterated the same narrative: We have a new Respiratory Virus. Those of us who looked closely at the disease from the beginning knew that wasn’t the true picture – or at least not the full picture.
Then, slowly, over the course of the first year of
3) the pandemic, everyone else began to realize this, too. Finally, towards the end of 2020, COVID-19 was universally recognized as a Vascular Disease. However, I have realized the virus’ deception does not stop there.
IT IS ONLY A VASCULAR DISEASE INSOMUCH AS THE SPIKE PROTEIN
Read 17 tweets
8 Jan
1) AN EXPLANATION FOR THE SUDDEN CARDIAC DEATHS:
I believe the endocytosis of the Spike Protein is interfering with Ryanodine Receptors causing a calcium channelopathy resulting in Catecholaminergic Polymorphic Ventricular Tachycardia. This is a potentially fatal arrhythmia
2) which is induced when the heart rate exceeds 120 bpm.
The intracellular actions of the Spike Protein with Cathepsin L, a protease on the plasma membrane of host cells, increases Ca2+ release from the endoplasmic reticulum (ER) via the ryanodine receptors (RyRs). The associated
3) elevation of cytosolic Ca2+ concentration, in turn, increases cathepsin L activity. Cathepsin L promotes virus fusion with host cells by cleaving and activating the spike (S) protein. High levels of extracellular and cytosolic Ca2+ concentrations are also necessary for virus
Read 6 tweets
1 Jan
1) THE SPIKE PROTEIN OF SARS-CoV-2 RECOGNIZES THE BODY’S ENTIRE ENDOTHELIUM AS TUMOR ENDOTHELIUM. IT CAUSES THE BODY TO “TREAT” THE ENDOTHELIUM PRECISELY AS A CANCER THERAPY – BY DESTROYING IT WHILE CAUSING MASSIVE INLAMMATION AND IMMUNE CELL INFILTRATION.
First, let us look at
2) what Cancer does. Cancer wants an Anergic Endothelium. It wants an Endothelium which WILL NOT REACT. It will not cause the Endothelium to activate so that immune cells will not be “attracted” to it and remove it. It also wants to create new blood vessels to feed it.
3) This combination of “problems” it has can be easily remedied – by destroying it. Indeed, THIS IS A RECENT THERAPEUTIC UNDER DEVELOPMENT.
The following certainly is COVID-19. Would you not agree?
One important physiological function of normal endothelial cells is quiescence of
Read 16 tweets
31 Dec 21
1) A HYPOTHETICAL FATAL PATHOGENIC FEEDBACK LOOP OF THE SARS-CoV-2 SPIKE PROTEIN
Heretofore the global medical community has been mainly concerned with the Acute phase of COVID-19. This is in both aspects of treatment and prevention. However, I believe that the Acute phase of
2) COVID-19 is only the very tip of a massive pathogenic iceberg. And one that continues to “build” upon itself.
A paper published November 12 identified kidney disease as the leading risk factor for hospitalization in confirmed COVID-19 patients. The phenome-wide association
3) study also identified Type 2 Diabetes, Congestive Heart Failure, COPD in addition to Chronic Kidney Disease.
What is it that ties all of these diseases together? Please remember that Schizophrenia is also considered to be a major risk factor for severe COVID. All of the above
Read 7 tweets
27 Dec 21
1) My Christmas gift of understanding to the world. May it help.
Breakthrough. Finally. Mechanisms found.
Eexecutive (non-techincal) Summary: The Spike Protein of COVID-19 most likely cases the body to reject iteself.
COVID-19 mimics Graft vs Host Disease via Anti-Angiotenisn II
2) Type 1 Receptor Antibody induction and complement/immune complex deposition in the microvasculature mediated by the Spike Protein.
If you have been following my posts, you know that for the better part of the past year I have been focused on the observation that COVID-19 and
3) its sequelae look VERY MUCH like Graft vs Host disease. We see identical endothelial/multisystemic destruction in both COVID-19 and GvHD. However, as you know, I have been working to find the mechanism for this parallel.
I believe I can now explain why COVID-19 and GvHD are
Read 10 tweets

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