Walter M Chesnut Profile picture
Apr 8, 2022 6 tweets 2 min read Read on X
1) THE SPIKE PROTEIN IS THE “AMYLOID” BEING DEPOSITED AND INDUCING AMYLOIDOSES: A MAJOR FINDING MISSED
SEVERE COVID MAY BE DUE TO THE ADDED DEPOSITION OF COMPLEMENT WITH THE SPIKE
The paper “The histologic and molecular correlates of COVID-19 vaccine-induced changes in the skin” ImageImageImage
2) made a case for the immune response to the Spike Protein causing self-limited hypersensitivity reactions to the vaccine. However, if you study the paper carefully, you notice that the authors have missed a far more important finding.
The biopsy specimens of normal skin post
3) vaccine and of skin affected by the post-vaccine eruption showed rare deep microvessels positive for spike glycoprotein with no complement deposition contrasting with greater vascular deposition of spike protein and complement in skin biopsies from patients experiencing severe
4) coronavirus disease 2019 (COVID-19).
The histology exactly recapitulated perniosis including COVID-19–associated perniosis, which provides evidence that the Spike Protein alone induces COVID-19 pathology. More importantly, the fact that IN NORMAL SKIN, the Spike Protein was
5) found to be deposited in DEEP VESSELS, MICROVESSELS, BLOOD VESSELS AND DEEP ENDOTHELIAL CELLS.
Why would one assume that this deposition is only occurring in NORMAL SKIN TISSUE? I believe it is most certainly occurring in NORMAL TISSUE. PERIOD.

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More from @Parsifaler

Jan 23, 2023
1) Although decreased translation fidelity causes protein misfolding and aggregation in both cardiomyocytes and Purkinje cells, the downstream pathways that lead to cell death in the two cell types may be quite different. Interestingly, a point mutation in the editing domain of
2) human mitochondrial AlaRS (AARS2) has been associated with infantile mitochondrial cardiomyopathy, suggesting increased errors in either cytoplasmic or mitochondrial protein synthesis can lead to cell death in the heart. In summary, our data show that a global reduction in
3) translational fidelity, rather than disruption of a specific protein, can induce defects in proteostasis in numerous cell types in the mouse. It is thus possible that proteinopathy can occur in the brain, heart, or even other tissues as a combination of genetic and/or
Read 4 tweets
Jan 3, 2023
On Damar Hamlin and Dr. Sutterer's "diagnosis" of Commodio Cordis
I am shocked that Commodio Cordis (CC) is named as the cause. Please examine the images carefully. In CC Ventricular fibrillation can be triggered by chest wall IMPACT ONLY OVER THE HEART, and predominantly occurs
with impact over the center of the left ventricle. Although CC usually involves impact from a baseball, it has also been reported during hockey, softball, lacrosse, karate, and other sports activities in which a relatively hard and compact projectile or bodily contact caused
impact to the person's precordium. Are we to believe that with the padding that professional football players wear, the location of the heart and the location of the impact, that this was actually CC?

Nonsense. Rapidly stated, narrative, ARRANT NONSENSE.
Read 6 tweets
Apr 11, 2022
1) UPDATED SYNTHESIS: EXTREMELY URGENT
IT’S COMPLICATED, BUT CLEAR – LOOKING ONE MOVE DEEPER
THE SPIKE PROTEIN PATHOGENIC ALGORITHM – DUAL PATHS TO TERMINAL SYSTEMIC FIBROSIS: IMMEDIATE FOR THOSE WITH SIGNIFICANT COMORBIDITIES, INDUCED FOR THOSE WITHOUT
The Spike Protein is Image
2) inducing terminal systemic fibrosis of all organs, including the blood, via two principal mechanisms.
The first is a direct, immediate path via binding to RGD-binding integrins, which includes several TGF-β -activating integrins. This this activates Myofibroblasts which ImageImageImage
3) induces Fibrosis. Indeed, in autopsies of COVID-19 patients with advanced disease, 38% collagen deposition was found in their lungs.
This is a rapid and certainly fatal circumstance.
But, this is not limited to the lungs. In a series of cardiac autopsies conducted in
Read 12 tweets
Apr 8, 2022
1) MORE THAN AMYLOIDOSES. ALL HUMAN TISSUES ARE BEING TRANSFORMED INTO FIBROUS MASSES. INCLUDING. THE. BLOOD.
AMYLOIDOSIS. FIBROSIS. | AUTOPSIES. INCREASED ORGAN WEIGHT. | CLOTS. AMYLOIDS.
The tissues of the body, INCLUDING THE BLOOD, are being either DEPOSITED WITH FIRBILS OR ImageImage
2) BEING TRANSFORMED INTO FIBRILS. Amyloidosis > Deposition of Fibrils. Fibrosis > Transformation into Fibrils.
The Spike Protein is transforming (at least) all human tissue (perhaps other species?) into non-functioning fibrous masses.
I was reading papers in the solarium after
3) dinner while having a cigar. And I kept thinking about Amyloidosis and Fibrosis. I have seen both. Is it one? Is it the other? After reading more papers – AND SOME AUTOPSY REPORTS - I came to a startling and incredibly disturbing conclusion. It is BOTH!
THE CLOTS WE ARE
Read 9 tweets
Apr 4, 2022
1) PLEASE READ THIS ENTIRE THREAD. IT IS URGENT.
Building on all recent work. By far my most important synthesis:
A NEW KIND OF CANCER: THE MARRIAGE OF ONCOGENESIS AND PRIONOPATHY
THE PROGRESSIVE DESTRUCTION OF TISSUE AND ORGANS BY THE AMYLOIDOGENIC AND OLIGOMERIC PROPERTIES OF Image
2) THE SARS-CoV-2 SPIKE PROTEIN
It has been proposed that Alzheimer's disease might be a 'whole body' problem, as amyloid-beta can travel, cancer-like, to brain from other parts of body.
Normal mice that had been joined (via bloodstreams) to genetically modified partners for a
3) year "contracted" Alzheimer's disease. The researcher song says the amyloid-beta traveled from the genetically-modified mice to the brains of their normal partners, where it accumulated and began to inflict damage.
Not only did the normal mice develop plaques, but also a
Read 11 tweets
Mar 31, 2022
1) THIS. CHANGES. EVERYTHING.
THE SPIKE PROTEIN FORMS COMPLEXES WITH ALL AMYLOIDOGENIC PROTEINS!
EXAMPLE: ATHLETES DIE FIRST BECAUSE ATHLETE’S HEARTS ALREADY HAVE HYPERTROPHY!!!
THE AVERAGE CANNOT PERCEIVE. ONLY THOSE AT PHYSICAL EXTREMES ARE AFFECTED INITIALLY. EXTREMES OF SIZE ImageImage
2) AGE AND ACTIVITY! MAKE NO MISTAKE IT WILL TAKE THE AVERAGE SOON ENOUGH AS THEY ARE “FIBRILLED”!
THE SPIKE PROTEIN IS A FOLDAMER AND A SELF-REPLICATOR: THE INDUCTION OF MULTIPLE LETHAL PROTEINOPATHIES
The more I go down the proteinopathy rabbit hole, the more trouble I see that
3) we are in. Even I am in shock and awe, and I have been often, recently. I keep thinking I have found the worst that the Spike Protein can do, but I keep looking one move deeper. And what I see is utterly terrifying and without an effective therapeutic will, in a very few
Read 14 tweets

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