1) A 🧵 providing a whistle stop tour of Frozen Shoulder (FS)
2) FS is associated with stiffness of the capsule which restricts GHJ A/PROM within a capsular pattern coupled with a normal XR finding (Zuckerman 2011). Previously thought to be self-limiting, now ++ evidence highlighting the significance it has on pain, disability and function
3) Epidemiology: Thought to affect 2-5% of the gen pop between the age of 40-70yrs. Women are more susceptible & it is thought that 6-17% of patients are unfortunate enough to develop it in the contralateral arm within the first 5yrs (Dyer et al., 2021).
4) Exact pathophysiology is not fully understood, yet histological findings highlight a thickened & tight capsule w chronic inflammatory cells & fibroblasts found in the joint capsule which suggest an autoimmune component (Date & Rahman 2020). This table shows the 4 stages;
5) It is split into two types, a) primary - idiopathic onset of Sx & b) secondary - ? associated with metabolic syndrome as a large % are obese, there is a x5 more chance of FS if diabetic. Thyroid problems, previous surgery, dupuytrens etc are also related (Date + Rahman 2020).
6) Diabetes & FS; T1DM = causal risk factor for developing FS (Green et al., 2021). 13.4% prevalence of FS in diabetics & 30% of FS population are diabetic (Dyer et al., 2021). ? glycemic changes influence capsular stiffness through collagen & release proinflammatory cytokines
7) Features; Pain is often severe, disabling and impedes sleep during the initial stage. Locality can vary, described as deep & may refer to the bicep/elbow. As the capsule stiffens, ROM is affected & this causes significant functional limitations. This paper summarises it nicely
8) Dx; ‘Shoulder pain for 1/12, unable to lie on affected side with restricted A/PROM, with a minimum of 50% reduction in passive external rotation vs contralateral side (Kelley et al., 2013). *Often assessing in supine, w a towel under the elbow helps prevent any compensation*
9) Imaging: Often not needed for a Dx. XR is recommended to rule out OA, # / dislocation or more sinister pathology. US may detect thickening of the rotator interval / surrounding soft tissue or teno/synovitis.
10) Prognosis; Will vary from pt-pt. Previous thoughts were that it has a favourable natural Hx. Yet Schaffer et al (1992) found 50% had Sx still at 7yrs. Whereas Wong et al (2017) showed ER remained limited by approx 50% at 1yr f/u if left untreated.
11) Treatment; UK Frost Trial. They concluded injection & PT worked well in unison and this had similar favourable outcomes at 1yr f/u compared to MUA & capsular release. Surgery had the best outcome @ 12/12 but not clinically superior & is associated with risks.
Always keep on the radar 🚨 in any adolescent presenting with acute pelvic pain OR recalcitrant Sx which are not responding to conservative efforts
2) They account for approx 3-5% of adolescent injuries involve the groin (Di Maria et al., 2022). Avulsion fractures typically occur w forceful contraction +/- passive stretching & this is due to secondary ossification centres being weaker than the MTU
3) This table by Schiller et al., (2017) highlights the ages where ossification centres fuse.
▪️Females tend to be 1-2yrs sooner vs male counterparts.
Note how ASIS & ischial tuberosity may not ossify until as late as 25yrs
These are the most common 🚩 of the spine. But what are some of the risk factors which ⬆️ likelihood⁉️
2) Typically affects the thoracolumbar spine (as seen). Be mindful a large % are asymptomatic & picked up incidentally during other investigations (e.g CXR)
Some features
▪️Localised pain & tenderness
▪️Pain supine & sitting hard chair
▪️+’ve percussion
▪️⬇️ ROM
3) Henschke et al., (2009) in a primary care cohort illustrated 4 key features which raised suspicion of it
1. Female sex 2. Age > 70yrs 3. Prolonged steroid use 4. Trauma (minor in elderly)
Glucocorticoids was enough to ⬆️ post-test probability by 25%
1) Approx. 5-11% of patients undergoing MRI for suspected Cauda Equina Syndrome (CES) will have it confirmed (Metcalfe et al., 2023) 💡
Once differentials are ruled out 🕵️♂️
The remaining % are then considered to be ‘Scan-Negative CES’. But what does this actually mean?
2) Scan-Negative CES ‼️
Pt’s have 1+ symptoms suggestive of CES, yet without the radiological evidence of compression. One possible cause is that it may occur secondary to an almost functional neuro disorder 🧠
3) Pt’s in this subgroup tend to present with more of the following…
▪️Psychiatric diagnoses
▪️Pre-existing B/B disorder
▪️Non-cardiac chest pain
▪️Higher % are female
▪️IBS
▪️FM (Hoeritzauer et al., 2018)
1) A whistle stop tour of muscle cramps. How can we tell if they are idiopathic vs something more systemic?
2) Thought to be influenced by both the PNS (weighted more heavily) and the CNS. Majority of them are benign
3) The most common we see in clinic, these are some simple ways of helping to highlight to patients they are no cause for concern. Rather than ‘drink more’