Rob Buttner Profile picture
May 20 10 tweets 4 min read
Writing an #ECG talk for @ICEM2022 and couldn't find an algorithm for approaching the "fixed" regular narrow complex tachycardia. Thought I would create my own.

#FOAMed @acemonline
@ICEM2022 @acemonline All this talk about VT versus SVT and it seems no one talks about an approach to regular narrow complex tachycardia.

Well, basic principles. Regular narrow complex tachycardias are either re-entrant, automatic, or due to triggered automaticity. (1)
Those due to re-entrant circuits include atrial flutter, AVNRT, orthodromic AVRT, and some focal atrial tachycardias. These are paroxysmal, have a fixed rate, and all respond to electricity (2)
Those due to enhanced automaticity include most focal atrial tachycardias, junctional tachycardia (AJR), and of course sinus tachycardia. These have a gradual onset and exhibit beat-to-beat variability. (3)
The 3 questions I ask myself in any regular narrow complex tachycardia:

1) Sudden versus gradual onset?
2) Beat-to-beat variability?
3) Presence, location (R-P interval), and morphology of P waves?

Oh, and don't forget to look for STE suggestive of underlying infarction (4)
Having trouble seeing P waves in the midst of the flashing fluorescent lighting, loud beeping, and interruptions of ED?

There are a couple of other tricks still (5)
Double the paper speed -- this is the rate at which the ECG machine produces a trace. Standard output is 25mm/sec.

Doubling the rate to 50mm/sec causes the ECG trace to appear "drawn out". Particularly useful for revealing flutter waves in 2:1 block (6)
At the same time, try the Lewis Lead configuration, which better detects atrial activity in relation to ventricles.

Place the RA & LA electrodes just to the right of the sternum (2nd & 4th ICS respectively), and monitor lead I. Also useful for detecting AV dissoc. in WCT (7)
Lastly, adenosine is useful as both a diagnostic and therapeutic tool in stable patients.

It will almost always work for AVNRT and orthodromic AVRT, and aborts some paroxysmal FATs.

Whilst it won't revert atrial flutter, it will often reveal flutter waves (8)
One more thing.

Try to avoid the term SVT when it comes to narrow complex tachycardias. The mere fact that they are narrow complex already tells us they are supraventricular in origin. I reserve this term for describing VT versus SVT with aberrancy. (9)

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More from @rob_buttner

May 22
Differentiating regular narrow and wide complex tachycardias can be a challenge.

Can't see any P waves?

Here are three handy tricks to make things easier
🧵👇
#1: Double the paper speed

Paper output speed is the rate at which the ECG machine produces a trace. Standard output is 25mm per second.

Doubling the standard rate causes the ECG to appear drawn out. 1 small square now becomes 0.02 seconds.
Here, atrial flutter with 2:1 block becomes more apparent once the trace is drawn out Image
Read 10 tweets
May 3
🔪🔪 Killer ECG Patterns 🔪🔪

Part Two -- acute coronary occlusion.

litfl.com/killer-ecg-pat…

Deadly diagnoses not to miss when you are asked to quickly “sign off” an #ECG.
A couple of things.

What about Wellen’s and ST elevation in aVR you say?
Well, STE in aVR simply represents O2 supply-demand mismatch, and can be due to cardiac & non-cardiac causes.

Whilst it can be seen in LMCA/LAD critical stenosis, it very rarely represents occlusion of these vessels. See a more in-depth dive here...

Read 9 tweets
Sep 22, 2021
Crushing chest pain and diaphoresis.

The computer reads "inferior ischaemia"

You, the astute twitter-reading clinician, read "normal"

Here's why 👇🏾🧵

litfl.com/ecg-case-134
No, you are not smarter than a computer. But computers don’t allow for human error.

This ECG looks a bit odd. That should prompt us to shift to our system 2 thinking (systematic, conscious, analytical interpretation). (1/11)
Immediately, we notice an abnormal P wave axis, with an upright P wave in aVR and negative in II.

Whilst on its own this can represent an ectopic atrial rhythm, the QRS axis is also abnormal and is northwest. (2/11)
Read 12 tweets
Sep 20, 2021
Tox ECGs CAN be made easy 💊💊💊

Understanding these ECG changes make them easier to recognise 👇
@LITFLblog We may have heard that Na channel blockade causes “right axis deviation of the terminal QRS”.

But what does this actually mean? (1/13)
Well, the right-sided intraventricular conduction system is more susceptible to Na channel blocker toxicity than the left.

This leads to delayed depolarization of the RV. As is the case in RBBB, delayed RV conduction manifests as deep, slurred S waves in leads I, II, V5-6 (2/13)
Read 14 tweets

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