The vulnerable population is not a fixed group of individuals to be expunged in some misguided pursuit of societal health.
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The concept of “culling” the vulnerable population with SARS-CoV-2 is a grotesque distortion of reality.
And a reflection of a profoundly flawed understanding of both biology and human society.
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The vulnerable population is a dynamic and evolving demographic.
It is constantly replenished by the very effects of the virus and the structural inequalities that exacerbate its impact.
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SARS-CoV-2 doesn’t just affect a pre-determined group of immunocompromised or elderly individuals.
It systematically creates new vulnerabilities within the population.
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With each wave, SC2 leaves in its wake long-term complications, damage to the cardiovascular, neurological, and immune systems, and chronic conditions
Those who might have been considered “healthy” before infection can rapidly transition into the category of the vulnerable
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This biological reality is compounded by the socio-economic structures that disproportionately expose certain populations to the virus.
Inequality—whether economic, racial, or geographic—plays a decisive role in determining who becomes vulnerable.
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Marginalized communities, those with less access to healthcare, proper housing, and protective measures.
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Essential workers, often in low-wage, precarious jobs, are thrust into environments where exposure is inevitable.
And where the consequences of infection—whether through the inability to afford healthcare or the long-term economic consequences of disability—are catastrophic
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To speak of “culling” the vulnerable without addressing the systemic inequalities that constantly create and sustain this vulnerability is a moral failure.
A perverse fantasy born from ignorance and a profound lack of empathy.
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The logic of eliminating the weak becomes a mechanism for perpetuating injustice.
The poor, the marginalized, and those disadvantaged by society’s inequities are continually pushed to the front lines of exposure and consequence.
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This isn’t a problem that ends with any number of deaths.
It’s a feedback loop in which inequality ensures that the vulnerable population is endlessly replenished.
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The notion that society could somehow “rid itself” of vulnerability through exposure or infection presupposes a brutal, survival-of-the-fittest worldview.
Scientifically ignorant and ethically abhorrent.
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The idea of “fitness” in the context of SC2 is a fallacy.
Variants emerge, adapt, and evade immunity, constantly shifting the goalposts of who is at risk.
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What was once a manageable infection for one group becomes deadly for another as SC2 evolves
As long as SARS-CoV-2 remains in circulation, the vulnerable population is not a static group of individuals to be culled, but a dynamic demographic
A reflection of the virus’s power to inflict continuous and compounding harm on society
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The brutal truth is this:
As long as we accept the virus as a tool for weeding out the vulnerable, we are accepting an unending cycle of human suffering, reinforced by the very inequalities that define modern society.
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This is not a strategy for survival or societal resilience.
It is a strategy for perpetuating exploitation and creating ever-deepening divides between the protected few and the expendable many.
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In the end, no one—regardless of their present “health” or “fitness”—is truly exempt from this cycle.
Vulnerability, in the context of a virus that causes long-term systemic damage, is everyone’s eventual fate.
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Humanity shouldn’t be stripped of empathy and understanding.
The value of human life shouldn’t be reduced to a brutal calculus of short-term survival.
The solution does not lie in “culling” the vulnerable.
It lies in protecting and uplifting them.
19/19
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What we now recognize as post-viral fatigue syndrome had a name a century ago.
As a society, we have not learned from history because we are seduced by the fallacy of immediate recovery.
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Our focus remains relentlessly fixed on short-term goals.
Economic revival, political stability, and a return to normalcy.
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In the aftermath of the 1918 Spanish Flu, much of the world’s attention shifted away from the dead and the sick, toward rebuilding shattered economies and societies.
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SARS-CoV-2 has drawn comparisons to many past pandemics.
Perhaps one of the most instructive parallels lies with the HIV/AIDS crisis.
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While these two viruses differ in their modes of transmission, their long-term health impacts, and the ways society has responded to them, they share a key characteristic.
Both have challenged our understanding of viruses and their lasting effects on the human body.
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What makes these comparisons compelling is that, much like HIV in the early 1980s, we are still in the early stages of truly grasping the long-term consequences of SARS-CoV-2.
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Thinking out loud about SARS-CoV-2 and cancer again.
We’ve been sitting on a ticking time bomb for decades, and now the alarm is finally going off.
Epstein-Barr Virus + SARS-CoV-2 = ?
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EBV has quietly embedded itself in the bodies of nearly 90% of the population.
We’ve let this virus slip through the cracks for years, never fully addressing the catastrophic potential it carries, and now, thanks to COVID-19, that negligence is coming back to haunt us.
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The danger EBV poses was never benign.
It has been tied to several malignancies.
Burkitt’s lymphoma, nasopharyngeal carcinoma, Hodgkin’s lymphoma, and gastric cancer.
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I’m seeing people discuss the oncogenic properties of SARS-CoV-2
It reminds me of something
It took humanity decades (far too long) to understand the brutal truth that Cytomegalovirus, this seemingly unremarkable virus, could be quietly nurturing the seeds of cancer.
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Discovered in the early 1900s, CMV was first seen as just another infection.
Something dangerous only to the vulnerable, the sick, the newborn.
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We didn’t realize that lurking within it was something far more insidious.
Something that could slowly, quietly tip the scales towards cancer in ways we barely understand even now.
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A generation marked not only by the immediate fallout of a pandemic but by the lingering specter of accelerated neurodegeneration.
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This research elucidates a brutal aspect of SARS-CoV-2’s pathogenesis:
The ability to enact molecular alterations that precipitate synaptic dysfunction and neurodegeneration.
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This interference with synaptic integrity and function is coupled with sustained neuroinflammation.
It bears striking similarities to early stages of Alzheimer’s and Parkinson’s pathologies, where synaptic dysfunction precedes overt neurodegeneration.
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