🧬2. There is only one virus that's known to be engineered that's currently infecting humans, OPV, the LAV against Polio which has been reverting for decades where there's poor sanitation.
🌎3. LAV's have been linked to multiple global pandemics in the past, including 1977's H1N1 which was engineered by serial passage to be a LAV, and 2009's H1N1 outbreak which was linked to LAVs on Mexican pork farms.
👹4. The chimeras created in Baric's controversial 2015 paper manipulating SARS-like viruses are perfectly consistent with the chimeras you'd want to create to attenuate a LAV:
"Viral chimeras—recombinants incorporating genes from close or distant relatives—are widely used in research and offer several different possibilities for attenuation... After 10 mouse–mouse transfers (by intracerebral injection), the chimeric virus exhibited the greatest stability of attenuation."
💩6. Since the development of OPV we've known that LAVs need to bind to intestinal tissue to work, SARS-CoV-2's affinity for ACE2 isn't about airborne transmission, it's about binding to the gut - where ACE2 is the most expressed:
❄️7. SARS-CoV-2’s behavior indicates it may have been cold-adapted, a common approach with LAVs to make sure they can replicate in the upper respiratory tract but not the lungs.
🧬8. SARS-CoV-2 has a novel gene named CaORF15 which encodes a protein which has never been seen before other than in RaTG13, which is a consensus sequence with no natural providence either, and seems to have the immunosuppressive activity that you would expect for a vaccine.
🧐10. Live-attenuated vaccines are the most well-documented engineered viruses known to man, which have been known to spread from their original recipient for decades, as well as revert back to full virulence.
The near complete silence around LAVs is the only direction you need.
🫧WHY IS NASAL IRRIGATION THE MOST IMPORTANT FIRST-LINE OF DEFENSE?
"The scientists inoculated the cultures by incubating them in the same dish with SARS-CoV-2. With light and electron microscopy and immunochemical staining, they monitored the epithelial cells for viral entry, replication and exit..."
Only ciliated cells became infected. Electron microscopy showed that the virus initially attaches only to cilia. Even 24 hours after inoculation, the virus was replicating only in a few cells. It took 48 hours for massive replication to occur.
SARS-CoV-2 needs a full day or two to start replicating full-tilt in real life, too. "It's clear that human ciliated nasal epithelial cells are the primary entry site for SARS-CoV-2 in nasal epithelial tissue," Jackson said.
🚨EVERYONE PUSHING "DEFUSE" AS THE SOURCE OF THIS PANDEMIC IS WORKING DIRECTLY FOR CHINA'S PLA TO HELP COVER-UP THE WORST CRIME AGAINST HUMANITY IN HISTORY!!
There is a massive discrepancy between what the DARPA employee who leaked DEFUSE claims it says and the truth...
Here's what's actually proposed within DEFUSE, you'll notice that no mention whatsoever is made about live-attenuated vaccines, they're proposing novel immune-boosting molecules - not LAVs even once at any point at all:
So then you might be asking yourself, why does a DARPA employee create all this exposition about live-attenuated vaccines when he "leaks" DEFUSE out to the world - when live-attenuated vaccines aren't actually proposed within DEFUSE at all?
💀THE EMERGENCE OF MONEKYPOX IS JUST THE BEGINNING!!
"And while the virus is growing sturdier, it’s also regaining more of its highly-pathogenic capabilities and gaining the ability to infect new mammalian hosts. Newer variants are beginning to show the ability to infect hosts ranging from mice, marmosets, and koalas all the way to rats and civets...
Jumping into rats is an inevitability that will turn every urban sewer on earth into white-hot forges for new swarming variants, other mutations waiting to happen have the potential to increase binding by one-thousand fold. And beginning to explore a jump into civets is a reminder that they were incorrectly identified as the original host for the first SARS - all samples jumped from humans into civets, not the other way around - a chimeric virus with roughly a 15% mortality rate.
There is nothing slowing this reversion down, the futility of all the vaccination programs is even more apparent than it was a year ago, as new studies “suggest that SARS-CoV-2 is diverging into distinct serological phenotypes and that vaccines tailored to one variant may become vulnerable to infections with another." And due to the emergence of new serotypes, the first step for ADE, it already appears as if troublesome infection-enhancing antibodies are emerging in spike-protein gene-therapy recipients who are exposed to newer variants - while ADE didn’t appear with exposure to the original Wuhan strain, even just the Delta variant was already beginning to use ADE against us.
"This paradoxical ability, for attenuated viral strains to deattenuate back into their original fully-virulent parent strain – much like the T-1000 was able to mercurially reassemble itself in the sequel to The Terminator – can be explained by examining polio...
...and other RNA viruses not as discrete linear genotypes transmitted on by discrete strains, but instead as quasispecies swarms of virions which carry distinct but complimentary sets of alleles, which work in concert in real-time to establish and expand infections.
One of the first empirical changes that comes once you consider an RNA virus as a quasispecies swarm is that at any point in time, all the extant variants’ genomes form a collective probability cloud that serves as the smallest selective unit...
😱WHO KNEW COVID WOULD INFECT EVERY MAJOR ORGAN SYSTEM!?
"An intriguing clinical correlate is that FCSes within influenza viruses are associated with lymphopenia in infected mice, and with neurological conditions following replication in the brains of ferrets...
...both of which are clinical manifestations observed in hospitalized patients infected by SARS‐CoV‐2 and suffering from COVID‐19.
Moreover, when a furin cleavage site was introduced to the IBV coronavirus spike‐protein via recombination, just like influenza viruses hosting this feature, it appeared to impart it with increased lethality as well as inflict neurological symptoms...