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Wondering whether axonal input *drives* fMRI-based functional connectivity? Us too!
Here is what we found using #chemogenetic deconstruction of rsfMRI in the mouse🐁🐭
➡️Cortical silencing results in paradoxical fMRI overconnectivity tinyurl.com/yy269j92
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Here is what we found using #chemogenetic deconstruction of rsfMRI in the mouse🐁🐭
➡️Cortical silencing results in paradoxical fMRI overconnectivity tinyurl.com/yy269j92
Thread below 1/n
Computational and empirical evidence suggest that structural and functional connectivity are robustly related
➡️ this recent review from @richardfbetzel @misicbata summarizes it all tinyurl.com/yyy5u3zm
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➡️ this recent review from @richardfbetzel @misicbata summarizes it all tinyurl.com/yyy5u3zm
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⚠️A key prediction of structurally based models of fMRI coupling is that *inactivation* of a brain node would result in reduced rsfMRI connectivity with its targets⚠️
But is that really the case?
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But is that really the case?
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