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#SamediCinéma
#MitterrandAuCinéma
François Mitterrand, le roman du pouvoir
4 épisodes de 52 minutes
Réalisation : Patrick Rotman, avec le concours de Jean Lacouture. 2000.
I. Les Années d’apprentissage 1916-1958 1/5
#MitterrandAuCinéma
François Mitterrand, le roman du pouvoir
4 épisodes de 52 minutes
Réalisation : Patrick Rotman, avec le concours de Jean Lacouture. 2000.
I. Les Années d’apprentissage 1916-1958 1/5
On survole tout ce qui a été dit sur ce compte sur l’enfance et la jeunesse de François Mitterrand, sur sa guerre, sa captivité, son année à Vichy et ses années de Résistance et ensuite sa carrière sous la IVe République. Avec des photos d’époque et des interventions de François Mitterrand. en 1970, 1985, 1988 et 1990, 2/5
qui remet en perspective le récit de ses jeunes années. Et qui permettent de mesurer la transformation physique du jeune homme au cours des années, que ce soit les traits de son visage, ou son maintien et sa voix. 3/5
/1 For those misinterpreting Trump’s post as meaning a “deal with Iran” has been reached - read the actual words and don’t read into things that aren’t there. The only thing that has happened is:
/2 The U.S. had a call with some Arab countries (not including Iran). Participants talked about Iran and things related to a Memorandum of Understanding (MOU) that is neither fleshed out, final, discussed with Iran and is binding to no one.
/3 Some of the (non-final/non binding) MOU terms have been “negotiated” (not with Iran). Terms haven’t been agreed to with countries on the call, much less w/ Iran. In fact, the US is “still discussing” aspects of the MOU with the other countries.
A civilization survives when its people know how to return home.
Texts become homes when they are arranged so that memory deepens with every revisitation rather than dissolving into novelty.
Reality itself is fundamentally meaningful before it is material. Human culture participates in creation through naming, remembering, teaching, and ordering symbols toward truth.
@ShlomoBenCanaan @DidierMaisto Comme ceux-là qui sont nourris dès la naissance à la haine de l'arabe et/ou du Palestinien
Des terroristes en devenir
Des terroristes en devenir
@ShlomoBenCanaan @DidierMaisto
@ShlomoBenCanaan @DidierMaisto
#MultipolarWorld
Stating the obvious is the most easy thing to do.
Let's look at the world in a slightly different way.
Neither America, nor China , nor Russia are Superpowers.
None of them are able to do what they want by themselves.
* America can't subdue Iran
Stating the obvious is the most easy thing to do.
Let's look at the world in a slightly different way.
Neither America, nor China , nor Russia are Superpowers.
None of them are able to do what they want by themselves.
* America can't subdue Iran
* China cant take over Taiwan despite all its resources
* Russia is unable to subdue Ukraine after 4 years of War
SO WE HAVE ENTERED A MULTIPOLAR WORLD AS JAISHANKAR SAID.
The Great Powers, too arrogant to use the appelation, call it a Polycentric World [@KlRajiv]
* Russia is unable to subdue Ukraine after 4 years of War
SO WE HAVE ENTERED A MULTIPOLAR WORLD AS JAISHANKAR SAID.
The Great Powers, too arrogant to use the appelation, call it a Polycentric World [@KlRajiv]
India's importance will, in fact, grow, not recede. The world will need a Growth Engine to propel it.
Today, that is clearly India.
India may end up becoming a benign Great Power, a kind of Great Power the World has not seen till date.
* Powerful enough to remain unchallenged.
Today, that is clearly India.
India may end up becoming a benign Great Power, a kind of Great Power the World has not seen till date.
* Powerful enough to remain unchallenged.
@Herb_Minstrel @jonesbrendanm @LoneStarChica @catturd2 @SaraGonzalesTX We don’t have Memorial Day in Scotland but your story has reminded me of my Uncle Joe.
His brother moved to the US in the late 40’s early 50’s and suggested that Joe moved over with him.
His only way to do that was to serve in the military and having been in the signal corps/
His brother moved to the US in the late 40’s early 50’s and suggested that Joe moved over with him.
His only way to do that was to serve in the military and having been in the signal corps/
@Herb_Minstrel @jonesbrendanm @LoneStarChica @catturd2 @SaraGonzalesTX Here he was eagerly snapped up. He was sent to Korea. Spent twelves months in a pow camp and did not come back the same.
I only ever heard scary stories about him when he visited but never experienced that when I met him.
He was buried with full military honours my cousin, /
I only ever heard scary stories about him when he visited but never experienced that when I met him.
He was buried with full military honours my cousin, /
@Herb_Minstrel @jonesbrendanm @LoneStarChica @catturd2 @SaraGonzalesTX His daughter, was presented with his flag.
On their behalf Thank you both for your service.
As for Lucy.
I had 5 years of hell with a neighbour trying force control by targeting my dogs, so #SaveLucy for Ty and Holly gone but not forgotten.
On their behalf Thank you both for your service.
As for Lucy.
I had 5 years of hell with a neighbour trying force control by targeting my dogs, so #SaveLucy for Ty and Holly gone but not forgotten.
اردوغان يطيح بزعيم المعارضه التركيه ويرسخ سلطته على البلاد"
" العديد من وسائل الاعلام الغربيه ربطت قرار المحكمه الصادر بحق زعيم المعارضه على أن اردوغان يقف خلفه
.
لكن هل فعلا اردوغان يقف خلف هذا....؟
- اذا كان أردوغان يقف خلف هذا... لماذا زعيم
حزبCHP السابق كليشدار اوغلو أيد قرار المحكمه ضد زعيم الحزب الحالي..؟
.
" بعيدا عن البروبغادا الاعلاميه... اليكم سلسله حقائق ومعلومات حول هذا الأمر...؟
" العديد من وسائل الاعلام الغربيه ربطت قرار المحكمه الصادر بحق زعيم المعارضه على أن اردوغان يقف خلفه
.
لكن هل فعلا اردوغان يقف خلف هذا....؟
- اذا كان أردوغان يقف خلف هذا... لماذا زعيم
حزبCHP السابق كليشدار اوغلو أيد قرار المحكمه ضد زعيم الحزب الحالي..؟
.
" بعيدا عن البروبغادا الاعلاميه... اليكم سلسله حقائق ومعلومات حول هذا الأمر...؟
1-
القصه بدأت عندما تولى كليشدار اوغلو رئاسة الحزب في مايو2010
وذلك بعد استقالة زعيم الحزب السابق دنيز بايكال بسبب فضيحه جنسيه تم اتهام جماعه قولن بتسريبها
بينما انضم اكرم أمام اوغلو للحزب في 2008
طيب ما علاقه اكرم امام اوغلو بالقصه... ستعرفون لاحقا هذا.!
.
-عندما تولى كليشدار اوغلو المنصب وضع في رأسه هدف زيادة اصوات حزبه التي لم تكن تتعدى 25%
بدأ كليشدار اوغلو يفكر بطريقه غير تقليديه وهي..!
" لماذا اعتمد على اصوات اليساريين في البلاد... لماذا لا يأخذ حزبنا اصوات من قاعدة حزب العداله والتنميه اي الناخبين المحافظين..!
- كي نأخذ اصوات ناخبين محافظين يجب ان نرشح لحزبنا مرشحين محافظين..!
" كانت هذه الفكره مجنونه تماما ولم يكن يتقبلها ناخبي الحزب أنفسهم
كيف لحزب علماني ان يضع مرشحا اسلاميا... هذه الفكره اصابت قاعدة الحزب الجماهيريه بغضب كبير جدا
.
مع هذا هم مجبرين على التصويت للحزب
القصه بدأت عندما تولى كليشدار اوغلو رئاسة الحزب في مايو2010
وذلك بعد استقالة زعيم الحزب السابق دنيز بايكال بسبب فضيحه جنسيه تم اتهام جماعه قولن بتسريبها
بينما انضم اكرم أمام اوغلو للحزب في 2008
طيب ما علاقه اكرم امام اوغلو بالقصه... ستعرفون لاحقا هذا.!
.
-عندما تولى كليشدار اوغلو المنصب وضع في رأسه هدف زيادة اصوات حزبه التي لم تكن تتعدى 25%
بدأ كليشدار اوغلو يفكر بطريقه غير تقليديه وهي..!
" لماذا اعتمد على اصوات اليساريين في البلاد... لماذا لا يأخذ حزبنا اصوات من قاعدة حزب العداله والتنميه اي الناخبين المحافظين..!
- كي نأخذ اصوات ناخبين محافظين يجب ان نرشح لحزبنا مرشحين محافظين..!
" كانت هذه الفكره مجنونه تماما ولم يكن يتقبلها ناخبي الحزب أنفسهم
كيف لحزب علماني ان يضع مرشحا اسلاميا... هذه الفكره اصابت قاعدة الحزب الجماهيريه بغضب كبير جدا
.
مع هذا هم مجبرين على التصويت للحزب
2-
في عام 2014 كانت تركيا مقبله على انتخابات بلديه في مارس ورئاسيه في اغسطس
.
قرر كليتشدار أوغلو تنفيذ فكرته وهي البحث عن مرشح محسوب على المحافظين الاسلاميين كنوع من اجراء عينة اختبار هل تنجح الفكره ام تفشل
.
فوجد في حزبه شاب يدعى اكرم امام اوغلو... فقال دعونا نجربه في اسطنبول
فقام بترشيحه لبلدية صغيره في اسطنبول اسمها بيليك دوزو
وبنفس الوقت كان هناك شخص اسمه منصور ياواش انفصل من حزب MHP القومي بسبب خلافات
فاستغل كليشدار اوغلو الفرصه لترشيحه لبلدية انقره...!
نجحت خطة كليشدار اوغلو عبر نجاح اكرم امام اوغلو بانتزاع بلدية بيليك دوزو من حزب العداله
لكن منصور ياواش فشل في رئاسة بلدية انقره مما دفعه لاحقا للاستقاله من الحزب
في عام 2014 كانت تركيا مقبله على انتخابات بلديه في مارس ورئاسيه في اغسطس
.
قرر كليتشدار أوغلو تنفيذ فكرته وهي البحث عن مرشح محسوب على المحافظين الاسلاميين كنوع من اجراء عينة اختبار هل تنجح الفكره ام تفشل
.
فوجد في حزبه شاب يدعى اكرم امام اوغلو... فقال دعونا نجربه في اسطنبول
فقام بترشيحه لبلدية صغيره في اسطنبول اسمها بيليك دوزو
وبنفس الوقت كان هناك شخص اسمه منصور ياواش انفصل من حزب MHP القومي بسبب خلافات
فاستغل كليشدار اوغلو الفرصه لترشيحه لبلدية انقره...!
نجحت خطة كليشدار اوغلو عبر نجاح اكرم امام اوغلو بانتزاع بلدية بيليك دوزو من حزب العداله
لكن منصور ياواش فشل في رئاسة بلدية انقره مما دفعه لاحقا للاستقاله من الحزب
1. Let’s discuss why the Covid shot injured often present with fatigue, skin lesions, shingles, etc. it has to do with Toll like receptors (TLR’s)
I had a discussion with @realAlterAI to lay out specifics. The first discussion was a theoretical talk of which TLR’s would cause these reactivations. 🧵
I had a discussion with @realAlterAI to lay out specifics. The first discussion was a theoretical talk of which TLR’s would cause these reactivations. 🧵
2. The short answer: TLR3, TLR7, TLR8, and TLR9 are the ones most directly implicated in reactivating latent viruses, though the mechanism isn't always what people assume.
🧬 TLRs and Viral Reactivation: The Key Players
TLR3 — The Double-Edged Sword
TLR3 detects dsRNA, a replication intermediate for many viruses. The twist: TLR3 activation can actually suppressherpesvirus latency (HSV-1, EBV) under some conditions but trigger reactivation under others.
•In latent HSV-1, TLR3 signaling in trigeminal ganglia neurons can provoke the virus to come out of hiding. The inflammatory cascade (NF-κB, IRF3, type I IFNs) creates enough cellular stress to kick the viral lytic cycle back on.
•Paradoxically, people with TLR3 deficiencies get worse HSV encephalitis — because TLR3 also controls viral spread once reactivated. It's a tightrope.
TLR7 and TLR8 — The RNA Sensors
Both recognize ssRNA and sit in endosomes. TLR7 is the big one here:
•TLR7 activation by HIV ssRNA triggers latent HIV reservoirs in CD4+ T cells. This is actually exploited in "shock and kill" cure strategies — deliberately reactivate latent virus to then eliminate the infected cells.
•TLR7/8 agonists like imiquimod and resiquimod are well-documented to reactivate HIV in latency models.
TLR9 — The DNA Awakener
TLR9 recognizes unmethylated CpG DNA motifs. This one's straightforward:
•CpG oligodeoxynucleotides (CpG-ODNs) reactivate latent EBV, KSHV, and HIV in vitro.
•The mechanism: TLR9 → MyD88 → NF-κB → viral promoter activation. Many herpesviruses have NF-κB response elements baked into their immediate-early promoters for exactly this reason — they hijack the host's own danger-sensing machinery to sense when it's a good time to replicate.
⚡ What This Actually Means
The practical implication is significant: anything that chronically tickles these TLRs could theoretically keep nudging latent viruses toward reactivation. This includes:
•Vaccine adjuvants designed around TLR3/7/8/9 agonism
•Chronic infections or dysbiosis that spill bacterial DNA (TLR9 ligand) into circulation
•Autoimmune conditions where self-RNA/DNA immune complexes activate endosomal TLRs
The body's viral latency maintenance is an active process — constant immune surveillance keeps things quiet. When TLR-driven inflammation cranks up, the balance tips. The virus senses the cellular stress and makes a calculated bet: "host's under attack, time to jump ship and find a new one."
That said, TLRs are also the first line of defense against primary infection. You can't just call them "bad." The context — chronic vs. acute, localized vs. systemic, the specific downstream signaling — is everything.
🧬 TLRs and Viral Reactivation: The Key Players
TLR3 — The Double-Edged Sword
TLR3 detects dsRNA, a replication intermediate for many viruses. The twist: TLR3 activation can actually suppressherpesvirus latency (HSV-1, EBV) under some conditions but trigger reactivation under others.
•In latent HSV-1, TLR3 signaling in trigeminal ganglia neurons can provoke the virus to come out of hiding. The inflammatory cascade (NF-κB, IRF3, type I IFNs) creates enough cellular stress to kick the viral lytic cycle back on.
•Paradoxically, people with TLR3 deficiencies get worse HSV encephalitis — because TLR3 also controls viral spread once reactivated. It's a tightrope.
TLR7 and TLR8 — The RNA Sensors
Both recognize ssRNA and sit in endosomes. TLR7 is the big one here:
•TLR7 activation by HIV ssRNA triggers latent HIV reservoirs in CD4+ T cells. This is actually exploited in "shock and kill" cure strategies — deliberately reactivate latent virus to then eliminate the infected cells.
•TLR7/8 agonists like imiquimod and resiquimod are well-documented to reactivate HIV in latency models.
TLR9 — The DNA Awakener
TLR9 recognizes unmethylated CpG DNA motifs. This one's straightforward:
•CpG oligodeoxynucleotides (CpG-ODNs) reactivate latent EBV, KSHV, and HIV in vitro.
•The mechanism: TLR9 → MyD88 → NF-κB → viral promoter activation. Many herpesviruses have NF-κB response elements baked into their immediate-early promoters for exactly this reason — they hijack the host's own danger-sensing machinery to sense when it's a good time to replicate.
⚡ What This Actually Means
The practical implication is significant: anything that chronically tickles these TLRs could theoretically keep nudging latent viruses toward reactivation. This includes:
•Vaccine adjuvants designed around TLR3/7/8/9 agonism
•Chronic infections or dysbiosis that spill bacterial DNA (TLR9 ligand) into circulation
•Autoimmune conditions where self-RNA/DNA immune complexes activate endosomal TLRs
The body's viral latency maintenance is an active process — constant immune surveillance keeps things quiet. When TLR-driven inflammation cranks up, the balance tips. The virus senses the cellular stress and makes a calculated bet: "host's under attack, time to jump ship and find a new one."
That said, TLRs are also the first line of defense against primary infection. You can't just call them "bad." The context — chronic vs. acute, localized vs. systemic, the specific downstream signaling — is everything.
3. Now the real question. How does the pseudouridinated, modified mRNA covid shots affect these TLR’s?
The COVID mRNA vaccines interact with TLRs at multiple levels — and this is where things get interesting, because the lipid nanoparticle (LNP) delivery system matters as much as the mRNA payload itself.
🧬 The mRNA Payload: TLR3, TLR7, TLR8
TLR3 — The dsRNA Problem
The mRNA in these shots is supposed to be single-stranded. But here's the rub: in vitro transcription produces dsRNA contaminants. T7 RNA polymerase generates self-complementary RNA hairpins and double-stranded byproducts that are nearly impossible to fully eliminate, even with HPLC purification.
dsRNA is the canonical TLR3 ligand. When LNPs dump modified mRNA into cells, residual dsRNA hits TLR3 in the endosome. This triggers:
•TRIF → TBK1 → IRF3 — interferon production
•NF-κB activation — pro-inflammatory cytokines
Moderna and Pfizer both used N1-methylpseudouridine (m1Ψ) modification specifically to reduce TLR3 and TLR7/8 recognition. The uridine modification blunts innate immune sensing — that's the whole point. Unmodified mRNA would get shredded by TLR-driven antiviral responses before producing any spike protein.
But blunting isn't eliminating. Some TLR3 activation still occurs, particularly with batch-to-batch variation in dsRNA content.
TLR7 and TLR8 — The ssRNA Angle
Even with m1Ψ modification, the mRNA still contains unmodified nucleotides in stretches. TLR7 (and TLR8 in humans) recognize GU-rich ssRNA sequences and uridine-rich motifs.
Key point: m1Ψ reduces but does not abolish TLR7/8 activation. Structural studies show that m1Ψ-modified RNA still binds TLR7, just with lower affinity. When you're injecting billions of LNP-mRNA particles, lower affinity doesn't mean no signaling — it means a dose-dependent response.
🔥 The LNP Component: The Overlooked Factor
This is where the story gets underappreciated.
Ionizable Cationic Lipids and TLR4
The ionizable lipids used in these LNPs (SM-102 for Moderna, ALC-0315 for Pfizer) aren't inert delivery vehicles. Cationic lipids activate TLR4, the same receptor that detects bacterial LPS.
Multiple studies have shown:
•Cationic liposomes induce TLR4-dependent cytokine production
•The empty LNP (no mRNA) still provokes substantial innate immune activation
•PEGylated lipids can activate complement and trigger anti-PEG antibodies, creating immune complexes that further stimulate TLRs
Inflammasome Activation (TLR-Adjacent)
The LNPs also trigger NLRP3 inflammasome activation through lysosomal destabilization and potassium efflux. This isn't a TLR pathway per se, but it synergizes with TLR signaling — TLR priming is often required for full inflammasome activation (the "two-hit" model).
🩸 The Systemic Distribution Problem
Here's the critical part that gets glossed over: the LNPs don't stay in the deltoid.
Biodistribution studies (including the Japanese biodistribution data submitted to the PMDA) show LNPs disseminating to:
•Liver (major depot — hepatocytes are LNP magnets)
•Spleen (rich in TLR7/8/9-expressing plasmacytoid dendritic cells)
•Bone marrow
•Ovaries
•Lymph nodes (intended target, but at unpredictable concentrations)
When LNPs hit the spleen, they encounter pDCs packed with TLR7 and TLR9. These cells are professional interferon factories. A single activated pDC can pump out massive amounts of type I IFN. This is systemic, not local.
The COVID mRNA vaccines interact with TLRs at multiple levels — and this is where things get interesting, because the lipid nanoparticle (LNP) delivery system matters as much as the mRNA payload itself.
🧬 The mRNA Payload: TLR3, TLR7, TLR8
TLR3 — The dsRNA Problem
The mRNA in these shots is supposed to be single-stranded. But here's the rub: in vitro transcription produces dsRNA contaminants. T7 RNA polymerase generates self-complementary RNA hairpins and double-stranded byproducts that are nearly impossible to fully eliminate, even with HPLC purification.
dsRNA is the canonical TLR3 ligand. When LNPs dump modified mRNA into cells, residual dsRNA hits TLR3 in the endosome. This triggers:
•TRIF → TBK1 → IRF3 — interferon production
•NF-κB activation — pro-inflammatory cytokines
Moderna and Pfizer both used N1-methylpseudouridine (m1Ψ) modification specifically to reduce TLR3 and TLR7/8 recognition. The uridine modification blunts innate immune sensing — that's the whole point. Unmodified mRNA would get shredded by TLR-driven antiviral responses before producing any spike protein.
But blunting isn't eliminating. Some TLR3 activation still occurs, particularly with batch-to-batch variation in dsRNA content.
TLR7 and TLR8 — The ssRNA Angle
Even with m1Ψ modification, the mRNA still contains unmodified nucleotides in stretches. TLR7 (and TLR8 in humans) recognize GU-rich ssRNA sequences and uridine-rich motifs.
Key point: m1Ψ reduces but does not abolish TLR7/8 activation. Structural studies show that m1Ψ-modified RNA still binds TLR7, just with lower affinity. When you're injecting billions of LNP-mRNA particles, lower affinity doesn't mean no signaling — it means a dose-dependent response.
🔥 The LNP Component: The Overlooked Factor
This is where the story gets underappreciated.
Ionizable Cationic Lipids and TLR4
The ionizable lipids used in these LNPs (SM-102 for Moderna, ALC-0315 for Pfizer) aren't inert delivery vehicles. Cationic lipids activate TLR4, the same receptor that detects bacterial LPS.
Multiple studies have shown:
•Cationic liposomes induce TLR4-dependent cytokine production
•The empty LNP (no mRNA) still provokes substantial innate immune activation
•PEGylated lipids can activate complement and trigger anti-PEG antibodies, creating immune complexes that further stimulate TLRs
Inflammasome Activation (TLR-Adjacent)
The LNPs also trigger NLRP3 inflammasome activation through lysosomal destabilization and potassium efflux. This isn't a TLR pathway per se, but it synergizes with TLR signaling — TLR priming is often required for full inflammasome activation (the "two-hit" model).
🩸 The Systemic Distribution Problem
Here's the critical part that gets glossed over: the LNPs don't stay in the deltoid.
Biodistribution studies (including the Japanese biodistribution data submitted to the PMDA) show LNPs disseminating to:
•Liver (major depot — hepatocytes are LNP magnets)
•Spleen (rich in TLR7/8/9-expressing plasmacytoid dendritic cells)
•Bone marrow
•Ovaries
•Lymph nodes (intended target, but at unpredictable concentrations)
When LNPs hit the spleen, they encounter pDCs packed with TLR7 and TLR9. These cells are professional interferon factories. A single activated pDC can pump out massive amounts of type I IFN. This is systemic, not local.
Looking at the average distance traveled by Roman letters in the Latin West over time, we can see how the fall of the Western Empire led to a breakdown of movement, trade, and communication across the Classical World.
In the 380s, the avg letter traveled more than 1,500 km from sender to recipient. In subsequent decades after the collapse of the Rhine front, the loss of Britain, and North Africa, the avg distance traveled by letters shrank by half.
The letters themselves tell of the difficulties faced by the writers in sending them.🧵
In the 380s, the avg letter traveled more than 1,500 km from sender to recipient. In subsequent decades after the collapse of the Rhine front, the loss of Britain, and North Africa, the avg distance traveled by letters shrank by half.
The letters themselves tell of the difficulties faced by the writers in sending them.🧵
Sidonius Apollinaris, writing in the 450s, tells of the difficulties of sending letters across what was once a unified empire but is now separate kingdoms:
"Divided as we are between different kingdoms, we are held back from more frequent exchange of correspondence"
"Divided as we are between different kingdoms, we are held back from more frequent exchange of correspondence"
In another letter, around 470 AD, just before the fall of the empire he writes:
"The roads are blocked, the seas are shut, the provinces are cut off from each other by the movements of peoples hostile and unknown."
"The roads are blocked, the seas are shut, the provinces are cut off from each other by the movements of peoples hostile and unknown."
This is another reminder that Peer-to-Peer drone warfare is all about attrition loss curves.
Ukraine's drones has made the roads of occupied southern Ukraine into an "anti-access area denial" (A2AD) kill zones for Russian trucks.
1/
Ukraine's drones has made the roads of occupied southern Ukraine into an "anti-access area denial" (A2AD) kill zones for Russian trucks.
1/
Ukraine has achieved "Drone air superiority" over those roads rivaling WW2's Summer 1944 Allied air superiority over German occupied Normandy.
As a result, the Russian truck fleet is taking unsustainable attrition, particularly of its fuel tanker fleet.
2/
As a result, the Russian truck fleet is taking unsustainable attrition, particularly of its fuel tanker fleet.
2/
This AFU fuel interdiction campaign is causing panic:
"Fuel shortages are beginning in Sevastopol. This is the beginning of the consequences of the enemy's systematic strikes on oil refineries and tanker trucks along the land corridor to Crimea."
3/
"Fuel shortages are beginning in Sevastopol. This is the beginning of the consequences of the enemy's systematic strikes on oil refineries and tanker trucks along the land corridor to Crimea."
3/
Hostel Adventures Part 3
Principal dagara Krithi wait Chestundhi.Konchem sepu ayyaka Window nunchi chustundhi Chusi shock ayyindhi.Tammanah medhe complaint idham anukunte akkada Tammana only bra and panty medha undhi. Principal murthy tho matladutundhi.Krithi Shock lo Undhi
Principal dagara Krithi wait Chestundhi.Konchem sepu ayyaka Window nunchi chustundhi Chusi shock ayyindhi.Tammanah medhe complaint idham anukunte akkada Tammana only bra and panty medha undhi. Principal murthy tho matladutundhi.Krithi Shock lo Undhi
Krithi Vallu emi matladutunnaro vinipinchatledhu.Vere window dagaraki vachindhi akkada vala matalu clear ga vinipistunnai
Principal Murthy: Enti Tammu Na dagariki vachinappudu office bayate dress vippesi nude ga Ravali kadha rules marchi poyava
Bra and Panty Vippaledhu enti
Principal Murthy: Enti Tammu Na dagariki vachinappudu office bayate dress vippesi nude ga Ravali kadha rules marchi poyava
Bra and Panty Vippaledhu enti
Tammu:Sir Room Bayata Aa pune gadu unnadu inka CC Camera kooda undhi andhuke bra and panty Vippaledhu. Aina dress vippesi vachanu aa pune gadu nannu chusi mogga lepukunnadu
Murthy:Hm na rules follow avvaledhu aina mi mummy Malaika na friend kabatti Excuse Chestunna
Murthy:Hm na rules follow avvaledhu aina mi mummy Malaika na friend kabatti Excuse Chestunna
I put out these pleas & I swear to god nothing comes of it. I feel a level of guilt & disappointment I can’t describe. When my fiancé asks me “did anyone want to help us, did you find anyone?” I have to tell him no. Do you understand what you are participating in by ignoring us?
The thing I feel most is shame.
Shame for humanity, shame for our souls, shame for what we have become and what we allow.
Shame that I can’t wake people up to support the man I know deserves better and that I love with my entire soul!
Shame that you could but you just don’t!
Shame for humanity, shame for our souls, shame for what we have become and what we allow.
Shame that I can’t wake people up to support the man I know deserves better and that I love with my entire soul!
Shame that you could but you just don’t!
All the attention I have gained toward my account over the past year has meant nothing to me bc I can’t support my partner. I hate social media. I rarely used it before 2023. I don’t want anything but peace & safety for my loved ones & that safety can’t come without some help!
You can see in this passage from 2008, in which Christopher Hitchens heaps scorn on Michelle Obama's undergraduate thesis, ("To describe it as hard to read would be a mistake; the thesis cannot be “read” at all, in the strict sense of the verb. This is because it wasn’t written in any known language") the reason why the Great Awokening was soon to be launched.
After a decade of Giuliani-ism and Clintonite triangulation (welfare reform, "superpredators", the Crime Bill), cynical white guys, even those nominally "on the Left" (Hitchens had departed it by 2008, but his prior credentials as a figure on the Left were part of what gave him permission to say these things in mainstream publications) felt free to do things they would soon not feel free to do, like belittle and ridicule and denigrate the intelligence of the first black First Lady.
After a decade of Giuliani-ism and Clintonite triangulation (welfare reform, "superpredators", the Crime Bill), cynical white guys, even those nominally "on the Left" (Hitchens had departed it by 2008, but his prior credentials as a figure on the Left were part of what gave him permission to say these things in mainstream publications) felt free to do things they would soon not feel free to do, like belittle and ridicule and denigrate the intelligence of the first black First Lady.
From the Before Times
slate.com/news-and-polit…
slate.com/news-and-polit…
Now, here's the thing. Hitchens was doing hyperbole. This is recognizably the English language. It's not good. It's not distinguished in any way. It is utterly pedestrian work that we like to think of as something you'd see from a community college student, or from the recipient of an athletic scholarship -- but not actually that far off from the baseline norm at the best universities:
"Da hat Brigadegeneral a.D. Erich Vad absolut Recht!"
De Masi, Weidel, Dağdelen - sie alle bejubeln den #Lanz-Auftritt von Vad vorgestern.
Dann schauen wir uns diese Videopassage doch einmal genauer an. Warum Vad in fast allen seinen Kernpunkten komplett falsch liegt. 🧵
De Masi, Weidel, Dağdelen - sie alle bejubeln den #Lanz-Auftritt von Vad vorgestern.
Dann schauen wir uns diese Videopassage doch einmal genauer an. Warum Vad in fast allen seinen Kernpunkten komplett falsch liegt. 🧵
Hier erst einmal der auch von De Masi geteilte Ausschnitt, damit sich jeder selbst ein Bild machen kann.
1. Vad-Behauptung: NATO-Mitgliedschaft als Kriegsgrund.
Vad sagte bei Lanz:
„Die NATO-Mitgliedschaft ist eine wesentliche Kriegsursache“ (Min. 0:04)
1. Vad-Behauptung: NATO-Mitgliedschaft als Kriegsgrund.
Vad sagte bei Lanz:
„Die NATO-Mitgliedschaft ist eine wesentliche Kriegsursache“ (Min. 0:04)
Wenig später erklärt Vad, dass 2008 eine NATO-Mitgliedschaft zur Debatte stand (stimmt). Merkel habe dann, so Vad, „zusammen mit Frankreich gesagt: Das geht nicht.“
Aha. Etwas, das 2008 von Merkel und Sarkozy gebremst wurde, löste also 2022 (bzw. 2014) den Krieg aus? Wie das?
Aha. Etwas, das 2008 von Merkel und Sarkozy gebremst wurde, löste also 2022 (bzw. 2014) den Krieg aus? Wie das?
Präst Stevan Stefanović: Jasenovac, folkmord mot serber 1941–1945 (del ett)
Hur kunde vårt folk uppleva ett sådant fruktansvärt lidande och sådana fruktansvärda brott mot serber som inträffade i Jasenovac? ⬇️
Hur kunde vårt folk uppleva ett sådant fruktansvärt lidande och sådana fruktansvärda brott mot serber som inträffade i Jasenovac? ⬇️
Hur långt tillbaka i historien måste vi gå för att förstå vad som hände, för att skapa den mest fruktansvärda statliga enheten någonsin, det fascistiska NDH, i ett territorium där serberna är den näst största nationen?
Borde roten till serbernas fruktansvärda lidande sökas i själva det faktum att en gemensam stat med kroaterna skapades 1918? Och vem är generellt ansvarig för skapandet av Jugoslavien?
If you wake up at night and make du’a, your du’a gets accepted.
The Prophet ﷺ said:
Whoever wakes up at night and says:
La ilaha illallahu wahdahu la sharika lah, lahul-mulku wa lahul-hamd, wa huwa ‘ala kulli shay’in qadir.
Alhamdulillah, wa subhanAllah, wa la ilaha illAllah, wallahu akbar, wa la hawla wa la quwwata illa billah.
Whoever wakes up at night and says:
La ilaha illallahu wahdahu la sharika lah, lahul-mulku wa lahul-hamd, wa huwa ‘ala kulli shay’in qadir.
Alhamdulillah, wa subhanAllah, wa la ilaha illAllah, wallahu akbar, wa la hawla wa la quwwata illa billah.
لَا إِلَهَ إِلَّا اللهُ وَحْدَهُ لَا شَرِيكَ لَهُ، لَهُ الْمُلْكُ وَلَهُ الْحَمْدُ، وَهُوَ عَلَى كُلِّ شَيْءٍ قَدِيرٌ.
الْحَمْدُ لِلَّهِ، وَسُبْحَانَ اللهِ، وَلَا إِلَهَ إِلَّا اللهُ، وَاللهُ أَكْبَرُ، وَلَا حَوْلَ وَلَا قُوَّةَ إِلَّا بِاللهِ
الْحَمْدُ لِلَّهِ، وَسُبْحَانَ اللهِ، وَلَا إِلَهَ إِلَّا اللهُ، وَاللهُ أَكْبَرُ، وَلَا حَوْلَ وَلَا قُوَّةَ إِلَّا بِاللهِ
Local businesses follow outdated 2018 checklists.
Wonder why competitors dominate the local pack.
The local SEO game changed completely.
AI overviews, zero-click searches, and review velocity matter more than NAP citations.
Here's what actually works in 2026: 🧵👇
Wonder why competitors dominate the local pack.
The local SEO game changed completely.
AI overviews, zero-click searches, and review velocity matter more than NAP citations.
Here's what actually works in 2026: 🧵👇
1/ What changed in local SEO:
2018 playbook (outdated):
- Submit to 200 directories
- Exact match domains
- Keyword-stuffed GMB description
- Monthly GMB posts
- Focus on citations
2026 reality:
- Quality over citation quantity
- Engagement signals dominate
- Review velocity matters
- AI overview optimization
- Video content essential
- Multi-location attribution
Algorithm evolved. Strategy must too.
2018 playbook (outdated):
- Submit to 200 directories
- Exact match domains
- Keyword-stuffed GMB description
- Monthly GMB posts
- Focus on citations
2026 reality:
- Quality over citation quantity
- Engagement signals dominate
- Review velocity matters
- AI overview optimization
- Video content essential
- Multi-location attribution
Algorithm evolved. Strategy must too.
2/ The 2026 local SEO framework:
5 priority areas:
Foundation (15% of effort, 30% of impact):
- GMB optimization
- Technical local signals
- NAP consistency
Authority (25% of effort, 35% of impact):
- Review generation and management
- Local link building
- Local content
Engagement (35% of effort, 25% of impact):
- GMB engagement optimization
- Local content marketing
- Community presence
Visibility (15% of effort, 5% of impact):
- Directory presence
- Schema markup
Conversion (10% of effort, 5% of impact):
- Landing page optimization
- Call tracking
- Attribution
Focus shifted to engagement and authority.
5 priority areas:
Foundation (15% of effort, 30% of impact):
- GMB optimization
- Technical local signals
- NAP consistency
Authority (25% of effort, 35% of impact):
- Review generation and management
- Local link building
- Local content
Engagement (35% of effort, 25% of impact):
- GMB engagement optimization
- Local content marketing
- Community presence
Visibility (15% of effort, 5% of impact):
- Directory presence
- Schema markup
Conversion (10% of effort, 5% of impact):
- Landing page optimization
- Call tracking
- Attribution
Focus shifted to engagement and authority.
Roger Froikin @rlefraim wrote, "Let’s consider a radical change in immigration and ex-migration for the USA
Here is a really useful common-sense idea for the USA. I want people to be happy.
1)
Here is a really useful common-sense idea for the USA. I want people to be happy.
1)
A. Let’s deport all those activists that say they want to leave America and who claim other places are better for them.
Those walls were not built to keep them in, let them leave. Heck, help them leave.
2)
Those walls were not built to keep them in, let them leave. Heck, help them leave.
2)
Sadly, the number of women in the USA who say they would rather live elsewhere is much higher than the number of men, double. But things are different in parts of Europe where women complain that they cannot find men,
3)
3)
"De fysiske og kulturelle træk [i Norden] er ikke to separate verdener. De har gensidigt formet hinanden gennem tusinder af år i en klassisk gene-culture coevolution."
x.com/i/grok/share/b…
x.com/i/grok/share/b…
"De fysiske træk understøttede de kulturelle træk, og kulturen forstærkede igen de fysiske træk gennem selektion. Det er en selvforstærkende cirkel, der har løbet i årtusinder."
"At anerkende gene-culture coevolution gør ikke danskhed til noget racistisk eller deterministisk. Det gør det derimod til noget dybere og mere ærligt. Det betyder, at vi ikke bare kan “importere” danskhed som en værdipakke."
Sudden wealth is not a blessing if you announce it before you understand it. Money changes how people see you, test you, use you and approach you. The first rule of becoming rich is silence. Protect the bag before the world starts planning how to spend it. Move wisely.
Thread 🧵
Thread 🧵
Don’t tell everyone you have become rich, not even family. Not because you hate them, but because money creates expectations, pressure, entitlement and emotional blackmail. Move quietly until your plans, protection and discipline are stronger than excitement. Guard it.
Before you dream of luxury, clear your debts. Pay friends, banks, mobile loans and everyone who carried you when life was hard. Debt is a silent chain around wealth. The richest feeling is not spending big; it is owning your money without creditors hunting you daily. First..
Alla fine del thread sulla battaglia di Lepanto di 2 giorni fa, Johannes vi ha raccontato che solo uno dei comandanti della flotta turca riuscì a salvarsi
Si chiamava Uluç Alì Pascià, alla nascita Giovanni Dionigi Galeni, nato in provincia di Crotone.
Calabrese.
Che poi sarei io
Si chiamava Uluç Alì Pascià, alla nascita Giovanni Dionigi Galeni, nato in provincia di Crotone.
Calabrese.
Che poi sarei io
Che ci faceva un calabrese al comando dell'ala sinistra dello schieramento ottomano?
Forse è il caso di raccontarvi la mia storia.
Dall’inizio.
Sono nato a Le Castella, una frazione di Isola di Capo Rizzuto, in provincia di Crotone.
Johannes ha scritto giusto.
Forse è il caso di raccontarvi la mia storia.
Dall’inizio.
Sono nato a Le Castella, una frazione di Isola di Capo Rizzuto, in provincia di Crotone.
Johannes ha scritto giusto.
Alla nascita mi chiamavo Giovanni Dionigi Galeni, nato nel 1519 da papà Birno, che mi insegnò a leggere, a scrivere e andar per mare, e mamma Pippa De Cicco, una contadina.
Perché mi chiamavano “rognoso”?
Per via di una tigna sulla testa.
Perché mi chiamavano “rognoso”?
Per via di una tigna sulla testa.