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The findings of this one suggest that persistent de novo lipogenesis during fasting may at least partly explain impaired fasting ketogenesis in metabolic syndrome, which appears to be the consequence of reciprocal regulation of DNL and β-oxidation. 
- Forty non-diabetic individuals with and without a history of NAFL were recruited for this study.
- Lipogenesis remained detectable in a subset of individuals after a 24 hour fast, including some subjects without hepatic steatosis or other hallmarks of metabolic syndrome.
