Abdel-Wahab Lab Profile picture
The Abdel-Wahab Lab studies the functional genomics of hematologic malignancies, focusing on mutations in members of the spliceosomal complex.

Apr 12, 2021, 10 tweets

We are excited to announce our paper in @NatureGenet uncovering a role for minor introns in clonal hematopoietic disorders, Noonan Syndrome, and a diverse number of cancers. A collaboration between our lab (@sloan_kettering) and @bradleybio (@fredhutch): nature.com/articles/s4158…

2.Most eukaryotes have 2 splicing machineries: the major & minor spliceosome. The minor spliceosome recognizes <0.5% of the introns in the human genome. Since their discovery, biological roles for minor introns have been enigmatic.

3.In work co-led by @DaichiInoue5, Jacob Polaski, and @TaylorJ_MD we identify that deletion of the minor spliceosome regulatory protein ZRSR2, frequently mutated in MDS and AML, enhances hematopoietic stem cell self-renewal.

4.We mapped ZRSR2 responsive introns across patients and mouse hematopoietic cells using RNA-seq and anti-ZRSR2 eCLIP-seq to find a subset of ZRSR2 regulated introns.

5.Beautiful work from Jose Pineda @bradleybio identified unique branchpoint features of ZRSR2 regulated introns. Please see Jose’s prior great paper in @GenesDev:
genesdev.cshlp.org/content/early/…

6.We then performed a CRISPR functional enrichment screen to identify those ZRSR2 regulated splicing events which may be associated with clonal advantage in hematopoietic cells.

7.This revealed a surprising discovery of mis-splicing of the minor intron of the CUL3 adaptor regulating RAS GTPas abundance LZTR1 in ZRSR2-mutant MDS and AML.

8.With wonderful help from @castel_pau & the Frank McCormick lab (@frankpmccormick) we found that mutations within LZTR1’s minor intron itself was transforming to hematopoietic cells and present in Noonan Syndrome (@noonansyndrome, @Noonan_Syndrome) as well as Schwannomatosis.

9.Moreover, in analyzing LZTR1 minor intron regulation across the pan-can #TCGA dataset, @chewomics (@csi_singapore) identified widespread impaired LZTR1 minor intron excision across cancers (the basis for which we are actively working to understand now):

10.Thanks to funding for Rob and I from the @EdwardPEvansFdn, @theNCI, @nih_nhlbi, and @CDMRP

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