Thread by @CharlesRixey

[1]
How interesting.
The parts of the env trimer for HlV found in the SARS-CoV-2 genome happen to match the epitopes targeted for vx studies since ~2015.
They target the CD4 binding site.
& DC-SIGN [CD209].
This is what was blocked by Fαuci in Jan 2020
@GigaohmResist @TyCardon

@GigaohmResist @TyCardon [2]
Fauci knew what these meant, because his VRC had been working on such candidates since at least 2006.
He knew that Furin triggers cancer cell growth, and that HlV is enhanced by/enhances amyloid dev.
The cascade of both would look just like what @EthicalSkeptic has shown us

@GigaohmResist @TyCardon @EthicalSkeptic [3]
This is what has become clear to me after reading 400 studies just looking at the homologous elements between HlV/SARS-CoV-2.
It's the end result of the findings laid out in⬇️
It's what they must hide until after the election
researchgate.net/publication/35…

@GigaohmResist @TyCardon @EthicalSkeptic [4]
All of which was sparked by the pre-print from a group of researchers in India.
They are heroes, who sought to warn the world of what was coming [@asrayagiriraj].
Foul-chi is now blocking research on l0ng C0vid - because he knows it's his fault, after hiding the homology

@GigaohmResist @TyCardon [1.5]
The fusion peptide [FP] of both viruses was already almost identical, which is key for understanding the promise of fusion inhib.
The 1st pic above describes the need for CD4bs + at least 2 other epitopes. 1 is the FP, & another is the V2 loop.
Homologous 'enough'
but wait-

@GigaohmResist @TyCardon [1.6]
The 2 regions identified by Montagnier & @JCPEREZCODEX stretch across the 1st part of the Spike protein, increasing this homology.
Perhaps even allowing for tests with certain primers to detect the presence of certain things