Who Are the Mystery Girls?
Scientists found a mystery gene sequence in a Pfzer COVID ๐ lot. What could this do in the body? ๐งต
Geneticist @Kevin_Mckernan explains in his sstack Nepetalactone Newsletter:
The protein expressed is similar to the protein profile for silk protein, and to bacterial collagen and fibroin.
The "uncharacterized protein" is from Oscillibacter,
which is a butyrate producing bacteria. >
Oscillibacter is a bacteria found in the gut microbiome. It is involved in insulin homeostasis. Recall that Dr. Sabine Hazan found that the COVID ๐ can wipe out the good gut bacteria. Is this protein (or the others) related to her finding, or is loss from something else? >
Human cells cannot make a spider's silk protein, but they can make a similar amyloid (misfolded) protein. >
Spider silk can self-assemble into nano fibrils. >
Spiker silk is the strongest fiber found in nature. Scientists use bacteria to try to reproduce synthetic spiker silks. >
These proteins are studied for the fibrillar structures. >
Given the amyloid clotting properties of the SARS2 spike protein, well documented in several papers (see #TeamClots Pretorius and Kell) these additional mystery proteins might give a gain of function effect of amplifying the spike's amyloidosis. >
Here Jessica explains the amyloidosis.
The spike protein has a prion genesis site on it so it is already highly efficient at misfolding proteins.
The SARS S1 subunit can activate the complement system (ITR) even without the presence of the virus.
The #ITR #InflammoThromboticResponse is described here by Dr. Flemming. This response attacks and destroys cells of the endothelium (lining of vessels and organs) as they express spike protein. This causes a release of collagen and cell debris. >
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If the mystery proteins produced are also a form of collagen, then this would add to the amount of collagen released when the tunica layers are destroyed. In the presence of PEG, this would aid in the possible formation of a hydrogel like substance. >
Multiple factors implicated in clotting from the ๐: the lipid nanoparticles. see @_HeartofGrace_, and the free spike see @lasikeyecenter1 #StringTheory
The mystery proteins might aid in stabilizing the clots formed by the spike protein, perhaps giving durability and redundancy.
#WhoAreTheMysteryGirls #InvissibleThreadTheory #InvissibleTouch #MysteryProtein #ClotClotBaby #pandemicplaylist
Can't forget about the n1-methyl-pseudoU
Dear X, this ๐งต is a THEORY.
Please stop ghost banning me for asking questions.
Here is Christie Grace's explanation of how Zeta potential (charge on an LNP) causes clots to be produced in the body by the LNPs.
This effect ALONE can explain the #CalimarClots. >
Now imagine if billions of charged LNPs are introduced into the bloodstream (not the deltoid) >
If billions of LNPs increase clotting by 1000xs and we add that ability to the spike protein's ability to form amyloid micro clots, then the use of a LNP casing for an mRNA expressing a spike protein is a GAIN OF FUNCTION, increasing its amyloid clotting formation. >
This is a similar #BaitAndSwitch the drug makers of the 1976 Swine Flu vaxx used which caused thousands of cases of Guillane Barre Syndrome. >
Manufacturers would have known about this effect from the clinical trials and a simple blood analysis. And Pfzer knew from their leaked biodistribution study. #TheyKnew >
This is why Speicher and McKernan's study is so important. More bacterial contamination = more adverse events.
Correlation, not causation indicates this contamination is having some effect, perhaps causing clotting? >
Does PEG play a role in the #CalimariClots and to what effect?
Less PEG = clotting OR more PEG = more clotting?
We need reproducible studies on this. >
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Great research, thanks @_HeartofGrace_! >
Between Christie's research and Greg's research showing metals in the body act as catalysts for clotting and Dr. Lee's showing free spike binds many more antibodies into chains, there are many redundant explanations explaining post ๐ aberrant clot formation. #CalimariClots
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