Statins increases human Coronary Artery Calcium Score and promotes vascular calcification, and the plaque gets harder and the stent becomes more useless but it is great for centralized cardiologists and heart surgeons.
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3. https://consultqd.clevelandclinic.o...coronary-atheromas-even-while-shrinking-them/
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The use of statins increases the calcific atherosclerosis in T2DM and increases risk of CVD morbidity and mortality - acc.org/latest-in-card…
nature.com/articles/s4151…
tctmd.com/news/statins-.…
care.diabetesjournals.org/content/35/11/…
2. Statins are mitochondrial toxins because of their effect on CoEnzQ10. There are many ways in which they increase coronary artery calcification. One way is depletion of Vitamin K2 from the gut, another is lack of sun to control calcium flows, and another is direct arterial melanopsin damage liberating Vitamin A to cause intimal damage and a loss of arterial NO.
Sufficient production of vital biochemicals such as Geranylgeraniol (GGPP) is required to maintain endotoxin tolerance in macrophages in our arteries once the damage occurs. Macrophages are the hallmarks of CVD/Atherosclerosis, contributing to plaque development, inflammation, and the promotion of thrombosis. Geranylgeraniol is downstream of Mevalonate in the cholesterol synthesis pathway, and GGPP synthesis is inhibited by Statins, as is CoQ10 and K2. Vitamin K2 is the cofactor for matrix Gla-protein activation, which PROTECTS arteries from calcification.
Statin use is independently associated with increased calcification in patients, & using an animal model of hypercholesterolemia, we present a molecular mechanism whereby statins promote the calcification of atherosclerotic plaque. ahajournals.org/doi/10.1161/AT…
3. @DrJackKruse has "taught" his tribe -> Statins are a mitochondria toxin.
linkedin.com/pulse/why-do-s…
described in <- Cholesterol – what is it, what does it do? An investigation into the mechanism of action of cholesterol in the human biological function.4.As
forum.jackkruse.com/index.php?post…
5. Statins affect the cataplerosis and anaplerosis pathways in the urea and TCA cycle. That is what Quantum Thermodynamics blog #8 on Patreon is all about.
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2. optimalklubs.com/qt8-krebs-bicy…
forum.jackkruse.com/index.p...r-th…
6. Gaining an understanding as to why employing a toxin into the mitochondria may not be a good option for health, specifically for patients who have cardiovascular disease.
The triglyceride paradox in the mortality of coronary artery disease -
Bottom line - the higher your cholesterol the higher your probability of survival from a heart attack -
Decentralized science takes are always more informed than the centralized ones pushing DRUGs.sci-hub.se/10.1186/s12944…
forum.jackkruse.com/index.php?post…
7. Ok, so there’s a riff in the fabric of cholesterol reporting. But that doesn’t change the hypothesis that cholesterol is associated with cardiovascular disease. -> Right my centralized lemmings waiting for your atatin Rx?
We know cholesterol is on the first line of defense against inflammation as it serves as an antioxidant within our cells as well as external of our cells. All antioxidants whether it is vitamin E or other antioxidant become damaged as they try to fix the inflammatory issue through the bio-chemical redox process. How many studies now show vitamin E has been associated with cancer?
…erpreventionresearch.aacrjournals.org/content/5/5/701
8. How about this? news.cancerconnect.com/head-neck-canc…
9. “Vitamin E supplements may raise lung cancer risk” -> Why is that? -> oxidative damage of the vitamin E lipid molecule.medicinenet.com/script/main/ar…
10. Name the study, antioxidants become damaged and must be shuttled out of the body just like ox-LDL. Does that make antioxidants bad? -> No
Those publishing the riff in the fabric of cholesterol reporting are being very careful not to cut off their funding support from the pharmaceutical industry. They use words like: Paradox, “We currently don’t know the mechanism of action”, but they also putting in the important qualification: “We know cholesterol is associated with cardiovascular disease.”
So since we are on the thread Educating Doctors, what’s a clinician to do with her CVD patients? -> Glycation due to blue light and nnEMF
It’s the low hanging fruit and it’s the easiest to work on but it does support the narrative does it?
11. Endothelial dysfunction can be assisted with both a Coronary Calcium score and an Ultrasound Bilateral Carotid Arteries analysis. If these are issues, Melanin chelation and/or Pulsed Ultrasound therapies have proven to be successful. pubmed.ncbi.nlm.nih.gov/26578361/
12. However, when these oxidated molecules are broken loose, it is important to provide binders and melanin to collect, clean up and help transport them out of the body. These can include: charcoal and/or chlorella. But nothing is better than endogenous melanin production fro the prevention of heart disease. Get a tan inside and out.
13. Blood pressure is primarily managed by the renin-angiotensin-aldosterone system (RAAS) system. Did you know light and dark cycles control the RAAS system?
A Renin Activity and Aldosterone lab analysis will show where in the RASS system it maybe misfunctioning. aacc.org/science-and-re…
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