1. Today's lesson is an ass kicker. It begins with one of my long term members asking a question on my website forum.

CITE
A. forum.jackkruse.com/threads/climat… 2. All one needs to know about the climate scam in one slide. Humans are the penultimate primate. We need more CO2 so plants make more 02 in our environment. Coupled oscillators, are C02 and 02 for the clade of silly talking monkeys.

The text in my slide below claims a "40-year hoax" and "CO₂ famine," arguing we need CO₂ at 1,200 ppm to avoid calamity, with humans as "penultimate primates" benefiting from more CO₂ for plant growth and thus more O₂.

This echoes decentralized climate views held by me: Earth has been in a relative CO₂ "famine" for millennia compared to geological highs, and rising CO₂ (now ~420 ppm as of 2025) has driven global greening, increased crop yields, and reduced famine risks.

For example, India's shift from famine-prone in the 1960s to agricultural exporter is partly attributed to CO₂ fertilization alongside green revolution tech. Some argue past low CO₂ contributed to events like the Carboniferous rainforest collapse or even the Permian extinction via "phytoplankton blackout."

Every single thing the government is behind is a bullshit story to enslave silly talking monkeys.

So why have I been traveling to the highest points on Earth and visiting many deserts along my path? A new blog covering it.

SPACE & EARTH ARE ENTANGLED AND LEAD YOU TO THE DISEASES Astronauts get. All Chronic diseases of man have the same etiology but few see it.

This blog starts at the magneto-heliosphere for the lesson and then I introduce you to an astronaut who has experienced this nnEMF in his own life so you can learn about your disease from his experience.

Learn the real ticket that moves the needle. If you want to know why your kid has childhood diseases and cancers I guarrantee you after this master class in quantum biology you'll know why.

We are the product of stardust that has burned us using oxygen. It light is the anvil which forged me which gave the will to make me formibile for my misfits because I will not break under force. All living things are electric and magnetic and they use the electromagnetic radiation of the sun and the earths magnetic field to create life. Man made radiation disrupts the natural order of atoms in our cells. The electromagnetic signals that come from the organization of these cells in mtDNA creates our health span or chronic diseases epidemics. This is plants and animals.

Humans are diurnal animals who need sunlight and darkness after sunset to function. This makes them fully decentralized. They cannot be optimal with too much of either. Indoor living, sun avoidance, constant exposure to artificial lights and man made electromagnetic radiation = a shifted metabolism from light that leads to all chronic diseases. Optimizing melatonin, dopamine, and melatonin (sunlight/darkness) and solar derived vitamin D, melanin, and NO is how you avoid chronic disease.

patreon.com/posts/decentra… 2. You don’t need more time.
You need more clarity in how you approach learning

Ask yourself 3 questions every morning:

1. What really matters most right now?
2. Who needs to hear from me today?
3. Am I living in alignment with what I say I value?

These 3 questions cut through the noise to get you to the signal of things that matter - at work, at home, in life.

If you think the Federal government is working for you you are an idiot. First they banned light bulbs that could help reduce oncogenesis. A peer-reviewed study showed that 734 nm near-infrared (NIR) can do this. Then the DOD, Fauci and Baric made a virus and jab designed to give you cancer. Then DARPA is supporting Gates research to block the sun at rhe same time they are inflating you money away as M2 rises. Every part of the government is involved in TIME THEFT.

Thomas Jefferson told us about clowns like this and in his time he told people to shoot the King over things way worse than what Americans are being force fed.


2. ^^^^^that was my second to last slide in the latest talk. This was another from the slide deck.

What did the paper miss? Fasting makes them bigger.

Implications?

What happens to energy resistance in your ankle when you sprain it? It increases and it SWELLS because of energy loss.

What happens to energy resistance in the heart when it fails? IT increases in sizes and hypertropies because of energy loss.

What happens to a G class star like our sun when it it dies? It increases its energy resistance because it can no longer burn hydrogen and helium and burns all the elements to be come a red giant. It increases and becomes larger, because of this energy loss.

See the trend..........

What did the paper miss? Fasting causes mitochondria to get larger..........

It happens because of energy loss. The implications are vast for cell biology. Few.

I bet when @msahsorin gets better technology and can measure endogenous UPEs from these mitochondria it will show the UPEs spectra widens and becomes less coherent. Few.

@MitoPsychoBio 2. What happens in cells when UPEs output changes. Let us just look at one system to get a small picture of what light is capable of in information transfer? @msahsorin

These guys dying are just like the autism story is being sold by RFK Jr to give people breathing room. None of this is the truth. Everyone wants to blame one thing but it is a combination effect that assaults their colony of mitochondria slowly reducing the delta psi to cause electrical resistance in our semiconductive circuits to rise. This destroys the heat sink of metabolic water and directly alters information transfer in UPEs. It is obvious why this happens in decentralized medicine. It is shocking to the morons in centralized healthcare.
2. Number one cornerstone risk that raises energy resistance most is blue light and nnEMF that destroys the endogenous photolitholithograpghy biology needs to main entropy in the cell. Today biophysicists keep shitting the bed on what UPEs are. They are information qubits we use to stay healthy and alive and how they are controlled is the story of decentralized biology. Anyone who does not get this idea should be IGNORED.

“Previous studies showed that the plasma of a healthy individual contains up to 50,000 times more mitochondrial DNA than nuclear DNA,” said Dr. Alain Thierry, a researcher at the Montpellier Cancer Research Institute.

Biophysicists keep saying they do not know.........
Why do I know what it means? Because I understand what Shannon taught us about information. The uniqueness is the signal, not the noise.

In 2019, I said I expected viral epidemics breakout in certain zip codes more significantly because viral infections react to the environment that is suboptimal. I also reminded my readers that these viral infections will cause rapid significant mitochondrial damage which in turn will liberate Vitamin A from opsins in cells and this puts downward pressure on endogenous production of Vitamin D. Then in September 2020, I said one thing is clear.........those who got C19 all had low levels of Vitamin D and immunodeficiency at some level that impacted their care and outcomes. This will affect their mitochondrial delta psi and lead to many other complications. I spoke about all those links here in this blog work. patreon.com/posts/22117599

TODAY IN 2025 I AM TELLING YOU IF YOU TOOK THE JAB AND HAVE A HIGH SPIKE PROTEIN ARRAY, YOU NEED TO MAXIMIZE THE VITAMIN D AND A cycles in your body and then the more controversial idea. If you are woman, even if you have infertility you should continue to try to become pregnant because there is a change the placenta from the pregnancy will retain the ability to clear a lot of the genetic damage from the jabs and limit intercalation. Let me be clear, the pregnancy will help you, it may not help the child so think about this before action.

The human placenta acts like plasmapharesis for DNA RNA and mtDNA damage. Few centralized MDs know this. My tribe does.

Recently scientists at the Wellcome Sanger Institute and the University of Cambridge conducted whole genome sequencing of 86 biopsies and 106 microdissections from 42 placentas, with samples taken from different areas of each organ.
The link between genetic aberrations in the placenta and birth outcomes has been established in the literature by this study but few MDs read outside their expertise, further studies using larger sample sizes could help to uncover the causes of complications and diseases that arise during pregnancy.

This study confirms for the first time in the PEER literature that the placenta is organized differently to every other human organ, and in fact resembles a patchwork of tumors collected from the circulatory system of Mother and fetus. The rates and patterns of genetic mutations were also incredibly high compared to other healthy human tissues.

The human placenta is akin to the ‘wild west’ of the human genome, completely different in its structure from any other healthy human tissue. It helps to protect humans from flaws in our genetic code, but equally there remains a high burden of disease associated with the placenta. This is why the DOD engineered spike protein to affect the uterus and hinder pregnancy creation. These findings now provide a rationale for my tribe for studying the association between genetic aberrations in the placenta and birth outcomes at the high resolution we deployed and at massive scale by the jab. I have been telling jabbed members who still bleed they should seek continuous pregnancy to rid their bodies of the genetic defects these bioweapons bring to people. No one knows how effective this information is but I guarrantee you no one is getting this information but my FARM members.

I encourage you to read the paper before deciding what to do. No one can legislate your sexual activity so having sex to get pregnant to save your life or improve it will not be interrupted by the state as yet.

The paper was fascinating because it allowed us to observe how such serious genetic flaws like a chromosomal copy number error could be ironed out by the baby tissues, but not by the placenta. the placenta seemed to be built to absorb all the genetic mistakes. I want to remind all my female savages that this is a way to lower heteroplasmy rate in your germ line eggs too. These errors would have been present in the fertilized egg. The paper showed us that derivative cell populations, and most importantly those that went on to form the child, had the correct number of copies of chromosome 10, whereas parts of the placenta failed to make this correction and it COLLECTED THE DEFECTS LIKE A FILTER. The placenta also provided a clue that the baby had inherited both copies of the chromosome from one parent, which can itself be associated with problems. Share this info with female savages who complied with tyranny.


2. Publication:

Tim H. H. Coorens, Thomas R. W. Oliver and Rashesh Sanghvi et al. (2021). Inherent mosaicism and extensive mutation of human placentas. Nature. DOI: 10.1038/s41586-021-03345-1

doi.org/10.1038/s41586…

How many savages know that use of tylenol cause destruction of endogenous glutathione recycling?

Few.

https://twitter.com/EthicalSkeptic/status/1970234575260709187

ROOM silent as I spoke. It's a true story........

It was Decemeber 2010 and a my team of anesthesia people asked me to sit in on a continuing education class in the surgery department on one my surgery days.

I realized in 1 minute this was a BigHarma circle jerk.

The primary brand name for intravenous (IV) acetaminophen in the United States is Ofirmev. The medication is used to treat mild to moderate pain, fever, and moderate to severe pain in combination with opioid analgesics. They wanted my opinion on if I was OK using this as part of the anesthesia alogrithm for post op pain management in brain and spine cases and because I did most of the neurosurgery volume in the hospital in 2010 they wanted me to make the call. They knew I knew a lot of pain, POMc, and tylenol, because I had banned the use of this drug from all my patients in 2002.

I listened to the drug reps and reviewed what they brought me. First question I asked the reps what did they think about the 1998 actions of the UK on this drug.

Dead silence in the conference room filled with free BigHarma merch and food. 5 reps to cover the entire anestheisa and surgery dept. At the time, the anesthesia and general surgery depts were filled with employed MDs who worked for the hospital system. No one in the neurosurgery or orthopod dept were employed. At this time I noted the COO of the hospital was sitting in the back of the room.

I told the room filled with MDs and CRNAs that I banned the use of this drug after the 1998 UK black box warning. The published stories of the UK action was that typical UK pack size became restricted. In 1998, the UK limited the pack sizes of paracetamol available over-the-counter to reduce cases of intentional self-poisoning. If you could self poison yourself with an OTC medicine I said I need to find out why that happens.

ROOM silent as I spoke because they knew about how I fact check everything.

You see what Dr. Grimm sees in this paper, but what do I see in this paper?

This answers, definitively the question I posed to Huberman on the Tetragrammaton podcast, that the KT event drove internalization of melanin in eutherian mammals but not so much in birds. Perioxisomes burn fat and release UPEs. See the slide below in #7 and 18. That is the real story here. Light sculpted many changes with a rejection of chromophores in eutherians mammals and push for endothermy that lead to optimaization of the leptin melanocortin pathways.

Expanded Biophoton Sources: Explosion Post K-T

My list of 20 intracellular biophoton sources (e.g., NADH/NADPH, ROS, mitochondrial chain, melanin) shows an "explosion" post K-T, driven by adaptations like mitochondrial/peroxisomal amplification and melanocortin internalization.

This Fits ideally into my photobiolelectric thesis. Post K-T event we had a surge of changes that lead to more UPE creation: Reduced external UV forced internal UPE reliance, with sources like peroxisomal oxidation adding to mitochondrial ROS.

This ties to leptin-melanocortin (e.g., melanin in pigment cells emitting UPEs), myelin (membrane potential changes triggering UPEs), and quantum endothermy (IR as biophotons structuring water). The list expands our UPE narrative: post K-T, mammals/birds amplified endogenous light for signaling, reducing entropy via Shannon's theorem, enabling complex behaviors and longevity (e.g., human fur loss for more UV-to-UPE conversion). I see targets that others do not even know are there. 2. What were the only two animals that made is past the 5th extinction? Therapod dinosaurs became birds and eutherian mammals became humans.

Differences as Adaptations:

Birds' higher temperature/metabolic rate suits flight, while mammals' BAT-focused NST (with peroxisome augmentation) emphasized grounded endurance underground.

The unappreciated metric, quantum-tuned endothermy, directly affected endogenous water chemistry in cells, as IR from both mitochondria/peroxisomes structures cytoplasmic water, enhancing coherence for UPE signaling. This was a critical step in evolutionary history of the mammals post KT.

Photoreceptors are useless without retinal, which is produced in the eye. When retinal is liberated oxygen becomes a toxin and Fe is a wrecking ball.
phys.org/news/2018-08-c…

nnEMF activates VGCCs, flooding cells with Ca²⁺ and ROS/RNS, damaging mtDNA and heme proteins (cytochromes, CYP11A1, CYP19A1). Blue light liberates retinal, impairing melanopsin and dehydrating melanin, lowering éR and DDW production. This stalls StAR (STARD1)-mediated cholesterol import via TOM/TIMM/VDAC2, blocking pregnenolone synthesis.

Dehydrated melanin disrupts microtubule dynamics and protein synthesis, exacerbating "pregnenolone steal" toward cortisol, depleting sex steroids. The retina, as a "window to the brain," thins under suboptimal light causung blindness, degenerating brain tissue (e.g., frontotemporal dementia precursors). nnEMF/lack of sun alters AMO in skin/eye, changing central retinal pathways. Oxidative stress increases peroxide (ROS) levels, altering UPE signatures and heme construction, and is linked to hormone-related disorders by disordered UPE transformation in the CYP enzyme system.

In humans, iron is a double-edged sword, especially in the brain. It’s essential for energy production in mitochondria and for synthesizing neurotransmitters like dopamine, which governs movement and reward. Iron accumulates naturally in the brain as we age, particularly in regions like the basal ganglia, globus pallidus, and substantia nigra. These areas, rich in gray matter, hold two to four times more iron than white matter, where myelin insulates nerve fibers.

But when iron accumulates excessively, trouble brews. In neurodegenerative diseases such as Parkinson’s, Alzheimer’s, and Friedreich’s ataxia, iron overload in neurons triggers oxidative stress, resulting in the production of reactive oxygen species (ROS) that damage lipids, proteins, and DNA. This process, known as ferroptosis, is particularly devastating in the retina, where photoreceptors and intrinsically photosensitive retinal ganglion cells (ipRGCs) are particularly vulnerable.

These cells, which detect light to regulate our circadian rhythms, rely on iron-containing proteins and melanin, a pigment that chelates iron to protect against oxidative damage. When this balance falters, ferroptosis destroys neurons, disrupting circadian signaling and contributing to diseases such as Parkinson’s, where the loss of dopamine-producing neurons in the substantia nigra is linked to iron overload and reactive oxygen species (ROS).

Why does iron accumulate in sick or dying neurons? The answer echoes the stars. Just as a star amasses iron in its core as it runs out of energy, neurons hoard iron when their energy production falters, often due to mitochondrial dysfunction. In both cases, iron signals a system on the brink, teetering between stability and collapse. 2. 95% of melatonin is made from mitochondria. So what happens when mitochondria are damaged from blu elight toxicity? You get high blood glucose and insulin and flatlined cortisol.

What happens when you knock out melatonin receptors via melanopsin dysfunction from the liberation of Vitamin A? You get insulin resistance and set the stage for many mitochondrial diseases like diabetes and cancer.
Do you know what I'm talking about? Light shapes life. How is it shaping yours? The link below should open your mind.
onlinelibrary.wiley.com/doi/full/10.10…

Deuterium's Quantum Dual Nature: Bosons vs. Fermions

At the core is deuterium (D or ²H), an isotope of hydrogen with an extra neutron. My blog explains its particle statistics: Nucleus: A boson (spin 1, even parity), following Bose-Einstein statistics. Bosons can occupy the same quantum state, enabling phenomena like superfluidity or Bose-Einstein condensates at low temperatures.

Atom: A fermion (half-integer spin, like 1/2 or 3/2 ħ when combined with an electron), obeying Fermi-Dirac statistics and the Pauli exclusion principle. This makes deuterium atoms behave like "individualists" that can't pile up in the same state. This is why biology is racist against their use in mitochondria. The raise energy resistance.

Molecule (D₂): A boson again, due to symmetric nuclear spins in the ground state. It forms antisymmetric (para-deuterium, spin 0 or 2 ħ, even L) and symmetric (ortho-deuterium, spin 1 ħ, odd L) states.

This contrasts with ordinary hydrogen (H₂), a fermion in its para form (spin 0) and boson in ortho (spin 1), leading to different behaviors at low temperatures. Cooling deuterium near absolute zero produces pure ortho-deuterium with residual spin-2 fractions that can't be fully removed, unlike hydrogen's para form with zero angular momentum.

The takeaway: Deuterium's bosonic tendencies make cold D₂ act like a collective fluid (Bose-Einstein-like), while H₂ follows fermionic exclusion. This affects heat capacities, neutron interactions, and low-energy systems—relevant for fusion, cryogenics, and, as we'll see, biology------> All covered here. optimalklubs.com/kruse-for-dumm… 2. Biochemical Implications: Hydrogen Isotopes in Melanin and Metabolic Pathways

The diagram links this to biology, showing a pathway from acetyl-CoA (a ketone precursor) through melanin dissociation, involving clock genes, sunlight, and electromagnetic spectra. Key points:Oxygen (O₂) from NADP⁺/NADPH cycles feeds into tryptophan hydroxylation, producing intermediates like 5-hydroxytryptophan (5-HTP) and 5-hydroxytryptamine (5-HT, serotonin).

Hydrogen ions (H⁺) are central, explicitly noted as "THIS HYDROGEN CAN BE H+ or D." Protons or deuterons from the pentose phosphate pathway (PPP) influence steps like aromatic L-amino acid decarboxylase, leading to glucogenic amino acids (e.g., alanine) or neurotransmitters.

Deuterium's heavier mass (twice hydrogen's) slows kinetic reactions (kinetic isotope effect), disrupting enzyme kinetics, and the dielectric constant of the metabolic pathways altering UPE function, mitochondrial function, and water structuring in cells. The diagram implies sunlight-driven melanin creates a "plasma" for harvesting electromagnetic energy, with H/D isotopes affecting unique codes in water and biomolecules.

The retina thins from the effect of man-made light and a lack of sunlight, and then your risk of Frontotemporal dementia rises. The OCT test is the best screening test for FTD. We have familial and mtDNA FTD.

The retina acts as a ‘window to the brain," and that window changes the photolithography of the retinal cells. When the light is not optimal, the retina thins and the brain degenerates. Retinal degeneration has been detectable in mutation carriers prior to the onset of cognitive symptoms, establishing retinal thinning as one of the earliest observable signs of familial (frontotemporal dementia).

nnEMF and a lack of sun lead to neural degeneration, which is primarily driven by the abnormal accumulation of misfolded proteins within neurons. Light controls the confirmation bending of proteins. The DNA code controls the AA sequence and secondary bending, but quantum processing determines tertiary and quaternary bending by UPE transformation in cells. The most common protein abnormality in FTD is the tau protein, also known as TDP-43. These protein aggregates damage and kill nerve cells, leading to brain atrophy (shrinkage) and the resulting behavioral, personality, language, and movement problems characteristic of FTD. I believe this protein also thins the retina. Light is the offender.

Blue light increases RBC turnover in the retina and changes the oxidation state of iron, and this affects the synthesis of new heme proteins in the retina and brain. Oxidative stress is a key trigger in this disease because it increases peroxide production (ROS), which alters the UPE signature of cells in the construction of the heme proteins, which changes the photolithography inside the retina and brain. Changing the photolithography = altered protein folding via the Golgi apparatus and RER. TDP-43 is a biomarker of redox imbalance in FTD-related UPEs.

The path of energy in life, based on the evidence we have as a species now, is that Optical photonics began 13.8 billion years ago. The photoelectric effect was born from the rudimentary physics present in the early universe. Photonics precedes all chemistry of all types. Functional medicine does not realize this or teach it. Molecules were and are innovated by redox chemistry, and each one has an electromagnetic barcode to program its possible states around the star it evolved in.

The TDP-43 molecule is codified by light in its absorption and emission spectra. This links it to aberrant UPE production in the central retinal pathways, as blue light increases heme turnover to alter its biophotonic signal. Look at all the places a UPE can be made from in a cell to cause this disease on the slide. You'll also see that light carries spin information that can create this disease.

Where there is a change in matter (TDP-43), there must also be a change in atomic molecular geometry in that tissue. Where normal cellular geometry ceases to exist, we will find light as the key agent of change. When both exist in simultaneity, you'd better understand Fermat's law and the photoelectric effect to understand what a disease really is. Right now, in centralized ophthalmology, none of them do. See Bruce Willis.

THE BIOPHYSICAL CODA of FTD: When Turing realized morphology in all living things had a photonic timeline in Nature, he wrote a key paper on morphogenesis. Light signaling predates biochemistry and molecular creation from redox chemistry. This paper suggested that a system of chemical substances, called morphogens, could react together as a symphony does and subsequently diffuse their electromagnetic, electrochemical, and photobiolelectric information through a tissue to sculpt it, changing its morphology without using the nuclear genome.

Turing stated in 1951 that this set of circumstances is adequate to account for the main phenomena of morphogenesis. In such a dissipative system, although the cells may originally be quite homogeneous at the embryo stage, this information upload may later develop a pattern or structure in the embryo due to the instability of the homogeneous equilibrium caused by light pollution in the parents' germ line, which is triggered by random disturbances in the tissue. Those random disturbances are UPEs. The random disturbance he hypothesized about turned out not to be random at all. UPEs are created in cells by light AMO interactions via redox chemistry that directs their own sculpting from the nuclear genome. The UPE energy transformation is driven by the mitochondrial genome, which is subject to light in the environment to create UPEs and eventually by reactive chemicals like ROS/RNS inside all cells. Any disease linked to aberrant UPEs will always present as having a spectrum of maladies. FTD has that optical signature. ncbi.nlm.nih.gov/books/NBK55928…


2. The GLP-1 Drug Connection: Weight Loss at a Cost to Your Eyes and Ears

GLP-1 drugs like semaglutide (Ozempic, Wegovy) and liraglutide (Victoza) are prescribed for type 2 diabetes and weight loss, helping millions shed pounds by curbing hunger and stabilizing blood sugar. However, literature suggests that they can affect the eyes and even the inner ear (cochlea), which ties into the light biology disruptions we just discussed.

Effects on the Eyes

These drugs cause rapid weight loss, which triggers fluid shifts and reduced blood volume in the body.📷This can stress the delicate blood vessels in your retina, leading to:
Worsening diabetic retinopathy: In people with diabetes, GLP-1s can make existing eye damage worse, causing blurred vision or macular issues.

Increased risk of vision loss diseases: Studies link them to a doubled risk of neovascular age-related macular degeneration (nAMD), a leading cause of blindness.

There's also a higher chance of non-arteritic anterior ischemic optic neuropathy (NAION), where blood flow to the optic nerve drops, potentially causing sudden vision loss.

Blurred vision and other issues: Common side effects include visual impairment, which can start as early as 10 days after use. While not everyone experiences this (risks are low but real), regular eye exams are recommended for users.

How does this relate to light biology? The fluid shifts and oxidative stress from GLP-1s can amplify ROS production, disrupting UPEs and protein folding in the retina, much like exposure to bad light. This thins the retina, mimicking the early FTD signs, and could raise dementia risk indirectly through disrupted circadian rhythms.

Interestingly, while some research suggests GLP-1s might protect against general dementia by reducing inflammation, their eye effects could counteract that, especially in FTD-prone folks. My TED talk was banned because BigHarma was afraid I was going to obliterate their GLP 1 train by showing the world why they banned stopped the leptin trials early on weight loss.

I tried to pull the veil back on the layers of the Deep State and where they are linked and why they are linked. It is a nasty story of deceit and ideological fascism where science was stolen to control modern humans with Light, drugs, and jabs.

The historical links you must know.  Why is Israel so misunderstood? Propaganda is its shield, so you can never know who is really behind it.

It is British Imperialism dressed in drag to protect its ideology of fascism, which is what a Fabian really is.

The billionaires of today are not the architects of Zionism
They’re the actors. Elon. Thiel. Gates.
They run infrastructure, but they don’t own the world.
They were groomed by systems older than corporations.
The real owners?
• BlackRock, Vanguard, BIS — own the assets
• Rockefeller, Rothschild, DuPont — own the timeline
• WEF, CFR, Chatham House, Fabians — write the script
• Think tanks + foundations — enforce ideology
Occult orders + Straussian elites — justify it all as “destiny”
This is not capitalism.
This is a technocratic theocracy—where power is hidden in algorithms, law, debt, and narrative.
They don’t fear elections.
They fear recognition.
They fear you finding out what their plan really is.
Why must we decentralize medicine?
Because it eliminates centralized Rockefeller Medicine from the World.
That is a world where drugs and jabs are used as weapons of control.
Time to wake up and do your diagnosing better today than you did in the past.

x.com/JuanGutiCA714/… 2. True Evil with assorted atrocities most often occurs when the most respectable and intelligent of people fall for it. The wise never do. They sniff out and diagnose the problem of centralization to avoid collateral damage. Be careful who packs your parachutes.

I started a huge Barista FIRE on the boat it appears with my Bitcoin talks with him every AM. He apparently was quite influential with the young staff and told them all they were working communist hours for communist pay, and when his manager heard it spill over to our morning conversations and how many of the krewe I am Orange pilling, they axed him. He was escorted off the book quickly in Peru, and I never knew it until this AM. Some of his barista folks confirmed it got hot quickly.

What Is the FIRE Movement, and Where Does Barista FIRE Fit In?

FIRE stands for “financial independence, retire early.” The FIRE movement puts forth the idea that becoming work-optional isn’t about reaching a certain retirement age; it’s about having enough money invested that the compound interest gains can sufficiently cover your annual expenses. Bitcoin really changes the mix for young people locked into communist like employee environments.

This is achieved by reaching what is called your FIRE number. A (very) rough calculation of FIRE number is to multiply expected annual expenses when no longer working by 25. This is the amount of retirement savings you’ll want to have ready to tap, but know that performance on investment accounts can vary widely from year to year. A traditional FIRE number is typically well north of $1 million.

There are many people who don’t actually want to stop working and enter full retirement. Instead, they want to downshift to doing more fulfilling work, or they want to be able to work part-time so that they can spend more time pursuing hobbies or passions. In jobs like medicine and cruiseship workers the math works rapidly but for different reasons. So when I explained this to Christopher he seemed to catch fire like I had poured diesel fuel on him. Never thought he'd get canned for it that fast. I guess capitalism is a bad antidote for the communist boat life.


2. The Origins of the FIRE Movement

In 1998, three researchers at Trinity University published the results of a study on retirement savings.

Their projections found that, if an investor had a certain multiple of their income saved up and withdrew 4% or less of their nest egg each year, their chances of depleting their savings in a 30-year period were zero. I re-did this calculation in 2013 and added Bitcoin CAGR to the mix, and the results were more stunning. That is when I realized I could bail on centralized medicine and began teaching this method to my MD clients. Sadly, most of them crumbled because they could not fathom walking away from their jobs because of their programming. The Trinity research group was based on rates of return since the invention of the 401(k) and other tax-advantaged retirement accounts, and later became known as the "Trinity study."

1. ANSWER: In ALAN contexts, blue light suppresses melatonin, which normally inhibits AVP and cortisol, leading to unchecked vasopressin release during "light stress." This causes a reactive hyponatremia in the posterior pituitary, activating brain osmoreceptors and RAAS, raising blood pressure and stress hormones, which fragment sleep and impair daytime recovery. They also chronically degrade the mitochondria in these neural tracts.
2. Now for the dirty details.
Precise Mechanisms of Sodium Regulation in the Brain and Circulatory SystemSodium (Na+) homeostasis is tightly regulated to maintain osmotic balance, blood pressure, and neuronal function, with disruptions like hyponatremia (serum Na+ <135 mEq/L) directly impacting sleep via arousal, cognitive fog, and fragmented rest. The brain acts as the central sensor and regulator, while the circulatory system executes adjustments through hormonal and renal pathways.

Brain's Role in Sodium Sensing and Control: The hypothalamus, particularly osmoreceptor neurons in the supraoptic and paraventricular nuclei, continuously monitors plasma osmolality and Na+ levels via stretch-sensitive ion channels. When hyponatremia occurs (e.g., from ALAN-induced vasopressin overrelease diluting blood Na+), these neurons trigger arginine vasopressin (AVP, or antidiuretic hormone) secretion from the posterior pituitary.

AVP promotes renal water reabsorption via aquaporin-2 channels in the collecting ducts, conserving water but exacerbating dilutional hyponatremia if unchecked. In the brain, low Na+ also activates the subfornical organ and organum vasculosum of the lamina terminalis (circumventricular organs lacking a blood-brain barrier), which integrate signals from baroreceptors and chemoreceptors to modulate thirst and salt appetite. Chronic hyponatremia impairs neuronal excitability, leading to symptoms like headaches and insomnia, as it alters membrane potentials and synaptic transmission.

Circulatory System Integration with Hormones: In the periphery, low Na+ (hyponatremia) is sensed by renal juxtaglomerular cells, triggering renin release, initiating RAAS: renin converts angiotensinogen to angiotensin I, then II, which stimulates aldosterone from the adrenal cortex to enhance Na+ reabsorption in the distal nephron and potassium excretion. This raises blood pressure to restore volume but can cause nocturnal hypertension, disrupting sleep architecture (e.g., reduced REM). Cortisol, released from the adrenal cortex in response to stress signals (including ALAN and hyponatremia), amplifies this by promoting Na+ retention via mineralocorticoid receptors and enhancing sympathetic activity, increasing heart rate and arousal. Vasopressin interacts with cortisol and RAAS; for instance, AVP potentiates cortisol's effects on water balance, while cortisol feedback inhibits AVP in healthy states, but ALAN disrupts this, leading to unchecked stress responses. Atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP) counterbalance by promoting natriuresis (Na+ excretion) when blood volume rises, but in hyponatremia from SIAD (syndrome of inappropriate antidiuresis), this balance fails, perpetuating low Na+ and sleep disturbances like frequent awakenings.

A couple of points: Not all human cells have mtDNA. See adult blood cells. This is a big deal when you consider their absorption spectra. 200-600 nm light with a sharp cut-off. Also, blood still transforms energy into UPEs. And since blood fills 20% of our CO to the CNS, this also has implications. The retina and gut increase their blood flow with light use and feeding. Feeding is a light-based phenomenon since all food webs link back to photosynthesis.

In humans, light stress does not force mitochondria to react exactly the same way as infection (no ATF4 surge, different folate handling), but there are partial overlaps in ISR activation, 1C remodeling, and mtDNA signaling.
This suggests UV light acts more as an external folate depleter for DNA protection/photorepair, while infection is an internal ATF4-orchestrated defense. If environments lack natural light controls (as in modern human life), it should exacerbate mismatches in folate biology. For deeper dives, I'd recommend reviewing the ATF4-UV papers for experimental details.

Folate as a Light-Responsive Switch: My patreon has made this point over and over again, natural folate (not folic acid) is photosensitive, degraded by UVB, and shows seasonal lows in summer (higher at low latitudes, with gender differences showing men have lower levels). This would act as a "switch" for mitochondrial/genome control, linking to melatonin/tryptophan pathways and neurotransmitter synthesis (e.g., serotonin, dopamine). In low-light/artificial blue light environments (e.g., indoors), folate stability increases unnaturally, disrupting methylation/DNA synthesis and contributing to diseases like Alzheimer's, Parkinson's, or diabetes (via melanin quenching loss and superoxide buildup). People with MTHFR and COMT defects are supersensitive to ALAN and a LACK OF SUNLIGHT. This is echoed all throughout my decentralized photo-bioelectric thesis: light refines folate via photosynthesis/food webs, but artificial setups bypass this, leading to transgenerational effects.

No Seasonal/Light Controls: absent natural light cycles (e.g., constant artificial light), retinal/CNS signaling should be expected to dysregulate—e.g., excess folic acid fortification (since 1996 in North America), which overloads 1C pathways, causing cognitive haze, sleep issues, or neural migration problems in lit environments. This photonic effect is completely missed in centralized medicine and is really missed in functional medicine, which pushes for excessive folate use with SNPs. These people need more sun and less ALAN, not drugs.

Black Swan Mitochondriac View: Melanin (from UV-absorbing aromatic amino acids) quenches mitochondrial superoxide, protecting against neurodegeneration. Blue light destroys melanin and heme proteins, amplifying risks from ALAN and a lack of sunlight which aligns perfectly with UV's mitochondrial stress but without the adaptive folate restriction seen in infection.

Now the picture is full. patreon.com/posts/decentra… 2. Why is this tweet important to the jabbed? If you follow the work of @Kevin_McKernan you'll find in his blogs many mentions of pseudouracil and jab injuries. Then you look at my blogs on jab repair, and you'll notice I recommend Tropical relocation as the best risk reduction for the compliant. WHY?

Did you know that in humans, UVR depletes folate in skin and blood, inducing S-phase arrest, affecting uracil misincorporation (frame-shift mutations), and sensitizing apoptosis in keratinocytes. This isn't "damage" but a permissive environment for seasonal phenotypic changes, e.g., increased melanin production to protect folate stores. When you understand the photorepair mechanism, you will see this is how humans can block the DoD and BigHarma death mechanism from uracil replacement induced by the jabs. @MdBreathe None of the COVID experts have this sophistication. They are still pushing detox answers when it is crystal clear that redox biology is the path for the Savage trying to stay alive and away from disease.

If Einstein was correct that energy = mass times the speed of light squared (E = MC2), then how did biology make all life from that simple equation? As I looked up, the sun hit me in my eyes. I realized the link, right there. I had to reverse Einstein’s equation to see how life made sense of the chaos on our planet, to create life from it. E = MC2 is a simple math equation using simple variables we all know.
Truth Bomb #2: Life is energy and energy is life according to this equation.
It also implies that the equation can also be reversed mathematically. The “Commutative Laws” say you can swap numbers over and still get the same answer. A + B = B + A. Biologic sciences have pretty much ignored E = MC^2 for much of the last 108 years since its discovery. It has been felt to be the domain of subatomic physics and of theoretical physics, and astronomy. Physicists and chemists have always read Einstein’s masterpiece equation from left to right. This helped them explain the massive power generated from the nuclear fission of atomic blasts. As a surgeon, I realized there is were no nuclear explosions occurring in cells to generate energy. I reasoned, biology could not use the reaction that way, so life had to find another way to do it. I thought, that maybe, life at its origin on the chaotic Earth, sought order from the environments it had to endure to survive. If that was correct, then all biology must start with the speed of light, squared and not with energy. I felt I had to reverse Einstein’s equation in my head to figure the riddle out. It made sense because today we know all life makes energy from the sun or from electrons in our mitochondria. Plantlife uses the photons or electrons of the sun to make its energy efficiently. Animals use electrons to make fuel in the mitochondria in the form of ATP.
The M, the ‘mass’ part of the equation brings in the elements of space/time and gravity from physics. This is a topic biology rarely deals in. This is where the riddle got complex for me. This implies the way the mitochondria account for energy has to include a very precise timing procedure as a part of energy generation. I knew from mitochondrial biology that is precisely what the “Rolex” in our head does at the SCN by responding to the magnetic field of the Schumann resonance of the Earth.
But there is more.
Food is information of light from the sun. It is also energy. This means that info and energy are linked. It turns out they are linked via the concept of mass.
Shannon took the vague concepts of information and pinned them down to what its essence was all about.
He asked what was the minimum requirement needed to create a message that could still be deciphered well.
The equation he came up with looks identical to Boltzmann's equation for entropy. This means Information truly links to thermodynamics.
Boltzman gave us the statistical explanation of the second law of thermodynamics. In 1877 he provided the current definition of entropy,
In 1948 Shannon figured this out by mathematically learning how to measure the information in the messages
Shannon realized that the quantity of the message had ZERO to do with its true meaning.
Physicist John Wheeler extended Shannon's ideas further.
Wheeler tells us every particle in the universe emanates from the information locked inside it.
My idea at my KLC clinic: This idea has massive implications for mitochondrion who deal exclusively with light, electrons, and protons in cells.
Wheeler’s ideas have been expanded recently by Vopson (dark energy non-believer)
His paper says that not only is information the essential unit of the universe but also that it is energy and it has to have mass.
To support this claim, he unifies and coordinates special relativity (1905) with the Landauer Principle (1961)
Landauer predicted that erasing even one bit of information would release a tiny amount of heat, a figure which he calculated = mtDNA is a heat engine
If information is energy, Information, once created has to "finite and quantifiable mass."
This connects information directly to energy. E-mc^2
When information is lost in the system there has to be a change in mass in the system..............
These ideas fueled the EMF 2 blog post at jackkruse.com 2. It amazes me how ignorant the paradigm is about AM sunlight. AM sunlight is filled with high intensty red light and a smaller amount of blue light. This is why the color temperature of AM sunlight at sunrise is around 1600K and it is 16,000 K at sunset. High intensity red light in the AM is the light that Nature uses to stimulate testosterone release in men. UV light is a potent "off switch" for testosterone action in the blood. It amazes me that this PEER reviewed article has no clue that AM SUNLIGHT is the circadian controller of testerone level in MEN.

They want to use fake light.........to do the job of the sun.
Ridiculous.

The use of light therapy dates back to ancient civilizations, going as far back as the ancient Egyptians and Indians, who used sun- light (heliotherapy) for healing and promoting health. The therapeutic use of light energy was more fully appreciated in the late 19th century when a Danish physician-scientist, Niels Ryberg Finsen, demonstrated the benefits of red and blue light in the treatment of lupus vulgaris and was recognized with the 1903 Nobel Prize in Medicine and Physiology. In 1960, the L.A.S.E.R. (Light Amplification by Stimulated Emission of Radiation) by Theodore Maiman was invented, based on theoretical work by Albert Einstein in 1917.

This brought renewed attention to the therapeutic light energy field. The monochromatic, coherent, and collimated nature of lasers led to immediate interest in their biologic effects. In 1967, Endre Mester, a Hungarian physician-scientist, reported that low-dose laser treatments were capable of promoting wound healing and hair regrowth in mice. He termed this phenomenon photostimulation and went on to demonstrate the efficacy of this treatment in human patients with skin ulcers. medicalnewstoday.com/articles/31296…

1. Today's lesson: Your longevity experts are RETARDS.
If you know you know. This is the lady below that shows you every longevity expert out there is FOS. She is and was the ultimate wellness rule breaker who lived 122 years and 164 days. Longer than anyone in recorded history. 2. Her daily routine:

↳ Smoked 2 cigarettes daily until age 117
↳ Ate 2 pounds of chocolate per week
↳ Drank Port wine regularly
↳ Doused everything from the Sea/land in animal fats and olive oil

She also took up fencing at 85. Rode her bike until 100. Walked until she was 110. She only quit smoking because she went blind and could not see her cigarette to light it. She was the opposite of this rich moron below.

1. This interaction is governed by the Chiral-Induced Spin Selectivity (CISSS) effect, where spin-polarized photons or electrons preferentially match the protein's chirality. In photosynthesis, this spin selectivity may guide exciton transport through protein complexes. If a biophoton's spin aligns with the protein's symmetry, it can trigger excitation or release; otherwise, the signal may be lost, suggesting spin could be nature's way of encoding selective bio-communication. 2. Mitochondria convert food into electrons via the electron transport chain, where cytochromes (especially cytochrome c oxidase) pump protons out, reducing oxygen to water. Proton tunneling, not just bulk diffusion, enables this rapid proton movement (over 10³ protons per second). ATP production peaks during awake hours and initially rises during sleep onset due to free fatty acid (FFA) release, but drops sharply in REM sleep to support a quantum state for brain recycling.

During uncoupling in deeper sleep, mitochondria release infrared heat, condensing surrounding water and coupling to quantum processes.

This mirrors spintronics, where electron spin, manipulated by electric and magnetic fields, stores information, much like mitochondria control electron spin for energy and life processes. Free radicals, with unpaired electrons spinning in the same direction, play a key role, defying the singlet state (paired spins) governed by the Pauli exclusion principle. This quantum spin manipulation underscores mitochondria's unique bioenergetic role.

Why dentistry needs to be decentralized badly: instagram.com/reel/DNDlNR1Ok… 2. Decentralized Answer:
My thesis leverages first principles in your question to me about LPR because light as energy, POMC as a UV switch, melanin as a charge modulator all link to propose that sunglasses disrupt UV-driven biology, reducing melanin and charge, and potentially causing LPR via glycation and neural crest effects. The UV-POMC-melanin link is solid; the LPR and charge extensions are cutting-edge hypotheses needing research.Sunglasses,

UV Light, and Melanin Production: Principle: Light is electromagnetic radiation, and UV photons (e.g., 280–315 nm UVB) carry energy that excites molecular chromophores in skin cells, triggering biochemical cascades. POMC (proopiomelanocortin) expression is a photochemical process driven by UV light, as confirmed by studies (e.g., Slominski et al., 2018) showing UV-induced upregulation in keratinocytes and melanocytes.

Mechanism: UV photons activate POMC transcription, leading to α-MSH production via the melanocortin 1 receptor (MC1R) pathway. α-MSH stimulates melanogenesis, producing melanin, which absorbs and dissipates UV energy, protecting tissues. Sunglasses Effect: Sunglasses with UV400 protection block 100% of UVA/UVB. If UV is the sole trigger for POMC translation, blocking this spectrum halts the signal. This reduces α-MSH and melanin synthesis. The extent depends on exposure time, but chronic use could significantly lower baseline levels, especially in low-pigment individuals. The claim holds, sunglasses directly reduce melanin by cutting the UV-POMC- α-MSH pathway.

While the tweet is correct, my tribe knows Dr. B is someone's advice to avoid. He has no idea about the quantum biology of NO. His advice was so bad I had to cancel a member's membership over this.

https://twitter.com/ValerieAnne1970/status/1950434524740939907

2. NO to excess is a danger. I saw this first hand in New Orleans during HIV.

Listening in here is hilarious to hear a few lawyers who have no historical background talk shit about things they do not know. Hard to fathom they are Bitcoiners. Verify do not trust only works for Bitcoin with these guys. x.com/i/spaces/1BdxY… 2. Simon Dixon, who got popped in the Mashinsky scam, so why would anyone listen to his opinion on anything money? He knows Mashinsky is also a Zionist scammer, so he hates anything Zionists too. The diagnosis of his thought pattern is not too hard to figure out. He is a shitcoiner and happens to own Bitcoin. Everyone knows who paid to get Trump elected: Fairshake Super Pac.