Thread by @DrJackKruse

1. New lesson today from my forum for the Savages to consider. This tweet below is the primer.

2. Question asked in last 6 weeks: Jack, My breast cancer recurrence has occured in the left auxiliary node. Currently taking Verzenio and tamoxifen. Declined ovarian suppression. Starting Dr. Makis protocol soon.

x.com/drjackkruse/st…

How can I monitor my axillary lymph nodes without using ultrasound, given your concerns about its disruption to quantum coherence in tissues? Especially since my traditional blood tumor markers (CA 15-3 and CA 27.29) have consistently tested negative?

Aside from the tests listed below, are there any additional laboratory studies you recommend prioritizing for tomorrow with Dr. G?

BUN/creatinine ratio
Vitamin D
Liver function
HsCRP

3. Back round info on U/S: westonaprice.org/health-topics/…

4. She asked the following Questions: How am I supposed to monitor my condition? In April I’m headed back into the oncology sausage grinder, where the default plan seems to be ripping out every lymph node in my armpit without hesitation.

At the same time, @MakisMedicine is not addressing reports of disease progression when ivermectin and fenbendazole doses are reduced.

Is the treatment sustainable long term?

@MakisMedicine 5. She then lists a bunch of superfluous labs her centralized care team ordered and then asks this question: Is this a Valid claim? theconversation.com/dont-cut-them-…

6. My Answer: It depends on the surgeon and oncologist. Most of them are still working on old data and ideas.
You should ask them since they are your cancer experts. If the disease is progressing the treatment plan should be adapted.

I believe Dr. Makis and your other treatment team should be looped in on this and asked for their opinions. They are your treating doctors here.

@MakisMedicine 7. She asks me this directly after the response above: Do you not want to help me because you think I’m going to die?

@MakisMedicine 8. Answer: I have tried to help you by educating you and your husband on decentralized medicine. You have chosen your experts, now use them.

9. She then asks me this: I’m here in Shalpa doing everything you said to stop feeding the fire. I doubt Ivermectin and fen/ben is going to put out the fire. What lesson are you trying to teach me?

Then she posts this comment followed by the picture below. She said, "This must be it."

10. My answer to the above comment and picture is.....

When you have an increase in the change of your tumor and you are using another doctor's protocol you have a duty to discuss your new adaptations with them. You should not force your wishes onto someone else.

Generally when you have measured your environment and you have VERIFIED there is no polarized light around you then you might ask the question, is this other doctor's protocol doing something to cause the size and shape changes in my tumor or in their mitochondria.

What centralized MDs should be worried about in this case is the tumor is going further into the GOE state and de-differentiating into a more malignant state. But decentralized clinicians know there is another possibility as well. Decentrlaized clinicians can educate you on this topic generally. Let me be clear. I am not your doctor. You and your husband told me you have other doctors specific to your cancer care. USE THEM and begin to question them ASAP.

I go on.........

11. When size changes occur it is evidence of thermodynamic change. This could be good or bad but it needs investigation with imaging. You want to see if the growth is associated with invasion or not. The best imaging here is MRI and/or PET/SPECT scaning. What should they be looking for? Whether there is extension into nodes or adjacent tissue planes or not. If tissue planes are destroyed you have to worry about a change in differentiation. Since you have cancer experts and Makis on your cases these are the things they should be DISCUSSING with you.

If those things aren't present then you want to see if there is a change in shadowing or melanin adjacent to the tumors to show there is actions happening on the mitochondria of the cancer cells specifically.

Since you are using the OTHER doctor's protocol these would be WISE things to consider.

I go further......

12. Ivermectin, Fenbendazole, Mebendazole are key parts of his protocol. These anthelmintics act as
redox-disruptors that target the very metal-dependent pathways I’ve highlighted in my decentralized thesis. I know Makis has no idea what that implies and means. You should because you are supposed to be reading and understanding. While centralized fucks think these drugs are only viewed purely as "parasite killers," their molecular interactions with Fe, Cu, Mn, and Mo reveal a profound impact on mitochondrial biology and UPE signaling. Melanin chelates all of them so they remain controlled to make sure mitochondrial respond with a coherent message.

This implies that the protocol drugs might cause changes in your tumor doesn't it. This is why Dr. Makis needs to follow up with you.

I go further.......

13. 1. Ivermectin: The Mitochondrial "Uncoupler" and ROS Generator

Ivermectin (IVM) primarily interacts with the system by inducing mitochondrial dysfunction and altering redox balance.

Copper (Cu) & Iron (Fe) Interaction: IVM has been shown to induce mitochondrial damage and apoptosis, specifically leading to the release of cytochrome C which is a heme protein whose function is entirely dependent on its Copper and Iron centers.

Manganese (Mn) & SOD2: IVM treatment upregulates the production of Reactive Oxygen Species (ROS). In my framework, this would place a massive demand on Mn-SOD (SOD2) to manage the resulting "oxidative noise." If the Mn-SOD shield is overwhelmed, the resulting ROS flux drives cell apoptosis and this would induce a change in the tumor as it loses energy, a mechanism being explored for its antitumor effects.

UPE Signaling: By decreasing mitochondrial membrane potential and respiration in cancer cells, IVM disrupts the coherent "light" (UPE) signaling required for cellular adaptation.

2. Mebendazole & Fenbendazole: The Iron-Dependent "Starvers"

The benzimidazoles (MBZ and FBZ) have a distinct relationship with iron status and mitochondrial integrity.
Iron (Fe) Dependence: Mebendazole is significantly less effective in iron-deficient environments. Clinical studies suggest that for infections like hookworm, iron supplementation is often required alongside MBZ to clear anemia and restore efficacy. This suggests MBZ may leverage or require the host's iron-dependent pathways to finalize its anthelmintic or antitumor effects.

Manganese (Mn) & Mitochondrial Stability: Both drugs target the mitochondria of the germinal layer in parasites, leading to decreased ATP production. In mammalian cells, FBZ has been noted to inhibit tumor growth specifically in models used to study mitochondrial genes, indicating a direct interaction with the organelle's metal-tuned hardware.

Molybdenum (Mo) and Sulfur: There is no direct documented interaction between these drugs and Molybdenum. However, since MBZ and FBZ can impact liver function (the site of major Mo-dependent enzyme activity in humans), prolonged high-dose therapy requires monitoring for metabolic "interference".

14. I ended the conversation with the following comments and reference.

Cancer is a GOE state of health. In a "Cambrian" state of health, these drugs would act as massive stressors by increasing ROS (UPE noise) and potentially sequestering or demanding metals like Iron and Copper. This makes them potent against "noisy" systems like cancer or parasites but highlights why they must be used carefully in a system already struggling with DHA-depletion or pseudohypoxia. You should continue your education to supplement your experts wisdom if you are interesting in adding time to your own time bank account.

Here is a reference that is topical to your problem. forum.jackkruse.com/threads...nce-…

@MakisMedicine 15. END OF LESSON