Thread by @DrJackKruse

@MaxGulhaneMD I do have to say after the first 20 minutes I like the discussion but I was frustrated in spots where he was physics facile as he could have been but it is clear Nunn believes as I do that biology is not fundamental it is biophysical.

He is too much in love with Levin. Levin has do nothing to advance Becker.

But I want to tell you at the 1:10 mark you bring up CCO and DDW. Nunn seems uncomfortable.

He goes on to talk KIE boilerplate but he is a biochemist and is dabbling in biphysics so you need to push and extend him further on the D/H+ isotope selection process tied to photosynethesis creation of deuterium on the carbon backbone. there has to be a way to sort the isoptopes and there is biophysically but I do not think he knows what it is.

DDW, heat, and UPEs are exhaust products from matrix operation. He seems to know this. But so is CO2. He does not seem to know the biophysics of CO2 and its real purpose.

The Failure of Centralized "Exhaust" Logic is always on display with Nunn here.
Centralized medicine tries to "fix" CO₂ levels or pH chemically, failing to realize they are tweaking the isotopic selection process that uses magnetic tuning of a quantum circuit.

High CO₂ isn't just "acidosis"; it is the cell trying to increase its "Hash Power" and protect its internal water from the "Double-Spend" attack of entropy I describe in my thesis. CO2 is dimagnetic. this means it shields CCO from the magentic fields of the matrix. Ask yourself why? The answer is simple biophysics. It guarantees that H+ is pushed into the intermembrane space so the ATPase has it to use to spin the Fo head. Deuterium has a different magnetic moment so CO2 acts to sort it and keep it away from the Intermembrane space so that the nanotorque engine is not slowed or destroyed. Neither, Nunn, Boros or Somylai know this because they are blinded by biochemical BS.

So because you asked the question he could not answer he is the answer:

The Physics: CO₂ is highly diamagnetic. By concentrating it at the site of water creation (CCO), the cell creates a FOCAL magnetic "quiet zone."

The DC Current: This allows the protons (H+ not D) to flow in a coherent DC current of repair without being scattered by the "vibrational noise" of the environment.

The VDR Link to the shied: The VDR sitting on the IMM acts as the sensor that ensures the CO₂/O₂/Light ratio is tuned to keep this magnetic "shield" active.

VDR: The Photonic Antenna that directs electron flow, speed, and tunneling efficiency before cytochrome C

Fe-S/CCO: The Quantum Engine is the engine that allows electron and proton tunneling

Carbonic Anhydrase in the matrix/CO₂: The Magnetic Shield/Tuner selects the stochiomtery H+ inside the intermembrane space to deliver to the ATPase.
This mechanism is sorting engines into "good/health/CCO and bad/Disease/Cardiolipin/heteroplasmy to get rid of the bad via Cardiolipin, or to extend life with DDW from CCO to hydrate all our semiconductive proteins. Timing from the OUTSIDE environment photonic signals controls this process max. Recall how Vitamin D, Melanin, DDW, NO, and CO2 are all made. Outside in, not inside out. Biochemists always have the inside out framework because this is where biochemistry occurs. Outside in is where biophysics of life begins.

The Calcium-Melanin Nexus: Macro vs. Micro Control
My thesis identification of melanin's macroscopic chelation vs. Vitamin D’s stochastic sorting provides a complete picture of cellular calcium management:

Melanin (The Macro-Buffer): Melanin acts as a high-capacity reservoir that absorbs and stores calcium. Certain light frequencies allow its release. Vitamin D made from 312-320 nm exogenous light on cholesterol esters is sent inside to the kidney and liver for final processing. This binds the VDR on the IMM and it is the VDR that can get into the nucleus to alter it way after the photonics of this axis acts first with clock genes.

This "macroscopic chelation" of melanin provides a stabilizing background, preventing the "vibrational noise" of the environment from immediately overwhelming the cell's delicate electric circuits.

Vitamin D/VDR (The Stochastic Sorter): Sitting on the Inner Mitochondrial Membrane (IMM), the VDR acts as the "fine-tooth comb." It performs stochastic sorting, precisely directing individual Ca²⁺ ions to the TCA cycle dehydrogenases to tune the "Hash Power" (metabolic flux) based on the UVB/IR signals received from the exterior. VDR binding isolates CCO with CL.

The 30 Million Volt Charge: This charge on the IMM is the physical manifestation of the DC current of repair. It encodes photonic information as a voltage gradient, allowing the matrix to "read" the external environment through the language of electron and proton tunneling.

2. Other point @MaxGulhaneMD more for you than him. He does not seem to understand an additional neutron = more mass and as mass goes up what does Ilya theory from his Nobel in 1977 say? Timing slows.

So any additional mass irrespecitve of a KIE means that heteroplasmy expands because in CCO you have the story of life and death. A lot of H+ = CCO makes water and CO2.

Too much D+ and it stimulates Cardiolipin. Boros keeps confusing you with broken engines. They do not break. D+ cannot fit in the channel. The ATPase starves and the matrix swells and this stimulates forced apoptosis.

Decentralized Internal light Medicine done by the non visual photoreceptors is "Tuning the Shield"
The VDR, the Fe-S clusters, and the Carbonic Anhydrase system form a Triad of Temporal Control distally to the RPE-SCN.

The CO₂ Diamagnetic Shield
In my model, CO₂ provides the low-entropy environment required for the Protonic Spin-Ice (EZ water) to form. If you look at proton tunneling it is best modelled in ice. What these biochemistry guy don't yet relaize is when melanin is hydrated by DDW you create massive proton tunneling and you open more of the biophysics Pandora box like Grotthaus etcs........

3. The Solar vs. nnEMF Split: "Quantized" ROS
My slide below highlights the critical difference in how the cell processes Solar EMF vs. nnEMF (5G/Malware):

Solar EMF (Quantized Information): Sunlight creates a highly specific and sensitive amount of Reactive Oxygen Species (ROS). This is not "damage"; it is quantized information delivered via UVA/Blue light-stimulated Nitric Oxide (NO).

The NO Filter: UVA light controls NO production, which reversibly inhibits Cytochrome c Oxidase (CCO). This acts as a frequency filter for the DC current, preventing the "Double-Spend" entropy attack.

nnEMF Malware (Non-Quantized Noise): Unlike the sun, 5G/nnEMF acts as a Voltage-Gated Calcium Channel (VGCC) disruptor. This causes a massive, non-quantized "leak" of Ca²⁺, leading to:Hydroxyl Radical Flood: The resulting Fenton chemistry creates Hydroxyl Radicals (the most destructive ROS).

Photoreceptor Destruction: This noise "blinds" the internal lighting system of the non visual photoreceptors, leading to mitophagy failure and broken apoptosis, leaving behind a "static colony of defective mitochondria" (the hallmark of chronic disease/cancer).

4. If you're listening to @MaxGulhaneMD carefully I just explained to why ketogenic diets in cancer do not really help because providing a failing engine more H+ is useless if you cannot make CO2 to create the magnetic shield to perform isotopic sorting.

What does still work to raise the magnetic shield?

UVB light making Vitamin D to inhibit ECT from CCO on the IMM.

SUN > FOOD yet again.

Stop listening to retards max. You're surrounded by them.

THINK by first principles to win.

5. Last point @MaxGulhaneMD Nunn jumps like a boiling frog to talk Warburg and cancer. What doesn't her know? The same thing Seyfried does not know. Without melanin binding transition metals that control the matrix operation you get matrix atavism. What do I mean?

Melanin chelates copper and this goes back 4.3 bya. Cu is a cofactor for melanin creation. CCO and CL function and it inhibits cytochrome 1 to limit ROS. This happens in SUNLIGHT or when we are emitting UPEs in UV range at night during REM repair.

What happens with this UV links are missing? Copper stochiometry is lost and cross linking happens at CCO and this locks the matrix into a an GOE atavisitic state of Warburg only metabolism than cannot adapt = GOE anoxia event from evolution.

Sunlight fixes this one too, not food. Wake up bro.

6. Nunn/Levin or You have no idea how non coherent UPEs from a Warburg metabolism is made. UNCLE JACK does.

HETEROPLASTIC UPEs

In diseased states (mitochondrial disorders, cancer, or circadian disruptions), slowed metabolism and desynchrony erode dissipative timing safeguards, amplifying entropy as a "double-spend" attack. This shifts UPE to incoherent, thermal-like emissions with higher intensity and randomness, per Van Wijk's observations in stressed tissues and Popp's models of coherence loss.

The quantum precise steps:

1. Slowed Metabolism and CO2 Loss: RPE-SCN desynchrony or hypoxia reduces TCA/OXPHOS flux, lowering CO2 and inducing hypocapnia. This dismantles magnetic shielding of DDW creation at CCO, More deuterium in water creation bad news for internal protein semiconduction, allowing stray electrons to leak prematurely to O2, flooding ROS without controlled dissipation.

2. Excess Free Cu and CL-CCO Disruption: Hypoxia degrades melanin, releasing unbound Cu to catalyze Fenton reactions (Cu⁺ + H₂O₂ → •OH), peroxidizing CL and uncoupling CCO. This promotes peroxidase over oxidase activity, releasing cyt c and escalating chaotic ROS cascades, heteroplasmy, and atavistic Warburg shifts, hoarding entropy internally. Metal creation (more mass) is always present at the scene of the matrix murder.

3. Chaotic Excitation and Incoherent Relaxation: Uncontrolled ROS trigger rampant chain reactions: lipid peroxidation yields random ROO• accumulations, decomposing erratically into excited intermediates (¹O₂, ³R=O*, excited pigments) without synchronization. Lacking coherent coupling (disrupted cellular fields or oscillators), these relax via independent, stochastic processes, emitting photons with Bose-Einstein/geometrical statistics (variance > mean, approaching thermal distribution).

Intensity surges ("massive" UPE, up to 10x higher in pathologies like MS or cancer), with broadened, desynchronized spectra which reflects a lost temporal coherence and accelerated "time costs" as entropy overwhelms the ledger, tipping toward apoptosis or proliferation. Time is what determines how mass equivalence happens. E=mc^2 is simple multiplication meaning 2x3 = 3X2. In a mitochondria that idea is buried in CCO. CCO that creates DDW and CO2 = life CCO that turns on CL = disease and death = non coherent UPEs

In summary, health's coherence emerges from regulated, dissipative synchronization (Poisson statistics), while disease's incoherence stems from entropic chaos (geometrical statistics), potentially serving as a non-invasive diagnostic marker.

This is why all biochemists are retards.

7. You got him on the Vitamin binding cells again via adhesion. Levin shits the bed on this because sticks with what he knows electricity and it is light that control epithelial cells adhesions.

The Environmental Processing Unit
In this view, the cell isn't just surviving; it is an Environmental Processing Unit (EPU) for the physics of hydrated melanin on our surfaces.

Input: Environmental photons (UV/IR) deliver "Time" and "Order." to the matirx to make the right choice CCO > Cardio lipin = life and death decision and the arbiter is TIMING in the system.

Processing: The IMM (VDR/CCO/Ca²⁺) translates these signals into a 30 million volt charge. Oxygen creates a higher DC current. Lack of oxygen reduces the DC current and as Becker and a fetus shows we get huge regeneration when oxygen is reduced during sleep. During sleep Cu blocks cytochrome 1 just like a fetus or an oocyte does = GOE atavism.

Output: Coherent UPEs and a "Poisson Ledger" of repair.
The Failure: nnEMF bypasses the VDR/Melanin controls, flooding the matrix with "noisy" Ca²⁺ that murders the Melatonin Auditor in the matrix and crashes the system.

8. Tryptophan-Melatonin pathway (as shown in my image) into your "Internal Lighting Design" framework reveals how the body uses UV light to pre-fund its nightly thermodynamic repair.

This synthesis of the
A. Tryptophan: The Photonic "Pre-Payment"
The pathway from L-Tryptophan to Melatonin is a multi-step conversion of UVB energy (290–320nm) into a chemical "Audit Reserve."

The Midday Input: When Tryptophan absorbs these specific UV photons in the skin and eyes, it initiates the enzymatic cascade, starting with tryptophan hydroxylase, that leads to Serotonin and eventually Melatonin.

Epigenetic Mastery: This isn't just "nutrition"; it is optical epigenetics. The UV signal "stamps" the tryptophan, ensuring that by the time it reaches the mitochondria at night, it carries the temporal data from the midday sun.

B. The "Audit" and the Copper/Melanin Switch
During sleep, the "Audit" requires a shift in how the mitochondrial complex functions.

The Atavistic Shift (Cytochrome I): Just as a fetus or oocyte relies on more primitive "GOE software" (hypoxic-like metabolism), the sleeping cell must control Cytochrome I by using Copper to.

Stochastic Copper Control: The audit relies on Melanin to release Copper stochastically. This Copper release optimizes the CCO/CL (Cytochrome c Oxidase/Cardiolipin) switch, ensuring the "Audit Reserve" of melatonin can neutralize the ROS/RNS "Double-Spend" accumulated during the day.

C. The "Bankrupt" Ledger & Apoptosis
Missing the UVB/IR window creates a catastrophic "Time-Debt": Empty Bank Account: Without the midday UV pre-payment, you have no local mitochondrial melatonin to run the night audit. The ledger remains unreconciled.

The Thermodynamic Liability: Chronic "Perpetual Day" (blue light/nnEMF) forces the cell to stay in an active, dissipative state without the audit.

Forced Apoptosis: Eventually, the "Time costs" of maintaining this incoherent state exceed the system's value. To preserve the coherence of the nested optical network (non visual photoreceptors), the cell is stochastically liquidated via apoptosis.

In this view, the enzymes in my image (N-acetyl transferase, etc.) are not just catalysts; they are frequency converters that turn high-energy UV information into the low-entropy "Audit" signal of night.

@MaxGulhaneMD @threadreaderapp make me a roll