Laurence Badgley, M.D. Profile picture
Aug 29, 2019 9 tweets 2 min read Read on X
Unilateral dominance of chronic pain & disability is something I commonly see in the #fibromyalgia patients I study. My theory is that they injure one side of their low back. The explanation seems to be in the pelvis...
The pelvic girdle is a bony ring & is platform for the spine. When the pelvis is fractured it commonly breaks in two places on the ring. However, forces that are not strong enough to fracture bone can tear ligaments.
When pelvic joint ligaments are injured, it usually occurs to one of the sacroiliac joints (SIJ). My guess is that impact of injury springs one joint first; tearing ligaments here first & this defuses/decompresses force from injuring other SIJ; protecting it.
Once a unilateral SIJ becomes injured, this evokes an unstable pelvic platform & spine assumes a functional scoliosis to maintain a medial body center of gravity. Usually, shoulder on injured SIJ side tilts lower (seen in mirror); evoking a tilted head.
Over time, chronic low back pain becomes accompanied by upper back pain, Thoracic Outlet Syndrome, TMJ & associated muscle tension HA, & Migraines.
An injured pelvic bony ring hurts when lying in bed due to mattress ground force transiting around ring to the loose SIJ; stretching ligaments. This does not happen with lumbar spine injury pain, which improves lying down.
Even the knee under a loose SIJ takes a beating (supporting a loose hemi-pelvis) & oft develops a chronic painful knee compartment syndrome on same side. The elbow on the tilted side gets lateral epicondylitis (tennis elbow).
Persons with this constellation of aches & pains sleep poorly & non-restoratively; leading to depression & cognitive disarray (fogginess). This constellation of symptoms has been labeled, “Fibromyalgia”.
SUMMARY: if the vector of gravity did not direct from heavens toward center of earth, we would not see predominantly unilateral disabilities in persons with #HSD & #Fibromyalgia.

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More from @BadgleyLaurence

May 6
SHOULD WE BE MEASURING BLOOD LEVELS OF THYMUS HORMONE IN LONG- COVID?
THYMUS HORMONES INCLUDE: Thymosin, Thymulin, and Thymopoietin. There are alpha and beta types of Thymosin.
PITUITARY PRO-HORMONE THAT STIMULATES THYMUS is not known to exist. Therefore, taking exogenous Thymus hormone would not be expected to cause biofeedback and Thymus dysregulation.
Read 6 tweets
Apr 16
LOOSE JOINTS, HYPERMOBITY? Gravity is at play in these women with tissue variations. When joint are unstable the obvious compensation is “gorilla” muscles to take over lax ligament efforts. A wise Physical Therapist is key. Lots of dedicated exercise also.
LOOSE JOINTS INHERITED (-15% women) is normal. These woman’s have easier births & indigo-smart children. These women are often quite attractive and smart.
ABORIGINAL SOCIETIES
CHERISHED HYPERMOBILE
WOMEN BECAUSE THEY WERE SMART & PROLIFIC BIRTHERS. INTELLIGENCE GENES LIKELY GROUP WITH HYPERMOBILITY GENES & GENES FOR BEAUTY. MEN WANTED FECUND ATTRACTIVE WOMEN SO THEY SELECTED THEM OUT.
Read 6 tweets
Apr 14
HYPOPERFUSION? Yes, maybe sympathetic nervous system dominance + decreased pituitary vasopressin causing excess free water elimination in urine ⏩️ decreased blood circulation of hyperosmolar blood.
COVID-CAUSED decreased blood volume of blood that is too thick might explain orthodtstic hypotension, POTS. Might also explain the wormy blood clots.
COVID-CAUSED diminished total blood volume might evoke sympathetic nervous system activation to prevent hypotension and prevent syncope. Diminished blood volume would be expected to deliver less oxygen to tissues ⏩️ fatigue & exercise intolerance.
Read 5 tweets
Apr 9
TO BE GAY ON NOT, THE QUESTION? In ancient days of the mid last century, the proper medical terminology was “homosexual”. Homo is a Greek prefix for “man” such as used in our species name, homosapiens, I think. Along the way a new adjective/noun, a euphemism, was created, “gay”.
“GAYNESS” might be adverb. I don’t know, grammar not my long suit. In any case, Anthropologist of last century seemed to be of consensus that a fixed ~15% of the population was homosexual as a transcultural genetic characteristic of homosapiens. Indeed, genes direct hormones.
TO BE GAY OR NOT: THE “GREAT H & E DEBATE” (heredity vs. environment) is a dynamic that has ever lent unending entertainment and fascination to the study of the human condition & especially to the world of medical studies.
Read 7 tweets
Apr 6
MCAS IN LONG COVID: is possibly etiologic via biomechanisms known about MCAS, HYPERMOBILITY (HS) & FIBROMYALGIA (FM). Persons with HS & FM commonly suffer MCAS. Reason has been theorized that mast cells reside proximate connective tissue.
MCAS, HS, & FM: hyperflexible connective tissue evokes mast cell degranulation. Reactive substances provoke proximate neural tissues. Some experimental data for this dynamic. It might be that those with long COVID & MCAS expression might have hypermobility (about 15% women).
Read 11 tweets
Mar 12
IC CAUSE DISCOVERED: for years clinicians have been mystified by etiology of IC. Genes, microbes, toxins, & biochemistry have failed to provide insights. IC oft occurs in women who have Hypermobility Syndrome and/or IBS. These circumstances suggest that IC is a dysautonomia.
IC A DYSAUTONOMIA? parasympathetic part of autonomic system stimulates smooth tissues of bladder & bowel to contract/evacuate those organs. Over activity of this neural network is unmeasurable, as yet, in current medical sciences. Absence of evidence is not evidence of absence.
IC CLINICAL DATA: hypermobile women are potentiated for developing pelvic girdle instability from loose sacroiliac joint (SIJ) ligaments & are prone to Mast Cell Activation Syndrome (MCAS). Dr. Afrin’s book explains MCAS. I published case studies of IC remission via SIJ fusion.
Read 9 tweets

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