ANXIETY AND PANIC ATTACKS:
I have my own theories about these disorders in those with #HSD. Panic attacks are common in persons with #Fibromyalgia, the patient population I study.
Often people with #Fibromyalgia come to me by way of federally-funded clinic Psychologists, who are treating them for psychologic disorders. They want me to prescribe major psych meds.
Many of these patients have been diagnosed with major depression & sent for anti-depression meds. Their underlying #Fibromyalgia has not been detected & no one has asked them about their average total nightly sleep time nor number of awakenings.
Patients with #Fibromyalgia sleep like “rotisserie chickens”. They toss and turn & sleep about 3-5 hours total; with several awakenings. Their sleep is interrupted by pain & is non-restorative of brain function. Daytime fatigue & naps are common.
Many of these patients have been diagnosed with major depression & sent for anti-depression meds. Their underlying #Fibromyalgia has not been detected & no one has asked them about their average total nightly sleep time nor number of awakenings.
Many of these patients surface sleep & rarely dream.
Patients with restless sleep are cognitively confused (foggy) & will go into rooms forgetting why. The major consequence of non-restorative sleep is Depression. Neurologists know this, or should. Loss of sleep induces anxiety & worry over inability to reverse downward mood slides
Many persons with #HSD have vertebral spine scoliosis when upright (disks are ligaments). I regard that functional scoliosis might induce impingements of contiguous ANS sympathetic chain, resulting in cardiac rhythm changes & gastric dysfunctions, i.e., Gastroparesis.
Functional scoliosis can be observed by companions of patients. Simply slide 2 fingers down sides of spine. Patient can look in mirror to see one shoulder lower.
Many persons with #HSD have Mitral Valve Prolapse (MVP). Mitral valves of heart are made of cartilage/connective tissue; when floppy can lead to incompetence & loss of forward blood flow when going upright. MVP is not a disease, but a functional disorder.
Another #HSD tissue variation is loss of venous vessel wall cartilage/connective tissue/muscle firmness & contractility. When patients go upright, blood pools in lower extremities depriving brain; causing head rushes & even syncope.
When the brain senses low pressure of blood flow it has options: cause syncope & render body tower horizontal so blood more easily flows to brain. Dystonic flailing are common with syncope & can be misinterpreted as “seizures”; later studied as “pseudoseizures”.
Another option of brain being deprived of oxygenated blood is for autonomic NS (sympathetic) to race heart & lungs, with symptoms experienced as shortness of breath & tachycardia. The person feels like they are about to die. This is called a Panic Attack.
Persons wPanic Attacks & absent laboratory findings (anemia, thyroid, etc.) worry doctors, who then consider psychologic issues; with Psychologist/Psychiatrist referrals to investigate. What these patients really need is cardiac ultrasound to visualize mitral valve in real time.
Those who have frequent syncope need to be referred to a Cardiologist & evaluated for Postural Orthostatic Tachycardia Syndrome (POTS). Psychologic issues are diagnoses of exclusion.
Once branded with psychological problems, many patients have difficulty ever finding a doctor to consider alternative diagnoses; much less to elicit history & stigmata of hypermobility.
A suggestion for patients is to bring wiki-type print outs of problems to doctor. If Dr. dismisses your input & takes not time to describe reasoning for diagnosis, you need to regard excess doctor-ego & find another physician (means “teacher” in Greek).
If the reader has interest in clinical medical references there are none; except for my previous publications. The theories and physiological dynamics offered above are my own novel inventions, and based on countless empirical clinical observations.
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these symptoms are due to intermittent impingement of neural branches within the brachial neural plexus, between the collar bone and the first rib. Women with FM often have tightness of this “Thoracic Outlet” d/t hyper flexible ligaments around the shoulder girdles.
THORACIC OUTLET IMPINGEMENT CORRECTION: Avoid surgeons. Physical therapists have ideas. Build muscles of military posture. Wear small backpack backwards by hang on chest. Fill pack with ~15-20% body wt. (bag sugar/rice). Wear when shopping, walking, working in yard.
THORACIC OUTLET IMPINGEMENT CORRECTION: the idea is to use gravity magnification imposed on muscles that lift shoulder bones up-back & thereby reduce slouching, even when pack is disused.
This is Dr. Krause, Neurosurgeon, of an El Salvador healing center he set up contemporaneously. Dr. Delgado, Neurosurgeon he mentions, was one of my Professors at Yale Medical School in 1966 & famous then for implanting radio frequency controlled electrode in a bull’s brain.
MIND CONTROL WITH EMF & LIGHT: Dr. Delgado, or surrogate, got into bull ring. As bull charged for the kill a flick of a switch stopped him in his tracks. ‘‘Twas a powerful demonstration. Dr. Delgado was an handsome Surgeon & the coeds, my own same-time fancy, swooned.
IBS IN VETERANS: As a medical doctor I have clinically studied this disorder. My clinical impressions are as follows:
My prediction is that this disorder will be found in more female veterans than males. My Threadreader to follows explains why …
VETERANS WITH IBS, PREFACE TO THIS THREAD: Over >2 decades, I have studied pathophysiology/etiology of IBS. Amongst my >300 threadreaders on Twitter/X, I have discussed IBS issues.
VETERANS WITH IBS, MY AUTHORITY: Since 2007, I have published clinical data internationally in “Proceedings of Interdisciplinary World Congress on Low Back & Pelvic Pain”, which Congress meets every three years.
HYPERMOBILITY SYNDROME (~15% women) predisposes ▶️ Dysmenorrhea due to Relaxin hormone each menses ▶️ Abdominal Endoscopy whereby 100% women ▶️ intestinal adhesion from #2-3 Scopes ▶️ IBS symptoms of partial obstruction = “Endometriosis” …
After several endoscopic procedures iatrogenic-caused bowel adhesions induce dysfunctional bowel manifest as IBS. Now the time for widespread ablation of peritoneal tissue. I am curious about the long term effects of this mutilating procedure.
FIBROMYALGIA SLEEP SOLUTION? Very simply, the solution is increased REM sleep. FM sufferers are surface sleepers aroused, by musculoskeletal pain, from deeper levels of sleep. Ask them if they regularly dream, and most admit they don’t.
FIBROMYALGIA & SLEEP: A sine qua non of Fibromyalgia (FM) is chronic low back pain. Commonly, etiology of FM low back pain is unilateral sacroiliac (SIJ) dysfunction/subluxation, whereat integrity of the SIJ capsule has been compromised.
DAMAGED LIGAMENTS HURT, as the Spanish Inquisition’s use of “The Rack” taught us. Each sacroiliac is 17 cm sq., largest joint in axial spine, & has a large ligament capsule. Torsion of this capsule occurs at night …
DYSAUTONOMIAS: etiologic via major joint subluxations (shoulder/pelvic girdles) proximate autonomic tracts/plexi become impinged, due to titled upright body tower, due to ⏬️ musculoskeletal tone, due to prolonged (>2 wks) bedrest, due to significant viral illness. GRAVITY RULES
FIBROMYALGIA BEGETS ITSELF: Once chronic widespread muscle spasms begin, chronic pain & restless sleep induce sedentary life, reclusiveness, daytime fatigue and depression. These changes lead to inadequate sunlight, low Vit D, weaker spastic muscles, & greater sedentariness.
FIBROMYALGIA BEGETS ITSELF: weak spastic muscles in persons with Hypermobility Syndrome (~15% of women) induce joint subluxations. These subluxations impinge the Autonomic Nervous System plexi at shoulder & pelvic girdle joints….