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Gabor Erdosi 🧩📄📊🧭 Profile picture
@gerdosi
Jul 8, 2020 • 4 tweets • 3 min read • Read on X
I keep hearing that SARS-CoV-2 is not like the flu, it’s a totally different virus. Well, can you spot the differences & similarities? I highlighted the most important commonality, i.e. how they typically kill.
mdpi.com/1999-4915/12/4…
sciencedirect.com/science/articl… ImageImage
cdc.gov/flu/symptoms/s… Image
The majority of SARS transmissions was linked to hospitals (intubation and ventilation as prime suspects) and to superspreading events. Sounds familiar? Why did it not to our infectious disease experts?
sciencedirect.com/science/articl… Image
Even though some of the immunity and R0 parameters used are already outdated a bit, this comparative review is a really good read. May I draw your attention to Table 1. thelancet.com/journals/lanin…

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More from @gerdosi

Gabor Erdosi 🧩📄📊🧭 Profile picture
Feb 18, 2023
Here I was called a COVID denier (what is that?), even though almost everything I stood for has been very close to reality since about March 2020. In fact, the only thing I was wrong about was the real world CFR of the virus. Back then I didn’t understand that poor management…
…was responsible for many deaths. Too early and unnecessary use of ventilators, too early and too high dose use of dexamethasone, remdesivir administration in hospitalized patients, avoidance of adding cheap and very low risk prophylactics, etc.
I only recognized these later.
I do remember when ventilator use was cut in half from one week to the other, following a protocol change in May 2020. Then I read this review from 2017 & realized that a lot more had been known about disease treatment than what was happening in hospitals. How could this happen?
Read 5 tweets
Gabor Erdosi 🧩📄📊🧭 Profile picture
Feb 15, 2022
Dude either has no clue about what ‘immunity’ is, or has a very narrow tunnel view of it. Alternatively he’s deliberately spreading misinformation here.
NB, more typical courses have been:
Infection with Wuhan + Omicron
Infection with Alpha + Omicron
Infection with Wuhan + Delta + Omicron
Etc.
Guess how these typical scenarios fair against vaccination + Omicron? Yep, you’re absolutely right.
Immunology noobs question my assessments above.
Please consider that the study only looked at systemic humoral immunity and Ab binding to the spike protein. Then think about all the layers of immunity omitted from the analysis, e.g. innate, mucosal, T cells, to all antigens, etc.
Read 4 tweets
Gabor Erdosi 🧩📄📊🧭 Profile picture
Dec 20, 2021
If you’re heading to the hospital with COVID make sure to declare that you’re allergic to remdesivir. It’s ineffective anyway.

Kidney disorders as serious adverse drug reactions of remdesivir in coronavirus disease 2019: a retrospective case–noncase study kidney-international.org/article/S0085-… Text Shot: Our findings, ba...
While remdesivir is still touted as ‘safe and effecrive’ in the COVID response of many countries, public health officials start removing health insurance reimbursement for vitamins and minerals due to lack of efficacy shown… Could you make this up? businessinsider.co.za/medical-scheme…
My sad personal experience echoes this. theepochtimes.com/controversial-…
Read 4 tweets
Gabor Erdosi 🧩📄📊🧭 Profile picture
Dec 10, 2021
‘Pandemic’ (changed by WHO in 2009)
‘Vaccine’ (changed by CDC in 2021, where kefir or vitamin D now fully qualify)
‘Peer reviewed’ (clueless people repeat it as mantra)
‘Breakthrough infection’ (if there’s robust immunity
‘T cells’ (ignored and ridiculed until Omicron appeared)
Oops, some editing issue at ‘peer reviewed’
If there’s robust immunity infecfion doesn’t occur. It only does if immunization was unsuccessful.
I think that we can safely add
‘hybrid immunity’
when it’s used as some superhuman state instead of natural immunity with repeated pathogen/antigen exposure.
Read 4 tweets
Gabor Erdosi 🧩📄📊🧭 Profile picture
Dec 6, 2021
A recently developed method, that expresses full spike trimers, enables more accurate measurement of binding IgG1 antibodies. Quite a few interesting results in this study.
First of all, the second dose of mRNA vaccines adds very little extra binding. medrxiv.org/content/10.110…
More importantly, the absolute increase in Delta variant binding antibodies following administration of the 2nd dose to previously infected people is limited to a small subset of adults. At the same time, the relative number of Delta binding Ab to non-binding (old Wuhan) Ab is…
…terribly low. What’s the use of that thick soup of sky high antibodies then? Triggering autoimmunity? 🤔
The authors are not impressed either, although expected such results from shotgun injecting the same antigen, leaving no time for maturation.
Read 4 tweets
Gabor Erdosi 🧩📄📊🧭 Profile picture
Oct 11, 2021
Let me strip the unnecessary narrative out of this otherwise nice study/abstract to get a clear picture. Short thread 🧵

Anti-SARS-CoV-2 receptor binding domain antibody evolution after mRNA vaccination
nature.com/articles/s4158…
How infection elicits broad protection
“SARS-CoV-2 infection produces B cell responses that continue to evolve for at least one year. During that time, memory B cells express increasingly broad and potent antibodies that are resistant to mutations found in variants of concern”
Following vaccination
“Between prime and boost, memory B cells produce antibodies that evolve increased neutralizing activity, but there is no further increase in potency or breadth thereafter.”
= the booster is too close to the priming dose and prevents affinity maturation.
Read 8 tweets

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