Laurence Badgley, M.D. Profile picture
Sep 17, 2020 9 tweets 2 min read Read on X
Botox yes! In TOS 1 Superior trapezius usually spastic/hyperdynamic; inducing same-side Pectoralis minor muscle to be spastic/hyperdynamic. These muscles control directional shift of distal clavicle; which, as drawn inferior, closes Thoracic Outlet (1 cm vs. clavicle & 1st rib).
The physical exam should note shoulder asymmetry; one shoulder lower with head usually tilted toward lower shoulder. The tilted 10# head often induces spasm & hypertrophy of Superior trapezius associated with opposite & higher shoulder.
Superior trapezius hyperdynamic function then induces spasm/hypertrophy within same-side opposing (shoulder lift & draw-down are complementary functions) Pectoralis minor (PM) muscle. It is PM hyperdynamic function that draws distal clavicle excessively inferior in TOS disorder.
The clavicle is about 18 cm long & 1 cm of excess inferior shift can close the Thoracic Outlet (TO) by ~30%; impinging brachial neurovascular bundle contents of the TO.
That part of the brachial neural plexus that gives rise to the ulnar nerve is the first part of the brachial neural plexus impinged as Thoracic Outlet closes; anatomical explanation why ulnar paresthesias are most common dysesthesia aroused by arm-shoulder abduction with TOS.
After surgeons failed to cure TOS with flayed/excision of Scalenes (~1980’s theory of TOS etiology), they discovered that Pectoralis muscle release (off corocoid process) & tie-down of first rib to 2nd (axilla surgical entrance) was therapeutic; opened up the Thoracic Outlet.
Yet this surgical approach seems somewhat butchery. Better to reduce “crossed muscle” asymmetry of Superior trapezium (strengthen weaker Superior trapezius with isometric & shrug exercises) & induce muscle relaxation of spastic shoulder muscles with massage, Botox & biofeedback.
The theories and practices referenced in this thread are my own novel insights and inventions; gained from years toiling in the clinical trenches. I have created several other threadreaders about TOS.

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More from @BadgleyLaurence

May 6
SHOULD WE BE MEASURING BLOOD LEVELS OF THYMUS HORMONE IN LONG- COVID?
THYMUS HORMONES INCLUDE: Thymosin, Thymulin, and Thymopoietin. There are alpha and beta types of Thymosin.
PITUITARY PRO-HORMONE THAT STIMULATES THYMUS is not known to exist. Therefore, taking exogenous Thymus hormone would not be expected to cause biofeedback and Thymus dysregulation.
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Apr 16
LOOSE JOINTS, HYPERMOBITY? Gravity is at play in these women with tissue variations. When joint are unstable the obvious compensation is “gorilla” muscles to take over lax ligament efforts. A wise Physical Therapist is key. Lots of dedicated exercise also.
LOOSE JOINTS INHERITED (-15% women) is normal. These woman’s have easier births & indigo-smart children. These women are often quite attractive and smart.
ABORIGINAL SOCIETIES
CHERISHED HYPERMOBILE
WOMEN BECAUSE THEY WERE SMART & PROLIFIC BIRTHERS. INTELLIGENCE GENES LIKELY GROUP WITH HYPERMOBILITY GENES & GENES FOR BEAUTY. MEN WANTED FECUND ATTRACTIVE WOMEN SO THEY SELECTED THEM OUT.
Read 6 tweets
Apr 14
HYPOPERFUSION? Yes, maybe sympathetic nervous system dominance + decreased pituitary vasopressin causing excess free water elimination in urine ⏩️ decreased blood circulation of hyperosmolar blood.
COVID-CAUSED decreased blood volume of blood that is too thick might explain orthodtstic hypotension, POTS. Might also explain the wormy blood clots.
COVID-CAUSED diminished total blood volume might evoke sympathetic nervous system activation to prevent hypotension and prevent syncope. Diminished blood volume would be expected to deliver less oxygen to tissues ⏩️ fatigue & exercise intolerance.
Read 5 tweets
Apr 9
TO BE GAY ON NOT, THE QUESTION? In ancient days of the mid last century, the proper medical terminology was “homosexual”. Homo is a Greek prefix for “man” such as used in our species name, homosapiens, I think. Along the way a new adjective/noun, a euphemism, was created, “gay”.
“GAYNESS” might be adverb. I don’t know, grammar not my long suit. In any case, Anthropologist of last century seemed to be of consensus that a fixed ~15% of the population was homosexual as a transcultural genetic characteristic of homosapiens. Indeed, genes direct hormones.
TO BE GAY OR NOT: THE “GREAT H & E DEBATE” (heredity vs. environment) is a dynamic that has ever lent unending entertainment and fascination to the study of the human condition & especially to the world of medical studies.
Read 7 tweets
Apr 6
MCAS IN LONG COVID: is possibly etiologic via biomechanisms known about MCAS, HYPERMOBILITY (HS) & FIBROMYALGIA (FM). Persons with HS & FM commonly suffer MCAS. Reason has been theorized that mast cells reside proximate connective tissue.
MCAS, HS, & FM: hyperflexible connective tissue evokes mast cell degranulation. Reactive substances provoke proximate neural tissues. Some experimental data for this dynamic. It might be that those with long COVID & MCAS expression might have hypermobility (about 15% women).
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Mar 12
IC CAUSE DISCOVERED: for years clinicians have been mystified by etiology of IC. Genes, microbes, toxins, & biochemistry have failed to provide insights. IC oft occurs in women who have Hypermobility Syndrome and/or IBS. These circumstances suggest that IC is a dysautonomia.
IC A DYSAUTONOMIA? parasympathetic part of autonomic system stimulates smooth tissues of bladder & bowel to contract/evacuate those organs. Over activity of this neural network is unmeasurable, as yet, in current medical sciences. Absence of evidence is not evidence of absence.
IC CLINICAL DATA: hypermobile women are potentiated for developing pelvic girdle instability from loose sacroiliac joint (SIJ) ligaments & are prone to Mast Cell Activation Syndrome (MCAS). Dr. Afrin’s book explains MCAS. I published case studies of IC remission via SIJ fusion.
Read 9 tweets

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