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Marion Holman Profile picture
@holmanm
Sep 19, 2020 • 5 tweets • 1 min read • Read on X
1/5 Rather than stop their use altogether, the modality of the health profession is to simply change statins when dangerous symptoms are reported. The most common and immediate statin use 'side effect' is muscle soreness and pain.
2/5 It is inevitable that everyone taking statins long term will develop one or more of these direct effects of statin use. Despite all the hype of the drug company-engineered study results on the so-called benefits of statins, the hard truth, the hard science proves otherwise
3/5 Other effects of statin use are too many to list but here are just a few:

Arthritis, Rhabdomyolysis, Type 2 Diabetes, Renal failure, Myopathy, Cardiomyopathy, Cancer, MS, ALS, Lupus, Parkinson's disease, Heart failure
4/5 Hepatitis, Diverticulosis, Memory loss, Mental disorders, Dementia, Cataracts, Tendonitis, Hair loss, Dermatitis, Pancreatitis, Peripheral Neuropathy, Weight Gain, Hearing Loss, Balance loss, Loss of libido, Chronic fatigue, Interstitial Lung disease
5/5 There is no such thing as a statin side effect. Statin poisoning is a direct & predictable effect of blocking mevalonate by inhibiting the enzyme reductase, which makes it.

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More from @holmanm

Marion Holman Profile picture
Feb 22
1/5 Statins are Mycotoxins:
Statins do their damage in two ways. By their Mevalonate Blockade and their Toxic content.
The lactone ring of Mycotoxins such as Ochratoxin A mimic the HMG-CoA Reductase molecule in order to inhibit it, which in turn enables Statins to block the /2
2/5 Mevalonate pathway. Ochratoxin A induces programmed cell death both in SH-SY5Y cells and in primary Neurons. Taking into account that most Neurodegenerative diseases including Alzheimer’s ALS, and Parkinson’s disease are related to neuronal cell death and that the central /3
3/5 nervous system tissue has limited regenerative capacity, it is obvious that Statins are responsible for a huge explosion in such serious autoimmune conditions, and should have never made it to market. Statins do not prevent heart disease,
Read 5 tweets
Marion Holman Profile picture
Feb 18
1/4 Describing all conceivable oxidations in our body is impossible. Even covering only oxidation of LDL-carriers is complex: lipids, proteins, DNA, & other molecules can oxidise.

There are more than 200 types of cholesteryl esters that can oxidise by numerous free
/2
2/4 radicals & oxidative reactions. Cholesteryl esters that incorporate Polyunsaturated fatty acids (PUFA) are most vulnerable to oxidation. Oxidised products can be very destructive.

LDL-carriers must have 1 functioning apoB100 to deliver fats to cells throughout the body.
/3
3/4 An oxidised apoB100 lipoprotein cannot dock with LDL-receptors, which contributes to Atherosclerosis, i.e. the OxLDL-carrier can no longer deliver. The consequences can be minimal i.e. the carrier is dissolved by a scavenger, or substantial (the OxLDL settles in
/4
Read 4 tweets
Marion Holman Profile picture
Feb 18
1/5 Engage in regular periods of intermittent fasting to lower LDL particles
Wait. Did you just say fasting ? As in not consuming any food for a period of time ? Yep. Sure did. The idea of fasting puts a lot of people off but regular intermittent fasting has been shown /2
2/5 to lower the level of LDL particles in your blood. My anecdotal experience has shown me that people with exaggerated LDL values benefit more than most from intermittent fasting, which mimics the natural human experience of deprivation. When you are doing /3
3/5 intermittent fasting, you might see a rise in Triglycerides for several days afterwards. This is a good thing because these Triglycerides are released from stored body fat. That’s a totally natural response. People sometimes get upset when they’ve just lost a /4
Read 5 tweets
Marion Holman Profile picture
Feb 17
1/4 Animals will keep eating until they satisfy their protein requirements, so a diet low in protein naturally stimulates overeating. This is why the most effective strategy for reversing Metabolic Syndrome is increasing protein while reducing carbohydrates. /2
2/4 This way of eating tends to stabilize hunger and satiety hormones incredibly well. It also decouples your mitochondria so they become active and eager to burn off energy as heat, increases your lean muscle mass, your metabolism, and your bone density. /3
3/4 It also lowers Insulin, drops your Triglycerides and raises your HDL-C.
The Carbohydrates you eat, with the exception of indigestible forms like most fibers, eventually become glucose in your blood. Assuming your metabolism is functioning normally,
/4
Read 4 tweets
Marion Holman Profile picture
Feb 17
1/6 Insulin has a profound effect on Cholesterol. It turns up the Cholesterol making machinery by turbocharging the activity of the enzyme that actually controls cholesterol manufacturing in your body. This enzyme is called HMG-CoA Reductase. /2
2/6 You can improve your lipid profile by simply lowering your insulin levels. By doing so you avoid all of the serious "direct effects" of taking Statin drugs, and will experience improved cardiovascular health. /3
3/6 To reduce your risk of heart disease: - Lower Triglycerides, Increase HDL, and Lower insulin. Do this by eliminating sugar, processed carbs/seed oils/trans fats. Intermittent fasting will also help, as does increasing exercise. /4
Read 6 tweets
Marion Holman Profile picture
Feb 16
1/10 If you read the patient leaflet for Lipitor (Atorvastatin) it states:

“The name of your medicine is LIPITOR. It comes as white oval film-coated tablets containing Atorvastatin Calcium Trihydrate equivalent to 10, 20, 40 or 80 mg of the active ingredient Atorvastatin” /2
2/10 Are you any wiser ? Does it tell you PRECISELY what that “active ingredient” is ? Of course not. First, you need to be aware that in order for Statins to inhibit Reductase a Mycotoxin needs to be present (as the active ingredient). The reason being - /3
3/10 Mycotoxins mimic a portion of the HMG-CoA enzyme which enable Statins to inhibit this enzyme, dysregulate the Mevalonate pathway and upregulate LDL receptors to shift blood cholesterol into organ and muscle cells until those cells undergo apoptosis. /4
Read 10 tweets

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