1/5 Statins are Mycotoxins:
Statins do their damage in two ways. By their Mevalonate Blockade and their Toxic content.
The lactone ring of Mycotoxins such as Ochratoxin A mimic the HMG-CoA Reductase molecule in order to inhibit it, which in turn enables Statins to block the /2

https://twitter.com/dramerling/status/1760760323571622279

2/5 Mevalonate pathway. Ochratoxin A induces programmed cell death both in SH-SY5Y cells and in primary Neurons. Taking into account that most Neurodegenerative diseases including Alzheimer’s ALS, and Parkinson’s disease are related to neuronal cell death and that the central /3

1/4 Describing all conceivable oxidations in our body is impossible. Even covering only oxidation of LDL-carriers is complex: lipids, proteins, DNA, & other molecules can oxidise.

There are more than 200 types of cholesteryl esters that can oxidise by numerous free
/2 2/4 radicals & oxidative reactions. Cholesteryl esters that incorporate Polyunsaturated fatty acids (PUFA) are most vulnerable to oxidation. Oxidised products can be very destructive.

LDL-carriers must have 1 functioning apoB100 to deliver fats to cells throughout the body.
/3

1/5 Engage in regular periods of intermittent fasting to lower LDL particles
Wait. Did you just say fasting ? As in not consuming any food for a period of time ? Yep. Sure did. The idea of fasting puts a lot of people off but regular intermittent fasting has been shown /2 2/5 to lower the level of LDL particles in your blood. My anecdotal experience has shown me that people with exaggerated LDL values benefit more than most from intermittent fasting, which mimics the natural human experience of deprivation. When you are doing /3

1/4 Animals will keep eating until they satisfy their protein requirements, so a diet low in protein naturally stimulates overeating. This is why the most effective strategy for reversing Metabolic Syndrome is increasing protein while reducing carbohydrates. /2 2/4 This way of eating tends to stabilize hunger and satiety hormones incredibly well. It also decouples your mitochondria so they become active and eager to burn off energy as heat, increases your lean muscle mass, your metabolism, and your bone density. /3

1/6 Insulin has a profound effect on Cholesterol. It turns up the Cholesterol making machinery by turbocharging the activity of the enzyme that actually controls cholesterol manufacturing in your body. This enzyme is called HMG-CoA Reductase. /2 2/6 You can improve your lipid profile by simply lowering your insulin levels. By doing so you avoid all of the serious "direct effects" of taking Statin drugs, and will experience improved cardiovascular health. /3

1/10 If you read the patient leaflet for Lipitor (Atorvastatin) it states:

“The name of your medicine is LIPITOR. It comes as white oval film-coated tablets containing Atorvastatin Calcium Trihydrate equivalent to 10, 20, 40 or 80 mg of the active ingredient Atorvastatin” /2 2/10 Are you any wiser ? Does it tell you PRECISELY what that “active ingredient” is ? Of course not. First, you need to be aware that in order for Statins to inhibit Reductase a Mycotoxin needs to be present (as the active ingredient). The reason being - /3

1/9 Most mycotoxins come from 4 species of fungi. Aspergillus, Penicillium, Proteus & Monascus. These same fungal species are used to make Statins. Statins are secondary fungal metabolites made to kill competing microbes. Statins 'look like' the precursor to Mevalonate /2 2/9 so Reductase is "tricked" into bonding with the Statin. Reductase in turn is inhibited. The Scientist working on Statins, Akira Endo reasoned that fungi make secondary metabolites that block Reductase, conveniently overlooking that cholesterol and Isoprenoids are, /3

1/5 When I was a child I remember looking forward to my Mother baking a chocolate cake. It didn’t happen very often, it was a rare treat. The ingredients in that cake were: Flour, sugar, butter, eggs, milk, and cocoa powder.
/2 2/5 Last week I was on a train full of children that had just finished school. I sat next to a young boy who opened a 4 pack of chocolate cup cakes. Within 15 minutes he’d eaten the lot. When he got off the train he left the cake wrapper on the seat next to me.
/3

1/4 Metabolic syndrome is a cluster of risk factors for Cardiovascular disease and Type 2 diabetes which occur together more often than by chance alone.

These risk factors include:
a) raised blood pressure;

/2 2/4

b) a distinct dyslipidemic phenotype (raised Triglycerides and lowered HDL high-density lipoprotein cholesterol);
c) altered glucose metabolism;
d) central obesity.
/3

1/5 Statin research was founded on scientific assumption and an AGENDA. Statins are dangerous drugs developed in error for profit, Scientific evidence had proven that Statins are lethal to cells years before the approval of Lovastatin. /2 2/5 Merck corrupted the science and the scientists. Merck LIED !
The approval process was so corrupted by Merck that Lovastatin was bound to be approved. Bought and paid for by the pharmaceutical industry, the FDA shirked its duty; neither safety nor efficacy was proven /3

1/7 Nobel Prize winning neuroscientist Eric Kandel says - we are who we are because of what we learn and what we remember. Who am I, then, if my memory is impaired ?

People who take Statins very often complain about “brain fog’, inability to think clearly, “memory loss”,
/2 2/7 “depression” etc. Even the FDA warns on Statin labels that people have developed memory loss or confusion while taking Statins.

Statins are - Thief of memory (here’s how)

The basic unit of communication in the nervous system is the nerve cell (neuron). Each nerve cell
/3

1/4 Statin toxicity & the mechanism of action of Statin drugs is VERY clear. However, Statin trials very often claim that fewer than half of patients who experience side effects on Statins can truly attribute them to their medications, & thus it is worth urging patients /2 2/4 to disregard these adverse effects and continue 'therapy'. How insane given the clear evidence that Statins damage every cell in your body. Hardly surprising that patients feel quite debilitated and discouraged by Statins and are /3

1/5 Anyone else think the practice of medicine has completely lost its way ? Remedies that treat symptoms and only offer some relief have replaced the search for cures. No one noticed. The problem is much more pervasive and sinister than that.
/2 2/5 The first & most basic tenant of medicine is "to do no harm". That tenant has been so utterly corrupted by the pharmaceutical industry that it no longer plays into the practice of medicine. Bribes are not necessary. The purchase of influence is done in full public view
/3

1/6 Another innovative and interesting application of Serrapeptase is in the treatment of Alzheimer’s disease via reduction of amyloidosis. Serrapeptase was found to be as effective as nattokinase (an enzyme shown to degrade amyloid fibroid) in relieving Alzheimer’s disease /2 2/6 pathophysiology in a rat model. Oral administration of this enzyme decreased brain acetylcholinesterase activity, as well as levels of transforming growth factor ß, Fas, and interleukin-6, all of which were significantly increased in patients with Alzheimer’s disease. /3

1/5 Interesting: A drug recommended for prevention of heart attacks and strokes is Aspirin. Estimates suggest that 20 million people are taking aspirin daily for prevention of vascular accidents. Yet at least 4 studies have shown no benefit.
/2 2/5 A study using Bufferin (aspirin and magnesium) showed no reduction in fatal heart attacks and no improvement in survival rate but a 40 percent decrease in the number of non-fatal heart attacks. Commentators reported these results as showing the benefit of
/3

1/8 Why might Statins affect nerves adversely? It turns out that nerve cells need cholesterol.
I got permission to share this little gem from someone on a "Stopped our Statins" Forum:

"I’ve been on Statins for about 30 years. My grandfather, mother & several cousins have a 2/8 history of high cholesterol. With an inherited trait, I thought I had to be medicated. Of course I didn’t listen to my Mother when she told me that her doctor never put her on medication for her high cholesterol. She is 89 and to this day hasn’t taken a Statin nor does she /3

1/9 Chronic inflammatory diseases, such as rheumatoid arthritis, lupus, psoriasis, and infections such as periodontal disease (p.gingivalis), & HIV, are associated with an increased risk of heart disease. Patients with these disorders also have an increase in coronary artery /2 2/9 calcium measured by CT & carotid intima media thickness measured by ultrasound. WHY ?? Because inflammation & infections induce a variety of alterations in lipid metabolism that may initially DAMPEN inflammation or fight infection, but if chronic, can contribute to the /3

1/4 The good, the bad and the ugly Killesterol 😂

Remember when “high” Total cholesterol was public enemy number one ? Then they found out that High-Density cholesterol (HDL-C) was “good,” so to maintain the FICTION that cholesterol is bad, we started hearing about the /2 2/4 villainous Low-Density cholesterol (LDL-cholesterol). Now it turns out that LDL is not a bad guy either. So the spotlight moves to Lp(a) as being the evil lipoprotein. When that fails, there’s always the dreaded ApoB, which it turns out isn’t as bad as we once thought /3

1/10 Much maligned Lp(a) 🙄
The presence of Lp(a) in species that have lost the ability for endogenous vitamin C production has been one of the unsolved puzzles of science. Particularly compelling is the fact that the appearance of Lp(a) about 40 million years ago coincided /2 2/10 with the loss of endogenous Vitamin C synthesis by the ancestor of man. Based on this & other observations it is proposed that Lp(a) functions as a substitute for vitamin C in stabilizing the Extracellular Matrix, particularly at times of prolonged nutritional scarcity. /3

1/7 I've seen Warfarin prescribed alongside a Statin on many occasions. What a great way to calcify arteries. A long-term adverse effect of warfarin therapy is vascular calcification. The proven mechanism is warfarin’s near-shutdown of the body’s vitamin K recycling capacity. /2 2/7 As the availability of functional (that is, electronically reduced) vitamin K via the K cycle becomes limited, whether from warfarin therapy, from dietary inadequacies, or from other factors, the body progressively is deprived of vitamin K’s carboxylation capacity.

1/6 Failure to disclose known drug risks by the FDA & MHRA must be prosecuted. Failure to provide raw data from Statin trials should be illegal. (what are they
hiding ?). Studies on safety and efficacy of Statin drugs must be done by truly INDEPENDENT entities without ties to /2 2/6 the pharma industry. With clear evidence of a totally corrupt process being so patently clear, nobody should sit on an FDA approval committee who has vested interest in the outcome. All approval-related meetings, & papers,should be completely transparent and free /3