Let us have a brainstorming session shall we?
Real patient with real management from me 😉
Scenario
40 years old
- obese
- came with back carbuncle
- operated with no complication
This is his cxr prior to admission
Day 4 of admission
- Having shortness of breath
- suddenly desaturated, needing oxygen support
- Hypotensive needing inotropic support
- no fever, cough
- lungs: clear
- S1S2 no murmur
Blood investigation
- no leucocytosis
This is his repeated chest xray on day 4 of admission
What you guys can see from this chest xray?
Yes , oligemic lung field. If you guys cant see the changes, please refer to previous cxr.
it is also called Westermark sign
Westermark sign is a chest x-ray finding of oligaemia (clarified area) distal to a large vessel that is occluded by a pulmonary embolus.
The focal area of increased translucency due to oligaemia is caused by impaired vascularisation of the lung due to primary mechanical obstruction or reflex vasoconstriction
So how do we confirm our diagnosis?
Yes CTPA was done and it showed massive pulmonary embolism. Patient is diagnosed to have a provoke pulmonary embolism.
Pulmonary embolism (PE) is a life-threatening condition resulting from dislodged thrombi occluding the pulmonary vasculature; right heart failure and cardiac arrest may ensue if not aggressively treated.
Clinical probability, assessed by a validated prediction rule and clinical judgement, is the basis for all diagnostic strategies for PE. Computed tomographic pulmonary angiography is the definitive diagnostic investigation.
Management?
First you need to classify whether the patient is hemodynamically stable or unstable
hemodynamically unstable
- Thrombolysis or thrombectomy
hemodynamically stable
- Anticoagulant
Easy right?
That is all from me tonight
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Topic: The Pathophysiology of Thyroid Hormones and Anti thyroid Medications
Remember guys, to master the topic you first need to understand the pathophysiology of
-Thyroid hormones production
-Anti thyroid medications
Follow this thread
Thyroid synthesis
1)Iodine transport
Iodide (I-) from the blood is actively transported into the follicular lumen
2)Thyroglobulin synthesis
Thyroglobulin is a protein that contains large numbers of tyrosine amino acids that go on to become individual thyroid hormone molecules. Thyroglobulin is synthesized within the follicular epithelial cell and secreted into the follicular lumen.
Anda tahu apa itu penyakit Tetanus? (kancing gigi)
Bagaimana ianya berlaku?
Bangaimana untuk megelak dari mendapat tetanus?
Sila follow bebenang ini
Pengenalan
Penyakit tetanus adalah penyakit yang serius berpunca daripada toksin bakteria yang merosakkan sistem saraf dan menyebabkan kekejangan otot yang menyakitkan terutama di bahagian rahang dan otot leher.
Tetanus boleh mengganggu sistem pernafasan anda dan membuat anda susah untuk bernafas.
Sakit Tetanus adalah sangat jarang berlaku di Malaysia disebabkan oleh program immunisasi yang diberikan oleh kerajaan Malaysia.
Atria fibrillation adalah masalah rentak jantung yang tidak sekata dan ianya boleh menyebabkan rasa jantung berdenyut laju, berdebar dan rasa jantung turun-naik dengan cepat.
Simptom Atrial flutter adalah
- Jantung rasa berdebar secara tiba-tiba
- Cepat rasa penat
- Sesak nafas
- Rasa hendak pitam
- atau sesetengah pesakit tidak mempunyai apa- apa simptom
62 y.o guy with
- PTB on intensive phase day 40
- dm
- hypertension
- dyslipidemia
Came with right hypochondriac and epigastric tenderness
- associated with nausea and vomiting
- no fever
- reduced oral intake
- able to pass flatus and BO
- no aggravating or relieving factor
Cxr
- showed consolidation over right upper zone
- bowel shadow below right diaphragm
Patient was treated for chiladaiti syndrome
- ryles tube free flow
- keep nil by mouth
- lactulose 15 mls tds
- IVD for hydration