Thread by @ReachEchoVoice

When I asked for proof of #COVID19 isolation (+ satisfied Koch's Postulates), I was given the following links. Let's take a look.
Note this article comes from McMaster University.
"Multiple researchers had predicted a coronavirus that would evolve and..
theconversation.com/i-study-viruse…

jump into humans."

Here's where the anti-parasitic theories (re such as IVM) would come from:
"These viruses are obligate intracellular parasites... they can only replicate in cells."

"We can use tricks to draw out a virus."
"In this case, the researchers tried a method Banerjee [& team worked on with MERS]: culturing the virus on immunodeficient cells." The claim is that immunodeficiency allows it to spread unchecked. "It worked." This lends to the basis of..

the HIV / AIDS claim. Which immunodeficient cells were used, and what implication(s) does that have for the gene jab?

Further:
ncbi.nlm.nih.gov/labs/pmc/artic…

"We isolated virus [Which virus?] from nasopharyngeal and oropharyngeal specimens [From the human?]..."

Virus replicated to high titer in Vero-CCL81 cells and Vero E6 cells in the absence of trypsin.
Were those chimeric?

en.m.wikipedia.org/wiki/Vero_cell
CCL-81

"As research is initiated to study and respond to SARS-CoV-2, information about cell lines and types susceptible to infection is needed. Therefore, we examined the capacity of SARS-CoV-2 to infect and replicate in several common primate and human cell lines, including human...

adenocarcinoma cells (A549), human liver cells (HUH7.0), and human embryonic kidney cells (HEK-293T), in addition to Vero E6 and Vero CCL81. We also examined an available big brown bat kidney cell line (EFK3B) for SARS-CoV-2 replication capacity.

Each cell line was inoculated with at high MOI and examined 24 hours post infection (Figure 3A). No cytopathic effect was observed in any of the cell lines except in Vero cells which grew to >107 PFU at 24 hours post infection. In contrast, both HUH7.0 and 293T cells showed only

modest viral replication and A549 cells were incompatible with SARS-CoV-2 infection. These results are consistent with previous susceptibility findings for SARS-CoV and suggest other common culture systems including MDCK, HeLa, HEP-2, MRC-5 cells, and embryonated eggs are

unlikely to support SARS-CoV-2 replication (14–16). In addition, SARS-CoV-2 failed to replicate in the bat EFK3B cells which are susceptible to MERS-CoV. Together, the results indicate that SARS-CoV-2 maintain a similar profile to SARS-CoV in terms of susceptible cell lines."

Note: "Virus isolation from patient samples was deemed to be non-human subjects research by [CDC]." 0900f3eb81ab4b6e
"Clinical specimens.. acquired during travel to china, were collected as described."
Hmm.

Also of note:
sciencedirect.com/science/articl…

"This furin-like cleavage site is supposed to be cleaved during virus egress for S-protein 'priming' and may provide a gain-of-function to the 2019 nCoV for efficient spreading in the human population..."
Big emphasis on "antivirals" & furin.

Also: IBV, Cheng.

Post regarding primate adaptation, and a Human-specific binding mechanism:

https://twitter.com/ReachEchoVoice/status/1440487922612924425?s=19

I haven't seen anything to contradict this:

https://twitter.com/ReachEchoVoice/status/1440539481694617600?s=09

There's also this claim (I haven't heard it refuted & the FDA has recalled the tests).

https://twitter.com/FRConclusions/status/1430527639748370435?s=19

https://twitter.com/FRConclusions/status/1433040222095171587?s=09

H/t to .@1Mirror1978