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Mg2+ Papi, the BowTied Mg2+ Profile picture
@Grimhood
Mar 6, 2022 • 5 tweets • 1 min read • Read on X
If you actually review the unbiased literature on seed oils (not your little food guru blog articles, actual studies), this is exactly what those papers suggest is harmful, rather than seed oils inherently.

Food for thought.
The Japanese eat a good amount of seed oils to compose their fat macro dietary intake, along with saturated fats in meats, O3 fats in seafood, and carbohydrates from rice, etc.

Pretty excellent longevity all things considered.
Most also eat much less fast food than the West.
Black seed oil has been consumed for a few thousand years, but never refried. Curious.
Now I’m not saying to go crazy with cooking/consuming industrialized vegetable/seed oils, just offering a newer perspective on the matter. Getting you to think beyond what others tell you to think.

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More from @Grimhood

Mg2+ Papi, the BowTied Mg2+ Profile picture
Dec 15
One of the key factors I notice with those with flagrant digestive issues are a severe aversion to any sort of bitter foods or herbs/vegetables.

These two factors will often go hand in hand, typically dating back to a childish palate and/or downregulated taste receptors that require a reset after a lifetime of a Stupid American or ultra processed diet.

Aside from chloride, zinc, trimethylglycine/betaine, thiamine, and potassium, tasting bitter foods/herbs on the tongue regularly helps to keep stomach acid production high via the bitter taste receptor on the tongue. Activating the bitter taste receptors stimulates the vagus nerve, which goes on to stimulate salivation (digestive enzymes begin during proper mastication/chewing), and gastric secretions (stomach acid and digestive enzymes in the stomach).

This is absolutely CRUCIAL to optimal digestion of proteins, fibers, and fats.
This is one key reason why I highly recommend to stop being a pussy and to taste herbs like black seed oil directly on the tongue, capsules and softgels eliminating a good portion of the potential benefits you could be experiencing.

Speaking of which, you can find a phenomenal quality black seed oil here: 100percentnaturals.co/products/100-p…Image
Image
This is equally important in those who need to eat frequent, generally protein-rich meals throughout the day to stimulate and maintain muscle protein synthesis, i.e. bulking, such as bodybuilders and power lifters.

You really need to ensure you’re getting sufficient micronutrient and herbal bitter/vegetable intake. Especially important in the off season and even during prep for contests so you’re getting the most out of your food intake while avoiding potential bloating and related digestive issues that can negatively impact performance and aesthetics.
Read 4 tweets
Mg2+ Papi, the BowTied Mg2+ Profile picture
Dec 13
The problem I have with natural T boosters is they’re largely squeezing out additional adrenal androgen output, vs gonadal/testicular. Adrenal androgen production making up only about 3% of total testosterone and DHT production systemically. Shilajit being a prime example of this phenomena.

An exception to this obviously being micronutrients that directly influence antioxidant capacity or hypothalamic-pituitary-testicular axis in the gonads and natural aromatase inhibitors, which reduces total systemic estradiol and interrupts the negative feedback loop on gonadotrophin production (luteinizing hormone/LH and follicle stimulating hormone/FSH) via HPTA.

I’ve yet to see this having a sustained effect on the HPTA and resulting androgen production without using that acute energy and motivation to make serious dietary, lifestyle, and environmental changes.
Such as subcutaneous and visceral fat loss, which downregulates aromatase expression, thus having a more sustained effect on relieving that negative feedback loop on HPTA function.

Providing a surplus of micronutrients and sufficient macronutrients/calories that will enable the testicles to produce gonadotrophins without interference from lack of energy or excessive reactive oxygen species production (ROS/oxidative stress).

Or seriously altering indoor light environment and sunlight exposure practices to provide the proper stimulus to HPTA via the retinohypothalamic tract and removing the negative stimulus that interferes with this and produces excessive ROS, that is artificial blue light and nnEMF/RF.
Any so-called “testosterone boosters” will only have an acute effect on androgen hormones at best, and more generally will have a greater effect on catecholamine or adrenal steroid production.
Read 5 tweets
Mg2+ Papi, the BowTied Mg2+ Profile picture
Nov 16
If you ignored my yearly warnings to START NOW on midday UV sunlight exposure and last winter ended up turning out rough for you, you need to read this so you don’t have to repeat it again.

You should only ever resort to supplements acutely to restore levels if deficient and below 30ng/mL on blood work, otherwise even if you just buy the Phillips bulbs and a basic set up instead of Sperti or a Chroma (safety and protection of the bulb is a highly concerning matter), you should be using UV light and I’ll tell you exactly why.
Cholecalciferol (vitamin D3 and what you think of when you hear “vitamin D”) is only ONE vitamin D complex metabolite of literally dozens.

There are dozens of different metabolites, both active and used as storage, of vitamin D that are highly important factors for vitamin D receptor (VDR) binding, organ receptor affinity and specificity, storage capacity, interchangeability throughout different contexts (i.e. low external UV, external temp dropping, healthy people demonstrating their own UV spectrum emission internally to influence the metabolites produced from dietary intake via cold exposure, etc.), and antiviral effects.Image
UVB exposure to the 7-dehydrocholesterol content of the skin forms pre-vitamin D3, which is unstable and undergoes thermal isomerization to vitamin D3.

Supplements won’t generate lumisterol and tachysterol from further UVB exposure to the pre-vitamin D3 formed in the skin.

In the presence of ROS, photosensitizing agents, and longer UV exposure, we also get 5,6-trans-vitamin D3, suprasterols, isotachysterols, and cholesta-5,7,9(11)-triene.
Read 6 tweets
Mg2+ Papi, the BowTied Mg2+ Profile picture
Nov 6
Try 97%. Only 3% of diseases are ever truly “genetic”.
x.com/grimhood/statu… x.com/grimhood/statu…
x.com/oliviercantin/… x.com/oliviercantin/…
Read 11 tweets
Mg2+ Papi, the BowTied Mg2+ Profile picture
Jul 26
Omega-3 fatty acids also generate endocannabinoids, yet aren’t associated with obesity. Quite the opposite in fact.

Cannabis usage is associated with lower BMI than non-users.
I’ve seen the theory that this association with cannabis usage and lower BMI is caused by a downregulation of CB1 receptors via chronic intake, but the same downregulation would occur with linoleic acid that gets stored in subcutaneous fat as they’re released and metabolized: a chronic supraphysiological activation of CB1R by endocannabinoids.
That’s not to mention the numerous other herbs and spices with agonistic activity at CB1R that are associated with lower BMI and reduced risk of obesity as well.

Lots of holes in this theory, back to the drawing board.
Read 4 tweets
Mg2+ Papi, the BowTied Mg2+ Profile picture
Jun 16
Start with 1-4 weeks of just sunrise and sunset exposure at 5-15 minutes if the ectoderm and neuroectoderm are that severely atrophied.

Then introduce 5 min midday sun exposure, and gradually build from there.
Including O3 PUFA (DHA specifically), vitamin A, copper, magnesium, riboflavin, zinc, and taurine as well.
I’ve worked with people who couldn’t even spend 5 minutes outside during midday sun without burning severely.

Mitigating your indoor light environment is also crucial to this restoration process.
Read 4 tweets

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