Skyler Lentz Profile picture
Apr 1 8 tweets 2 min read
Important lessons 📜 from ICU case:

🚨Not everything is septic shock
🚨A limited echo has limitations

Older patient in shock on 3 vasopressors, who initially presented with dyspnea and a CXR like 👇

What the diagnosis?

#FOAMed #FOAMcc

Image credit: ahajournals.org/doi/10.1161/CI…
Presented to the ED with acute onset dyspnea, new O2 requirement 6 L NC, minimal PMH

BP 95/70, HR ~ 100, afebrile

Hypoxemia worsened during ED stay requiring intubation

Now hypotensive

CT obtained with unilateral right sided pulm infiltrates, small pleural effusions, no PE
Labs 🧪

WBC 20k

Troponin mild elevation

Lactic acid 5.0

Cr mild elevation

U/A with > 50 WBC

Antibiotics, 2 L IVF given and admitted to ICU
🛌Arrival to ICU

B: 50% FiO2 PEEP 10

C: BP 80/60 HR 120s on
Norepi 40 mcg/min
Vasopressin .03 u/min
Epi 5 mcg/min

Distal extremities cool, oliguric, systolic heart murmur heard over vent (old per notes)
Middle of night POCUS 🫀

RV mildly dilated but systolic function looks ok
IVC 2.5 cm minimal variation
LVEF is normal
AS suspected on visual assessment

Comprehensive echo ordered for better valve assessment in AM
Must be pneumonia and sepsis, right?

Time, antibiotics, source control...
Echo next morning gives the diagnosis:

Severe AS
+
Acute chordae tendineae rupture with severe MR
📜Lessons:

✔️Not everything is septic shock
✅Normal LV/RV function does NOT rule cardiogenic cause of shock-low cardiac output from MR+AS despite normal EF in this case
☑️Severe MR can cause unilateral pulmonary edema
✔️Narrow pulse pressure (BP 80/60) hints at cardiac cause

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More from @SkylerLentz

Jun 18, 2021
New heart failure presentation and a BP like this 114/33?

👇

😯🚨

#FOAMed #FOAMcc Image
We often see a large pulse pressure in distributive shock (sepsis) from a low SVR (vasodilation)

Not everything is sepsis, don't be fooled! 🧐

When seen with an acute heart failure presentation 🫀🫁 it may be acute aortic valve regurgitation

Endocarditis is a common cause
Diastolic back flow into the LV through an incompetent aortic valve = low diastolic BP & elevated pulse pressure

Regurgitation can lead to pulmonary edema (LVEDP ⬆️, Left atrial pressure ⬆️ = pulmonary edema)

And poor forward cardiac output may cause hypoperfusion (Shock)
Read 5 tweets
Apr 18, 2020
Can you be in shock and HYPERtensive?

I think so. Our patients teach us many lessons

#FOAMed
#FOAMcc
@UVMEmergencyMed

👇
A patient presents with 7 days of dyspnea, LE edema and fatigue. They have run out of all meds 2 weeks ago #COVID. They had an MI with ishcemic cardiomyopathy EF ~30%, also has a-fib.
Exam:
Afebrile, BP consistently >160/110, HR 110 afib, RR 30, POx 86%-> 94% 4LNC

Mild confusion
JVD+
Mild resp distress, crackles BL
No murmur
Hands, legs COLD and mottled to knees, toes and fingers purple, cap refill delayed
Pitting LE edema
Read 8 tweets
Apr 16, 2020
Massive variceal bleed and no EGD at your hospital?

Be ready to place a blakemore/balloon tamponade device in uncontrolled variceal hemorrhage.

Tips from prior cases

#foamcc
#foammed
After ABC's...

1. Place the device like an OG

2. Check position by xray (may need little air ~50 ml in balloon to see)

3. Inflate gastric balloon with ~ 250 mL air

4. X ray

Don't over inflate

👇

Image: overinflated and improperly positioned causing gastric distension 😯
Deflated & repositioned w/ balloon at the GE junction to tamponade the variceal origins

Apply light traction w/ kerlix->IV pull->1 L fluid
Read 4 tweets
Jul 22, 2019
Tips on the mechanically ventilated ARDS and sick hypoxemic patient.

Protect the lungs from the start! See the basics below.

#Tweetorial #foamcc #foamed

Based on prior work with @mattroginski @roo_atchinson

@UVM_EM

1/
What is the definition of ARDS?

The Berlin criteria: Acute onset within one-week, bilateral opacities on CXR not explained by cardiogenic pulmonary edema, pleural effusion etc. and a PaO2/FiO2 ratio of <300 mm Hg with PEEP 5 cm H2O.

jamanetwork.com/journals/jama/…

2/
More simply in the ED or acute setting I consider anyone with bilateral infiltrates + inflammation (sepsis, pneumonia, trauma etc) + hypoxemia to be at risk for ARDS and if intubated manage them with lung protective ventilation.
3/
Read 16 tweets
Jul 10, 2019
You intubated the asthmatic! What to look for on the vent and what to do about it.

It gets complicated, but the basics are in the thread below

#foamcc #foamed #tweetorial mini
1/
The problem is bronchospasm and secretions narrow the airways and lead to obstruction, limitations in exhalation and high airway resistance.

On the vent, this is seen as a high peak pressure (high resistance) and a prolonged expiratory flow or incomplete exhalation.
2/
The high peak pressure isn’t really a problem unless the plateau (obtained by an end insp hold) is also high. The delicate alveoli only feel the plateau pressure. Best to keep the plateau pressure < 30 cm H20 by minimizing auto-PEEP as the auto-PEEP contributes to plat press
3/
Read 11 tweets

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