This concerted disinformation campaign has been given life once again by some virologists who fear a prospective ban on research & are more concerned about their own funding than on the safety and welfare of humanity.
"Experts at the WHO, however, are reluctant to take the new research as the final word on the matter. the WHO’s emergencies director Michael Ryan.. said that “all hypotheses remain on the table. This is a scientific detective story that goes on."
As published, the paper is now a damp squib. Gone is all the certainty of the preprint. Where the preprint claimed ‘dispositive evidence for the emergence of Sars-CoV-2’, the paper now cites ‘insufficient evidence to define upstream events’
Ground Control to Major Tom
Take your protein pills and put your helmet on
Ashes to ashes, funk to funky
We know Major Tom's a junkie
My mother said, to get things done
You'd better not mess with Major Tom
2. Tutorial on how to fight & win the next space war ;)
The demented logic of Daszak & Holmes in all its glory
They missed this:
1. Dr. Eloit and colleagues decided against going as far as 11 transfers, stopping at 6:
“we wanted to do more than 6 passages..but we did not want to open the risk to adapting a bat virus to humans”
2. They also missed this:
Dr. Peacock was reluctant to draw strong conclusions from such small-scale experiments. “I think it’s quite a difficult thing to ask to get a furin site after a few passages”
Weird take. Even the researchers publishing this study suggest this makes an intermediate host less likely. So we probably need to look at places in Wuhan where non-Wuhan bats could have infected a human. Any ideas?
"From an evolutionary perspective, it is still unknown if the sialic acid-binding features for SARS-CoV-2 spike are specific to SARS-CoV-2 or cov adaptation to humans or if this spike property is also retained by other sarbecoviruses"
"it is also interesting that ORF10 and ORF8 are the only two coded proteins present in SARS-CoV-2 which do not have a homologue in SARS-CoV-1, perhaps suggesting a unique amyloid etiology for COVID-19."