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@1goodtern

Mar 22

https://twitter.com/TeamPostX/status/2035592120887607735

Five things about this study.

First, even mild Covid infection screws your immune system so you're 60% more likely to be hospitalised by EBV/mono/glandular fever for and the effect lasts ages.

Covid infection can screw up your immune system.

https://twitter.com/TeamPostX/status/2035592120887607735

Second, we're talking about *hospitalisation* by EBV after the covid infection, so it's not just getting extra 'mild' bugs afterwards.

Third, the hospital data doesn't say whether the hospitalisations were *reactivations* or *new infections* of EBV.

But it's probably a bit of both.

I've been hospitalised by reactivated EBV. It was unpleasant.

Read 16 tweets

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@1goodtern

Mar 21

I've seen this gotcha quite a few times now:
"If the Kent meningitis outbreak was caused by Covid, why is it just in Kent?"
Which completely misses the point of what people mean when they say that outbreaks like this are made more likely by the damage caused by covid infections.

Wildfires aren't a perfect analogy for infection outbreaks - but they can help us understand certain aspects.

Think of a whole country made more prone to wildfires by a drought.

The whole country doesn't suddenly burst into flames.

In a drought ridden country, what happens it that you get wildfires happening locally sporadically.

Read 59 tweets

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@1goodtern

Mar 20

Enormously massively huge studies have shown that each wave of Covid infections causes damage to people's immune systems. The science is incontrovertible.

And yet you will not find a single media article about the current meningitis outbreak that mentions that.

It's really simple.
It's been established science for decades that "a low CD4 count... has been shown to be associated with an increased risk of Invasive Meningococcal Disease"
Governments base policies on this established science.
pmc.ncbi.nlm.nih.gov/articles/PMC10…

And Covid infection... Covid infection hammers your lymphocytes including CD4 T cells... and the rest.

Read 11 tweets

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@1goodtern

Mar 20

Don't you get it?

If lots of people in your population have lower ability to fight infection, it doesn't just mean those people are more likely to *catch* infections.

It means they are more likely to *spread* them too.

Let me explain.
This is important.

Jack has a metal lunchbox.
No ants can get into his lunchbox, so when he leaves the park, no ants fall out of his lunchbox.

Annie has a lunch bag made of wool.
Ants can climb into it, and they can also fall out easily too.
And that's what they do.

Obviously it's more complex than that. But if you don't catch something in the first place then you don't spread it.

But there's much more to it than that.

Read 24 tweets

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@1goodtern

Mar 19

Ten things they'll be telling us about meningitis before the end of the week:

1
It's mild

2
Kids can't get it.

Read 56 tweets

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@1goodtern

Mar 19

I'm sitting at my computer with 46 tabs open with media stories about the meningitis outbreak from the last 3 days.

Following mainstream coverage, govt statements and UKHSA briefings on the meningitis outbreak has been surprisingly tiring.

Here are a few of the inconsistencies:

"Outbreak has been contained." Then within 16 hours: "It is too soon to say the outbreak is contained."

UKHSA said this looked like a "single event cluster" linked to a nightclub. Then they started saying there was likely "ongoing spread" in university halls. A contained exposure event and active transmission through accommodation networks are not the same thing, Susan.

Read 21 tweets

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