Just imagine for a moment that you are infected with a virus that harms the lining of your arteries. The virus doesn't damage the artery walls in every part of your body to exactly the same degree. Some parts will be more damaged, some less.
At your next infection, will the parts of your artery wall that were more damaged first time round be more or less vulnerable to the virus?
It's an interesting question, isn't it.
Will those damaged parts be better equipped the next time round?
Sadly the answer is no
Those harder-hit patches do not reset to factory condition once the acute infection passes.
The minimisers would have you believe that every bad health condition develops immediately, symptoms of it appear immediately, medical attention is found immediately, and the condition is diagnosed immediately.
The truth is very different.
It can take years for conditions to develop after they have been triggered.
It can take years for the symptoms to become bad enough to need medical attention.
I think we've let the damage that covid infections do to *linings* slip into the background of all the other problems that covid infections cause.
I think this may be a *big* problem.
Across all of these, the pattern is the same: structural cell loss is followed by repair that restores structural continuity but not precision of purpose.
The tissue remains present, but its behaviour changes.
Function becomes uneven.
Symptoms emerge from loss of fine control