Thread by @NutriDetect

"vitamin" A Deficiency Doesn't Exist: What I Learned By Examining Human Studies

It is claimed that "vit" A is *essential* & required thru diet

That said, if even *1* person can be healthy w/o any of it, then it's NOT essential

Let us explore this topic IN DETAIL

/ thread /

What I'll be covering today:
- Types of studies we prefer
- Supposed signs & symptoms of "vit" A deficiency
- Animal & human studies debunking "vit" A deficiency
- Nutrient deficiencies that cause the EXACT SAME symptoms
- Current research on low "vit" A diets in animals & humans

How I prioritize the value of "vit" A research studies:

Much *LESS* Valuable:
- In animals
- Short-term
- In vitro
- Mechanism
- Blood test only

Much *MORE* Valuable:
- In humans
- Long-term
- In vivo
- “Signs & symptoms”, present OR lacking
- Actual human function observations

What are classic signs & symptoms of "vit" A deficiency?

Ocular (eyes):
- Poor dark adaptation, aka “night blindness”
- Dry eye disease aka xerophthalmia, can progress to keratomalacia
- Macro- & microscopic changes in eye

But ⬆️ carrots actually ⬇️ night vision, see thread

https://twitter.com/1318332497160138752/status/1593346155626332160

Continued

Dermatological (skin):
- Macro- & microscopic changes in skin aka skin diseases

Hematological (blood):
- Changes in white blood cell counts

With that out of the way, let's get to the studies

Animals first...the question here:

Is "vit" A ESSENTIAL or not?

From: PATHOGENESIS DUE TO VITAMIN DEFICIENCY IN THE RAT

"rats were fed on definite dietary planes which would produce normal growth except for the *lack of either vitamin A or B*. From these groups, animals were selected which were representative..."

drive.google.com/file/d/14FauCD…

"Care was taken to see that the animals were free from infection. Control tests were made on tissues from rats fed a complete diet
[...]
***In the rats which lacked vitamin A in their diet, there were no special outstanding pathological findings***..."

"In marked contrast w lack of vit B ***the livers showed no fatty changes, the adrenals no hypertrophy, & the thymus no atrophy***. The control animals proved to be normal as far as histological examination showed."

The answer so far is, "vit" A is NOT essential

Moving on

ALL retinol aka "animal vit A" derives from carotenoids

There is NO "vit" A anywhere w/o a carotenoid foundation

"Animal vit A" comes from animals eating carotenoids & detoxing them 😉 into retinoids, then you eat half-detoxed retinoids

Question: Are carotenoids essential?

"None of the carotenoids are considered essential nutrients. No carotenoid is directly involved in a vital metabolic pathway, nor has relative absence of a carotenoid been linked exclusively to induction of a specific deficiency or chronic disease”

Hmmm

academic.oup.com/advances/artic…

On to human studies

"23 'conscientious objectors', 20 men & 3 women, volunteered to live on a diet designed to be deficient in these substances but complete in every other respect...absence of vit A & its precursors was confirmed spectrophotometrically & biologically..."

"..disappearance of carotene from blood plasma of deprived volunteers confirmed absence of these substances from diet
...
W efficiency for dark adaptation & plasma values for vit A as criteria, none of the 16 deprived subjects became depleted w/in a year"

nature.com/articles/15601…

Paper: Vit A & epilepsy: a dietary contretemps

“In order to investigate further a possible connection bw vit A & epilepsy, Sharman then studied 8 epileptic patients given a vit A-depleted diet for 2 yrs.

NO cases of night blindness were observed..."

It gets better

"***patients reported fewer [epileptic] fits during the depletion period***
...
The author called his experiment a “dietary contretemps.””

What does contretemps mean, you wonder?

👇

Contretemps definition: an inopportune or ***embarrassing occurrence*** or situation; an ***unforeseen event that disrupts the normal course of things***

Now understand the author admitted his BIAS

A proper low "vit" A diet ⬇️ seizures, I've seen it

pubmed.ncbi.nlm.nih.gov/4795151/

Next study

“The great majority of clinical examinations revealed ***no significant differences*** between the deprived and a non-deprived group, or in the same person before & after deprivation of vit A..."

"...In several of the subjects follicular hyperkeratosis was present at the start & varied during the experiment, but ***the variations bore no relation to the vit A intake***”

Something else caused the follicular hyperkeratosis then, eh?

jamanetwork.com/journals/jamai…

Now, what if there were HUGE populations that were diagnosably "vitamin A deficient" (VAD) by blood test...

Yet the deficiency symptoms never showed up en masse?

Enter Exhibit A:
Vitamin A deficiency in Latin America and the Caribbean: an overview (1998)

👇

“Vit A deficiency (VAD) has been known to exist in Latin America & the Caribbean since the mid-1960s
...
there was little interest in controlling it because of the low frequency of clinical findings”

Where's all the PROBLEMS that should be going on?

"...VAD was NOT considered a problem of public health significance because ocular [eye] lesions leading to permanent blindness were NOT frequently observed despite prevalences of low serum retinol ["vit" A] hovering near the cutoff of 15% then recommended by WHO..."

"...On the other hand, ocular [EYE] signs attributable to clinical VAD were EXTREMELY RARE in the 1980s & they have not been assessed more recently..."

OTOH, ~10% of Guatemalans were ***gold standard liver-biopsy "vit" A toxic*** in mid-1970s, see below

pdfs.semanticscholar.org/df83/af00b6811…

https://twitter.com/NutriDetect/status/1594364705254301697

Another study:

"68% of subjects in lower socioeconomic group had serum retinol levels bw 10-19 ug%, & 4% below 10 ug%, but NONE of them showed any symptoms of retinol deficiency"

Below 10 ug% is considered SEVERE deficiency...yet NO problems?

Weird 😉

ncbi.nlm.nih.gov/pubmed/8316814

This one closes the case

“...the mean liver vit A stores in ***apparently healthy infants*** is lower, ranging from ***0*** to 320 μg/g of liver.”

ZERO "vit" A in the liver of HEALTHY INFANTS

Apparently NOT essential!!!

Let that sink into your noggin

nap.edu/read/10026

This is a BIG study in JAMA (of all places) that blows "vit" A out of the water:

“5 young adults, 3 women & 2 men, were placed on a vit A-low diet for 7 1/2 & 4 1/2 months respectively. At suitable intervals measurements were made of dark adaptation, blood vit A & carotene,..."

"..total white blood cell counts & differential blood counts & changes in structure of skin & conjunctivas. NONE of these measurements showed any definite changes from those found prior to deficiency for length of time studied”

NO negative changes

Ouch

jamanetwork.com/journals/jamai…

I say that the problems attributed to "vit" A deficiency are actually caused by deficiencies of OTHER THINGS

What if there was a study to demonstrate this?

What if the SAME disease condition showed up w deficient "vit" A...

& w EXCESSIVE "vit" A???

That would be weird...😉

From:
THE PATHOLOGICAL ANATOMY OF OPHTHALMIA PRODUCED BY DIETS CONTAINING FAT-SOLUBLE A, BUT UNFAVORABLE CONTENTS OF CERTAIN INORGANIC ELEMENTS

Ophthalmia = eye inflammation

Fat-soluble A = "vit" A

Unfavorable = could be excess OR deficient

Inorganic elements = minerals

👇

"...diets containing an ABUNDANT supply of fat-soluble A may produce an ophthalmia in rats
...
The disease is very similar clinically to that which develops in animals which are fed diets DEFICIENT in fat-soluble A
...
Their diets contained different salt [mineral] mixtures"

🤯

"Xerophthalmia [dry eye] & keratomalacia [untreated progression of xerophthalmia] are produced in rats by feeding them diets which are DEFICIENT in fat-soluble A..."

Keep going...

"The same changes may result from consumption of diets which have (caeteris paribus) [“all other things being equal”] an ABUNDANT supply of fat-soluble A, but which contain certain UNFAVORABLE salt [MINERAL] mixtures..."

Some mineral(s) must be pretty darn important, right?!?!

"...The 2 conditions are clinically & pathologically IDENTICAL & are identical w human xerophthalmia & keratomalacia
...
It would seem...that...factors OTHER THAN fat-soluble A may have to be considered in regard to etiology & treatment of the condition”

How'd they fix it?

👇

“1 rat, in which keratomalacia had been produced, was placed on a diet containing a GOOD salt [MINERALS] mixture, after both corneas had been perforated by the ulcers. The activity of the pathological process was checked [STOPPED] at once clinically, as a result of the change..."

"...Photophobia disappeared, the discharge diminished, & the conjunctiva became moistened”

Therefore, "vit" A DEFICIENCY has NOTHING to do w xerophthalmia (dry eye) or keratomalacia (dryness w ulceration & perforation of cornea)

It's a MINERAL problem

babel.hathitrust.org/cgi/pt?id=uiug…

Here's the defining characteristic of where supposed "vit" A deficiency symptoms shows up

It's always in MALNOURISHED people (Africa is common)

FYI, gold standard liver biopsy "vit" A TOXICITY has been found in multiple African countries since 1960s

Ghana, Zambia, S Africa 👇

https://twitter.com/1318332497160138752/status/1594364556050313216

FDA says:
"Vit A deficiency is rare in the US because most people get enough vit A from foods they eat"
ods.od.nih.gov/factsheets/vit…

They changed labeling requirements:
"FDA does not require food labels to list vit A content unless vit A has been added"
ods.od.nih.gov/factsheets/Vit…

Question: If "vit" A deficiency is RARE in US...why are we FORTIFYING FOODS?!?!

On the other hand, what happens in well-nourished, *muscle-meat-consuming* peoples (this is KEY), as they consume more "vit" A?

See quote below

PUT IT TOGETHER

Are we being poisoned?

https://twitter.com/1318332497160138752/status/1590078725068296193

What is the main FOOD difference bw WELL-NOURISHED & MALNOURISHED people?

I say it's MUSCLE MEAT

Seems obvious

Key factors in MEAT we're going to connect:
- Protein
- Zinc
- Taurine

Are they involved w supposed "vit" A deficiency?

Do we need these to PROTECT us from "vit" A?

There is *retinol binding protein* (RBP) that is necessary to protect us from "vit" A toxicity

"Hypervitaminosis A occurs when amount of plasma retinol exceeds availability of RBP to bind it..."

⬇️ RBP = ⬆️ "vit" A toxicity

sciencedirect.com/science/articl…

"...leading to unbound & elevated levels of free retinol in plasma, & formation of excess retinoid metabolites"

⬆️ free "vit" A = ⬆️ TOXICITY

Are RBP levels related to protein deficiency &/or zinc deficiency?

You already know it's coming

We will first establish that blood "vit" A = blood RBP

"The use of RBP results in the classification of essentially the same children with VAD ["vit" A deficiency] as does [serum] retinol, & ***RBP is an excellent surrogate for serum retinol***"

pubmed.ncbi.nlm.nih.gov/11237937/

Next, we establish that BLOOD "vit" A does NOT equal LIVER "vit" A:

“SERUM ["blood"] retinol concentrations do NOT reflect HEPATIC [liver] vit A conc's over a wide range of liver values...Thus SERUM retinol concentrations vary LITTLE despite MAJOR alterations in vit A INTAKE.."

"Case reports of hypervitaminosis A [diagnosed by HISTORY & SYMPTOMS] often show SERUM retinol conc's w/in NORMAL limits, indicating that SERUM retinol is NOT a VALID measure of vitamin A STATUS during TOXICITY”

You can have a TOXIC LIVER w NORMAL BLOOD

hindawi.com/journals/isrn/…

So...
- retinol = RBP
- blood "vit" A or RBP ≠ liver "vit" A

We're about to connect
- protein deficiency (def) to supposed "vit" A def symptoms
- protein def to ⬇️ RBP
&
- zinc def to supposed "vit" A def symptoms
- zinc def to ⬇️ RBP

Then we'll do similar things w taurine

Let's quickly review some supposed classic "vit" A deficiency symptoms, in the order we'll debunk them:
- Dry eye disease aka xerophthalmia
- Poor dark adaptation, aka “night blindness”
- Skin diseases

PROTEIN DEFICIENCY & XEROPHTHALMIA (Dry Eye)

Study titles from the search linked below:
- Xerophthalmia & protein malnutrition
- Xerophthalmia & protein malnutrition in Bantu children
- Xerophthalmia in protein calorie malnutrition

They're connected 😉

pubmed.ncbi.nlm.nih.gov/?term=xerophth…

PROTEIN DEFICIENCY & NIGHT BLINDNESS

"Our results showed that night blindness was associated w protein energy malnutrition when using the mid-upper arm circumference (MUAC) as a measure of nutritional status"

Connected

ncbi.nlm.nih.gov/pubmed/8985529

PROTEIN & RETINOL-BINDING PROTEIN (RBP)

"Like retinol, RBP may not accurately identify true vit A status under all conditions, bc acute phase response & ***protein malnutrition depress RBP concentrations***"

⬇️ dietary protein = ⬇️ RBP

academic.oup.com/ajcn/article/7…

I just showed how lack of dietary PROTEIN causes a lack of RBP...

& MEAT is the best source of PROTEIN (not gonna argue this)...

& lack of RBP leads to worse "vit" A toxicity...

MEAT is also the best source of ZINC...

Is ZINC deficiency connected to RBP deficiency too?

😉 👇

ZINC & RETINOL-BINDING PROTEIN (RBP) #1

"zinc deficiency interferes with the synthesis of RBP by the liver & probably w the synthesization of some other plasma proteins as well"

Connected

semanticscholar.org/paper/The-effe…

ZINC & RETINOL-BINDING PROTEIN (RBP) #2

"experimental group [of rats] received a zinc-deficient liquid diet...Hepatic [liver] cellular RBP was ⬇️ by >50% in the experimental group as compared to each of 3 control groups"

Ditto

pubmed.ncbi.nlm.nih.gov/1517037/

ZINC & NIGHT BLINDNESS

“Symptoms resulting from zinc deficiency are as diverse as the enzymes w which the metal is assoc. If chronic, severe, & untreated, zinc deficiency can be fatal. **Less drastic symptoms include...night blindness**"

Seems important

pubmed.ncbi.nlm.nih.gov/3514057/

ZINC & SKIN PROBLEMS

Skin issues connected to zinc deficiency:
- Acne
- Hair loss
- Atopic dermatitis
- Dandruff
- Delayed healing
- Dermatitis on hands, feet, face, &/or anogenital region
- Diaper rash

In practice, if I see skin issues, I think "vit" A toxicity & Zn deficiency

We've now connected a dietary zinc deficiency to both:
- lack of protective RBP
- symptoms of supposed "vit" A deficiency

Supposed "vit" A deficiency in people ALWAYS shows up in MALNOURISHED people (can include VEGANS d/t lack of MEAT)

Can PROTEIN & ZINC ⬆️ "vit" A levels?

This is an extremely important question

"sCiEnCe" diagnoses "vit" A deficiency SOLELY based on blood "vit" A or sometimes blood RBP levels

But...the LIVER can be full of "vit" A while the BLOOD is low

If PROTEIN or ZINC can ⬆️ blood "vit" A levels, where did it come from?

😉

FIXING PROTEIN DEFICIENCY ⬆️ "vit" A LEVELS #1

Remember, protein does NOT implicitly contain "vit" A, so pay attention

"Plasma vit A, RBP, & prealbumin conc's have been studied in 38 northern Thai children with protein-calorie malnutrition (PCM)..."

"The 4-week study period consisted of 1 week of stabilization followed by 3 wks of treatment w formula diets varying in their protein & calorie content. The stabilization period comprised 7 days of initial treatment w fluids, antibiotics, and..."

"...a gradually ⬆️ intake of PROTEIN & calories to a final level of 1 g protein & 100 kcal per kg of body weight. During this period vit A, RBP & prealbumin levels all showed SIGNIFICANT INCREASES compared to admission values..."

But...let's confirm this

pubmed.ncbi.nlm.nih.gov/807094/

FIXING PROTEIN DEFICIENCY ⬆️ "vit" A LEVELS #2

"Plasma RBP is ⬇️ to...31.1% of normal, in acute stage of kwashiorkor [PCM]

RBP DOUBLED its plasma level after 1 wk & TRIPLED after 2 wk of appropriate refeeding"

Remember, retinol = RBP

pubmed.ncbi.nlm.nih.gov/805336/

FIXING PROTEIN DEFICIENCY ⬆️ "vit" A LEVELS #3

This will blow your mind & nail this coffin shut

"A 62-yr-oId white male presented w edema, PROTEIN MALNUTRITION, & abnormal liver function. He had ingested from 40,000-50,000 IU of vit A per day for 7 yr..."

"Examination of liver tissue by light & electron microscopy revealed findings characteristic of an EXCESSIVE ACCUMULATION of vit A. Liver tissue contained MASSIVE amounts of vit A (19,700 IU/g); however, both the serum conc of vit A & RBP were BELOW NORMAL [aka *deficient*]..."

"& serum RBP was not saturated. On a NORMAL DIET the patient was able to mobilize hepatic [liver] vit A, as indicated 71 days later by a repeat biopsy of the liver, which then contained 9000 IU of vit A/g..."

"During this time his NUTRITION IMPROVED, serum vit A ⬆️, & serum RBP became transiently saturated"

To clarify, this sick man:
- Was taking 40-50K IU of "vit" A/day
- Protein-deficient diet
- Serum retinol initally was 18 ug/dL, aka "DEFICIENT"

gastrojournal.org/article/0016-5…

The doctors had this man:
- STOP taking "vit" A supps
- I assume he stopped high "vit" A foods (though this isn't mentioned)
- ⬆️ PROTEIN intake

By wk 10, serum retinol ⬆️ to *62 ug/dL* [reference ranges below]

Liver "vit" A went ⬇️ from 19,700 ➡️ 9,000 IU/g

Protein = detox

What is this telling us?

Simplified...low protein diets HIDE "vit" A toxicity in the liver

The liver WILL NOT release its toxic stored "vit" A unless there is enough RBP present, which requires dietary protein

Does zinc do the same thing?

Yes. Yes, it does

I'm going to interject here with a personal client anecdote

I've seen a person on a low "vit" A diet working w me...

With an initial serum retinol test of 25 ug/dL (barely above "deficient")

W/in 6 months

Go up to 125 ug/dL (2x over blood "toxicity" threshold)

This is real

"plasma vit A began to ⬆️ significantly following zinc treatment. By 5th day, plasma vit A conc ⬆️ to w/in normal range...ADEQUATE FOOD INTAKE as well as ZINC appears necessary to totally reverse low plasma vit A conc's in zinc deficiency"

Done & dusted

pubmed.ncbi.nlm.nih.gov/1255272/

Review:
- PROTEIN & ZINC deficiency ⬇️ "vit" A & RBP in BLOOD
- "vit" A is getting stuck in LIVER
- as Zn deficiency corrected, "vit" A in BLOOD ⬆️
- d/t "vit" A being released aka detoxed from LIVER

PROTEIN & ZINC help protect us (RBP) & detox us from "vit" A!

Now to TAURINE

Taurine is a sulfonic amino acid that is ONLY found in ANIMAL FOODS

Red meats are BY FAR the highest in taurine content

Guess what type of protein is used in mouse/rat studies to induce supposed "vit" A deficiency?

Dairy protein! ➡️ ALMOST ZERO TAURINE

Coincidence?

😬😬😬

TAURINE & NIGHT VISION

"It is now firmly established that a ⬇️ of taurine retinal [ie. retina of eye] levels beyond a critical point results in a retinal degeneration characterized by an *alteration in structural & functional integrity of photoreceptors* & other retinal cells"

Why is this important?

Photoreceptors are your specialized nerve cells able to detect & react to LIGHT

This shows that taurine deficiency ➡️ LIGHT receptor STRUCTURAL & FUNCTIONAL degeneration ➡️ night vision

Taurine deficiency = night blindness

google.com/books/edition/…

TAURINE & DRY EYE (XEROPHTHALMIA)

"Dry eye was induced by atropine sulfate & topical treatment was carried out w...0.4% sodium hyaluronate + 0.5% taurine
...
Taurine...prevents ocular surface [dry eye] damage"

Taurine prevents dry eye

pubmed.ncbi.nlm.nih.gov/28771380/

TAURINE & SKIN ISSUES

"Taurine...occurs naturally in food, especially in...meat. Taurine is used in topical *anti-acne* therapy. It was used to explore *anti-tumor* promoting activity in a skin carcinogenesis. It is used in *skin ulcers*

researchgate.net/publication/30…

"...it exerts an *antiwrinkle* effect on the skin. Taurine is effective in treating the underlying causes of *psoriasis*. It is also used in *atopic eczema*"

Those sure are a lot of skin connections!

See thread below for the "cause" of psoriasis..."vit" A toxicity in the skin

https://twitter.com/NutriDetect/status/1590783958731849729

TAURINE PROTECTS CELLS AGAINST "vit" A TOXICITY

"The presence of TAURINE (5-20 mM) & ZINC (50-100 microM) protected cells from RETINOL-induced injury"

Weird how zinc showed up here too, eh?

pubmed.ncbi.nlm.nih.gov/6502269/

TAURINE PROTECTS RATS AGAINST "vit" A TOXICITY

"symptoms of vit A toxicity in rats included...hepatotoxicity [liver damage] & nephrotoxicity [kidney damage]
...
these toxic effects of vit A were significantly ⬇️ when rats were fed the diet w...taurine"

sciencedirect.com/science/articl…

Doubters scream..."B..B..BUT VIT A *DOES STUFF*!!!

Short-term:
Caffeine ➡️ energy...NOT essential
Viagra ➡️ erections...NOT essential
Metformin ➡️ blood sugar...NOT essential
Statins ➡️ cholesterol...NOT essential

Pharma poisons *DO STUFF*...this does NOT make them ESSENTIAL!

Is there real-world evidence of near-zero blood "vit" A & ppl doing fine?

Yes

Grant Genereux of ggenereux.blog
- 8 yrs low "vit" A diet
- Most recent serum retinol = 0.1 umol/L (Range 1.04-2.77)
- CURED chronic kidney disease, jaundice, & eczema

He's doing well 😉

Dino did 2.5 years on a minimal "vit" A diet:

His serum retinol: 0.04 umol/L (Range 1.04-2.77)

"As far as I am concerned, I now am convinced that vitamin A is not a vitamin at all, otherwise I would have developed some problems"

ggenereux.blog/discussion/top…

Let's talk animals

The classic 1925 study of Wolbach & Howe that "proved vit A is an essential nutrient” had major diet problems

Casein protein contains NO TAURINE

Their mineral mixture contained NO ZINC

Most of their rat subjects DIED w/in 9-11 wks

Here comes the best part

Rat's diet contained casein, lard, & yeast

Casein contains "vit" A:
ncbi.nlm.nih.gov/pubmed/23265510
ncbi.nlm.nih.gov/pubmed/1897408

Lard contains "vit" A:
pubmed.ncbi.nlm.nih.gov/13203849/
pubmed.ncbi.nlm.nih.gov/13186866/

Yeast ⬆️ "vit" A toxicity:
nutritionrestored.com/blog-forum/top…

It was NOT a "vit" A free diet at all!

What happens when animals are put on a truly "vit" A free diet that has PROPER & ADEQUATE nutrition?

Grant Genereux kept a gerbil in perfect health on a "vit" A-free diet for 33 wks

An Australian man told Grant he kept mice on a "vit" A-free diet for 52 wks

We need new studies

Remember this?
“In the rats which lacked vit A in their diet, there were NO special outstanding pathological findings”

Someone, somewhere SCREWED UP BAD

We've been told "vit" A is essential

It's NOT

If it's TOXIC in high doses
Yet is NOT ESSENTIAL
Then it's a TOXIN

Why is this SO important?

Bc if studies looking at the SAME scientific question CANNOT be repeated w SIMILAR results, then this suggests an error, either by accident or ON PURPOSE, which creates huge doubts in reliability of research & its CONCLUSIONS

My take? We were duped

Here’s what is going on, re-using a previous quote here:
“It would seem...that in cases of xerophthalmia which occur in human beings other *factors other than fat-soluble A may have to be considered* in regard to the etiology & treatment of the condition"

We know the factors

Let us review what we've covered here today:
- "vit" A is not essential in animals
- "vit" A is not essential in humans
- A person can have LOW blood "vit" A w HIGH liver "vit" A
- Reworded--A person can look *blood DEFICIENT* while they're completely *liver TOXIC*

👇

- "vit" A in well-nourished ppl ⬇️ quality of life & hastens death
- "vit" A deficiency happens in malnourished ppl, aka lacking MEAT
- MEAT contains best sources of protein, zinc, & taurine
- LACK of protein, zinc, & taurine cause SAME diseases as supposed "vit" A deficiency

👇

- LACK of protein & zinc appears as ⬇️ serum retinol bc "vit" A is STUCK in liver
- LACK of protein & zinc results in ⬇️ RBP, which ⬆️ "vit" A toxicity
- Taurine protects against "vit" A toxicity
- There are MANY humans & animals doing EXTREMELY well on low "vit" A diets

👇

Now that you've read this beast of a thread (that I am quite proud of), here are the action items you can do:

- MINIMUM 50 grams of TOTAL protein/day
- Red MUSCLE meat is best, dark MUSCLE meat poultry 2nd best
- See thread below for BASICS on how to lower dietary "vit" A

https://twitter.com/NutriDetect/status/1509165907800514562

To conclude:
- "vit" A is NOT an ESSENTIAL nutrient
- supposed "vit" A deficiency & assoc diseases are caused by MUSCLE MEAT deficiency (protein, zinc, &/or taurine deficiencies)
- "vit" A toxicity is epidemic worldwide

*THEY* are not going to tell you this, ever

Q. E. D.

https://twitter.com/NutriDetect/status/1599523128518098944

If you believe you have health problems related to "vit" A toxicity & you want to know how you can get more help, see below

Work directly w me or @NathanWiens9 via Testing & Consultation packages:
nutritiondetective.com/collections/sc…

My DIY Love Your Liver program:
members.nutritiondetective.com

@NathanWiens9 That's a wrap!

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