- pronator drift (pyramidal drift) was the 1st to be described
- Dr. Barre was the 1st to report it
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Pronator drift (Barre’s sign)
progress from distal to proximal
1st downward arm drift
2nd forearm pronation
3rd flexion of the wrist and elbow
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Pronator drift – assessment
"patient extends both arms upright in the supinated position and hold them at shoulder height for at least 10 sec (patient should be asked to keep eyes open initially and later test again with eyes closed)"
via: daihocyduoc 4/
Response
“the examiner can simple wait for the response or hasten the process by tapping on the patient’s palms or having the patient turn the head back and forth, or both”
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Pronator drift development
The stronger muscles of the upper limbs are "pronators, biceps, and internal rotators of the shoulder"
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Clinical significance
a. can detect subtle upper motor neuron lesion which goes unrecognized by routine motor examination
b. included in initial examination of stroke
c. if only one motor test could be done in a patient – the best single test would be to examine the drift
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Mechanism
Why pronator drift occurs when eyes are closed?
Why pronator overcomes supinator in pyramidal lesion?
“downward drift without pronation of the paretic arm”
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Leg drift
“patient lies supine with the hips and knees flexed, the knees forming an angle of about 45 degrees”
-positive, heel will gradually slide downward, knee slowly extends, and the hip goes into extension, external rotation, and abduction
- no clear localization
17/
David Newman-Toker, neuro-otology
Senior author of "HINTS to diagnose stroke"
1/
What is dizziness?
-Dizziness→ impaired perception of spatial orientation without vertigo
-Vertigo→ illusion of motion (spinning/non-spinning)
>Subjective (person)→ MC peripheral
>Objective (environment)→ MC central
2/
-Oscillopsia→ 'world bounces'
>can’t read signs while walking
>B/L vestibular hypofunction
-Lightheadness, syncope→ LOC
-Imbalance→ severe truncal ataxia
3/
"Involuntary movements on one side that mirror voluntary actions on the other"
-Normal in <7–10yo (corpus callosum myelination), 70% healthy children (on speed-based task)
2/
Pathophysiology
Three teories
-Overflow
> signal 'overflows' to the other hand
-Weak interhemispheric inhibition
> both hemispheres fire together
-Abnormal crossing motor pathways
>same commands go to both hands
What EXACTLY happened before, during, and after the event?
-open-ended quest at begining, than close
-LOC, incotinence
-Witness and recurrence
*avoid term fainting
-Triggers (sleep dep, drugs, stand) vs premonitory symptoms