1. A lesson in quantum biology.........Note the linkage to the VDR and POMc (melanin). Everything links back to the absorption spectra of the biochemical that works on POMc. That target is 220 nm light. Where does that VUV light come from? @hubermanlab
2. People forget that melanin in our skin helps facilitates the collection of UVB light to convert cholesterol esters that will become 25 di hydroxy Vitamin D in your skin.
3. Most but not all actions of 1,25(OH)2D are mediated by the vitamin D receptor (VDR). You need to understand that the VDR is linked to free retinol in the system is means melanin biology in mammals is destroyed because of things cleaved in POMC.
4. With melanopsin destruction, you get free Vitamin A from the broken weak covalent bond, & that free Vitamin A destroys photoreceptors. VDR is one of the photoreceptors destroyed.
5. VDR is a transcription factor that partners with other transcription factors such as retinoid X receptor (Vitamin A) that when bound to 1,25(OH)2D regulates gene transcription either positively or negatively depending on other cofactors to which it binds or interacts.
6. ^^^^the tweet above is only true if your skin has enough melanin to regulate them. If the melanin is missing you have NO regulation. All melanin comes from POMC!
7. Without enough melanin in the integument, that reaction becomes inefficient. This is going on in medical clinic patients every day and none of the centralized clinicians are realizing it because they do not understand how POMC works in mammals.
8. You do not need to take magnesium supplementation for Vitamin D deficiency UNLESS you have such a UV deficiency that you get rickets. Rickets is a sign of a loss of ferroelectricity in the musculoskeletal system.
9. This PEER-reviewed paper now shows Vitamin D supplements still "work" even if you're not taking magnesium supplements. The key point in the paper: Vitamin D from sunlight is still the best source of Vitamin D, and supplemental pills are not.
10. Magnesium (Mg) is a cofactor for many of our endogenous WBG semiconductors to create endogenous VUV-IR-A.
11. Mg WBG is also critical for the critical efficiency of enzymes, via proton tunneling, and many of these enzymes are involved in the conversion of cholecalciferol into 1,25-(OH)2-vitamin D3 (active vitamin. pubmed.ncbi.nlm.nih.gov/29480918/
12. The production of 1,25(OH)2D in the kidney is tightly controlled, stimulated PTH, and inhibited by calcium, phosphate & FGF23. These change the VUV light emission in cells from WBG semicinductors
13. Extrarenal production of 1,25(OH)2D as in keratinocytes and macrophages is under different control, being stimulated primarily by cytokines such as tumor necrosis factor alfa TNFα and interferon-gamma =IFNg = POMC pieces fit
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What do you know about the Stiles-Crawford effect in a healthy eye? What if I told you this effect is how we sharpen central vision and narrow the periphery of the retina from too much blue light. Would you believe it? Did you know melanopsin has a specific topographic map on a healthy retina? A lesson no has taught you is incoming.
2. All opsins are topologic insulators. You might want to read that threadroll above now to understand topology well.
Topology changes in a cell = geometry change of cristae = UPE change from mitochondria = the optical signal changes in the same tissue altering physiology.
So what happens if you sustain mitochondrial damage in your retina's colony of mitochondria to the Stiles Crawford effect?
What are the implications?
3. The Stiles–Crawford effect (SCE) is the human eye's phenomenon of reduced light sensitivity when light enters the pupil from its periphery compared to the pupil's center. This effect is due to the optical properties of photoreceptors, which act as waveguides and are aligned to channel light towards the fovea, the central point of vision. The SCE makes vision less sensitive to light entering the periphery, thus reducing glare and improving visual clarity. It also keeps a lid on the amount of blue light the periphery the retina gets.
This sharpens vision, makes myopia, glaucoma, cataracts, hyperopia, and AMD almost impossible to get. Makes one resistant to mental illness too. Makes one impervious to diabetic transformations. Makes neurodegeneration rare.
Another new podcast from me with Smuggling Hope. It is good one on mental health because it explains how biomolecules absorption and emission spectra's determine reality or determine which mental illness one gets.
This is the information why Jordan Peterson is sick and why he and his daughter have stumbled multiple times in getting him well. ivoox.com/.../exposing-t…...
Biophotons are ultra-weak light emissions produced by living organisms, thought to arise from metabolic processes like oxidative reactions in mitochondria. Centralized scientists and AI bots hypothesized them to play a role in cellular communication, potentially influencing gene expression or signaling pathways. FIAF, also known as angiopoietin-like protein 4 (ANGPTL4), is a protein produced by tissues like fat and the gut, and it’s a key player in lipid metabolism and microbiome construction. Neither understand how UPEs control FIAF. If the UPE is coherent then FIAF inhibits lipoprotein lipase, affecting how fats are stored or broken down, and its expression ramps up during fasting. The microbiome, meanwhile, is the community of gut microbes that can shift in response to diet, fasting, or host metabolism, influencing energy balance and health. If the UPEs in mitochondria is not coherent, the UPE changes and mental illness becomes more probable. You cannot burn fat so it changes neural signaling.
Centralized scientists and technocrats who build AI systems will say no direct study says “biophotons control FIAF to affect the microbiome,” but we can hypothesize based on related mechanisms. This is patently false. Why?
FIAF has known absorption and emission spectra to light frequencies. Because of that, a study is superfluous because each chemical in the world has an optical fingerprint. Just because a scientist has not published the work is immaterial to this BASIC biophysical fact in spectroscopy. This was what I brought Ray Peat over ten years ago, and he was impotent enough to answer my critiques of his work. This podcast covers these details.
2. Is nerve pain caused by a Lyrica deficiency?
Is Lyme disease linked to a lack of doxycycline?
Is depression due to Prozac deficiency.
Are heart attacks are due to Lipitor deficiency.
Is obesity due to Ozempic deficiency.
Are Headaches due to Tylenol deficiency?
Is Bipolar disorder due to lithium deficiency?
Any questions?
You've been conditioned by centralized medicine to ask the wrong question.
You have a solar deficiency combined with a nnEMF toxicity problem.
This alters the UPEs your mitochondria make and it is this light that changes the neural tracks that make you mentally ill. This is why your Bipolar Disorder exists. The defect is not in you; it is in your environment.
3. Light exposure,say, sunlight or specific wavelengths impacts circadian rhythms via the suprachiasmatic nucleus in the brain, which regulates hormones like dopamine, GABA,melatonin and cortisol.
These hormones influence metabolism, including fat tissue activity where FIAF is expressed. Metabolism is what makes the key UPEs.
Fasting, which boosts FIAF, is also tied to light cycles, think of how daylight affects feeding patterns. If biophotons reflect or amplify these light-driven processes at a cellular level, they might indirectly tweak FIAF production by signaling energy states or oxidative stress in cells. If you cannot burn fat you are more likely to be mentally ill.
My decentralized ideas have always been spot-on that this topic doesn’t need a scientist to spell it out in a paper, absorption/emission spectra are measurable, and biophoton emissions are detectable.
The gap isn’t in the physics; it’s in tying the specific wavelengths of biophotons (which vary by cell type and state) to FIAF’s exact optical profile in vivo. Still, if gut cells emit biophotons in, say, the 300-400 nm range, and FIAF absorbs there, the optical photonics interaction’s real, study or not. It is first principle thinking. It is obvious what they problem. It’s like saying water absorbs infrared; we don’t need a new experiment to prove it gets hot under sunlight. Biology acts like it does, but in physics they use theoretical physics and first principle thinking to make predictions when the experiments are not done or cannot be done.
When matter experiences this topologic change do you know it become capable of emitting photons? In biology we call this UPEs. That is what does all the information transferring in life to keep you alive and kicking.
2. In astronomy and cosmology Spectroscopy is a form of remote sensing, meaning it allows scientists to determine the composition of an object without physically interacting with it. This is how we examine remote atoms in deep space.
How do we know what other worlds are made of? Planets we’ve never touched, stars we’ll never reach? By reading their light. Why can't quantum biologists realize the same opportunity exists in cells?
3. Quantum biology cannot yet apply the same spectroscopic "reading of light" as astronomy because cellular components are too small to be analyzed by light in the same way, and the inherent quantum effects within cells are not easily distinguishable from background noise in typical biological systems. They do not have photomultipliers small enough to sample UPEs yet.
Just because the technology is not available or studied means we should ignore the idea. Absense of evidence is not absence of effect. This is first principle thinking that is missing from most scientists today. In physics theoretical physicists provide a first principle lens to the Standard Model to innovate. We need to do the same in quantum biology. This is what I do.
Astronomers use spectroscopy to analyze the wavelengths of light absorbed or emitted by large celestial bodies, which reveal their chemical composition. However, cellular molecules are often too small to generate a detectable light signature, and the complex, noisy environment of a living cell makes it difficult to isolate and interpret the faint quantum signals associated with specific biological processes. there is no doubt today UPEs are real and carry information. We've know that AXIOMATICALLY since the Onion root experiment in 1922.
Cutaneous antimicrobial effects of sunlight on cholesterol conversion to Vitamin D components are today's PSA boys and girls.
1,25(OH)2D made in the liver and kidneys from 25 D(OH) from the skin by the sun and cholesterol and its receptor regulate the processing of the long-chain glycosylceramides that are critical for the skin barrier formation which is crucial in defending the skin.
Do you know how the heme protein enzymes CYP control this process?
The two Vitamin D biomolecules induce toll-like receptor 2 (TLR2) and its coreceptor CD14, which initiate the innate immune response in the skin. Activation of these receptors leads to the induction of CYP27B1 (heme protein), which in turn induces cathelicidin resulting in the killing of invasive organisms.
What happens when blue light and nnEMF destroy heme proteins when you know this connection? Innate immunity is destroyed. This is why Fauci wanted you indoors during COVID he and Baric made in Ukraine and China. ncbi.nlm.nih.gov/pmc/articles/P…
2. See how the heme photoreceptors are blown away?
3. Spine tumors are not common but most of them are associated with a poorly functioning immune arm and associated with low Vitamin D levels from poor solar exposure. That is something you can prevent to avoid this outcome.
Fire your centralized doctor by hiring nature for your reversal! Nature quantizes the precise amount of melatonin from the mitochondria needed to optimize autophagy and apoptosis. 95% of melatonin is made in human mitochondria. It is not your pineal or your gut. Your central retinal pathways have more mitochondrial density in it than any other part of the brain. The same is true with DHA to run your SCN faster than the other molecular clocks in your body to meet relativity needs. Want more info on exogenous melatonin? Use Yandex search with my name and mitohack #722. Your welcome in advance.
2. Shall we also say that sunlight plus natural darkness at night control melatonin, which in turn controls HIF-1a, which in turn controls sensing of O2?
Guess what happens to your mitochondria when oxygen tensions change? The IMJ geometry changes, metabolism morphs, geometry alters, and UPE become less common but more coherent. Mitochondria can change their physiology when the environment changes too. This is a remnant of the GOE when we had chronic hypoxia. this is why we innovated HIF1 and linked it to the PER clock genes.
Did you know UV light exposure raises oxygen tension in the venous plasma?
Guess what that all implies?
I know a lot more than any centralized MD or PhD about how we really operate.
If you have a T1D child you have a light problem. That light problem has manifested in your germ line.
If you're a Type 1 diabetic, by defintiion you have a chronic UVA and UVB deficiency and a chronic overdose of artificial blue light and nnEMF. It is also axiomatic.
Look at the chart below. T1D is almost nonexistent near the equator
2. By around 20 weeks of pregnancy, a baby girl’s ovaries already contain every egg she will ever carry.
Which means that when your grandmother was pregnant with your mother, the cell that would one day help form you was already there.
Three generations, held in one body.
This isn’t folklore. It’s embryology.
Pregnancy is sometimes called a three-generation event: grandmother, mother, child, all sharing the same environment in a single moment. Scientists call it multigenerational exposure. I call transgeneration biology.
3. But biology makes it hard to imagine these things don’t matter. The oocytes that hold potential life are shaped by the whole soup of environment, and so are the children they become:
☀️ Light and circadian rhythm
🌊 Water quality
🥬 Nutrition and minerals
🌬 The air we breathe
💊 Medications and substances
💤 The quality of rest and sleep
💭 The emotions we carry
🧲 Electromagnetic fields and magnetism
🧪 Toxins and chemicals
Even mitochondria, not just “batteries” but regulators of repair, signalling, and survival, are passed down the maternal line. It is an unbroken inheritance that centralized medicine continues to ignore at your peril.
Three generations are intertwined in every pregnancy in humans.
Biology is carrying echoes of what came before, and the possibility of restoration.
Parents need to become conscious of these risks to eradicate these diseases. The transhumanists seem to know, why don't the normies?