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☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
@DrJackKruse
Mar 15, 2023 • 13 tweets • 5 min read • Read on X
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1. A lesson in quantum biology.........Note the linkage to the VDR and POMc (melanin). Everything links back to the absorption spectra of the biochemical that works on POMc. That target is 220 nm light. Where does that VUV light come from? @hubermanlab
2. People forget that melanin in our skin helps facilitates the collection of UVB light to convert cholesterol esters that will become 25 di hydroxy Vitamin D in your skin.
3. Most but not all actions of 1,25(OH)2D are mediated by the vitamin D receptor (VDR). You need to understand that the VDR is linked to free retinol in the system is means melanin biology in mammals is destroyed because of things cleaved in POMC.
4. With melanopsin destruction, you get free Vitamin A from the broken weak covalent bond, & that free Vitamin A destroys photoreceptors. VDR is one of the photoreceptors destroyed.
5. VDR is a transcription factor that partners with other transcription factors such as retinoid X receptor (Vitamin A) that when bound to 1,25(OH)2D regulates gene transcription either positively or negatively depending on other cofactors to which it binds or interacts.
6. ^^^^the tweet above is only true if your skin has enough melanin to regulate them. If the melanin is missing you have NO regulation. All melanin comes from POMC!
7. Without enough melanin in the integument, that reaction becomes inefficient. This is going on in medical clinic patients every day and none of the centralized clinicians are realizing it because they do not understand how POMC works in mammals.
8. You do not need to take magnesium supplementation for Vitamin D deficiency UNLESS you have such a UV deficiency that you get rickets. Rickets is a sign of a loss of ferroelectricity in the musculoskeletal system.
9. This PEER-reviewed paper now shows Vitamin D supplements still "work" even if you're not taking magnesium supplements. The key point in the paper: Vitamin D from sunlight is still the best source of Vitamin D, and supplemental pills are not.
10. Magnesium (Mg) is a cofactor for many of our endogenous WBG semiconductors to create endogenous VUV-IR-A.
11. Mg WBG is also critical for the critical efficiency of enzymes, via proton tunneling, and many of these enzymes are involved in the conversion of cholecalciferol into 1,25-(OH)2-vitamin D3 (active vitamin. pubmed.ncbi.nlm.nih.gov/29480918/
12. The production of 1,25(OH)2D in the kidney is tightly controlled, stimulated PTH, and inhibited by calcium, phosphate & FGF23. These change the VUV light emission in cells from WBG semicinductors
13. Extrarenal production of 1,25(OH)2D as in keratinocytes and macrophages is under different control, being stimulated primarily by cytokines such as tumor necrosis factor alfa TNFα and interferon-gamma =IFNg = POMC pieces fit

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More from @DrJackKruse

☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
May 27
The 1942 Kenneth Mellanby paper from Nature below has been the absolute subatomic proof that the 2026 "100 g protein ceiling myth" study completely ignores. By focusing solely on classical "nitrogen balance" and labeled isotope tracers, the researchers missed the massive deuterium tax and water table collapse that comes with forcing a 100g protein load into the human semiconductor.
My decentralized framework in the Leptin Rx, is wholly backed by Mellanby's historical data, exposes why forcing massive protein loads into the system is a hidden "desiccation trap" that destroys the Inner Mitochondrial Junction (IMJ) particle accelerator.Image
2. Mellanby’s data establishes a clear, mathematical hierarchy of Metabolic Water Production during complete combustion. When the body combusts fat via beta-oxidation, Cytochrome c Oxidase (CCO) manufactures 110 grams of perfectly structured, deuterium-depleted water per 100g of substrate. This high-dielectric fluid (𝜖 ≈160) acts as the frictionless lubricant for the Brachistochrone curve of the cristae.

By contrast, ingesting 100g of protein cuts metabolic water production exactly in half. Worse, protein metabolism demands massive amounts of water for the urea cycle to clear toxic ammonia, creating a severe intracellular desiccation pull.Image
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3. The Biophysical Critique of the 12-Hour 100g Protein Study
The study celebrates that 100g of protein keeps muscle protein synthesis elevated for over 12 hours without increasing amino acid oxidation. In my vortex physics framework, this isn't a state of "infinite anabolism", it is a state of forced, high-entropy Lattice Lock. this is why exercise is harm so many high protein eaters.

Key sign they have is hair loss on the top of their head before they fry their neurons. Why? Entropic heat loss of the neuroectoderm. This is why you should look at their hair and the brain's cognition as two keys signs why hypertrophies muscles causes hair loss due to high heat entropy.

This explains why they stay in simpleton paradigms and do some questionable stuff----> dopamine destructions from melanin loss due to rises in signlet oxygen over triplet state oxygen.Image
Image
Read 9 tweets
☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
May 16
The Dolmen of Menga in Antequera, Spain, represents a profound challenge to mainstream, linear archaeology. Built around 3800–3600 BCE, more than a thousand years before the Great Pyramid of Giza, it features 32 colossal stones weighing a total of 1,140 tons, with a single ceiling capstone weighing an astonishing 150 to 180 tons.

The landmark Science Advances (2024) high-resolution laser scan study revealed that Menga’s builders possessed an incredibly advanced understanding of geology, physics, geometry, and astronomy. They embedded one-third of the vertical stones deep into the bedrock, angled them slightly inward to form a highly earthquake-resistant trapezoidal shape, and carved the massive capstones to be slightly convex, deploying the earliest known use of a stress-relief arch in human history.

When we decode why the ancients engineered this massive underground stone matrix, it becomes clear that they were not building a primitive cemetery; they were anchoring themselves against a planet-wide geomagnetic excursion event. Not hard to figure out IYKYK
2. Mainstream archeology is deeply puzzled by Menga's orientation. Standard European dolmens are aligned to track sunrise during the solstices or equinoxes. Menga breaks this cultural baseline entirely, shifting its central symmetry axis to a highly anomalous 45 degrees northeast.

I am not.

The Mountain Target: The axis points with millimetric precision directly to the northern cliff of La Peña de los Enamorados (Lover’s Rock), a nearby mountain that strikingly resembles a giant human face looking up at the sky.

The Tectonic Antenna: The mountain is not just an aesthetic landmark; it is a massive, exposed, high-susceptibility paramagnetic and limestone fault outcropping. By locking the central axis of the underground stone chamber to this specific geological mountain node, the builders created a macroscopic piezoelectric and telluric current antenna. IYKYKImage
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3. The Science Advances petrographic analyses revealed that Menga was built using calcarenite, a poorly cemented, porous detrital sedimentary rock. Centralized engineering labels this "soft stone" and questions why the builders chose it over harder rock variants.

Through my lens of sub-molecular physics, this choice represents masterful material selection by the Early Spanish humans:
The High-Surface-Area Sponge: Calcarenite is inherently packed with millions of microscopic, water-retaining pore cavities. Under the damp, earthen tumulus mound that encapsulates the dolmen, these soft stones absorb ground moisture, loading the internal mineral lattices with structured water.

The Solid-State Capacitor: As telluric currents surge up from the local Guadalhorce River valley fault zones, this wet, porous, mineralized calcarenite matrix acts as a massive dielectric capacitor. It draws in the erratic, high-voltage electrostatic charges tearing across the landscape during an active magnetic excursion, storing the energy and smoothing out the spikes so they do not short-circuit the biology of the humans inside. My thesis links this knowledge to the Maya via the Spanish conquests via oral history telling.
Read 25 tweets
☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
May 15
The Nature (2026) study confirming that the inner bird retina operates in a chronic state of total anoxia is the exact empirical proof of my framework. Centralized physiology looks at this and laments it as an "inefficient, primitive evolutionary compromise".

They completely miss the sub-molecular reality: the pecten oculi is not a failed oxygen radiator; it is an intentional, insulin-resistant quantum pump engineered to generate internal light for magnetic navigation. They need to read my work on the pectin oculi.

patreon.com/posts/decentra…
2. Centralized science is baffled by why a high-performance tissue would dump a 15-fold energy advantage by refusing oxygen. The answers lie in quantum magnetoreception.

Red blood vessels cast shadows and create magnetic turbulence. Stripping the inner retina of blood vessels creates a perfectly clear, non-magnetic optical medium.

Generating Internal UPEs: Anaerobic glycolysis running at 2.5 times the normal metabolic rate of the brain forces a massive, continuous release of Ultra-weak Photon Emissions (UPEs) inside the vitreous water.

The Cryptochrome Trigger: These coherent, internal UPEs act as an endogenous light source. They constantly excite the cryptochrome radical pairs in the eye, keeping the bird's quantum magnetic compass online even during pitch-black nights, high-altitude migrations, or global ash plumes.

The Insulin Resistance Shield: To keep this glucose pump running at maximum velocity without triggering metabolic collapse, birds maintain a baseline of physiological insulin resistance. Remember birds were once therapod dinosaurs who made it through the KT event with our ancestors the eutherian mammals. This quantum design ensures the eye gets priority access to the body's fuel lines. This allowed flying dinosaurs to find food when the sun could nto shine on much of the planet.

Proof that melanin is a super power and mammals who have no melanin become type 1, 2 and 3 diabetics. Get in the sun and ground to win.Image
Image
3. The K-T Impact: Internalizing the Sun via POMC
The K-T (K-Pg) asteroid impact 66 million years ago was not just a mechanical extinction event; it was a severe quantum light famine. When the ash plume extinguished the sun, it forced a radical biophysical pivot in the survivors. Your functional meds tard army has no idea insulin resistance is ho wbirds live their life and they tell you it is always pathological. They are wrong.

From Ectothermy to Endothermy: Dinosaurs were low-agency, centralized hardware giants dependent on direct, high-flux external solar radiation. The ancestors of birds and mammals survived because they had already initiated the POMC (Pro-opiomelanocortin) internalization strategy.

Skin as a Thermistor: By cleaving the POMC protein in the neuroectoderm, they stopped relying on external UV-A/B for heat. They transformed their melanin coatings into an active thermistor and semiconductor network.

Carrying the Inner Sun: This allowed them to maintain endogenous heat and mitochondrial electron tunneling internally, completely independent of the blocked sky.Image
Read 7 tweets
☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
May 6
1. Time to de-retard Chad. Since Chad won't read my work this will be the first and last time he will get educated using the tools on my website forum. Lesson begins. READ THE PAPER!!!! Image
2. The core issue is neither the declination angle itself nor its movement, but rather what a "magnetic stall" (a significant drop in the Earth's dipole moment) represents for the planet's atmospheric chemistry.

Your intuition about the poles being "static" in Siberia vs. "moving" hits on a key geodynamic reality: the alignment of magnetic and physical north (\(declination = 0\)) is a marker of a strong, stable geodynamo. When they decouple significantly, it often signals a weakening of the entire protective field.

Why Declination Matters (Biophysically)
The alignment of the magnetic dipole with the rotational axis (True North) is not just a convenience for human evolution; it is a byproduct of a strong, active core.

The Protective Shield: A strong magnetic dipole acts like a "filter" that prevents solar wind from stripping away the atmosphere.

The Oxygen Link: As shown in the Science Advances article I provided you for the 100th time now, there is a powerful correlation between magnetic field strength and atmospheric oxygen levels over the last 540 million years.

The "Magnetic Stall": If the magnetic north pole were to "permanently end up in Siberia" while remaining static and strong, it might not be a problem for humans. However, such a "static offset" is physically unlikely. In geophysics, when the pole drifts far from the rotational axis (high declination) or wanders erratically, it usually indicates the dipole is weakening or "stalling".

You have to put in some work here Chad. This has been covered elsewhere on the forum on my website for other tards like yourself.

youtube.com/watch?v=L2gf_z…
3. Nonzero Declination (Static)

If the field remained strong but tilted (e.g., North Pole in Siberia forever), the primary biological issue would be localized radiation spikes at the "new" poles. Most complex life would likely adapt.

The problem with the current erratic movement toward Siberia is that it reflects instability in the molten outer core not th einner one which got cooked 780,000 years ago just before the Cambrian.

Atmospheric Stripping: A "wandering" or weakening field allows more high-energy particles to penetrate the atmosphere. This can lead to the depletion of the ozone layer and, eventually, the loss of oxygen to space while deuterating and tritiating the food webs below on Earth. This is the real problem in NA and OZ now. Soon will be an African and EU issue due to AMOC collapse.
Read 10 tweets
☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
May 4
1. Ichthyosis vulgaris (IV) is a highly compelling case study for challenging the rigid boundary between pure genetics and epigenetics.

In standard centralized dermatology, IV is classified as a classic Mendelian, autosomal semi-dominant genetic disease caused by loss-of-function mutations in the filaggrin (FLG) gene.

However, from a systems-decentralized biology or quantum biology perspective, the skin’s physical presentation is driven heavily by the epigenetic and environmental constraints forced upon the tissue
2. The Filaggrin Gene is Not the Whole Story

In clinical medicine, finding a "spelling mistake" or null mutation in the FLG gene is considered the ultimate cause of IV. Filaggrin is the key protein that bundles keratin filaments and later breaks down into the "natural moisturizing factor" (NMF) and urocanic acid (which maintains skin pH and acts as a natural sunscreen).

The Epigenetic Disconnect: Many individuals carry heterozygous FLG mutations (haploinsufficiency) and display zero clinical symptoms or only very mild dry skin. Conversely, individuals can present with full clinical IV or severe atopic dermatitis with perfectly normal, unmutated FLG genes.

Promoter Methylation: Research into FLG expression shows that DNA methylation heavily regulates whether the gene is turned on or off. If methylation is deuterated this process is BROKEN. What looks like a genetic problem is really an epigenetic issue. Undifferentiated skin cells can actively suppress the healthy gene through non-CpG island promoter methylation. The genetic code is present, but the epigenetic software is refusing to run it.
3. The Mitochondrial Stress and Energy Tax

Connecting back to the previous discussion on high-turnover clones and the "isotopic tax": the epidermis is constantly renewing itself.

To successfully flatten, enucleate (spit out its nucleus), and form the stratum corneum (the brick-and-mortar skin barrier), a keratinocyte undergoes a massive, highly orchestrated energy-intensive process.

Acquired Ichthyosis: True genetic IV presents at birth or in early childhood. However, acquired ichthyosis occurs in adults who possess perfectly normal genetics.

It is heavily triggered by systemic conditions like Hodgkin's lymphoma, severe acute malnutrition, or new-onset diabetes.

What this reveals: This proves that the "fish scale" phenotype is not solely a product of a broken gene. It is a default failure state of the skin barrier when the body's systemic redox potential drops or metabolic wasting takes over, preventing cells from correctly processing lipids and proteins. Genes are not the be all end all in any disease. Rockefeller medicine wants MDs to believe it to write Rx's.
Read 5 tweets
☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 Profile picture
May 4
1. No Liz. Base chain of DNA/RNA has to be undeuterated to be coherent in its expression if not an aberrent phenotype results.

Let me give you an example. See the pic? It is called cradle cap.

Do you know what centralized pediatricians tell patients with kids who are born with this?Image
2. IS CRADLE CAP A SYMPTOM LIKE JAUNDICE IS, LEADING TO HIGHER HETEROPLAMSY LEVEL?

Children with cradle cap suffer from more metabolic problems later in life pointing out why this is not a temporary mothering hormone issue that centralized science tries to sell to MDs.

My observation highlights a critical pivot in how we understand chronic, systemic diseases. The argument is that if cradle cap (infantile seborrheic dermatitis) were merely a transient reaction to maternal hormones, it would not correlate with long-term metabolic dysfunction later in life.

While the standard centralized medical model focuses on the local, temporary symptom (excess oil and yeast), the perspective of quantum biology and bio-energetics argues that the skin is merely the outward map of internal mitochondrial efficiency and "isotopic tax."

The Conventional Medical View sold to parents is as follows......
In standard dermatology, cradle cap is known as infantile seborrheic dermatitis.
The Mechanism: It is a self-limiting condition characterized by yellowish, greasy plaques and scales on the scalp.
The Clinical Cause: It is primarily attributed to maternal hormones crossing the placenta before birth. These hormones temporarily hyperactivate the infant's sebaceous (oil) glands.
The Microbiome Role: The excess sebum acts as a lipid-rich feeding ground for commensal skin fungi, specifically Malassezia. The inflammatory response to the fungal byproducts causes hyperkeratosis (the sticking together of dead skin cells).Image
Image
3. The Quantum Biology / "Deuterium Burden" Hypothesis of Kruse
The perspective I've presented operates on the premise that disease and cellular dysfunction are problems of sub-molecular physics and energy transfer rather than just genetic hardwiring or hormonal shifts.

If we apply this specific framework to cradle cap, the hypothesis would be structured as follows: The Follicle as an Energy Sink: As I've noted many times before, hair follicles and their associated sebaceous glands are highly active "clones" demanding immense metabolic energy to fuel continuous cellular turnover.

The Mitochondrial Stutter: In my decentralized framework, mitochondria function as nano-motors. At the center of the ATP synthase motor is a rotating axle propelled by hydrogen ions (protium). Because deuterium is twice the mass of normal hydrogen, its presence in the mitochondrial matrix can physically damage or "stutter" the motor.

The Singlet Trap & Apoptosis: High local concentrations of deuterium within the rapidly dividing follicular cells would reduce the efficiency of the electron transport chain. This causes electron leakage, creates reactive oxygen species (the "singlet trap" heat/entropy), triggers inflammation, and interrupts normal cellular desquamation (shedding).

Here is an analysis of why viewing cradle cap as an early marker of metabolic/deuterium stress, rather than a simple hormonal fluke, that holds weight under my framework.Image
Read 8 tweets

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