Janiesaysyay Profile picture
Mar 19, 2023 35 tweets 9 min read Read on X
🧌 Its Alive! 🧌

Working theory, "Show replies" at the end for all.

Is SARS-CoV-2 a lab-created chimera?

A Frankenstein virus created from the genetic material of other lethal organisms, creating similar pathophysiological mechanisms in the body?

Gene sequences say YES >
The SC2 spike protein contains gene sequences that are homologous to those found in HIV, SEB, animal venoms, herpes, malaria, rabies and more.
The sequences from these organisms could release toxic peptides and pathophysiological mechanisms in the body similar to those found >
in the original lethal organisms.
Mechanisms are chemical recipes, like baking recipes.
A list of instructions from the genes that, when followed, produce a unique product like micro clots or a cytokine storm.

Just as a recipe for 🎃 pie produces 🎃 and not 🍎 pie. >
It is interesting to see the similarities between the mechanisms produced by SARS-CoV-2 and the original lethal organisms it shares gene sequences with. Several scientists have sequenced the the genome of the spike protein >
The Laboratory of Molecular Biology and Immunology, The U of Patras, @ konstantinospo7 identified a toxin-like sequence, similar to 🐍 venom in the genome of the spike protein >
pubmed.ncbi.nlm.nih.gov/32823591/
The Craniomed group of carlobrogna1 identified 🐍🐌 toxin-like peptides produced in COVID patients >
pubmed.ncbi.nlm.nih.gov/35106136/
Prashant Pradhan and teams from Kusuma School of Biological Sciences, Indian Inst. of Technology, U of New Delhi and SUNY Stonybrook found gene inserts similar to HIV in the spike. #PradhanWasRight > biorxiv.org/content/10.110…
Luc Montagnier, Pasteur Inst. and Jean Claude Perez, IBM European Research Center on AI, found gene sequences similar to HIV in the spike. >
osf.io/d9e5g/
These genes, and the toxic peptides they produce, could make SARS-CoV-2 (SC2) and its spike protein (SP) an aerosolized bioweapon.

A coronavirus that causes the severe endothelial damage, clots and immune disorder of COVID.
A lethal cold.

How might this manifest? >
Covid is a bi-phasic disease.

The 1st phase is a cold-like respiratory virus.
One which most immune systems can destroy without 💊.
Or it can be overcome with early treatments like the Zelenko and FLCCC protocols. >
The 2nd phase is Acute COVID (AC)

Here the pathology of the bioweapon commences,
the patient becomes severely hypoxic, losing 02 at a cellular level as the virus destroys the cells of the endothelium and the mitochondria and causes micro clots.

The lethal phase of COVID. >
SC2 causes a specific amyloid clotting cascade, and inflammo-thrombotic-response (see Doctor_I_am_The) /cytokine feedforward loop. This mechanism is quite similar to the one produced in the envenomed prey of Cobra and Krait snakes and Cone snails. >

National Geographic
Aquarium of The Pacific
Academy of Natural Sciences of Drexel University.
Amyloid clots are made of disordered, fibrous misfolded proteins, during AC and envenomation they are produced rapidly. >
en.wikipedia.org/wiki/Amyloid
The viral SP infects the cells of the endothelium. It permeablizes membranes and is expressed on the outside of cells.
The endothelium is the largest vital organ in the body. It lines, protects and regulates every 🩸 vessel, lymph node and organ. >
study.com/academy/lesson…
When the SP is expressed on endothelial cells it signals the start of a feed forward loop. The immune system is hyperactivated, a "cytokine storm" occurs.
It attacks the cells expressing foreign spike protein, and damages the endothelium. >
The spike and the immune system begin to destroy the membranes, vasculature, and all organs.
Organelles like mitochondria responsible for cellular respiration, and delicate cells/tissues like erythrocytes, alveoli, and capillaries are shredded.
This results in ferroptosis >
vessel leakage, bleeding.

O2 is lost in cells in many areas of the endothelium simultaneously.

Repair mechanisms begin amyloid clot formation to repair damaged vessels. This causes fibrosis and stiffening of the vessels and organs. Sepsis, lymphocytopenia and dysregulation >
of the autonomic nervous system also occur.
Shock, respiratory failure, pulmonary embolism, cardiac failure, or stroke cause death. > ccforum.biomedcentral.com/articles/10.11…
The pathophysiology caused by the genetic sequences in COVID is similar to the pathophysiology caused by the similar sequences found in the original lethal organisms.

Envenomation: amyloid micro clots, fibrosis, cytokine storm, bleeding, coagulation cascade, vasculitis >
sepsis, dysautonomia, shock.

SEB: toxic shock, sepsis, multiple organ failure, ARDS, cytokine storm, fibrosis, vasculitis, immune tolerance.

Rabies: encephalopathy, headache confusion, lymphocytopenia, dysautonomia, aerosol transmission transmission along the axons >
HIV: clotting cascade, DVT, cytokine storm, neurodegeneration (HAND), dysautonomia, lymphocytopenia, sepsis, genomic integration, latency
immune dysregulation >
Summary:

SARS-CoV-2 could be a lab created chimera.
A corona virus with a toxic spike protein, an aerosolized bioweapon.
Several researchers sequenced the genome of SC2.
They detected gene sequences similar to sequences found in other lethal organisms. >
spliced together in the spike protein. (HIV, SEB, Hepatitus, Herpes, Malaria, Rabies, Venoms).
These genes code for toxic peptides which are produced in the body, like a recipe.
The peptides produce mechanisms and symptoms similar to those found in the original organisms. >
#ItsAlive #Toxic #FrankensteinVirus #chimera #ItCameFromALab #Pie #PumpkinPie #SpikeProtein #HIV #SEB #Influenza #malaria #rabies #hepatitis #herpes #venom #ouroboros #Snakes #Bioweapon #Biowar #pandemicplaylist
Text👇 is a quote, I forgot to put quotation marks.
Descripton of the amyloid micro clotting mechanism seen in COVID. This is similar to the mechanism in envenomed prey of 🐍. The clots are ssimilar, the symptoms are ssimilar, the deaths are ssimilar. >
🧵explaining the #Frankenstein #chimera "Show replies" to see all.
It's simple, the DoD has been funding gain of function venoms research and coronavirus research for years in the U.S. and CCP bioweapons labs.
They recently reinstated the funding.
No one is hiding it. 🙄 #ItCameFromALab
Probably a more straight forward explanation of the chimeric nature of the spike protein is found in this thread.

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More from @janiesaysyay

Nov 14
It's Friday November 14 2025

Millions around the 🌎 are 💀 from a pandemic panic
caused by a chimeric 🦠 created in a 🧪 by 🇨🇳👩‍🔬👨‍🔬
with 🇺🇸 data & 💰
directed by

Dr. Anthony Fauci.
🧵
They are 💀 because of the disastrous pandemic response directed by Fauci.

Fauci gave conflicting medical advice and censored
life-saving early 💊s in order to shield DoD bi0weapons programs from scrutiny and enable a novel warp speed mRNA 💉 >
One of Fauci's NIH grantees and the premier U.S. coronavirus researcher Ralph Baric, initially gave evidence that SARS2 might have come from a lab. >
Read 17 tweets
Nov 13
🧬 From Grok to Scott

Grok explains the potential for oncogenesis from the C0VID 💉s based on lab results of bacterial DNA impurities and other published data. @ScottAdamsSays

🧵 Image
🧬
> Image
🧬
> Image
Read 10 tweets
Oct 31
💀 It's Friday 🎃ctober 31, 2025 💀

Millions around the 🌎 are 💀 from a pandemic panic caused by a chimeric 🦠 created in a ☣️🧪 by
🇨🇳 👩‍🔬🧑‍🔬 conducting Gain of Function research
with 🇺🇸 data and 💰 directed by
Dr. Anthony Fauci.

🧵
💀
Why is Tony Fauci called the most prolific serial killer?

Decades before Fauci & NIH/DoD helped fund the creation of GoF coronavirus in the 2000s,
the NIH/DoD funded the creation and spread of HIV. >
💀
Why did gay men suddenly start falling ill and dying at
an alarming rate in 1978 in major cities across America?
Read 16 tweets
Oct 21
The 🫀 Remembers,
SARS-CoV-2 & Cardiac Pathophysiology

How might viral 🦠 persistence and vascular biofilm + amyloid processes translate into cardiomyocyte damage, fibrosis, and chronic 🫀 dysfunction?

A theory 🧵 >
tinyurl.com/2fdj7j8a
SARS‑CoV‑2 enters through the ACE2 receptor,
which is abundantly expressed in cardiac pericytes, endothelial cells, and cardiomyocytes.

Studies demonstrate that infection of pericytes compromises capillary perfusion, producing micro‑ischemia.

Autopsies confirmed viral RNA and protein in about 40% of🫀s.

This evidence shows that SARS2 reaches the myocardium directly, setting the stage for injury. >Image
Once inside the vasculature, the 🦠 damages the endothelium — the inner cellular lining of 🩸vessels.
There is complement deposition and fibrin micro clots in small cardiac vessels consistent with inflammatory endotheliitis.

This process decreases nitric‑oxide signaling and damages pericytes, producing local edema and hypoperfusion.
This causes patches of perfusion defects and troponin elevations seen in COVID‑19 cardiomyopathy. >Image
Read 25 tweets
Oct 1
🚨Shocking data out of Finland.

A hypothesis with Grok about why it is occurring.

Using research of: Arne Burkhardt, Sucharit Bhakdi, Kevin McCairn, Kevin McKernan, Bruce Patterson,
Ram Yogendra, Ethical Skeptic, Lyndsey House,
James Thorpe, Janiesaysyay, Jessica Rose,
John Beaudoin Sr, Marc Girardot, Mary Talley Bowden, Nicolas Hulscher, Peter McCullough, Robert Malone, Resia Pretorius, Akiko Iwasaki, Lynn Fynn, Ryan Cole, Sabine Hazan, William Makis, and others.

🧵 (keep showing replies)Image
> Image
Here we look at how the injection goes wrong from the start, this is Girardot's #BolusTheory

The deltoid muscle is full of tiny 🩸 vessels, so even a careful 💉 can hit one, sending LNPs straight into the 🩸 like an EpiPen.

They hitch a ride on proteins like ApoE and head to organs hungry for lipids, like the adrenals.

In 🤰 hormones make everything leakier, helping LNPs slip through the placenta via receptor uptake and membrane fusion.

This is step one: getting to the fetus. >Image
Read 10 tweets
Sep 30
Image
Have you heard the story of the hot rod race 🎵 Image
where the Fords and Lincolns were settin' the pace?
> Image
Read 26 tweets

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