1. Longevity in humans is linked to optimal solar exposure. The reason is simple. This protects the 7 layers of energy generation inside a cell. The more sun human gets the more diseases they can avoid and the #1 risk of most diseases is AGE. Solar exposure effectively makes you younger because it lengthens the TET mechanism inside of cells to improve the HAyflick limit in all cell lines. It is not hard to understand when your perspective is decentralized.
2. From an evolutionary point of view, vitamin D and melatonin appeared very early and share functions related to the defense mechanisms of the mammalian powerplant. In the current clinical setting, vitamin D is exclusively associated with phosphocalcic metabolism when it is sulfated and in its reduced state. When it is not sulfated or reduced its role in calcium control is diminished. This usually happens in winter months with mammals when they are in the cold and LDL cholesterol production is upregulated by the light stress response of the POMC gene by a lack of 380nm light. This signal is via neuropsin & ACTH in mammals. When 380 nm light is missing mTOR signaling shifts mammalian biochemistry from anabolic to catabolic. This occurs via lipid raft electrical changes mediated by cholesterol biology and proteins embedded in the mammal's membranes.
3. Calcium flows are critical in mitochondrial control because they are a key dopant atom in semiconductive proteins in humans. Meanwhile, melatonin has chronobiological effects and influences the sleep-wake cycle. Scientific evidence, however, has identified new actions of both molecules in different physiological and pathological settings. In centralized science, there is a belief that melatonin and Vitamin D are inversely related to solar exposure. This perspective is wrong. The decentralized idea is both are controlled by the sun because melatonin absorption spectra tell us this is the case. Melatonin's spectra are 224nm & 290nm. This light is never present at night in the environment. The spectra reflect light made internally. Centralized medicine has no idea of this concept.
4. The biosynthetic pathways of vitamin D and melatonin are directly related relative to sun exposure. A deficiency of either of these molecules has been associated with the pathogenesis of cardiovascular diseases, including arterial hypertension, neurodegenerative diseases, sleep disorders, kidney diseases, cancer, psychiatric disorders, bone diseases, metabolic syndrome, and diabetes, among others. During aging, the intake and cutaneous synthesis of vitamin D, as well as the endogenous synthesis of melatonin is remarkably depleted, therefore, producing a state characterized by an increase of oxidative stress, inflammation, and mitochondrial dysfunction. Oxidation = lack of electrons = you cannot absorb solar light. Mitochondria also control the change program of mitochondria apoptosis and autophagy. Apoptosis efficiency is controlled by UV light and autophagy is controlled by IR-A light
5. For example with reference to the two major diseases killing modern humans heart disease and neurodegeneration both neurohormones protect humans from both. Sunlight controls heart disease by lowering APoE, Lpa, and calcium index scores. Neurologic function is protected and extended by sunlight via POMC, VDR, RXR signaling, BDNF, and neurotrophin synthesis. Both molecules are involved in the homeostatic functioning of the mitochondria. Given the presence of specific receptors in the organelle, the antagonism of the renin-angiotensin-aldosterone system (RAAS), the decrease of reactive species of oxygen (ROS), in conjunction with modifications in autophagy and apoptosis, anti-inflammatory properties inter alia, mitochondria clearly have emerged as the final common target for melatonin and vitamin D. ROS is controlled by melanin sheets The primary purpose of these Tweets is to show the non-believers how decentralized medicine elucidates the common molecular mechanisms by which vitamin D and melatonin might share a synergistic effect in the protection of proper mitochondrial functioning.
6. The skin is the melaninated sheets of solar panel for the brain to give it more energy from the sun to run the Ferrari engine in our head. This has to be optimized for neurological function. Most modern human disease is linked to a break in this quantum biologic connection.
7. The quantum connection between the skin and brain is this. You must become aware that NON-VISUAL PHOTORECEPTION is the key to most diseases in the human heart and in the brain. What links both organs? They are both impotent without cholesterol and light stimulus. How do cholesterol, neuropsin, mTOR, melanin, and vitamins A and D link in this decentralized dance to optimize longevity? Issue one. Taking a starting is among the most ignorant thing one can do when you understand how disordered the centralized paradigm around LDL cholesterol is. Non-VISUAL photoreception controls this entire system in humans. Most of the non-visual photoreceptors are weakly covalently bound to Vitamin A and when they decouple photoreceptors are degraded = biophysical physiology fails. Let's begin. The heart response to strong light on the chest by making adenosine. Adenosign stops all aberrant calcium flows hence why it is on every crash cart for ACLS as part of the algorithm for SVY. Note how this system immediately linked the brain's SCN optical lattice clock via the PER2 gene. This gene controls the biophysics of the lipid rafts that change seasonally. How?
8. BIOPHYSICS 101 OF THE SKIN related to the heart and the liver. Eating cholesterol is of zero consequence to mammals. Creating it in the liver is critical in understanding the biophysics of cholesterol non-visual photoreception. The lipid raft's ability to change in mammals occurs by seasonal light variation and collection via the non-visual photoreceptors via perception on the skin, eyes, and gut. That external light determines the reality the mammal faces. When the solar cycles change so do the lipid rafts. This photoelectric change alters biochemistry in mTOR, PPP, glycolysis, the TCA cycle, and POMC cleavage. When the lipid content changes they induce changes in the semiconductive proteins embedded in them. This changes the physiologic ability. This is why the clock mechanism in mammals is linked to light and temperature. Both signals change to the surfaces of mammals.
9. This change in the skin has massive implications for the circulatory system, arteries, blood, and especially the liver. Most people do not know the deuterium content of blood and the lumen in the gut is also plastic via light and temperature signaling for two reasons. Deuterium has an extra neutron so this heavier atomic mass means more energy is needed to move it. And Deuterium has a different magnetic moment than H+ so this means it reacts differently when the electric signal in mammalian membranes changes.
10. Because semiconductive proteins are embedded in the skin, what type of cholesterol and fatty acids matter to the functioning of VGCCs. Why? Because the lipid rafts are like Morse code for the Vitamin D system in the skin and Vitamin A signal in the opsin system. The rafts alter the functioning of voltage-gated channels that control the photoisomerization step of the conversion of cholesterol to 25D (OH). This chemical has to go to the kidney and/or liver for final conversion to act at target receptors in this system of the mitochondria. Sunlight increases NO and oxygen deliver to mitochondria to alter their function because sunlight controls the oxidation state of Fe and keeps it in the +3 state. This increases the sulfation of all things in the system and it makes them MORE WATER SOLUABLE. APOB and LpA drops and they cease to be an issue. It also thins the blood while lowering calcium flows in the mitochondria. Lowering calcium and raising NO both act to reduce mitochondrial power. What takes over when all this happens to create H+ and oxygen and electrons to run the system? POMC creates melanin and melanin makes all three things massively. This is why NO slows mitochondrial metabolism and lowers BP. Centralized medicine does not understand this wiring diagram in 2023. Their longevity experts are still advocating the use of statins which completely ruin the fidelity of this system. Sulfate platelets and GAGs in the vessel wall are less sticky and there is better laminar flow. This is why we have an epidemic of patients on blood thinners. No one is going outside enough. As a result, clots cause both heart and brain damage. This is why PAD is linked to both diseases. @hubermanlab
11. When the electric charge is altered in the skin and the membranes inside of your tissues, your tissues begin to become a net collector of the heavier isotope of hydrogen called deuterium. This occurs in the skin and your liver. Blue light/nnEMF NOT FOUND IN THE SUN CAUSES THIS ISSUES. Melanopsin is the blue light opsin of this nonvisual system. It has its highest density in the brain, arteries, and heart. All places are fed by the blood and why brain and heart diseases are always linked to PAD. This effect implies you cannot make D3 even with equatorial sun. All things centralized medicine is ignorant of.
12. Centralized healthcare's ignorance of the basics of this Tweet thread has led to incalculable errors for public health. I mentioned this to @RickRubin & @hubermanlab when we spoke about Dr. Changs' belief it made 50% of what is in the textbooks obsolete. I am telling you 99.9% is hot garbage. Why? The number one opsin in mammals is MELANOPSIN and we no longer live under the sun. We live inside under LED light that destroys this non-visual photoreceptive circuit. People want to blame glucose and insulin yet, when you look at your blood you see this. Does Nature make mistakes or has centralized medicine ignored a lot of facts they should have been asking questions about? When deuterium is let into the matrix this is what redox shift all biochemical pathways the longevity experts THINK never change. This is why none of them understand mTOR and UCP-2. Those proteins embedded in the lipid rafts or connected to them by the tensegrity system change how they respond. Why does NO fall as we age? Because modern humans live under an alien light. Why do Apo proteins and LpA look like a problem to the PEter Attias of the world? Because none of his patients in NYC or San Diego live in sunlight. If they did their LDL cholesterol would be low and their HDL would be high and he would not write a new book telling everyone to take a statin because it is a GOOD plan for longevity. This message is DEAD WRONG.
13. Because ideas like his are allowed to be considered expert opinion, that is why this information has been kept int he shadows by big pharma and big food. I promise you this is why all of you do not know it either. Cholesterol is another nonvisual photoreceptor of man that absorbs best in the UV range. When it is sulfated it's absorption spectra is in the 190-350nm range. When it's in its LDL it absorbs at 500-600nm (winter/blue light). For example, if you have the wrong type of cholesterol in your skin when the sun is strong you won't be able to make Vitamin D at all even at the equator. This explains why people who live indoors and work in offices all have high cholesterol. It also means they are all collecting deuterium in their systems instead of H+. Since your mito matric runs purely on H+ you might see the problem now why heart brain and PAD diseases are all linked. Cholesterol has to be sulfated and in the HDL format because those electrons are needed to absorb the 290-320 nm light. THIS IS THE REDUCED VERSION OF CHOLESTEROL mammals use in spring and summer. If your HDL is low it is because you LIVE MOSTLY IN BLUE LIGHT or nnEMF stress. REMEMBER LIGHT ONLY WORKS WITH ELECTRONS. LDL cholesterol is DEVOID of electrons and sulfur. when you have the wrong type of cholesterol in your skin, the lipid rafts change the voltage gate channel operation of proteins embedded in them to alter function to match the light. When the system is disordered, as it is in most people in California/NYC due to blue light and nnEMF, not even standing on the equator naked will raise your vitamin D level. It is Biophysics 101. Right now this is why people in California and NYC have record rates of LDL cholesterol levels, low vitamin D levels, metabolic syndrome in the liver, and higher rates of skin cancer, colon cancer, and melasma. It is fully explainable when you get how light controls mammals. Keep enjoying your tech and NYC/Cali and prep for a life filled with problems that centralized scheme will wallet biopsy with regularity.
14. I mentioned metabolic syndrome and liver disease. My new young protege, @MaxGulhaneMD is very concerned about fatty liver and is convinced that seed oils are behind it as most of the meat heads in carnivores seen are. Time to educate them.
15. There is strong class one evidence of a significant relation of 25(OH)D levels with the degree of liver dysfunction, considering that an inverse correlation of 25(OH)D levels with both Child-Pugh score and Model for End-Stage Liver Disease has been reported in the GI literature. In addition, vitamin D deficiency has been shown to increase the risk for overall mortality and infections in patients with cirrhosis. Vitamin D deficiency has been also associated with advanced stages of hepatocellular carcinoma and poor prognosis. Finally, there are studies suggesting that patients with chronic hepatitis C and normal vitamin D levels have higher virological responses to treatment. The sun is always the answer for liver disease = decentralized wisdom 101. It is not the meat diet. That solution is 4 steps below the sun.
16. #1 is sunlight ALWAYS. This is why Vitamin D is converted into an active neurohormone in the body. Key proxies to look at for decentralized clinicians = look at blood glucose, LDL cholesterol levels, B12, and any surface skin or colon color changes (endoscopy). If any of them are abnormal your liver is getting pounded and the melanin sheets at the organ of Zuckerkandl are being degraded. Women with melasma and men with melanosis coli you are in trouble and you are collecting deuterium in your liver to grow a fatty liver. Note the date on the paper and ask yourself why is that every time the GI guy sticks the black snake in my rectum he has never told me this if the data is 30 years old? WHY?
17. the organ of Zuckerlandl is a chromaffin body derived from the neural crest, loaded with melanin sheets that services the liver, intestines, stomach, pancreas, spleen, gallbladder, kidney, and adrenal medulla and is part of the melanin network that is located at the bifurcation of the aorta or at the origin of the inferior mesenteric artery. This nonvisual photoreceptive array connects with the enterochromaffin cells of the gut that contain massive stores of melanin and aromatic amino acids in the lumen of the gut and in the intestinal wall. Tryptophan is the key time crystal in the gut and the sympathetic nervous system allowing mammals to know precisely where the Earth is in relation to the sun during a revolution cycle on Earth. I wrote a blog on how that works on Patreon. Read it. This allows for the perfect planetary adaptation of the organism to change its skin and gut biology to absorb solar light PROPERLY.
18. This directs the turnover of enterocytes to a 24-48 cycle designed to remove deuterium from the blood and lumen so the liver does not get fatty. This same organ of Zuckerlandl controls your adrenal medulla on the top of your kidney. The POMC gene cleavage releases ACTH. This ACTH allows for high-flux mitochondrial cholesterol trafficking in tissues where POMC is located in post-mitotic cells in adult mammals. It turns out that in the heavily melanated adrenal cortex, this is a specialized function in the mammalian clade. Chromaffin cells migrate to the area adjacent to the sympathetic ganglia with neural crest-derived POMC neurons via the somites migration plan to the adrenal medulla where they're the most abundant type of cells in mammals. The largest extra-adrenal cluster of chromaffin cells in mammals is the organ of Zuckerkandl. Sunlight expands this organ and the adrenal medulla to improve liver and kidney functioning. This is skin 25 D(OH) is converted in both organs to the active format of D3. That vitamin D3 then binds to the VDR in the matrix to slow ECT to stop the need for food to run the ATPase. The 43% of red light in the solar spectrum can spin the ATPase and the liver becomes protected from the deuterium loads. If the load gets in because of bad mammalian ideas, the enterocytes can still slough off every day to protect the liver if the SCN clock is operational because the mammal is in the sun getting UV light. The 380 nm light hitting the RPE informs mTOR to be in its catabolic or anabolic state = which controls the flow of protons into mitochondria in the liver. That is the circle of control of the liver. NOTHING is better for liver diseases than the sun.
19. THE END OF THE LESSON
20. If you read my Patreon work on tryptophan’s role as a protein semiconductor and its seasonal role as a time crystal then understanding this post will be easy. Sunlight reduces inflammation by lowering the proton content in the cytosolic water and making sure protons stay inside the mitochondrial matrix. As a result negative charge density builds in the cytosolic regions of a cell. This high net negative charge is known as a high redox state. Persistent chronic inflammation slows the production of serotonin, steering it instead toward self-destructive quinolinic acid production.
This is thought to play a role in psychiatric symptoms associated with chronic inflammation and infections.
Without sunlight melanin is eventually degraded into quinolinic acid. This compound destroys charge density in a cell causing dielectric collapse. It mimics the effect of fluoride deposition in a cell. Sunlight exposure sets the metabolic efficiency of how the pathway operates. The image accurately represents the relative efficiency of the kynurenine pathway when solar redox is optimized.
For instance, the serotonin “branch” flows at a less efficient rate compared to the kynurenine “branch” (~98% vs ~2%). It also points out why exogenous supplementation of melatonin upsets the charge density of tissues like the retina where melanin is located. Here is the key. A lack of sunlight or melanin degradation by any cause leads to a change in how the pathway operates in neuroectoderm in humans. Chronic inflammation results from a lack of sun. It can also happen via hypoxia caused by a myriad of things such as during an infection or an autoimmune disease. Light fundamentally changes the kynurenine pathway.
The part of the pathway that normally synthesizes beneficial molecules slows to a trickle while the floodgates open for the harmful part of the pathway. Why is this?
Well, inflammation:
✅ increases the catalytic activity of enzyme IDO
Making more kynurenine and less serotonin and melatonin
AND
❌ decreases the catalytic activity of KAT
Making less kynurenine acid (protective) and more quinolinic acid (harmful) from melanin degradation. A lack of sun causes melanin degradation via hypoxia. Non native EMF via liberation of Vitamin A from the loose covalent bond is todays major cause of disruptions in this pathway. How does this happen? A lack of sun changes the catalytic efficiency of an enzyme called IDO in the pathway. This changes cytokine signaling which in turn changes the biochemistry of the pathway. Note a lack of sun or excess nnEMF is the key stimuli.
A lack of sun increases thesecytokines increase IDO activity:
✴️ IL-1b
✴️ INFg
✴️ IFNa
✴️ TNFa
✴️ IL-6
✴️ IL-12
While these cytokines decrease KAT activity:
✴️ IL-1b
✴️ INF-g
✴️ TNFa
This is how light changes disease phenotype. Hence, persistent chronic inflammation from a lack of sun or too much nnEMF slows the production of essential neurotransmitters, neurohormones, and neuroprotective substances, steering it instead toward self-destructive processes in neuroectodermal derivatives in mammals
In humans we have extra neuroectoderm to protect in our frontal lobes. That photonic switch is in the habenular nucleus. When melanin is degraded in this pathway all he’ll breaks loose in executive function. These alterations eventually lead to the disruption of limbic and paralimbic brain circuits, compromising emotional functioning. This explains how light plays the leading role in the development of psychiatric symptoms associated with altered solar redox and many mitochondrial illnesses. It’s no longer a mystery. You just need to read the literature and connect the dots to POMC biology and melanin production and degradation.
21. Please read the literature on BDNF. It is also increased by solar exposure and destroyed by nnEMF and build up of quinolinic acid from melanin degradation of the non visual photoreceptor system in neuroectodermal derivatives. Implications??
22. BDNF in humans is on chromosome 11.
BDNF: Brain-Derived Neurotrophic Factor
BDNF is paramount in the growth, development, and maintenance of neurons in the brain. It is linked to solar exposure via WNT signaling embryo logically. Recall that the Leptin melanocortin pathway controls fecundity and development in the human embryo.
It works to help existing neurons survive and impacts the growth and differentiation of new neurons and synapses. One can only imagine the consequences. Just think about autism for a moment and why it’s exploding since 1940 when humans began using light to communicate in tech gear.
Mutation or changes in expression could result in neurological, mood, and cognitive disorders.
It would be a terrible thing if somehow this mechanism was mutated in some way, by say, the presence of DNA plasmid contamination, that also carries an SV40 promoter, poor solar exposure, alien light, or as found in other instances, gene expression might have been acted upon by the pure presence of linear DNA plasmid pieces; don’t you think. Few are making these connections
******
There are 8 BDNF promoters. Never forget sunlight increases all of them properly.
23. You might want to read this paper after you read my Quantum engineering #45 blog on the link to melanin melanopsin and melatonin to autism. Why was I warning my tribe about the mRNA technology before 2020? I laid that story out to RFK Jr and Rick Rubin in 2023 Tetragrammaton podcast. Now look at this paper. I’ve known about these links for thirty years. link.springer.com/article/10.100…
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Melanin is a skeleton key to unlock the quantum mechanical abilities found in water. It is that simple.
2. What is one of the most amazing things that melanin does to water? It creates a massive source of electrons to be delocalized for semiconduction use, but it also changes the hydrogen bond angles of water.
3. Melanin also affects K+ levels and ATP levels in cells as temperture changes. When melanin degrades it also affects BG and insulin levels. Insulin does not operate well below 62F. This is why melanin becomes a different kind of electrical conductor as temperature varies. You can see this effect on the slide below. Heat is linked to NO production = links to biophoton release. More heat = less UV biophotons released. THe less UV biophotons released links directly to hydgrogen bond angles.
When K+ is low inside energy transduction goes down and bond angles in hydrogen change. When a lifeform dies 100% of water and K+ leaves the cell. When K+ leaves the cell so does light that is also stored at the electronic and vibrational level. This destroys the cell as a dissipative structure. When water is made in the mitochondria and leaches out it needs light creation. The light levels link to melanin granules right outside of the mitochondria during metabolism. Without melanin redox power drops, energy/information drops, light is lost and information transfer and biochemical fidelity drop.
1. Chiara D'Arcangelo asks me,
"Would you mind to elaborate on Mercury toxicity due to amalgam fillings from a Quantum biology/physics stand point? How does it disrupt the mitochondria machinery & therefore all the other body systems?
THANK YOU"
2. Why do you have to be careful with alternative functional memes/ideas and narratives that are linked to money? Often they are not true and people get "herded" into a belief system that they need expensive testing and therapies to remove their risk of heavy metal toxicity. I see it daily in my clinic. Functional medicine types are as guilty as the centralized allopathic MDs who believe BigHarma BS.
3. Mercury is prevalent in apex marine predators, and it can be a problem with eating these types of meats often. (tuna). However the lower in the food chain you go the "less prevalent it is". So eat your oysters as the Epi-paleo Rx tells you! The selenium in the seafood is what protects against metal poisoning. If you are worried, eat a Brazil nut every day because it is loaded with Se. Me, I'll pass on this. Hg is also cleared by melanin. It absorbs all heavy metals. When Melanin renovation is done it also raises the endogenous glutathione system in your live to act optimally. No functional practitioner wants to tell you this because it will dash their chances to sell you some magic supplement or potion from their closet/cellar. It is all BS.
Remember the battle for the Nobel Prize between the discovery of HIV from Montagnier versus Robert Gallo in 1983?
Do you know that Robert Gallo began studying HIV in the jungles of the Congo in chimpanzees during the oral polio virus trials.
The first oral polio vaccine was aerosolized oral spray that caused cancers all over the mouth and sinus system. A bioweapons company did the work for DoD and Robert Gallo was on that team. Things you just do not know when you do not know your history. Did you know that Fauci support the Nobel Prize for his friend Gallo over Luke Montagnier?
Coincidence or might it be related to why Reagan hired Fauci in 1982?
3. The first bioweapons lab on Magazine Street in NOLA and the work done at the US Public health hospital and Henry Clay Street in NOLA was transferred to the Tulane Primate Center in Covington Louisiana under the direction of Dr. Riopelle. When it became clear after the JFK killing in the mid 1960s that these simian viruses were behind many new human diseases like cancer and eventually HIV they were transferred to Fort Detrich as I laid out in my pod with RFK Jr. In the beginning Fort Detrich was an dual purpose base with the ARMY and CIA sharing thefacility and the bioweapons labs added to the mix as the science of Dr. Mary Stewart and Mary Sherman was developed by the CIA and industrial military complex.
This is why I wrote my hypoxia series. It explains why sunscreen, clothing, and glasses/contacts are the real problem in oxygen delivery. This affects heart attacks, fibromyalgia, and so many other diseases. You need UVA light exposure to run the mammalian cardiovascular system properly.
In trauma cases where I have used blood products I have always added in supplemental UVA light in the OR. No one knew what I was up to. This is why I have said forever that UV light exposure increase venous 02 saturation that helps lower tissue heteroplasmy rate. UVBI is something that should be standard of care in trauma centers.
Ever single thing Gates, dermatologists, and opthalmologists say about the SUN is 100% dead wrong. And they continue to say it because it makes them fiat money.
We run a three gas system and it turns out the free radical signal has a lot to with control of the oxidation state of iron.
Understanding the interaction of nitric oxide with RBCs is vital to elucidating the metabolic fate of NO in the vasculature. Because hemoglobin is the most abundant intraerythrocytic protein in RBCs and it reacts rapidly with NO, the interaction of NO with Hb has been studied extensively in the centralized literature. The data created has not been made sense of because we believe we already understand how we breath. This is why I wrote that Wim Hoff method of breathing is horribly flawed. Recent data has shown the NO reaction with RBCs is nearly 1,000-fold slower than the reaction with cell-free Hb.
The decentralized reality: The products of the NO Reaction with oxyHb Depend on local quantum mechanical concentrations of the environment. When that environment is altered by malillumination Dr. Kruse reaches for Methylene blue in his trauma patients. He also recommended it to his clients (Rick Rubin).
These are things "superstar centralized food and exercise guru" Peter Attia never knew before giving advice to Mr. Rubin and still does not understand the collateral effects.
2. The iron is itself attached on one side of the “heme pocket” to the amino acid residue histidine—the proximal histidine. Histidine is another aromatic amino acid that absorbs UV light. Another histidine is situated on the other side of the pocket. This second histidine is not directly linked to the ferrous atom and is called the distal histidine.
NO bonds to cysteine. This should make you realize why I wrote so eloquently about this amino acid as a photoswitch in the blogs.
Think of water as the ultimate molecular photoelectric adapter for life. Water’s behavior is a function of its microenvironment. An MRI is proof of this concept. Water looks different in different tissues of the human body, yet, it still remains H20 at a biochemical level of observation. This biochemical scale is not good enough to explain how life organizes. To understand how life makes use of water, you need a smaller scale: a quantum scale.
Water energy transfers are governed by something called “bandgap engineering.” This is a quantum mechanical action found in chemicals/proteins that are capable of semiconduction. Becker found this exact same mechanism in bone biology when he realized that two copper ions were the doping mechanism that rectified the photoelectric effect in the periosteum of bone to transfer bone’s photoelectric signal into a mechanical one for bone apposition and resorption.
Light and water also do the same thing as proteins like cysteine. It is the “potential” of the thiol groups in cysteine where your redox potential really lies. Cysteine is the rarest amino acid in nature, and this fact alone makes it an ideal signaling molecule in redox biochemistry. It links aging and neolithic disease risk, and it also marks where wellness begins and ends in us all.
3. The redox of the thiols in cysteine describes why blood works as it does.
Now that you have a basic idea of how a quantum cell works (see the November 2013 webinar), let’s discuss how this cell interacts with the first biologic protein force I mentioned in the Energy & Epigenetics #11 blog, the redox reactions in the Pentose Phosphate Pathway and beta-oxidation of fats.
If you go back and reread EMF4 blog, you will see that one of the major benefits of living within that pathway is to make NADPH that replenishes glutathione.
This pathway has only a few major endpoints, but they are all massively important for human life. It is designed to optimally restore RNA and DNA synthesis by providing maximum recycling of ATP; you now know the real physiologic function of ATP is to withdraw electrons from proteins’ electron clouds to cause them to fully expand and open its binding sites to water. This is Gilbert Ling’s work; he proved it all experimentally, yet no one listened to him.
The PPP also allows for the optimal formation of bio-energetic substrates to replenish the mitochondria’s ability to increase total adenine nucleotides to re-establish optimal ATP stores from beta-oxidation of fats. This is why we make 147 ATP from beta-oxidation of fats and only 36 ATP from carbohydrate metabolism. This difference is not just a calculation of ATP stoichiometry, it is a function of the electromagnetic spectrum energies during the seasons present on Earth as they change. It controls timing of metabolic pathways in use in our cells. NO time scales in our blood vessels is one such decision. I spoke about in the Vermont 2018 talk many times as the slides shows. Therefore, the electromagnetic spectrum of the sun carries information and energies of these seasonal factors directly to mitochondria. Mitochondrial proteins are used to decipher this “Seasonal Rosetta Stone” of energies.
After my Factor X webinar, people learned that the currency of survival for all eutherian mammals was a new biologic strategy to speed up DNA expression to adapt more quickly to the environment. POMC was that adaptation. Specifically how alpha MSH use changed in the mammalian family after sunlight was blocked by the asteroid.
The reasons eutherian mammals survived is because they use a placenta to birth their young. There is now strong evidence from molecular biology that the genes that coded for the evolution of the placenta were heavily selected for after the K-T event.
How do we know this?
The first mammals were rather old in an evolutionary scale, about 210 x10 to the 6th years before the present time (YBP). These early mammals (such as multituberculates) were small, furry, egg-laying, monotreme-like insect predators.
They were present prior to the advent of the dinosaurs, but became extinct about 35 x 10 to the 6th YBP. We know nothing about their molecular genomes, but what we do know, is that two other mammalian lines developed from them directly, the marsupials and the eutharian placentals.
Humans are in the later class. We also know that there is a massive phenotypic difference between chimps and man yet their genomes are almost identical. This is a clue that epigenetics is far more important to adaptative change and speciation than Darwin ever knew. This has huge implications for solving the modern disease epidemics. @GraduatedBen
How do we know placenta were heavily selected by a post K-T world? Well, the marsupials are only found in the southern hemisphere with the few exceptions. This fits perfectly with how Factor X occurred 65 million years ago as well as I laid out in my Webinar. Moreover, the placental mammals were far more successful because they radiated over 2000 genera, to about only 140 genera in the marsupial clade. @billgifford
Most of the surviving species left on the land of this planet is due to that formative moment in evolution. This is a big step in knowing who we really are and how we are built. Factor X, the KT event, was huge for any mammal that evolved after it. Why? It changed the way mammals used light because the sun was blocked.
In this way, the KT event mimics the advice of Bill Gates, dermotology, and opthalmology. Follow that advice and you'll face your own extinction event that will be preceded by modern chronic disease epidemics of diseases centralized medicine is becoming rich on.
That is the your inconvenient truth.
2. What is known as the “gut-first” — as opposed to “brain-first” — hypothesis states that Parkinson’s begins as abnormal proteins in the nerves of the gastrointestinal tract. While normal proteins fold into a specific three-dimensional shape, misfolded proteins fail to achieve this form.
What does brain first and gut hypothesis both miss?
BOTH CAN CAUSE PD because melanocytes migrate from the gut and the brain in their search UV light or lack there of. This is the story of the KT event collateral affects in the modern world.
CENTRALIZED SCIENCE is clueless about this reality.
Misfolded proteins, found excessively in the post-mortem brains of patients with Parkinson’s as well as Alzheimer’s, accumulate into large, toxic clumps that disrupt nerve cell function.
Anyone who has read my Quantum Engineering series on Patreon should get this insight rapidly. Melanocytes move when no UV light stimulus is present. They are looking for the light by design.
3. The link between Parkinson’s, traditionally considered a brain disorder, and the gastrointestinal tract is not new. The first detailed description of Parkinson’s disease, was published in 1817 by British physician James Parkinson. It is an interesting paper because bowel constitpation was described in the case reports. No one had an explanation for it until I did the Tetragrammaton podcast with Rick and huberman.
Based on his initial observation of six cases, Parkinson found that a patient starts by experiencing a slight sense of weakness and some bothersome trembling in one of their hands.
Constipation was also present in most of the patients and subsequently early was considered a risk factor for Parkinson’s disease development. This makes sense when you understand how much dopamine and melanin derivative are present in the human gut. Parkinson had no way of knowing that, but today's gastroenterologists and neurologists do not suffer from this lack of knowledge. They suffer from a lack of decentralized learning and connection of the effect of light in the gut and brain. Constipation remains one of the most prevalent non-motor symptoms, affecting up to two-thirds of all patients. Even Parkinson, in his long-ago essay, wrote that the patient’s bowels often “demand stimulating medicines of very considerable power.”
Any patient I see with PD is told about the POMC affect in the surface skin above the rectus abdominus muscle and how it affects the melanin levels in the neural crest derivatives that are filled with melanin in the splanchnic nerves. It is also why many patients on their way to diseases have melanosis coli when they have a colonoscopy. Most GI doctors have no idea that this finding is a pre malignant issue or an issue related to an impended neurodegenerative disease. Now you do.
During wakefullness our colony of mitochondria are predominantly using paramagnetic gases like oxygen and nitric oxide to live. At night time we are using diamagnetic atoms to sleep. The same thing can be found in anesthetics. @LucaTurin has been working at this puzzle for sometime. Becker opened the door with his use of magnets to put salamanders asleep with his limb regeneration studies. The key mechanism in Nature that disturbs quantum entanglement in a warm wet environment is how these atoms are used. This is what has Nick Lane's attention now but he has not put it all together as yet.
Remember in the melanin series of blogs I told you how I hacked the periodic table. This was a topic I covered in my own work 15 years ago. It is nice to see some brilliant guys like Turin and Lane work on it now.
How to tell if a substance is paramagnetic or diamagnetic. The magnetic form of a substance can be determined by examining its electron configuration: if it shows unpaired electrons, then the substance is paramagnetic; if all electrons are paired, the substance is diamagnetic.
The magnetic properties of atoms depend on whether they have unpaired electrons. Sulfur is paramagnetic, it has two unpaired electrons. Xenon is diamagnetic, due to its full shell of paired valence electrons. Melanin has some of the most unusual electric and magnetic properties in mammals. It is tied to what Becker found and it is also tied to the current work of Michael Levin at Tufts now.
2. This idea has long roots all the way back to the time of Gurwitsch and Warburg. None of your food gurus go down these rabbit holes for you.
VLECK, J. The Theory of the Paramagnetism of Oxygen and Nitric Oxide. Nature119, 670 (1927). doi.org/10.1038/119670…
3. Life, when it innovated consciousness also used specific elements to do it. Remember what I said in the COld Thermogenesis series. Sleep was the default state of life and we evolved wakefulness. How did we do it? We harnessed magnetic flux from the sun to do it. The paramagnetic atoms on the periodic table that are all favored by life all contain unpaired electrons; this means the details of life will be found in the electrons life uses. Thus, the paramagnetic atoms from Z = 1 to Z = 20 are: H, Li, B, C, N, O, F, Na, Al, Si, P, S, Cl, K.