The SILENT PANDEMIC: Emergent ANTIBIOTIC RESISTANCES following the global response to SARS-CoV-2
SUMMARY :
The overuse and misuse of antibiotics during the pandemic may lead to the development of resistant bacterial strains. This is particularly concerning as ...
2) ... antibiotic resistance is a growing global threat and could lead to a silent pandemic that is just as deadly as COVID-19. The study emphasizes the need for appropriate use of antibiotics and the development of new antimicrobial therapies to combat emerging resistant strains
4) Fig. Antimicrobial resistance and COVID-19 mortalities
5) Fig. Antibiotic therapeutic resistance and bacterial co-infections
6) DISCUSSION
The increased use of antibiotic therapeutics during this pandemic is anticipated to result in the rapid emergence of novel, deadly, AMR pathogens. This crisis will only be mitigated by an active communication to the public about the risk of these infections
NIPAH VIRUS
Jumping to conclusions during outbreaks only fuels panic and misunderstanding; NOT EVERY VIRUS OUTBREAK is a PANDEMIC.
We raised the alarm about Nipah in 2023, but it’s essential not to confuse an epidemic related to this virus, ...
2) ...like the one in India, with a pandemic risk or a situation like COVID-19.
Nipah virus is transmitted primarily from animals, especially bats, to humans. It spreads through direct contact with infected animals, their secretions, or contaminated foods.
3) Human-to-human transmission occurs only through close contact, such as touching or caring for an infected person, as well as sharing fluids like saliva or blood.
We cannot equate a virus transmitted through fluids with a respiratory virus like SARS-CoV-2.
"70% to 90% of our CELLS are completely RENEWED in less than 5 YEARS!
So If we’re mostly "New", why are so many still struggling with SARS-CoV-2?
We will tackle this question, which is more complex than it seems, in several posts.
2) First, let's say a few words about cell renewal.
70% to 90% of our cells are renewed over time. This turnover occurs in various cell types, including skin, blood, and immune cells, allowing the body to replace damaged or aged cells. sciencefocus.com/the-human-body…
3) For instance, red blood cells have a lifespan of about 120 days, while skin cells regenerate every few weeks.
Even with significant cell renewal, aging persists due to changes in our DNA. sciencefocus.com/the-human-body…
2) Co-infection occurs when a person is infected by more than one virus at the same time. This can lead to interesting and sometimes complex interactions between the viruses
3) In one study about SARS-CoV-2, researchers found that when different versions of this virus infect the same person, they can mix their genetic material through a process called recombination. This means that the new version of the virus can have traits from both parent viruses
For more than three years, we have been emphasizing that the envelope protein is an essential component and that we cannot limit our focus to just the spike protein. We are now uncovering its role in long COVID.
3) The Envelope (E) protein of SARS-CoV-2 plays a crucial role for several reasons:
▶️ Virus Assembly and Release: The E protein is essential for the virus's assembly and budding from infected cells, contributing to its stability and infectivity.
In 2022, COVID-19 was the second leading cause of death globally, with the repercussions of the virus far from over. The lasting impact of the pandemic continues to affect lives around the world.
Recent research may have identified a key factor contributing to long COVID: microscopic clots intertwined with immune system debris in the blood. These unusual structures were found to be nearly 20 times more prevalent ...
2) ...in long COVID patients compared to healthy individuals. The clots, associated with neutrophil extracellular traps (NETs), suggest a potential biological marker for persistent symptoms.
3) These microclots could impede blood flow in small vessels, leading to issues like brain fog, fatigue, and shortness of breath.