1/THE LNP/mRNA VACCINE KILLS THE CELL IT TRANSFECTS AND CREATES DANGEROUS INFLAMMATION. Imagine that it transfects cells that do not regenerate and you have no control over that. This is not safe for children especially. Pfizer admits that the cell dies. See thread.
2/The Pfizer diagrams that are in their marketing material admit that the cell dies after it has produced the Spike Protein. Diagrams are showing that some Spike is released first in exosomes and later the cell dies when Spike is consumed by macrophages that consume dead cells.
3/ Natural cell death apoptosis (cell is worn out and dies) or necrosis (caused by natural trauma) causes cell to blebb, contents protrude and leak out the cell membrane. This shows the process. And explanation provided. Spike protein is the contents. https://t.co/R0949ioL1wptglab.com/news/blog/what…
4/ Electron microscopy in this study shows exosomes of Spike Protein are formed. Instead of natural blebbing, encapsulted Spike is formed and expelled. Exosomes can be expelled fluids and in the breath. So guess what? Shedding has an real explanation. https://t.co/Zlf2ZbLYQHjournals.aai.org/jimmunol/artic…
5/ Manipulating a cell to produce a foreign protein has never been done so there isn't a totally accurate description of how the cell dies. I believe that necroptosis is probably the closest. It is highly INFLAMMATORY so everyone getting the jab is getting sick by definition.
6/Now think about that you know that Pfizer knows that it kills cells. LNPs go everywhere that it can be picked up by the blood stream. When you kill some cells, they are gone forever. All you get is scar tissue.. https://t.co/N7NHdQJqr2ncbi.nlm.nih.gov/pmc/articles/P…
7/ What about brain cells? Do you even want to think about killing your child's brain cells and whether they will re-generate? They may re-generate with time and under certain parameters but who wants to even go there. Astounding stupid! centreofthecell.org/blog/science-q…
8/You can take it from here. And know that they know that the mRNA vaccine format kills all transfected cells, which make the Spike Protein. This is INSANITY!!!
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We cannot allow data from any clinical trials outside the United States to be used for any licensing of any product to be marketed in the US. We have no jurisdiction over foreign physicians. There is no consequence for their malfeasance or incompetence.
For those who have followed me from real name, Ducky, to Clucky, you know that I have been telling you for 4 years that Sars2 is a vector vaccine. It is not a generic base but has been modified to be for HIV and contains a biological adjuvant specific to Chinese alleles. See👇
1/Furin cleavage insert PRRAR: All viruses must utilize protease cleavage in order to gain entry to a cell and replicate. Diagram 1 = sequences of similar viruses. Diagram 2 = PRRAR was positioned upstream of the cleavage sites of Sars1. RRAR is not optimal and needs TMPRSS.
2/There are both extracellular and intracellular proteases. Viruses have protease cleavage sites in their genome. Epigenetics creates viruses that use the location of the hosts proteases to adapt to replicating within different hosts.
SPURIOUS (FALSE) HYPOXEMIA: A previously (prior to Covid) known phenomena occurring when patients with leukocytosis or thrombocytosis have low PaO2 on arterial blood gas analysis (ABG) but actual oxygen saturation is normal. Who told hospitals to ventilate without investigation?
Example #1 - 2013: Spurious hypoxemia occurs when patients with leukocytosis or thrombocytosis have low PaO2 on arterial blood gas analysis (ABG), but actual oxygen saturation is normal. .journal.chestnet.org/article/S0012-…
Example #2 - 2005: Failure to recognize spurious hypoxemia can lead to unnecessary diagnostic tests and therapeutic interventions, exposing patients to avoidable risk. A diagnostic algorithm is proposed.pubmed.ncbi.nlm.nih.gov/16096465/
@JosephFraiman
1/Dr. Fraiman asks "Why do the mRNA vaccines cause myocarditis? Seriously does anyone understand the mechanism?" And the anonymous chicken answers. See the thread underneath the response.
@JosephFraiman 2/It doesn't happen to everyone. INFLAMMATION: occurs in those with gene polymorphisms in the IL-1 gene cluster (IL-1α, IL-1β, and IL-1 receptor antagonist) that result in greater expression of the agonists or reduced expression of the antagonist.ncbi.nlm.nih.gov/pmc/articles/P…
@JosephFraiman 3/Prolong inflammatory state will result in amyloid fibrosis of the heart muscle from the misfolded proteins. academic.oup.com/ckj/article/16…
@JosephFraiman 2/Then the role of IL-1 in heart disease. "Anakinra is a recombinant human interleukin-1 competitive receptor antagonist that blocks the biologic effects of interleukin-1, thereby reducing systemic inflammatory responses." ncbi.nlm.nih.gov/pmc/articles/P…
@JosephFraiman 3/Prolong inflammatory state will result in amyloid fibrosis of the heart muscle from the misfolded proteins. academic.oup.com/ckj/article/16…
@JosephFraiman Inflammatory cytokines Interleukin 1 and Interleukin 6 induce intracellular production of Amyloid A in cardiac tissues and vessels. Then clinically it manifests as "stiff heart syndrome." link.springer.com/article/10.100…
1/Anyone with a pharma background that is not bought could have told you the simple science of why you know these things. First thing you do when you meet a new virus, is to find out if it is similar to one we know about. Yes, Sars2 was 78% similar to Sars1. Next.
2/ Then you read all the Sars1 literature that you can find which revealed. It was a seemingly respiratory virus with strange happenings related to blood coagulation i.e. microclots. Death due to pneumonia, diffuse alveolar damage, hyaline membrane, edema (lungs filled up).
3/Blood profiles: High cytokine levels which were divergent in children vs. adults. Children produced high IL-1B but not IL-6; adults produced high IL-6 but not IL-1B.