We cannot allow data from any clinical trials outside the United States to be used for any licensing of any product to be marketed in the US. We have no jurisdiction over foreign physicians. There is no consequence for their malfeasance or incompetence. Here is Argentina.

For those who have followed me from real name, Ducky, to Clucky, you know that I have been telling you for 4 years that Sars2 is a vector vaccine. It is not a generic base but has been modified to be for HIV and contains a biological adjuvant specific to Chinese alleles. See👇

https://twitter.com/TheChiefNerd/status/1798737025593684114

1/Furin cleavage insert PRRAR: All viruses must utilize protease cleavage in order to gain entry to a cell and replicate. Diagram 1 = sequences of similar viruses. Diagram 2 = PRRAR was positioned upstream of the cleavage sites of Sars1. RRAR is not optimal and needs TMPRSS.

SPURIOUS (FALSE) HYPOXEMIA: A previously (prior to Covid) known phenomena occurring when patients with leukocytosis or thrombocytosis have low PaO2 on arterial blood gas analysis (ABG) but actual oxygen saturation is normal. Who told hospitals to ventilate without investigation?

https://twitter.com/michaelpsenger/status/1658646420541550592

Example #1 - 2013: Spurious hypoxemia occurs when patients with leukocytosis or thrombocytosis have low PaO2 on arterial blood gas analysis (ABG), but actual oxygen saturation is normal. .journal.chestnet.org/article/S0012-…

@JosephFraiman
1/Dr. Fraiman asks "Why do the mRNA vaccines cause myocarditis? Seriously does anyone understand the mechanism?" And the anonymous chicken answers. See the thread underneath the response. @JosephFraiman 2/It doesn't happen to everyone. INFLAMMATION: occurs in those with gene polymorphisms in the IL-1 gene cluster (IL-1α, IL-1β, and IL-1 receptor antagonist) that result in greater expression of the agonists or reduced expression of the antagonist.ncbi.nlm.nih.gov/pmc/articles/P…

@JosephFraiman 2/Then the role of IL-1 in heart disease. "Anakinra is a recombinant human interleukin-1 competitive receptor antagonist that blocks the biologic effects of interleukin-1, thereby reducing systemic inflammatory responses." ncbi.nlm.nih.gov/pmc/articles/P… @JosephFraiman 3/Prolong inflammatory state will result in amyloid fibrosis of the heart muscle from the misfolded proteins. academic.oup.com/ckj/article/16…

1/THE LNP/mRNA VACCINE KILLS THE CELL IT TRANSFECTS AND CREATES DANGEROUS INFLAMMATION. Imagine that it transfects cells that do not regenerate and you have no control over that. This is not safe for children especially. Pfizer admits that the cell dies. See thread. 2/The Pfizer diagrams that are in their marketing material admit that the cell dies after it has produced the Spike Protein. Diagrams are showing that some Spike is released first in exosomes and later the cell dies when Spike is consumed by macrophages that consume dead cells.

1/Anyone with a pharma background that is not bought could have told you the simple science of why you know these things. First thing you do when you meet a new virus, is to find out if it is similar to one we know about. Yes, Sars2 was 78% similar to Sars1. Next.

2/ Then you read all the Sars1 literature that you can find which revealed. It was a seemingly respiratory virus with strange happenings related to blood coagulation i.e. microclots. Death due to pneumonia, diffuse alveolar damage, hyaline membrane, edema (lungs filled up).

S1 is expelled at the point of cleavage by furin/TMPRSS. This was one of my "Ducky Diagrams" 2 years ago. Nobody agreed then. I am often alone for a while. I heard Dr. Ryan Cole mention this in his interview with Dr Joseph Mercola. Now I will go back without feeling so lonely. So I am going to start at the end and show how I came to the conclusion. As of Mar 2022 we know that the cytokine pattern of severe disease is consistent with what is produced by the monocyte. And that S1 is the inflammatory agent for that response. ncbi.nlm.nih.gov/pmc/articles/P…

How you know that this is just a "pure and simple" failed 💉design & variants have nothing to do with it? If you select a Spike sequence design, your target epitopes will be the proteins of the receptor binding domain which is the place where the virus binds to ACE2 receptors.1/ There are 4 immunogenic epitopes in the receptor binding domain IDD IDE IDF & IDG expected to cause an antibody response which would "block" the virus from gaining entry to cells. We know that the Spike v@xes do NOT work to prevent infection so these antibodies are useless.2/

1/QTNSPRRAR: peptide immunotherapy vaccine adjuvant, a biological enhancer of CTL instead of a chemical enhancer for a vector (coronavirus) vaccine for HIV (evidence of HIV sequence inserts). Start w/2004 development of peptide immunotherapy for cancer. aacrjournals.org/clincancerres/… 2/ Some traditional vaccines have adjuvants and some do not. cdc.gov/vaccinesafety/…

1/The parallels between COVID and snake venom aren't what you think. There is an explanation and it is scientific but it does not have anything to do with there being snake venom in the protein sequence. I do not find any protein homology to Sars2. See this thread. 👇 2/Yes, there is an enzyme, a serine protease, called elastase that is associated with acute lung injury. Elastase breaks down elastin. In your body, there is digestive elastase (pancreas) and neutrophil elastase (immune system) - Same enzyme different functions.

1) I want to tell you something that we are missing about the LNP (lipid nanoparticle) and mRNA translation. The LNP stays intact any where in the body at neutral pH. It would be unless if it ruptured before it reached the ribosome. The LNP can pass through any cell membrane. 2) The LPN upon passing through the cell membrane without puncturing it or causing cell material to be dumped into the extracellar space, it passes into the cytosol which has a pH of 7 and contains the ribosomes. But wait, it doesn't release the mRNA there because of pH.