1/THE LNP/mRNA VACCINE KILLS THE CELL IT TRANSFECTS AND CREATES DANGEROUS INFLAMMATION. Imagine that it transfects cells that do not regenerate and you have no control over that. This is not safe for children especially. Pfizer admits that the cell dies. See thread.
2/The Pfizer diagrams that are in their marketing material admit that the cell dies after it has produced the Spike Protein. Diagrams are showing that some Spike is released first in exosomes and later the cell dies when Spike is consumed by macrophages that consume dead cells.
3/ Natural cell death apoptosis (cell is worn out and dies) or necrosis (caused by natural trauma) causes cell to blebb, contents protrude and leak out the cell membrane. This shows the process. And explanation provided. Spike protein is the contents. https://t.co/R0949ioL1wptglab.com/news/blog/what…
4/ Electron microscopy in this study shows exosomes of Spike Protein are formed. Instead of natural blebbing, encapsulted Spike is formed and expelled. Exosomes can be expelled fluids and in the breath. So guess what? Shedding has an real explanation. https://t.co/Zlf2ZbLYQHjournals.aai.org/jimmunol/artic…
5/ Manipulating a cell to produce a foreign protein has never been done so there isn't a totally accurate description of how the cell dies. I believe that necroptosis is probably the closest. It is highly INFLAMMATORY so everyone getting the jab is getting sick by definition.
6/Now think about that you know that Pfizer knows that it kills cells. LNPs go everywhere that it can be picked up by the blood stream. When you kill some cells, they are gone forever. All you get is scar tissue.. https://t.co/N7NHdQJqr2ncbi.nlm.nih.gov/pmc/articles/P…
7/ What about brain cells? Do you even want to think about killing your child's brain cells and whether they will re-generate? They may re-generate with time and under certain parameters but who wants to even go there. Astounding stupid! centreofthecell.org/blog/science-q…
8/You can take it from here. And know that they know that the mRNA vaccine format kills all transfected cells, which make the Spike Protein. This is INSANITY!!!
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1/Anyone with a pharma background that is not bought could have told you the simple science of why you know these things. First thing you do when you meet a new virus, is to find out if it is similar to one we know about. Yes, Sars2 was 78% similar to Sars1. Next.
2/ Then you read all the Sars1 literature that you can find which revealed. It was a seemingly respiratory virus with strange happenings related to blood coagulation i.e. microclots. Death due to pneumonia, diffuse alveolar damage, hyaline membrane, edema (lungs filled up).
3/Blood profiles: High cytokine levels which were divergent in children vs. adults. Children produced high IL-1B but not IL-6; adults produced high IL-6 but not IL-1B.
S1 is expelled at the point of cleavage by furin/TMPRSS. This was one of my "Ducky Diagrams" 2 years ago. Nobody agreed then. I am often alone for a while. I heard Dr. Ryan Cole mention this in his interview with Dr Joseph Mercola. Now I will go back without feeling so lonely.
So I am going to start at the end and show how I came to the conclusion. As of Mar 2022 we know that the cytokine pattern of severe disease is consistent with what is produced by the monocyte. And that S1 is the inflammatory agent for that response. ncbi.nlm.nih.gov/pmc/articles/P…
We could hypothesis that S1 which stimulates cytokine response in monocytes has some areas that set off this excessive response in SOME people. I would look at the NTD epitopes. And compare to the Pradhan HIV inserts.
How you know that this is just a "pure and simple" failed 💉design & variants have nothing to do with it? If you select a Spike sequence design, your target epitopes will be the proteins of the receptor binding domain which is the place where the virus binds to ACE2 receptors.1/
There are 4 immunogenic epitopes in the receptor binding domain IDD IDE IDF & IDG expected to cause an antibody response which would "block" the virus from gaining entry to cells. We know that the Spike v@xes do NOT work to prevent infection so these antibodies are useless.2/
Pharma wants to blame the failure on mutations that are said to evade the antibodies- happens but not in this case. They want to make a new version of the failed design. Below is comparing alpha to omicron to see whether the mutation is in an epitope to see immune response. 3/
1/QTNSPRRAR: peptide immunotherapy vaccine adjuvant, a biological enhancer of CTL instead of a chemical enhancer for a vector (coronavirus) vaccine for HIV (evidence of HIV sequence inserts). Start w/2004 development of peptide immunotherapy for cancer. aacrjournals.org/clincancerres/…
3/ Why were they studying peptide immunotherapy for cancer? They were looking for a way to stimulate cytotoxic lymphocytes against cancer cells, i.e. make a vaccine. Category is "mainly" CD8 cells which are activated by MHC-1. So next I show you the bioinformatics on QTNSPRRAR
1/The parallels between COVID and snake venom aren't what you think. There is an explanation and it is scientific but it does not have anything to do with there being snake venom in the protein sequence. I do not find any protein homology to Sars2. See this thread. 👇
2/Yes, there is an enzyme, a serine protease, called elastase that is associated with acute lung injury. Elastase breaks down elastin. In your body, there is digestive elastase (pancreas) and neutrophil elastase (immune system) - Same enzyme different functions.
3/ Known since before Sars1 that neutrophil elastase causes severe lung injury. It damages the tissue, literally destroys the tight junctions between cells. Imagine lung tissue that allows fluids and air to flow in between cells. How could you breathe? pubmed.ncbi.nlm.nih.gov/12223222/
1) I want to tell you something that we are missing about the LNP (lipid nanoparticle) and mRNA translation. The LNP stays intact any where in the body at neutral pH. It would be unless if it ruptured before it reached the ribosome. The LNP can pass through any cell membrane.
2) The LPN upon passing through the cell membrane without puncturing it or causing cell material to be dumped into the extracellar space, it passes into the cytosol which has a pH of 7 and contains the ribosomes. But wait, it doesn't release the mRNA there because of pH.
3) The LNP doesn't rupture unless it reaches the acid areas of the cell which are the endosome and the lysosome. So it actually dumps the mRNA into the areas where there are no ribosomes.