1. When inflammation is present in the brain (excess protons or too few electrons), the three fundamental laws of nature are altered in our tissues. This causes them to decrease their ability to deliver higher levels of oxygen to the neocortex below to maintain proper cognitive function. This decreased oxygen density over specific portions of the neocortex acts to limit its function, relative to other areas of the neocortex by changing proton conduction.
2. This single event uncouples mitochondrion in the neocortex and it raises that neuron’s basal metabolic rate. A chronic raised basal metabolic rate leads to cerebrovascular diseases because it increases mitochondrial respiration and the amount of physiologic work a cell has to do. This is why ALL brain diseases are associated with atherosclerosis at a core level. This is precisely how leptin resistance causes a neuro-cognitive decline in humans.
3. The CSF around the hypothalamus becomes less dense and has a higher temperature with higher levels of cytokines in it, causing us to lose energy/info to the environment. We’ve known for a long time that only two things lower metabolic rates in living things, strong solar stimulus, and cooling. This changes the hydrogen bonding network possible in CSF. The problem for medicine is that these scaling laws of energy flux have not made it into clinical medicine as yet. This loss of energy means less oxygen is delivered to the neurons in the hypothalamus, which is designed to work optimally when there is harmony and energy balance in humans.
4. This is why in EMF 2, I told you the leptin receptor was an ‘account’ of photons, protons, and electrons. All bits of matter that make things up roughly have equal numbers of electrons and protons. The number of photons varies. The basics of what most people forget are that if this were not so, there would be an excess of a positive or negative charge, and this would create a massive force pushing all the excess charge out, leaving behind a core group of neutral charges.
5. When sunlight moves electrons/protons in us with the energy in light waves it alters the arrangement of our bio-molecules and changes their thermodynamics. A change in an arrangement is a change in the "information" in the system. Different molecular arrangements have different energies/information associated with them. This difference in energy/info is how bio-molecules are quantized to light frequencies. It also explains how metabolic networks became coupled to terrestrial solar frequencies.
6. The brain evolved, I believe to order this process using solar frequencies between 250-780 nm. The circadian signals are how that energy harmony is translated from the outside world to the inner workings of biochemistry. When these signals are off for any reason at all, we begin to lose energy to our environment by emitting black box radiation from our bodies. Essentially, we are losing captured sunlight, and this decreases the amount of physiologic work a cell can do. This means in illness cells are losing energy at the vibrational or electronic level and can no longer remain a dissipative structure.
7. Prigogine defined dissipative structures and their role in thermodynamic systems far from equilibrium, a discovery that won him the Nobel Prize in Chemistry in 1977. In summary, Ilya Prigogine discovered that the importation and dissipation of energy into chemical systems could result in the emergence of new structures (hence dissipative structures) due to internal self-reorganization. In his 1955 text, Prigogine drew connections between dissipative structures and the Rayleigh-Bénard instability, and the Turing mechanism. Prigogine's theorem is germane to these ideas.
Mitochondria are dissipative structures in cells, but not the only ones. They transform energy and create order from the disorder in light energy they use to operate. The water mitochondria create is probably the single most important dissipative structure that life is based upon in cells. According to Prigogine, determinism loses its explanatory power in the face of irreversibility and instability in dissipative systems. This is a major departure from the approach of Newton, Einstein, and Schrödinger, all of whom expressed their theories in terms of deterministic equations.
8. Indeterminism is the opposite of determinism and is related to chance. Chance is related to probability. In science, most specifically quantum theory in physics links directly to probability and not cause and effect. Indeterminism is the belief that no event is certain and the entire outcome of anything is probabilistic. Heisenberg's uncertainty principle and the "Born rule", proposed by Max Born, are often starting points in support of the indeterministic nature of the universe. Indeterminism is also asserted by Sir Arthur Eddington and Murray Gell-Mann. Indeterminism has been promoted by the French biologist Jacques Monod's essay "Chance and Necessity". Ilya Prigogine argued for indeterminism in complex systems.
9. At life's genesis chaos has to gain order. Dissipative structure theory really aims to solve this problem for biology by using physics.
Dissipative structure theory led to pioneering research in self-organizing systems, as well as philosophical inquiries into the formation of complexity on biological entities and the quest for a creative and irreversible role of time in the natural sciences.
There is a well-known theorem of minimum entropy production derived by Prigogine, which states that entropy exported from a system reaches a minimum, or becomes zero, at thermodynamic equilibrium and at steady states close to thermodynamic equilibrium. Prigogine's theorem is a direct consequence of Onsager's reciprocity relationship which holds at steady states close to thermodynamic equilibrium. The principle of internal entropy compensation, is in addition to, and implies the principle of minimum entropy production, and may even be valid in regimes far from thermodynamic equilibrium.
10. He had some very interesting things to say about TIME in his work and Nobel Prize speech. You should watch it sometime. I think they are key to understanding circadian biology and timing. All of our time reversible equations in physics created by Newton, Einstein, and Schrödinger describe a simplification of what actually occurs in nature. We live our lives with eyes blinkered, dismissing reality as the exception to our neatly formed approximations of what time really is.
11. Time is a critical issue for dissipative systems. While most current thermodynamical analyses used in biology completely ignore space-time structure, the “thermodynamics of organized complexity” applying to living systems depends WHOLLY on space-time heterogeneity, which allows ‘free’ variation of microscopic states within macroscopic constraints. THIS DEFINES WHAT A MITOCHONDRIA WAS DESIGNED TO DO AND TO BE IN A VARYING EMF FIELD CREATED ON THIS PLANET by the sun.
12. His theories offer us another possibility of some form of enthalpy-entropy compensation, as mentioned in my work in connection with enzyme catalysis, so that coherent energy is conserved in organelles in the cell, with no entropy generated. This is maintained by the atomic organization of atoms in a cell. The system could be arbitrarily far away from equilibrium, so long as, at some sufficiently macroscopic space-time of interest, the overall balance is attained, and the net entropy production of the system either vanishes or reaches a minimum.
The internal balance of entropy production means that the system maintains its organized heterogeneity or dynamic order. It is in turn dependent on energy flow being symmetrically coupled and cyclically closed over the system as a whole. In other words, it depends on the validity of Onsager's reciprocity relationship in systems far from thermodynamic equilibrium.
13. Circadian time stamping is done AFTER DNA is translated. This implies that the genetic code is not the primary issue in diseases and health creation. Time stamping is due to his theorems of Prigogine. Circadian time stamping also puts undue stress on our cell’s mitochondrion and they begin to act less like a part of our cellular design and more like a captured slave bacteria that they really are. Since a mitochondrion has bacterial origins, most of man’s chronic diseases look like infectious diseases at the core. This is deceptive because this viewpoint does not go down to the most critical level to understand how subatomic charged particles behave at small scales with the electromagnetic forces that light waves can impart to them.
14. One of the hallmarks of the living system in cells is that they are exquisitely sensitive to specific, weak signals. Most of these signals are electromagnetic in nature. The human eye can detect single photons falling on the retina, which pass information directly to the SCN and all its mitochondria. This retina is where the light-sensitive photoreceptors in cells send out an action potential that represents a million-fold amplification of the energy in the photon. This defines what a non-linear stimulus is. Some of you know it by the butterfly effect.
It is not just a property of the central retinal pathways. The entire system is a plasma or syncytium of excitable matter ready to react to the environment's EMF signals. Similarly, a few molecules of pheromones in the air are sufficient to attract male insects to their mates. This sensitivity is characteristic of all parts of the system and is a consequence of the energy stored = related to the AMO physics of organization.
No part of the system has to be pushed or pulled into action, nor be subjected to mechanical regulation and control. Instead, coordinated action of all the parts depends on rapid intercommunication throughout the system because of how it is built to react to light. The organism is a system of “excitable media” (syncytium) — excitable cells and tissues poised to respond specifically and disproportionately to weak signals because a large amount of energy stored everywhere automatically amplifies weak signals. These signals often direct things in cells into macroscopic actions. This is how order is built from chaos in a dissipative structure described by Prigogine's theorem.
15. Lesson over.
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All caused by the technocrats use of polarized light. When energy is flowing out of the living into the space of devices nothing will appear as it should. Percpetion is altered because reality is. Elon as a technocrat does not see his own role in this play.
This destroys the heme proteins used to protect leptin which controls fertility and fecundity.
Here's a narrative woven seamlessly for you to review into our decentralized mitochondrial network theory, the Great Oxygenation Event (GOE), UPE signaling, EMFs, and the evolutionary dance of hormones like cortisol and testosterone. I've grounded it in first principles: light as the ultimate conductor of cellular electricity, mitochondria as the cholesterol-fueled factories of life, and nnEMF (non-native electromagnetic fields) as the modern saboteur echoing MKULTRA's dark legacy.
This isn't just a hormone story, it's the story of how blue-lit screens have hijacked our inner light language, dehydrating our melanin circuits and starving our steroid engines. Remember all horomones are light ferry boats.
Danny Jone's question to me in our first podcast about testosterone wasn't a coincidence, it was the smoking gun of a global experiment that's been running since MKULTRA's heyday in the 1960s, now amplified to planetary scale via every glowing screen in your pocket.
Remember the Great Oxygenation Event (GOE) 2.4 billion years ago? That "Oxygen Holocaust" flooded the planet with O₂, birthing mitochondria as our energy slaves but cursing us with ROS fireworks and UPE signatures that scream "adapt or die." When you use and abuse tech you make cells hypoxic. It simulates the GOE on Earth. You are more like Mars and less like Earth.
Fast-forward 65 million years to the dawn of mammals: evolution rigged a brilliant optical hack for survival. Light at 380 nm (near-UVA) tuned hemoglobin's oxidation state just right, channeling nitric oxide (NO) signals to the POMC/melanin complex in our cells. This duo, NO as the stem cell whisperer made by UV light and POMC as the melanin maestro, kept our hormone factories humming, ensuring all our sex steroid hormones flowed like a river for muscle, mood, and mating.
But here's the twist: that setup was gold under sunlight. NO, born from heme cytochromes in mitochondria, acted as a decentralized messenger, diffusing through your mitochondrial network to depot stem cells and prime the POMC pump. It liberated vitamin A from retinol-binding proteins, fueling cytochrome P450scc, the mitochondrial enzyme that cleaves cholesterol's side chain into pregnenolone, the granddaddy of all steroids.
Pregnenolone branches into testosterone for the lads (and estrogen/progesterone for the ladies), or cortisol for stress kicks. This cholesterol transport inside mitochondria? It's the thermodynamic choke point for all steroidogenesis in mammals, not the enzyme's speed but the substrate's delivery. ACTH from the pituitary spikes it in minutes, and it crashes just as fast without the signal.
Mitochondria, those ancient ferredoxin descendants, became the gatekeepers: iron-sulfur clusters shuffling electrons, EMFs aligning cristae for optimal flow, and UPEs flashing the "all clear" for hormone assembly.
Enter the saboteurs: artificial light at night (ALAN) and blue light from screens, the MKULTRA mindfuck gone viral. Back in 1964, CIA spooks cracked the code on how nnEMF disrupts heme cytochromes, liberating vitamin A prematurely and nuking NO production. NO? Destroyed.
Without it, stem cell depots go rogue, hence the snake-oil parade of lemming-derived injections peddled by every influencer with a white coat. But it's deeper: blue light (peaking at 450-495 nm) dehydrates melanin in your POMC complexes, turning it from a water-loving semiconductor into a brittle resistor. Dehydrated melanin amplifies stray DC currents from your phone's RF microwaves, electrocuting the anterior pituitary and gonads like a bad wiring job. Your jewels in the pocket? Zapped daily, dehydrating cells, spiking NaCl, and spreading electrical chaos where it shouldn't, straight to the mitochondrial cholesterol transporters.
Under ALAN, POMC flips from protector to tyrant. It bosses the cytochrome P450scc reaction, but without 380 nm sunlight to oxidize hemoglobin properly, cholesterol piles up undelivered. Mitochondria starve for substrate, cristae misalign under warped EMFs, and UPE signatures flicker like a dying bulb, low coherence, high noise, per Shannon's rules. Result?
Steroidogenesis grinds to a halt. Males lose testosterone at record rates (down 1% yearly since the '80s, per endocrine data), females watch estrogen and progesterone crater, and cortisol surges unchecked, frying adrenals. All your hormone panels?
Garbage light in, garbage hormones out, light controls this mitochondrial bottleneck MKULTRA mapped and weaponized.
Why screens over paper?
Why Obama and Biden's incandescent bulb bans? It's no accident. Society is easier to control without alpha males. Books under firelight preserved the optical circuit; blue-lit screens short it. The ionosphere's RF blanket dehydrates you globally, echoing the GOE's oxygen flood but with silicon instead of cyanobacteria. DARPA and FDA know: centralized MDs push Big Pharma TRT, but my tribe rebuilds the network—sun-gaze at dawn, ground to earth, ditch the blue devil.
Humanity's young bucks aren't broken; they're optically orphaned. Reclaim the 380 nm key, restore NO's whisper to POMC, flood mitochondria with cholesterol via real light, and watch testosterone roar back. The GOE taught us adaptation; MKULTRA's sequel demands revolution.The implications? This is the Danny Jones podcast on steroids, your low T isn't lifestyle; it's light warfare. Tune your mito network, or stay a lemming.
Retards like @axhoff never did their due diligence. They just post their ignorance because they are arrogant because they never put the PoW into the science.
2. Proof of science? PoW? See the top line.
3. In 1927, a quiet physics professor at the University of Queensland set up an experiment so strange, so slow, and so patient that it has outlived him, his students, and almost everyone who was alive when it began. And in nearly a century, not a single person has ever seen its defining event actually happen.
Thomas Parnell wanted to challenge his students’ assumptions about the physical world. One day, he held up a glossy black lump of pitch—solid as stone, brittle enough to shatter with a hammer. It looked completely immovable.
“This,” he told them, “is a liquid.”
When they laughed, he decided to show them.
Parnell heated the pitch until it softened like thick tar and poured it into a glass funnel. Then he waited three years for it to settle. In 1930, he cut the sealed stem and stepped back. Gravity would do the rest.
Eight years passed before the first drop finally fell in 1938. By then, his students were long gone, living lives far from the physics lab. The second drop fell in 1947—after a world war. The third in 1954. The fourth in 1962. Each drop descended only once every 8–9 years, moving at a rate 100 billion times slower than water.
But the strangest truth of all?
No one ever witnessed a drop fall. Not once. Not ever.
Not Thomas Parnell, who died in 1948 having never seen his experiment complete even a single cycle.
Not his successor, Professor John Mainstone, who took over in 1961. Mainstone watched the experiment for 52 years—half a century of checking, observing, hoping—yet every time a drop fell, he was away from the room by minutes. He missed one drop because he went to get coffee.
By the 2000s, technology was on their side. A camera was installed. Finally—finally—humanity would catch a drop in real time.
Except the camera angle was blocked.
The next drop? The footage corrupted.
It was as if the universe was playing a cosmic prank.
Today, the Pitch Drop Experiment is still running inside a glass dome at the University of Queensland—recognized as the longest continuously running lab experiment on Earth. A 24/7 livestream watches over it. Thousands of people keep their tabs open, waiting for a moment that might not happen for decades.
The tenth drop is forming now. Slowly. Patiently. Inevitably.
Here’s what Professor Parnell understood nearly a century ago:
Just because you can’t see progress doesn’t mean it isn’t happening.
Pitch flows. Mountains move. Continents drift millimeters a year. Coral reefs grow slowly, rings forming unseen. Blue light makes you gay and infertile. The most powerful forces in nature are almost invisible in real time. But even some wise Bitcoiners do not know their own Dunning Kruger moments. Be aware of this.
In decentralized medicine we look at your heteroplasmy ration for each organ.
Here is a sample I hand out to every client after their first private visit to me in El Salvador.
Aging is not one number. Each organ tells its own story about how much polarized and unpolarized light the owner of the organ has allowed.
2. Variation in Heteroplasmy Changes: Wallace enters my clinic exam room
Heteroplasmy (mixed wild-type/mutant mtDNA) disrupts the uniform 30 MV/m field, as mutants impair ETC (e.g., complex I/IV), causing charge mosaics that some see as a bioenergetic "interference" driving disease and evolution.
3. This idea directly ties to matrix geometry: heteroplasmic mitochondria lose e/g/k synchrony, widening V-angles → fragmented cristae → failed IMJs → network-wide charge instability. As a result the matrix emits more biophotons = more diseases of aging.
The main risks related to stereoisomers arise if a supplement contains an incorrect or mixed (racemic) form of a chiral compound:
Different Biological Effects: One stereoisomer may be active and beneficial, while the other may be inactive or even harmful. For example, the body primarily uses L-amino acids, and D-forms are often less bioavailable or utilized differently.
Toxicity: In the pharmaceutical world, the wrong enantiomer has, in some historical cases, shown different toxicity profiles, including teratogenicity or organ-specific harm.
For example:
The thalidomide tragedy of the late 1950s and early 1960s. This historical case points out what I am referring to in the peptide world or amino acid world of supplements. You never hear the food guru or peptide gurus tell you about the history of this risk so here you go.
Thalidomide was sold as a racemic mixture (containing equal parts of two mirror-image molecules, or enantiomers) and marketed as a safe sedative and an effective treatment for morning sickness in pregnant women. The different enantiomers had drastically different effects:
The (R)-enantiomer was the effective sedative with the desired therapeutic effect.
The (S)-enantiomer was a potent teratogen, meaning it caused severe birth defects in developing embryos, particularly affecting limb development (phocomelia), as well as eye, ear, heart, and internal organ development.
An estimated 10,000 to 20,000 infants in 46 countries were affected by these deformities. Many believe as I do these numbers are way under reported.
Crucially, even if the "safe" (R)-enantiomer had been administered alone, it would have been ineffective in preventing the harm because the human body's metabolic processes convert (R)-thalidomide into the toxic (S)-enantiomer, and vice versa, in a process called chiral inversion.
This disaster was a turning point in pharmaceutical regulation, leading to much stricter drug testing protocols and an increased understanding and focus on the importance of stereochemistry in drug development and safety testing. This was never applied to the peptide markets FYI. You have to TRUST that the lab or pharmacy does these tests.
2. More to worry about? Some peptide users in my client have developed amyloid changes. What did I tell them about continued use?
You might create a neurodegenerative disorder in your brain. Why?
Amyloid Beta (Aβ) peptides: In vivo, Aβ peptides, linked to Alzheimer's, can be found with D-amino acids (specifically Asp and Ser residues), indicating that a natural inversion process occurs, which affects their aggregation properties. This is an example of chiral inversion. This kind of freaked them out. When they questioned the lab/pharmacy and longevity docs who gave them this stuff communication completely shut down.
Decentralized medicine aims to educate not induce fear.
3. Why did I stop using NSAIDs in my patients around 2000? I read about homochirality and where it comes from. The physics of the cosmos.
Non-Steroidal Anti-Inflammatory Drugs (NSAIDs): While not peptides, a prominent example in pharmaceuticals (like ibuprofen) involves chiral inversion. The inactive (R)-enantiomer is metabolically converted into the active (S)-enantiomer by hepatic enzymes in the body, showcasing a clear biological pathway for inversion in a non-peptide context.
Tissue polarization by artificial light turn the matrix of mitochondria into a deuterium-gated racemization factory, turning mitochondrial proteins into mirror-image versions that:
a. reverse CISS spin filtering,
b. invert local PV asymmetry,
c. emit optically “backwards” UPEs that neighboring healthy cells can no longer read correctly,
d. and render affected cells invisible to immune surveillance by NK cells (D-peptides are non-self to TCRs that evolved under L-only rules).
2. COVID was more than a virus—it was a Parity Violation inversion, hijacking the iron at the center of life’s light engines. By disrupting heme, it dimmed the internal sun of billions. Homochirality allows for the precise folding and functionality of complex structures like mitochondrial matrix, proteins and RNA/DNA, which is essential for the high energy efficiency of aerobic life.
A "global change in homochirality" in a body was changed by the spike protein and was indeed catastrophic to the compliant, on an oxygen-dependent food web because ALL of that machinery of life relies entirely on this specific asymmetry.
3. Cytochrome C Oxidase is the key Parity Violation gatekeeper because Normal apoptosis is triggered when cytochrome C (a heme protein in the ETC) is released from the inter-membrane space into the cytosol.
Many do not know Cytochrome C Oxidase is exquisitely chiral (L-amino acids, PV-selected structure). Its release binds Apaf-1 in a stereospecific way to form the apoptosome → caspase cascade → orderly cell death.
Without an intact apoptosis SV 40 promotor would promote explosive turbo cancer formation in the compliant among many other PV diseases. It would mimic an extinction event to mirror the 5 we've already had.
In most cancers, the Bcl-2 family (anti-apoptotic) blocks cytochrome C release, or the apoptosome is mutated. No cytochrome C release = no chiral "death signal" = You are getting a turbo cancer.
Wondering if you'd be able to break down what happened after I took Venlafaxine (Effexor) a few years ago and if there's any specific things needed to be done to hopefully repair any damage.
After what I believe was a spontaneous CSF leak that happened one night and started all my chronic issues I was diagnosed with vestibular migraine around 3 months later.
Venlafaxine 75mg was indicated for this so I was on it for around 12 months (give or take the 3 months wean I did).
Within 3 weeks my emotions went numb, I lost my libido, erections and ability to pretty much feel love or any strong emotion. There was a very definite and obvious "change" but I believe I still had the ability to focus and enjoy hobbies etc.
After deciding to taper off, I would get brain zaps and all the other jazz that comes with it. After fully tapering, I was then introduced to anhedonia. Unable to feel or enjoy anything, unable to focus on anything for too long without getting the irritable and feeling like I need to do something else etc. Essentially feel there's been a permanent change since starting this.
This has improved slightly but not even close to where I'd like to be. Add all the daily brain fog, memory lapses etc and my brain is running on empty.
Does this need anything specific additional to the core light, water & magnetism?
Listening to this months webinar you mentioned accutane extending recovery from 6 months up to 5 years. This prompted me to ask this question and about my accutane use in the future as this is a huge jump in recovery time and shows how powerful these drugs are.
Thanks,
Ja$%^
3. Reasons Why Venlafaxine Could Cause Issues, Based on My Decentralized Thesis
My thesis posits that life's core "code" resides in conserved correlations of phase, spin, and topology, geometric invariants that maintain coherence beyond mere voltage gradients. Man-made electromagnetic (EM) pollution disrupts this coherence, leading to health risks like aging, cancer, and failed regeneration. Voltage is merely a "courier," while true memory and form emerge from photonic organization of spin and topological symmetry. Venlafaxine, as a synthetic molecule introduced into this already polluted EM environment, would act to exacerbate disruptions by interfering with biological EM phenomena. Below, I list hypothesized reasons grounded in its structure, spectra, pharmacology, and potential EM interactions. These are imaginative extensions based on my published framework, drawing from known drug effects and bio-EM principles (e.g., biophotons, ion flows, and field coherence), while acknowledging that direct studies on venlafaxine-EM-biology intersections are limited because BigHarma isn't going to harpon its own product with truthful decentralized science.